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  • Disease Models, Animal  (18)
  • American Association for the Advancement of Science (AAAS)  (18)
  • Blackwell Publishing Ltd
  • 1980-1984  (18)
  • 1980  (18)
Collection
Keywords
Publisher
  • American Association for the Advancement of Science (AAAS)  (18)
  • Blackwell Publishing Ltd
Years
  • 1980-1984  (18)
Year
  • 1
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    Reorganization of the axon membrane in demyelinated peripheral nerve fibers: morphological evidence (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-11-07
    Description: Cytochemical staining of demyelinated peripheral axons revealed two types of axon membrane organization, one of which suggests that the demyelinated axolemma acquires a high density of sodium channels. Ferric ion-ferrocyanide stain was confined to a restricted region of axon membrane at the beginning of a demyelinated segment or was distributed throughout the demyelinated segment of axon. The latter pattern represents one possible morphological correlate of continuous conduction through a demyelinated segment and suggests a reorganization of the axolemma after demyelination.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Foster, R E -- Whalen, C C -- Waxman, S G -- NS-15320/NS/NINDS NIH HHS/ -- New York, N.Y. -- Science. 1980 Nov 7;210(4470):661-3.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6159685" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Demyelinating Diseases/metabolism/*pathology ; Disease Models, Animal ; Ion Channels/*metabolism ; Male ; Neural Conduction ; Neurilemma/*metabolism/pathology ; Rats ; Staining and Labeling
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 2
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    Survival of mice receiving melanoma transplants is promoted by hydroquinone (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-04-25
    Description: In BALB/c female mice with melanoma transplants, the incidence of "takes" is decreased and survival is increased by hydroquinone, a melanocytolytic agent. The mechanism of drug action is suggested by via DNA. The significant and high degree of positive response to hydroquinone treatment in vivo is encouraging for the clinical management of melanoma with melanocytolytic agents.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Chavin, W -- Jelonek, E J Jr -- Reed, A H -- Binder, L R -- New York, N.Y. -- Science. 1980 Apr 25;208(4442):408-10.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7367868" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Disease Models, Animal ; Dose-Response Relationship, Drug ; Female ; Hydroquinones/metabolism/*therapeutic use ; Melanocytes/metabolism ; Melanoma/*drug therapy ; Mice ; Neoplasm Transplantation ; Neoplasms, Experimental/drug therapy
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 3
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    Role of the spleen in the growth of a murine B cell leukemia (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-04-04
    Description: A spontaneous B cell leukemia (BCL1) grew progressively in normal BALB/c mice after injection of tumor cells but did not grow in splenectomized recipients. Despite the absence of progressive tumor growth, residual tumor cells with malignant potential were found in the peripheral blood of the splenectomized animals. Splenectomy performed after injection of tumor cells but before the development of marked leukocytosis also prevented progressive tumor growth and death of the host. Thus the spleen appears to be necessary for progressive proliferation of this lymphocytic leukemia early after passage in vivo.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Kotzin, B L -- Strober, S -- New York, N.Y. -- Science. 1980 Apr 4;208(4439):59-61.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6965803" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; B-Lymphocytes/*pathology ; Disease Models, Animal ; Female ; Leukemia, Experimental/etiology/physiopathology ; Leukemia, Lymphoid/*etiology/physiopathology ; Mice ; Mice, Inbred BALB C ; Spleen/*physiology ; Splenectomy
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 4
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    Mebendazole therapy of parenteral trichinellosis (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-03-14
    Description: Mebendazole was highly effective against the helminth parasite Trichinella spiralis in mice subjected to a 3-day course of treatment during the invasive and encystment phases of experimental trichinellosis. When treatment began either 2 or 4 weeks after the mice were inoculated with parasites, the number of larvae developing in the host musculature was greatly reduced by twice-daily oral administration of 3.125, 6.25, or 12.5 milligrams of mebendazole per kilogram of body weight.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉McCracken, R O -- Taylor, D D -- New York, N.Y. -- Science. 1980 Mar 14;207(4436):1220-2.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7355285" target="_blank"〉PubMed〈/a〉
    Keywords: Administration, Oral ; Animals ; Benzimidazoles/*therapeutic use ; Disease Models, Animal ; Dose-Response Relationship, Drug ; Larva ; Male ; Mebendazole/administration & dosage/*therapeutic use ; Mice ; Muscles/parasitology ; Trichinella/drug effects ; Trichinellosis/*drug therapy
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 5
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    Animal anorexias (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-02-22
    Description: Eating very little in the presence of food or failure to serach for food has been documented in various species during the hibernation season, incubation, molting, and defense of the territory or harem. At these times feeding competes with other, more important activities. One way to avoid conflicts between feeding and these other activities to lower the programmed weight or set-point for body fat. Experiments on mammalian hibernators and incubating birds provide evidence that set-points are indeed lowered. Failure to eat in these two examples depends on anorexia, loss of appetite. A review of other examples suggests that conceptualization in terms of lowered set-points provides a unified and testable way of understanding many naturally occurring instances of fasting in the animal kingdom. Finally, spontaneous animal anorexias are contrasted with attempts by people to lose weight.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Mrosovsky, N -- Sherry, D F -- New York, N.Y. -- Science. 1980 Feb 22;207(4433):837-42.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6928327" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Anorexia/*veterinary ; Anorexia Nervosa/physiopathology ; Behavior, Animal/*physiology ; Birds/physiology ; Body Weight ; Disease Models, Animal ; Energy Metabolism ; Feeding Behavior/*physiology ; Feeding and Eating Disorders/*veterinary ; Hibernation ; Humans ; Maternal Behavior/physiology ; Obesity/physiopathology ; Rodentia/physiology ; Territoriality
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 6
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    Latency of herpes simplex virus in absence of neutralizing antibody: model for reactivation (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-11-28
    Description: Mice inoculated with herpes simplex virus (type 1) by the lip or corneal route and then passively immunized with rabbit antibody to herpes simplex virus developed a latent infection in the trigeminal ganglia within 96 hours. Neutralizing antibody to herpes simplex virus was cleared from the circulation and could not be detected in most of these mice after 2 months. Examination of ganglia from the antibody-negative mice revealed latent virus in over 90 percent of the animals, indicating that serum neutralizing antibody is not necessary to maintain the latent state. When the lips or corneas of these mice were traumatized, viral reactivation occurred in up to 90 percent of the mice, as demonstrated by the appearance of neutralizing antibody. This study provides a model for identifying factors that trigger viral reactivation.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Sekizawa, T -- Openshaw, H -- Wohlenberg, C -- Notkins, A L -- New York, N.Y. -- Science. 1980 Nov 28;210(4473):1026-8.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6254149" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Antibodies, Viral/*metabolism ; Antigens, Viral/analysis ; Disease Models, Animal ; Ganglia/microbiology ; Herpes Simplex/*immunology ; Immune Tolerance ; Immunization, Passive ; Mice ; Simplexvirus/*growth & development/immunology ; *Virus Activation
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 7
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    Sparing of the brain in neonatal undernutrition: amino acid transport and incorporation into brain and muscle (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-02-22
    Description: Rates of tyrosine and lysine transport and incorporation into protein were measured in control and undernourished weanling rats. Undernutrition was induced by feeding lactating dams a low protein diet (12 percent casein) from birth to day 21. At weaning, body and brain weights of undernourished rats were 50 percent and 88 percent, respectively, of control values. Lysine and tyrosine transport rates into skeletal muscle were reduced by over 75 percent, more than twice the reduction seen in brain. Rates of amino acid incorporation into muscle protein were reduced by approximately 50 percent; the change in rate of incorporation into brain protein was not statistically significant. These data indicate that, in spite of marked retardation of amino acid transport into brain, the brain seems fully capable of maintaining normal rates of protein synthesis.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Freedman, L S -- Samuels, S -- Fish, I -- Schwartz, S A -- Lange, B -- Katz, M -- Morgano, L -- New York, N.Y. -- Science. 1980 Feb 22;207(4433):902-4.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6766565" target="_blank"〉PubMed〈/a〉
    Keywords: Amino Acids/*metabolism ; Animals ; Animals, Newborn/metabolism ; Biological Transport ; Body Weight ; Brain/growth & development/*metabolism ; Disease Models, Animal ; Female ; Lactation ; Male ; Muscles/*metabolism ; Pregnancy ; Protein-Energy Malnutrition/*metabolism ; Rats
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 8
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    Teratogenic effects of alcohol in humans and laboratory animals (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-07-18
    Description: The teratogenicity of alcohol has been demonstrated in humans through clinical studies, behavioral studies, and epidemiologic studies, and in animals through controlled laboratory experiments. In humans exposed to alcohol during gestation the effects can range from fetal alcohol syndrome in some offspring of chronic alcoholic women to reduced average birth weight in offspring of women reporting an average consumption of two to three drinks or more per day. The behavioral effects of such exposure may range from mental retardation in children with fetal alcohol syndrome to milder developmental and behavioral effects in infants born to social drinkers. In animals, exposure to alcohol in utero may result in death, malformation, and growth deficiency as well as behavioral and developmental abnormalities. The mechanisms of impairment and related risk factors are yet to be elucidated.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Streissguth, A P -- Landesman-Dwyer, S -- Martin, J C -- Smith, D W -- New York, N.Y. -- Science. 1980 Jul 18;209(4454):353-61.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6992275" target="_blank"〉PubMed〈/a〉
    Keywords: Abnormalities, Drug-Induced ; Alcohol Drinking ; Brain/pathology ; Disease Models, Animal ; *Ethanol/pharmacology ; Female ; Fetal Alcohol Spectrum Disorders/*physiopathology ; Humans ; Hyperkinesis/chemically induced ; Infant, Low Birth Weight ; Infant, Newborn ; Pregnancy ; Sucking Behavior/drug effects ; *Teratogens
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 9
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    Anticonvulsants specific for petit mal antagonize epileptogenic effect of leucine enkephalin (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-11-28
    Description: The anticonvulsants ethosuximide, sodium valproate, and trimethadione that are specific for petit mal epilepsy abolished in rats the electrical seizure activity and behavioral abnormalities produced by leucine enkephalin, whereas phenobarbital and phenytoin had no effect. The dose-response curve for naloxone against seizure activity induced by leucine enkephalin was the same as that in gamma-hydroxybutyrate-induced petit mal. These data indicate that the epileptic properties of leucine enkephalin are petit mal-like and raise the possibility of involvement of enkephalinergic systems in. The dose-response curve for naloxone against seizure activity induced by leucine enkephalin was the same as that in gamma-hydroxybutyrate-induced petit mal. These data indicate that the epileptic properties of leucine enkephalin are petit mal-like and raise the possibility of involvement of enkephalinergic systems in petit mal epilepsy.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Snead, O C 3rd -- Bearden, L J -- 1K07 NS 00 484-01/NS/NINDS NIH HHS/ -- New York, N.Y. -- Science. 1980 Nov 28;210(4473):1031-3.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6254150" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Anticonvulsants/*pharmacology ; Disease Models, Animal ; Endorphins/*antagonists & inhibitors ; Enkephalins/*antagonists & inhibitors ; Epilepsy, Absence/drug therapy/*physiopathology ; Ethosuximide/pharmacology ; Male ; Rats ; Receptors, Opioid/drug effects ; Seizures/*prevention & control ; Trimethadione/pharmacology ; Valproic Acid/pharmacology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 10
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    Rat model for carcinogenesis in ureterosigmoidostomy (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-03-07
    Description: A rat model is used to study the carcinogenesis that occurs when urine is surgically diverted into the fecal stream, as in ureterosigmoidostomy. Adenocarcinoma of the colon occurs adjacent to the urine inlet. It is completely prevented by proximal diversion of the feces, implying that fecal carcinogens are activated locally by the urine or the urothelium.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Crissey, M M -- Steele, G D -- Gittes, R F -- New York, N.Y. -- Science. 1980 Mar 7;207(4435):1079-80.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7355272" target="_blank"〉PubMed〈/a〉
    Keywords: Adenocarcinoma/*etiology ; Animals ; Biotransformation ; Carcinogens ; Colon, Sigmoid/metabolism/*surgery ; Disease Models, Animal ; Rats ; Sigmoid Neoplasms/*etiology ; Ureter/*surgery ; Urinary Diversion/*adverse effects
    Print ISSN: 0036-8075
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    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 11
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    Impaired brain growth in neonatal rats exposed to ethanol (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-05-16
    Description: Infant rat pups, fed through intragastric cannulas from postnatal day 4 through day 18, showed a 19 percent reduction in total brain weight when ethanol was included in their diet on days 4 through 7. This reduction in brain weight occurred even though body growth in the experimental rats was equal to that of their littermate controls. The ethanol-exposed animals were markedly hypoactive during the period of drug administration, then displayed gross body tremors for 3 to 5 days. Throughout the study, the animals treated with ethanol had poor motor coordination and were hyperresponsive. These brain and behavioral effects appear similar to those seen in fetal alcohol syndrome.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Diaz, J -- Samson, H H -- New York, N.Y. -- Science. 1980 May 16;208(4445):751-3.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7189297" target="_blank"〉PubMed〈/a〉
    Keywords: Age Factors ; Animals ; Animals, Newborn ; Behavior, Animal/physiology ; Brain/anatomy & histology/drug effects/*growth & development ; Cerebellum/growth & development ; Disease Models, Animal ; Ethanol/*pharmacology ; Female ; Fetal Alcohol Spectrum Disorders/*embryology ; Organ Size ; Pregnancy ; Rats
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 12
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    Human cutaneous lieshmania in a mouse macrophage line: propagation and isolation of intracellular parasites (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-09-12
    Description: A mouse macrophage line, J774G8, supports continuous and prolific intracellular growth of Leishmania mexicana amazonensis, the etiological agent of a South American cutaneous leishmaniasis. The intracellular parasites from these infected cultures can be isolated with high recovery rate and purity by simple Percoll gradient centrifugation.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Chang, K P -- New York, N.Y. -- Science. 1980 Sep 12;209(4462):1240-42.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7403880" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Cells, Cultured ; Disease Models, Animal ; Humans ; Leishmania/growth & development ; Leishmaniasis/*parasitology/pathology ; Macrophages/*parasitology ; Mice
    Print ISSN: 0036-8075
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  • 13
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    Cellular transplantation in the treatment of experimental hepatic failure (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-11-21
    Description: The survival of Lewis rats with D-galactosamine-induced fulminant hepatic failure was prolonged if they were given intraperitoneal injections of single-cell suspensions of liver or bone marrow cells from normal rats. Suspensions of liver cells were also effective in prolonging the survival of rats with ischemia-induced hepatic necrosis. The liver cells did not act by repopulating the recipient liver.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Makowka, L -- Falk, R E -- Rotstein, L E -- Falk, J A -- Nossal, N -- Langer, B -- Blendis, L M -- Phillips, M J -- New York, N.Y. -- Science. 1980 Nov 21;210(4472):901-3.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7001630" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Anoxia/complications ; *Bone Marrow Transplantation ; Disease Models, Animal ; Galactosamine ; Hepatectomy ; Liver/cytology ; Liver Diseases/*therapy ; *Liver Transplantation ; Necrosis ; Rats ; Transplantation, Homologous
    Print ISSN: 0036-8075
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  • 14
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    The regulation of carcinogenic hazards (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-04-18
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Gori, G B -- New York, N.Y. -- Science. 1980 Apr 18;208(4441):256-61.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6768129" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; *Carcinogens ; Cell Transformation, Neoplastic ; Cost-Benefit Analysis ; Diet ; Disease Models, Animal ; Humans ; Maximum Allowable Concentration ; Risk ; Socioeconomic Factors ; United States
    Print ISSN: 0036-8075
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  • 15
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    Functional development of grafted vasopressin neurons (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-12-19
    Description: Vasopressin neurons, transplanted from normal rat fetuses into the third ventricle of adult Brattleboro rats, alleviate the polydipsia and polyuria of the hosts. Determination of the antidiuretic activity of grafted neurons in hosts with congenital diabetes insipidus provides a convenient model for analyzing the development, plasticity, and function of transplanted central nervous system neurons in mammals.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Gash, D -- Sladek, J R Jr -- Sladek, C D -- AM 16166/AM/NIADDK NIH HHS/ -- NS 15109/NS/NINDS NIH HHS/ -- New York, N.Y. -- Science. 1980 Dec 19;210(4476):1367-9.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7434031" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Diabetes Insipidus/physiopathology/*therapy ; Disease Models, Animal ; Drinking Behavior/physiology ; Hypothalamus/cytology/embryology/*transplantation ; Kidney Concentrating Ability ; Rats ; Transplantation, Homologous ; Vasopressins/*physiology
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    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 16
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    Cross-linking of lens crystallins in a photodynamic system: a process mediated by singlet oxygen (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-06-13
    Description: In a dye-sensitized photooxidation system, lens crystallin polypeptides become cross-linked, and a blue fluorescence that is associated with the proteins is produced. These changes are similar to those seen in vivo in the aging human lens. Evidence implicating singlet oxygen as the causative agent of the effects in vitro is presented, and the possibility that this species may play a role in aging and cataractogenesis in vivo is discussed.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Goosey, J D -- Zigler, J S Jr -- Kinoshita, J H -- New York, N.Y. -- Science. 1980 Jun 13;208(4449):1278-80.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7375939" target="_blank"〉PubMed〈/a〉
    Keywords: Aging ; Animals ; Cataract/etiology ; Cattle ; Crystallins/*radiation effects ; Disease Models, Animal ; Fluorescence ; Light ; Methylene Blue ; Oxidation-Reduction ; Oxygen ; Photochemistry ; Riboflavin ; Rose Bengal
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  • 17
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    Sodium-induced elevation of blood pressure in the anephric state (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-08-22
    Description: Normotensive anephric rats infused with 2 milliliters of a hyperosmolar solution of either sodium chloride or mannitol showed an increase in arterial pressure that was very pronounced with the sodium chloride and that could be partly abolished by administration of an antagonist to the vasopressor action of antidiuretic hormone (ADH). Rats with congenital ADH deficiency subjected to the same treatment showed smaller increments in arterial pressure that remained unchanged after administration of the ADH antagonist. Expansion of intravascular fluid volume was similar in all four groups and bore no correlation to the change in arterial pressure. It is concluded that about half of the increase in blood pressure induced by saline was attributable to the vasopressor effect of stimulated ADH and the remainder to an additional sodium-related factor, since it was more pronounced in the saline-infused than in the mannitol-infused groups. Expansion of the intravascular volume per se could only account for a minimal part of the increment in pressure.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Hatzinikolaou, P -- Gavras, H -- Brunner, H R -- Gavras, I -- New York, N.Y. -- Science. 1980 Aug 22;209(4459):935-6.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7403861" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Blood Pressure ; *Blood Volume ; Disease Models, Animal ; Hypertension/*etiology/physiopathology ; Male ; *Nephrectomy ; Rats ; Sodium/*blood ; Vasoconstriction ; Vasopressins/antagonists & inhibitors/deficiency/physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 18
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    Persistence of crystallin messenger RNA's with reduced translation in hereditary cataracts in mice (1980)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1980-11-21
    Description: In vitro translation experiments showed that the lens fiber cells of two hereditary cataracts in mice (Nakano and Philly) possessed a full complement of crystallin messenger RNA's, despite severely reduced synthesis of crystallin in these cells. The reduction in synthesis in the lens fiber cells correlated with the increase in Na+ and the decrease in K+, which occurs during cataractogenesis. In contrast to the fiber cells, the epithelial cells continued to synthesize crystallins in the cataractous lenses. Crystallin synthesis was stimulated in the fiber cells by raising the K+ concentration and lowering the Na+ concentration in the cultured lenses. The reduction in crystallin synthesis in the initial stages of cataractogenesis in the Nakano and Philly lenses thus appears to be due to poor utilization of crystallin messenger RNA's in the fiber cells.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Shinohara, T -- Piatigorsky, J -- New York, N.Y. -- Science. 1980 Nov 21;210(4472):914-6.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7434006" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Cataract/genetics/*metabolism ; Crystallins/biosynthesis/*genetics ; Disease Models, Animal ; Mice ; Mice, Mutant Strains/metabolism ; Potassium/metabolism ; Protein Biosynthesis ; RNA, Messenger/*metabolism ; Sodium/metabolism
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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