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  • 1
    Publication Date: 2015-12-12
    Description: One-step Synthesis of Few-layer WS〈sub〉2〈/sub〉 by Pulsed Laser Deposition Scientific Reports, Published online: 11 December 2015; doi:10.1038/srep18116
    Electronic ISSN: 2045-2322
    Topics: Natural Sciences in General
    Published by Springer Nature
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  • 2
    Publication Date: 2013-12-11
    Description: The association between lymphopenia and autoimmunity is recognized, but the underlying mechanisms are poorly understood and have not been studied systematically in humans. People with multiple sclerosis treated with the lymphocyte-depleting monoclonal antibody alemtuzumab offer a unique opportunity to study this phenomenon; one in three people develops clinical autoimmunity, and...
    Print ISSN: 0027-8424
    Electronic ISSN: 1091-6490
    Topics: Biology , Medicine , Natural Sciences in General
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  • 3
    Publication Date: 2014-05-20
    Description: Organic Letters DOI: 10.1021/ol501093v
    Print ISSN: 1523-7060
    Electronic ISSN: 1523-7052
    Topics: Chemistry and Pharmacology
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  • 4
    Publication Date: 2014-09-17
    Description: We report the fabrication of a three-dimensional forest of highly crystalline two-dimensional (2D) molybdenum disulfide (MoS 2 ) nano-petals encapsulating vertically aligned carbon nanotubes (CNT) in a core-shell configuration. Growth was conducted via magnetron sputtering at room temperature and it was found that the nano-petal morphology was formed only when a critical threshold in sputter deposition time was reached. Below this threshold, an amorphous tubular structure composed of mainly molybdenum oxides dominates instead. The presence of the MoS 2 nano-petals was shown to impart photoluminescence to the CNTs, in addition to significantly enhancing their electron emission properties, where the turn-on field was lowered from 2.50 V μ m −1 for pristine CNTs to 0.80 V μ m −1 for MoS 2 -CNT heterostructures fabricated at 30 min sputter deposition time. Photoluminescence was detected at wavelengths of approximately 684 nm and 615 nm, with the band at 684 nm gradually blue-shifting as sputter time was increased. These results demonstrate that it is possible to synthesize 2D MoS 2 layers without the need for chemical routes and high growth temperatures.
    Print ISSN: 0021-8979
    Electronic ISSN: 1089-7550
    Topics: Physics
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  • 5
    Publication Date: 2018-10-04
    Description: An ultra-low thiourea catalyzed strain-release glycosylation and a multicatalytic diversification strategy An ultra-low thiourea catalyzed strain-release glycosylation and a multicatalytic diversification strategy, Published online: 03 October 2018; doi:10.1038/s41467-018-06329-4 Non-covalent glycosyl donor activation often requires high organocatalyst loadings. Here, the authors demonstrate that strain-release glycosylations can take place at very low thiourea catalyst loadings. In addition, the authors developed a one-pot multicatalytic strategy that can diversify glycosides rapidly.
    Electronic ISSN: 2041-1723
    Topics: Biology , Chemistry and Pharmacology , Natural Sciences in General , Physics
    Published by Springer Nature
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  • 6
    Publication Date: 2008-10-14
    Description: Susceptibility to Crohn's disease, a complex inflammatory disease involving the small intestine, is controlled by over 30 loci. One Crohn's disease risk allele is in ATG16L1, a gene homologous to the essential yeast autophagy gene ATG16 (ref. 2). It is not known how ATG16L1 or autophagy contributes to intestinal biology or Crohn's disease pathogenesis. To address these questions, we generated and characterized mice that are hypomorphic for ATG16L1 protein expression, and validated conclusions on the basis of studies in these mice by analysing intestinal tissues that we collected from Crohn's disease patients carrying the Crohn's disease risk allele of ATG16L1. Here we show that ATG16L1 is a bona fide autophagy protein. Within the ileal epithelium, both ATG16L1 and a second essential autophagy protein ATG5 are selectively important for the biology of the Paneth cell, a specialized epithelial cell that functions in part by secretion of granule contents containing antimicrobial peptides and other proteins that alter the intestinal environment. ATG16L1- and ATG5-deficient Paneth cells exhibited notable abnormalities in the granule exocytosis pathway. In addition, transcriptional analysis revealed an unexpected gain of function specific to ATG16L1-deficient Paneth cells including increased expression of genes involved in peroxisome proliferator-activated receptor (PPAR) signalling and lipid metabolism, of acute phase reactants and of two adipocytokines, leptin and adiponectin, known to directly influence intestinal injury responses. Importantly, Crohn's disease patients homozygous for the ATG16L1 Crohn's disease risk allele displayed Paneth cell granule abnormalities similar to those observed in autophagy-protein-deficient mice and expressed increased levels of leptin protein. Thus, ATG16L1, and probably the process of autophagy, have a role within the intestinal epithelium of mice and Crohn's disease patients by selective effects on the cell biology and specialized regulatory properties of Paneth cells.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2695978/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2695978/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Cadwell, Ken -- Liu, John Y -- Brown, Sarah L -- Miyoshi, Hiroyuki -- Loh, Joy -- Lennerz, Jochen K -- Kishi, Chieko -- Kc, Wumesh -- Carrero, Javier A -- Hunt, Steven -- Stone, Christian D -- Brunt, Elizabeth M -- Xavier, Ramnik J -- Sleckman, Barry P -- Li, Ellen -- Mizushima, Noboru -- Stappenbeck, Thaddeus S -- Virgin, Herbert W 4th -- AI062773/AI/NIAID NIH HHS/ -- DK43351/DK/NIDDK NIH HHS/ -- P30 DK040561/DK/NIDDK NIH HHS/ -- P30 DK040561-13/DK/NIDDK NIH HHS/ -- P30 DK043351/DK/NIDDK NIH HHS/ -- P30 DK043351-18/DK/NIDDK NIH HHS/ -- P30 DK052574-09/DK/NIDDK NIH HHS/ -- P30 DK52574/DK/NIDDK NIH HHS/ -- R01 AI062773/AI/NIAID NIH HHS/ -- R01 AI062773-01A1/AI/NIAID NIH HHS/ -- R01 AI062832/AI/NIAID NIH HHS/ -- R01 AI062832-04/AI/NIAID NIH HHS/ -- T32 AR007279/AR/NIAMS NIH HHS/ -- T32 AR007279-30/AR/NIAMS NIH HHS/ -- T32 AR07279/AR/NIAMS NIH HHS/ -- U54 AI057160/AI/NIAID NIH HHS/ -- U54 AI057160-010005/AI/NIAID NIH HHS/ -- U54 AI057160-05S10018/AI/NIAID NIH HHS/ -- England -- Nature. 2008 Nov 13;456(7219):259-63. doi: 10.1038/nature07416. Epub 2008 Oct 5.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Pathology and Immunology, Washington University School of Medicine, St Louis, Missouri 63110, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/18849966" target="_blank"〉PubMed〈/a〉
    Keywords: Alleles ; Animals ; Autophagy/*genetics ; Carrier Proteins/genetics/*metabolism ; Cell Line ; Crohn Disease/genetics/pathology ; Exocytosis/genetics ; Homozygote ; Humans ; Mice ; Mice, Inbred C57BL ; Mutation ; Paneth Cells/*metabolism/pathology
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
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  • 7
    Publication Date: 2012-01-20
    Description: Exercise has beneficial effects on human health, including protection against metabolic disorders such as diabetes. However, the cellular mechanisms underlying these effects are incompletely understood. The lysosomal degradation pathway, autophagy, is an intracellular recycling system that functions during basal conditions in organelle and protein quality control. During stress, increased levels of autophagy permit cells to adapt to changing nutritional and energy demands through protein catabolism. Moreover, in animal models, autophagy protects against diseases such as cancer, neurodegenerative disorders, infections, inflammatory diseases, ageing and insulin resistance. Here we show that acute exercise induces autophagy in skeletal and cardiac muscle of fed mice. To investigate the role of exercise-mediated autophagy in vivo, we generated mutant mice that show normal levels of basal autophagy but are deficient in stimulus (exercise- or starvation)-induced autophagy. These mice (termed BCL2 AAA mice) contain knock-in mutations in BCL2 phosphorylation sites (Thr69Ala, Ser70Ala and Ser84Ala) that prevent stimulus-induced disruption of the BCL2-beclin-1 complex and autophagy activation. BCL2 AAA mice show decreased endurance and altered glucose metabolism during acute exercise, as well as impaired chronic exercise-mediated protection against high-fat-diet-induced glucose intolerance. Thus, exercise induces autophagy, BCL2 is a crucial regulator of exercise- (and starvation)-induced autophagy in vivo, and autophagy induction may contribute to the beneficial metabolic effects of exercise.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3518436/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3518436/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉He, Congcong -- Bassik, Michael C -- Moresi, Viviana -- Sun, Kai -- Wei, Yongjie -- Zou, Zhongju -- An, Zhenyi -- Loh, Joy -- Fisher, Jill -- Sun, Qihua -- Korsmeyer, Stanley -- Packer, Milton -- May, Herman I -- Hill, Joseph A -- Virgin, Herbert W -- Gilpin, Christopher -- Xiao, Guanghua -- Bassel-Duby, Rhonda -- Scherer, Philipp E -- Levine, Beth -- 1P01 DK0887761/DK/NIDDK NIH HHS/ -- P01 DK088761/DK/NIDDK NIH HHS/ -- P30 CA142543/CA/NCI NIH HHS/ -- R01 CA109618/CA/NCI NIH HHS/ -- R01 CA112023/CA/NCI NIH HHS/ -- R01 DK055758/DK/NIDDK NIH HHS/ -- R0I AI084887/AI/NIAID NIH HHS/ -- R0I HL080244/HL/NHLBI NIH HHS/ -- R0I HL090842/HL/NHLBI NIH HHS/ -- RC1 DK086629/DK/NIDDK NIH HHS/ -- RCI DK086629/DK/NIDDK NIH HHS/ -- Howard Hughes Medical Institute/ -- England -- Nature. 2012 Jan 18;481(7382):511-5. doi: 10.1038/nature10758.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Center for Autophagy Research, University of Texas Southwestern Medical Center, 5323 Harry Hines Boulevard, Dallas, Texas 75390, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/22258505" target="_blank"〉PubMed〈/a〉
    Keywords: Adiponectin/blood ; Animals ; Apoptosis Regulatory Proteins/genetics/metabolism ; Autophagy/drug effects/genetics/*physiology ; Cells, Cultured ; Dietary Fats/adverse effects ; Food Deprivation/physiology ; Gene Knock-In Techniques ; Glucose/*metabolism ; Glucose Intolerance/chemically induced/prevention & control ; Glucose Tolerance Test ; *Homeostasis/drug effects ; Leptin/blood ; Male ; Mice ; Mice, Transgenic ; Muscle, Skeletal/cytology/drug effects/*metabolism ; Mutation ; Myocardium/cytology/*metabolism ; Phosphorylation/genetics ; Physical Conditioning, Animal/*physiology ; Physical Endurance/genetics/physiology ; Physical Exertion/genetics/physiology ; Protein Binding/genetics ; Proto-Oncogene Proteins/genetics/*metabolism ; Proto-Oncogene Proteins c-bcl-2 ; Running/physiology
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
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  • 8
    Publication Date: 2018-09-10
    Description: Sensors, Vol. 18, Pages 3015: Noncontact Strain Monitoring of Osseointegrated Prostheses Sensors doi: 10.3390/s18093015 Authors: Sumit Gupta Han-Joo Lee Kenneth J. Loh Michael D. Todd Joseph Reed A. Drew Barnett The objective of this study was to develop a noncontact, noninvasive, imaging system for monitoring the strain and deformation states of osseointegrated prostheses. The proposed sensing methodology comprised of two parts. First, a passive thin film was designed such that its electrical permittivity increases in tandem with applied tensile loading and decreases while unloading. It was found that patterning the thin films could enhance their dielectric property’s sensitivity to strain. The film can be deposited onto prosthesis surfaces as an external coating prior to implant. Second, an electrical capacitance tomography (ECT) measurement technique and reconstruction algorithm were implemented to capture strain-induced changes in the dielectric property of nanocomposite-coated prosthesis phantoms when subjected to different loading scenarios. The preliminary results showed that ECT, when coupled with strain-sensitive nanocomposites, could quantify the strain-induced changes in the dielectric property of thin film-coated prosthesis phantoms. The results suggested that ECT coupled with embedded thin films could serve as a new noncontact strain sensing method for scenarios when tethered strain sensors cannot be used or instrumented, especially in the case of osseointegrated prostheses.
    Electronic ISSN: 1424-8220
    Topics: Chemistry and Pharmacology , Electrical Engineering, Measurement and Control Technology
    Published by MDPI Publishing
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  • 9
    Publication Date: 2010-05-01
    Description: In 2002, world leaders committed, through the Convention on Biological Diversity, to achieve a significant reduction in the rate of biodiversity loss by 2010. We compiled 31 indicators to report on progress toward this target. Most indicators of the state of biodiversity (covering species' population trends, extinction risk, habitat extent and condition, and community composition) showed declines, with no significant recent reductions in rate, whereas indicators of pressures on biodiversity (including resource consumption, invasive alien species, nitrogen pollution, overexploitation, and climate change impacts) showed increases. Despite some local successes and increasing responses (including extent and biodiversity coverage of protected areas, sustainable forest management, policy responses to invasive alien species, and biodiversity-related aid), the rate of biodiversity loss does not appear to be slowing.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Butchart, Stuart H M -- Walpole, Matt -- Collen, Ben -- van Strien, Arco -- Scharlemann, Jorn P W -- Almond, Rosamunde E A -- Baillie, Jonathan E M -- Bomhard, Bastian -- Brown, Claire -- Bruno, John -- Carpenter, Kent E -- Carr, Genevieve M -- Chanson, Janice -- Chenery, Anna M -- Csirke, Jorge -- Davidson, Nick C -- Dentener, Frank -- Foster, Matt -- Galli, Alessandro -- Galloway, James N -- Genovesi, Piero -- Gregory, Richard D -- Hockings, Marc -- Kapos, Valerie -- Lamarque, Jean-Francois -- Leverington, Fiona -- Loh, Jonathan -- McGeoch, Melodie A -- McRae, Louise -- Minasyan, Anahit -- Hernandez Morcillo, Monica -- Oldfield, Thomasina E E -- Pauly, Daniel -- Quader, Suhel -- Revenga, Carmen -- Sauer, John R -- Skolnik, Benjamin -- Spear, Dian -- Stanwell-Smith, Damon -- Stuart, Simon N -- Symes, Andy -- Tierney, Megan -- Tyrrell, Tristan D -- Vie, Jean-Christophe -- Watson, Reg -- New York, N.Y. -- Science. 2010 May 28;328(5982):1164-8. doi: 10.1126/science.1187512. Epub 2010 Apr 29.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉United Nations Environment Programme World Conservation Monitoring Centre, 219 Huntingdon Road, Cambridge CB3 0DL, UK. stuart.butchart@birdlife.org〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/20430971" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Anthozoa ; *Biodiversity ; Conservation of Natural Resources/trends ; *Ecosystem ; Extinction, Biological ; Humans ; International Cooperation ; *Internationality ; Plants ; Population Dynamics ; Time Factors ; Trees ; Vertebrates
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 10
    Publication Date: 2014-10-04
    Description: In 2010, the international community, under the auspices of the Convention on Biological Diversity, agreed on 20 biodiversity-related "Aichi Targets" to be achieved within a decade. We provide a comprehensive mid-term assessment of progress toward these global targets using 55 indicator data sets. We projected indicator trends to 2020 using an adaptive statistical framework that incorporated the specific properties of individual time series. On current trajectories, results suggest that despite accelerating policy and management responses to the biodiversity crisis, the impacts of these efforts are unlikely to be reflected in improved trends in the state of biodiversity by 2020. We highlight areas of societal endeavor requiring additional efforts to achieve the Aichi Targets, and provide a baseline against which to assess future progress.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Tittensor, Derek P -- Walpole, Matt -- Hill, Samantha L L -- Boyce, Daniel G -- Britten, Gregory L -- Burgess, Neil D -- Butchart, Stuart H M -- Leadley, Paul W -- Regan, Eugenie C -- Alkemade, Rob -- Baumung, Roswitha -- Bellard, Celine -- Bouwman, Lex -- Bowles-Newark, Nadine J -- Chenery, Anna M -- Cheung, William W L -- Christensen, Villy -- Cooper, H David -- Crowther, Annabel R -- Dixon, Matthew J R -- Galli, Alessandro -- Gaveau, Valerie -- Gregory, Richard D -- Gutierrez, Nicolas L -- Hirsch, Tim L -- Hoft, Robert -- Januchowski-Hartley, Stephanie R -- Karmann, Marion -- Krug, Cornelia B -- Leverington, Fiona J -- Loh, Jonathan -- Lojenga, Rik Kutsch -- Malsch, Kelly -- Marques, Alexandra -- Morgan, David H W -- Mumby, Peter J -- Newbold, Tim -- Noonan-Mooney, Kieran -- Pagad, Shyama N -- Parks, Bradley C -- Pereira, Henrique M -- Robertson, Tim -- Rondinini, Carlo -- Santini, Luca -- Scharlemann, Jorn P W -- Schindler, Stefan -- Sumaila, U Rashid -- Teh, Louise S L -- van Kolck, Jennifer -- Visconti, Piero -- Ye, Yimin -- New York, N.Y. -- Science. 2014 Oct 10;346(6206):241-4. doi: 10.1126/science.1257484. Epub 2014 Oct 2.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉United Nations Environment Programme World Conservation Monitoring Centre (UNEP-WCMC), 219 Huntingdon Road, Cambridge CB3 0DL, UK. Department of Biology, Dalhousie University, 1355 Oxford Street, Halifax, NS B3H 4R2, Canada. derek.tittensor@unep-wcmc.org. ; United Nations Environment Programme World Conservation Monitoring Centre (UNEP-WCMC), 219 Huntingdon Road, Cambridge CB3 0DL, UK. ; Department of Biology, Queen's University, Kingston, ON K7L 3N6, Canada. Ocean Sciences Division, Bedford Institute of Oceanography, Post Office Box 1006, Dartmouth, NS B2Y 4A2, Canada. ; Department of Biology, Dalhousie University, 1355 Oxford Street, Halifax, NS B3H 4R2, Canada. ; United Nations Environment Programme World Conservation Monitoring Centre (UNEP-WCMC), 219 Huntingdon Road, Cambridge CB3 0DL, UK. Centre for Macroecology, Evolution and Climate, Natural History Museum, Copenhagen, DK-2100, Denmark. ; BirdLife International, Wellbrook Court, Cambridge CB3 0NA, UK. ; ESE Laboratory, Universite Paris-Sud, UMR 8079, CNRS-Universite Paris-Sud, 91405 Orsay, France. ; PBL Netherlands Environmental Assessment Agency, Post Office Box 303, 3720 AH, Bilthoven, Netherlands. ; Food and Agricultural Organization of the United Nations, Viale delle Terme di Caracalla, 00153 Rome, Italy. ; PBL Netherlands Environmental Assessment Agency, Post Office Box 303, 3720 AH, Bilthoven, Netherlands. Department of Earth Sciences-Geochemistry, Faculty of Geosciences, Utrecht University, Post Office Box 80021, 3508 TA Utrecht, Netherlands. ; Fisheries Centre, The University of British Columbia, 2202 Main Mall, Vancouver, BC V6T 1Z4, Canada. ; Secretariat of the Convention on Biological Diversity, 413, Saint Jacques Street, Suite 800, Montreal, QC H2Y 1N9, Canada. ; Global Footprint Network, 7-9 Chemin de Balexert, 1219 Geneva, Switzerland. ; Organisation for Economic Co-operation and Development, 2 rue Andre-Pascal, 75775 Paris Cedex 16, France. ; RSPB Centre for Conservation Science The Lodge, Sandy, Bedfordshire SG19 2DL, UK. ; Marine Stewardship Council, 1-3 Snow Hill, London EC1A 2DH, UK. ; The Global Biodiversity Information Facility (GBIF) Secretariat Universitetsparken 15, 2100 Copenhagen, Denmark. ; Center for Limnology, University of Wisconsin-Madison, 680 North Park Street, Madison, WI 53706, USA. ; Forest Stewardship Council (FSC) International, Charles-de-Gaulle Strasse 5, 53113 Bonn, Germany. ; ESE Laboratory, Universite Paris-Sud, UMR 8079, CNRS-Universite Paris-Sud, 91405 Orsay, France. DIVERSITAS, 57 rue Cuvier-CP 41, 75231 Paris Cedex 05, France. ; University of Queensland, Diamantina National Park via Winton, QLD 4735, Australia. ; Zoological Society of London, Regent's Park, London NW1 4RY, UK. ; Union for Ethical BioTrade, De Ruyterkade 6, 1013 AA, Amsterdam, Netherlands. ; German Centre for Integrative Biodiversity Research (iDiv) Halle-Jena-Leipzig, Deutscher Platz 5e, 04103 Leipzig, Germany. Institute of Biology, Martin Luther University Halle-Wittenberg, Am Kirchtor 1, 06108 Halle (Saale), Germany. ; Convention on International Trade in Endangered Species Secretariat, Maison internationale de l'environnement, 11-13 Chemin des Anemones, 1219 Chatelaine, Geneva, Switzerland. ; Marine Spatial Ecology Lab, School of Biological Sciences, University of Queensland, St. Lucia Brisbane, Qld 4072 Australia. ; The International Union for Conservation of Nature Species Survival Commission (IUCN SSC) Invasive Species Specialist Group, University of Auckland, Tamaki Campus, Auckland, New Zealand. ; AidData, The College of William and Mary, Post Office Box 8795, Williamsburg, VA 23187-8795, USA. ; Department of Biology and Biotechnologies, Sapienza-Universita di Roma, Viale dell' Universita 32, 00185 Rome, Italy. ; United Nations Environment Programme World Conservation Monitoring Centre (UNEP-WCMC), 219 Huntingdon Road, Cambridge CB3 0DL, UK. School of Life Sciences, University of Sussex, Brighton BN1 9QG, UK. ; Environment Agency Austria, Department of Biodiversity and Nature Conservation, Spittelauer Lande 5, 1090 Vienna, Austria. University of Vienna, Department of Botany and Biodiversity Research, Division of Conservation Biology, Vegetation Ecology and Landscape Ecology, Rennweg 14, 1030 Vienna, Austria. ; Microsoft Research, Computational Science Laboratory, 21 Station Road, Cambridge, CB1 2FB, UK.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/25278504" target="_blank"〉PubMed〈/a〉
    Keywords: *Biodiversity ; *Conservation of Natural Resources ; *Extinction, Biological
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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