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  • Humans  (1,371)
  • Chemistry
  • LUNAR AND PLANETARY EXPLORATION
  • 2020-2022
  • 2005-2009  (1,406)
  • 2008  (1,406)
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Keywords
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  • 2020-2022
  • 2005-2009  (1,406)
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  • 1
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    Calidad ambiental de la zona costera del Golfo de Batabanó, Cuba (2008)
    CONyMA
    Details
    Publication Date: 2021-05-19
    Description: The anthropogenic affectation was evaluated on the coast N of the Gulf of Batabanó in May 2003 (corresponding to the provinces of Matanzas and Havana), in areas located in the line of the coast. The results were compared with the historical information of the sector. In the coast N and the Ensenada of the Broa, the parameters oxygen saturation, DBO5 and DQO showed characteristic high values of eutrofication. The biggest contribution in the Cianoficies was in the near coastal areas to sources of organic contamination. In the case of the nutrients they show specific data of mesothrofic waters with tendency to the eutrofization and the silts presented a high affectation for toxic metals. The area near to Guanímar is distinguished to present conditions of organic contamination that favor heterothrofic conditions, corroborated by a prevalence of the processes of mineralization of the organic matter over primary production and lows values of fitoplankton concentration. On the contrary, in the region of Surgidero of Batabanó, the processes of synthesis of organic matter prevail suggested by a high primary production, and concentration of fitoplankton, with low breathing levels and mineralization of the organic matter, that indicates that the system is behaving autothrofically. In a general way, this sector is very affected by the anthropogenic impact. The information obtained is of great importance for the development of the fishing and tourist industries in the area.
    Description: Published
    Keywords: Phytoplankton ; Water quality ; Primary production ; Chemistry ; Environmental monitoring ; Phytoplankton ; Water quality ; Primary production ; Chemistry ; Environmental monitoring
    Repository Name: AquaDocs
    Type: Proceedings Paper
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  • 2
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    A stress-responsive RNA switch regulates VEGFA expression (2008)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2008-12-23
    Description: Ligand binding to structural elements in the non-coding regions of messenger RNA modulates gene expression. Ligands such as free metabolites or other small molecules directly bind and induce conformational changes in regulatory RNA elements known as riboswitches. Other types of RNA switches are activated by complexed metabolites-for example, RNA-ligated metabolites such as aminoacyl-charged transfer RNA in the T-box system, or protein-bound metabolites in the glucose- or amino-acid-stimulated terminator-anti-terminator systems. All of these switch types are found in bacteria, fungi and plants. Here we report an RNA switch in human vascular endothelial growth factor-A (VEGFA, also known as VEGF) mRNA 3' untranslated region (UTR) that integrates signals from interferon (IFN)-gamma and hypoxia to regulate VEGFA translation in myeloid cells. Analogous to riboswitches, the VEGFA 3' UTR undergoes a binary conformational change in response to environmental signals. However, the VEGFA 3' UTR switch is metabolite-independent, and the conformational change is dictated by mutually exclusive, stimulus-dependent binding of proteins, namely, the IFN-gamma-activated inhibitor of translation complex and heterogeneous nuclear ribonucleoprotein L (HNRNPL, also known as hnRNP L). We speculate that the VEGFA switch represents the founding member of a family of signal-mediated, protein-dependent RNA switches that evolved to regulate gene expression in multicellular animals in which the precise integration of disparate inputs may be more important than the rapidity of response.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2858559/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2858559/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Ray, Partho Sarothi -- Jia, Jie -- Yao, Peng -- Majumder, Mithu -- Hatzoglou, Maria -- Fox, Paul L -- P01 HL029582/HL/NHLBI NIH HHS/ -- P01 HL029582-26A18735/HL/NHLBI NIH HHS/ -- P01 HL076491/HL/NHLBI NIH HHS/ -- P01 HL076491-050002/HL/NHLBI NIH HHS/ -- P01 HL29582/HL/NHLBI NIH HHS/ -- P01 HL76491/HL/NHLBI NIH HHS/ -- R01 DK053307/DK/NIDDK NIH HHS/ -- R01 DK060596/DK/NIDDK NIH HHS/ -- R01 DK060596-08/DK/NIDDK NIH HHS/ -- R01 DK60596/DK/NIDDK NIH HHS/ -- R01 HL067725/HL/NHLBI NIH HHS/ -- R01 HL067725-04/HL/NHLBI NIH HHS/ -- R01 HL67725/HL/NHLBI NIH HHS/ -- England -- Nature. 2009 Feb 12;457(7231):915-9. doi: 10.1038/nature07598. Epub 2008 Dec 21.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Cell Biology, The Lerner Research Institute, Cleveland Clinic, 9500 Euclid Avenue, Cleveland, Ohio 44195, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19098893" target="_blank"〉PubMed〈/a〉
    Keywords: 3' Untranslated Regions ; Amino Acyl-tRNA Synthetases ; Anoxia/metabolism ; *Gene Expression Regulation ; Gene Silencing ; Heterogeneous-Nuclear Ribonucleoprotein L/metabolism ; Humans ; Interferon-gamma/metabolism ; Myeloid Cells/metabolism/physiology ; RNA/chemistry/*metabolism ; Signal Transduction ; Stress, Physiological/*physiology ; U937 Cells ; Vascular Endothelial Growth Factor A/genetics/*metabolism
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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  • 3
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    Induced pluripotent stem cells from a spinal muscular atrophy patient (2008)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2008-12-23
    Description: Spinal muscular atrophy is one of the most common inherited forms of neurological disease leading to infant mortality. Patients have selective loss of lower motor neurons resulting in muscle weakness, paralysis and often death. Although patient fibroblasts have been used extensively to study spinal muscular atrophy, motor neurons have a unique anatomy and physiology which may underlie their vulnerability to the disease process. Here we report the generation of induced pluripotent stem cells from skin fibroblast samples taken from a child with spinal muscular atrophy. These cells expanded robustly in culture, maintained the disease genotype and generated motor neurons that showed selective deficits compared to those derived from the child's unaffected mother. This is the first study to show that human induced pluripotent stem cells can be used to model the specific pathology seen in a genetically inherited disease. As such, it represents a promising resource to study disease mechanisms, screen new drug compounds and develop new therapies.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2659408/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2659408/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Ebert, Allison D -- Yu, Junying -- Rose, Ferrill F Jr -- Mattis, Virginia B -- Lorson, Christian L -- Thomson, James A -- Svendsen, Clive N -- P01 NS057778/NS/NINDS NIH HHS/ -- P01 NS057778-01A1/NS/NINDS NIH HHS/ -- P01 NS057778-01A10002/NS/NINDS NIH HHS/ -- P01 NS057778-01A18867/NS/NINDS NIH HHS/ -- P01 NS057778-01A19001/NS/NINDS NIH HHS/ -- P01 NS057778-02/NS/NINDS NIH HHS/ -- P01 NS057778-020002/NS/NINDS NIH HHS/ -- P01 NS057778-029001/NS/NINDS NIH HHS/ -- P01NS057778/NS/NINDS NIH HHS/ -- R01HD054413/HD/NICHD NIH HHS/ -- R01NS41584/NS/NINDS NIH HHS/ -- T32 AG027566/AG/NIA NIH HHS/ -- T32 AG027566-01/AG/NIA NIH HHS/ -- T32 AG027566-02/AG/NIA NIH HHS/ -- T32 AG027566-03/AG/NIA NIH HHS/ -- T32 AG027566-04/AG/NIA NIH HHS/ -- T32GM008396/GM/NIGMS NIH HHS/ -- England -- Nature. 2009 Jan 15;457(7227):277-80. doi: 10.1038/nature07677. Epub 2008 Dec 21.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉The Waisman Center, The Stem Cell and Regenerative Medicine Center, University of Wisconsin-Madison, 1500 Highland Avenue, Madison, Wisconsin 53705, USA. ebert@waisman.wisc.edu〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19098894" target="_blank"〉PubMed〈/a〉
    Keywords: Cell Differentiation/drug effects ; Cell Lineage ; Cell Separation ; Cells, Cultured ; *Cellular Reprogramming/drug effects ; Child ; Female ; Fibroblasts/*cytology/drug effects ; Gene Expression Regulation/drug effects ; Humans ; Male ; *Models, Biological ; Motor Neurons/drug effects/metabolism/*pathology ; Muscular Atrophy, Spinal/metabolism/*pathology ; Pluripotent Stem Cells/*cytology/drug effects/metabolism/*pathology ; Skin/cytology ; Survival of Motor Neuron 1 Protein/genetics/metabolism
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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  • 4
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    The state of global hunger (2008)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2008-12-20
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Sastre, Juan -- New York, N.Y. -- Science. 2008 Dec 19;322(5909):1788-9. doi: 10.1126/science.322.5909.1788b.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19095923" target="_blank"〉PubMed〈/a〉
    Keywords: *Agriculture ; Developed Countries ; *Food Supply ; *Global Health ; Humans ; *Hunger ; *International Cooperation ; Malnutrition/*epidemiology/prevention & control
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 5
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    Nuclear reprogramming in cells (2008)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2008-12-20
    Description: Nuclear reprogramming describes a switch in gene expression of one kind of cell to that of another unrelated cell type. Early studies in frog cloning provided some of the first experimental evidence for reprogramming. Subsequent procedures included mammalian somatic cell nuclear transfer, cell fusion, induction of pluripotency by ectopic gene expression, and direct reprogramming. Through these methods it becomes possible to derive one kind of specialized cell (such as a brain cell) from another, more accessible, tissue (such as skin) in the same individual. This has potential applications for cell replacement without the immunosuppression treatments that are required when cells are transferred between genetically different individuals. This article provides some background to this field, a discussion of mechanisms and efficiency, and comments on prospects for future nuclear reprogramming research.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Gurdon, J B -- Melton, D A -- Wellcome Trust/United Kingdom -- New York, N.Y. -- Science. 2008 Dec 19;322(5909):1811-5. doi: 10.1126/science.1160810.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Wellcome Trust/Cancer Research UK Gurdon Institute and Department of Zoology, University of Cambridge, Cambridge CB2 12N, UK.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19095934" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Cell Dedifferentiation ; Cell Differentiation ; Cell Fusion ; Cell Lineage ; *Cellular Reprogramming ; Cloning, Organism ; DNA/metabolism ; DNA-Binding Proteins/metabolism ; Embryonic Stem Cells/cytology/physiology ; Female ; Gene Expression ; Humans ; Male ; Nuclear Transfer Techniques ; Oocytes/cytology ; Pluripotent Stem Cells/cytology/physiology ; Regulatory Sequences, Nucleic Acid ; Transcription Factors/genetics/metabolism
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 6
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    Materials Research Society fall meeting. Protein chip promises cheaper diagnostics (2008)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2008-12-20
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Service, Robert F -- New York, N.Y. -- Science. 2008 Dec 19;322(5909):1784-5. doi: 10.1126/science.322.5909.1784b.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19095919" target="_blank"〉PubMed〈/a〉
    Keywords: Biomarkers, Tumor/*blood ; Blood Chemical Analysis/*methods ; Blood Proteins/*analysis ; Humans ; *Microfluidics ; Neoplasms/blood/diagnosis ; *Protein Array Analysis
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 7
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    Science policy. Report faults U.S. strategy for nanotoxicology research (2008)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2008-12-20
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Service, Robert F -- New York, N.Y. -- Science. 2008 Dec 19;322(5909):1779. doi: 10.1126/science.322.5909.1779a.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19095914" target="_blank"〉PubMed〈/a〉
    Keywords: Consumer Product Safety ; Humans ; Nanostructures/*toxicity ; *Nanotechnology ; National Academy of Sciences (U.S.) ; *Public Policy ; *Research ; Risk ; United States ; United States Government Agencies
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 8
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    Label-free biomedical imaging with high sensitivity by stimulated Raman scattering microscopy (2008)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2008-12-20
    Description: Label-free chemical contrast is highly desirable in biomedical imaging. Spontaneous Raman microscopy provides specific vibrational signatures of chemical bonds, but is often hindered by low sensitivity. Here we report a three-dimensional multiphoton vibrational imaging technique based on stimulated Raman scattering (SRS). The sensitivity of SRS imaging is significantly greater than that of spontaneous Raman microscopy, which is achieved by implementing high-frequency (megahertz) phase-sensitive detection. SRS microscopy has a major advantage over previous coherent Raman techniques in that it offers background-free and readily interpretable chemical contrast. We show a variety of biomedical applications, such as differentiating distributions of omega-3 fatty acids and saturated lipids in living cells, imaging of brain and skin tissues based on intrinsic lipid contrast, and monitoring drug delivery through the epidermis.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3576036/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3576036/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Freudiger, Christian W -- Min, Wei -- Saar, Brian G -- Lu, Sijia -- Holtom, Gary R -- He, Chengwei -- Tsai, Jason C -- Kang, Jing X -- Xie, X Sunney -- CA113605/CA/NCI NIH HHS/ -- DP1 OD000277/OD/NIH HHS/ -- DP1 OD000277-05/OD/NIH HHS/ -- R01 CA113605/CA/NCI NIH HHS/ -- R01 CA113605-01A2/CA/NCI NIH HHS/ -- New York, N.Y. -- Science. 2008 Dec 19;322(5909):1857-61. doi: 10.1126/science.1165758.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Chemistry and Chemical Biology, Harvard University, Cambridge, MA 02138, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19095943" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Cell Line, Tumor ; Corpus Callosum/chemistry/cytology ; Dimethyl Sulfoxide/administration & dosage/pharmacokinetics ; Eicosapentaenoic Acid/metabolism ; Epidermis/chemistry/metabolism/ultrastructure ; Humans ; Imaging, Three-Dimensional/*methods ; Lipids/*analysis ; Mice ; Microscopy/*methods ; Neurons/ultrastructure ; Sensitivity and Specificity ; Skin/chemistry/ultrastructure ; *Spectrum Analysis, Raman ; Tretinoin/administration & dosage/pharmacokinetics ; Vitamin A/analysis/chemistry
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 9
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    Leukemic cells create bone marrow niches that disrupt the behavior of normal hematopoietic progenitor cells (2008)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2008-12-20
    Description: The host tissue microenvironment influences malignant cell proliferation and metastasis, but little is known about how tumor-induced changes in the microenvironment affect benign cellular ecosystems. Applying dynamic in vivo imaging to a mouse model, we show that leukemic cell growth disrupts normal hematopoietic progenitor cell (HPC) bone marrow niches and creates abnormal microenvironments that sequester transplanted human CD34+ (HPC-enriched) cells. CD34+ cells in leukemic mice declined in number over time and failed to mobilize into the peripheral circulation in response to cytokine stimulation. Neutralization of stem cell factor (SCF) secreted by leukemic cells inhibited CD34+ cell migration into malignant niches, normalized CD34+ cell numbers, and restored CD34+ cell mobilization in leukemic mice. These data suggest that the tumor microenvironment causes HPC dysfunction by usurping normal HPC niches and that therapeutic inhibition of HPC interaction with tumor niches may help maintain normal progenitor cell function in the setting of malignancy.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Colmone, Angela -- Amorim, Maria -- Pontier, Andrea L -- Wang, Sheng -- Jablonski, Elizabeth -- Sipkins, Dorothy A -- 1DP2OD002160-01/OD/NIH HHS/ -- 5K08CA112126-02/CA/NCI NIH HHS/ -- New York, N.Y. -- Science. 2008 Dec 19;322(5909):1861-5. doi: 10.1126/science.1164390.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Medicine, Section of Hematology/Oncology, University of Chicago, 5841 South Maryland Avenue MC 2115, Chicago, IL 60637, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19095944" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Antigens, CD34/analysis ; Bone Marrow/*pathology ; Cell Count ; Cell Line, Tumor ; Cell Movement ; Chemokine CXCL12/metabolism ; Granulocyte Colony-Stimulating Factor/pharmacology ; Hematopoietic Stem Cell Transplantation ; Hematopoietic Stem Cells/metabolism/*physiology ; Heterocyclic Compounds/pharmacology ; Humans ; Leukemia, Myeloid, Acute/metabolism/*pathology ; Mice ; Mice, SCID ; Neoplasm Transplantation ; Precursor B-Cell Lymphoblastic ; Leukemia-Lymphoma/metabolism/*pathology/physiopathology ; Stem Cell Factor/genetics/metabolism ; Stem Cell Niche/*pathology/physiopathology ; Transplantation, Heterologous ; Tumor Cells, Cultured
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 10
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    Breakthrough of the year. The runners-up (2008)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2008-12-20
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉New York, N.Y. -- Science. 2008 Dec 19;322(5909):1768. doi: 10.1126/science.322.5909.1768.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19095904" target="_blank"〉PubMed〈/a〉
    Keywords: Adipocytes, Brown/cytology/physiology ; Animals ; Electric Conductivity ; Embryo, Nonmammalian ; Genes, Neoplasm ; Genome ; Humans ; Motion Pictures as Topic ; Planets ; Proteins/chemistry/metabolism ; Protons ; *Science ; Sequence Analysis, DNA ; Water/chemistry
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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