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  • 1
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    The endocast of MH1, Australopithecus sediba (2011)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2011-09-10
    Description: The virtual endocast of MH1 (Australopithecus sediba), obtained from high-quality synchrotron scanning, reveals generally australopith-like convolutional patterns on the frontal lobes but also some foreshadowing of features of the human frontal lobes, such as posterior repositioning of the olfactory bulbs. Principal component analysis of orbitofrontal dimensions on australopith endocasts (MH1, Sts 5, and Sts 60) indicates that among these, MH1 orbitofrontal shape and organization align most closely with human endocasts. These results are consistent with gradual neural reorganization of the orbitofrontal region in the transition from Australopithecus to Homo, but given the small volume of the MH1 endocast, they are not consistent with gradual brain enlargement before the transition.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Carlson, Kristian J -- Stout, Dietrich -- Jashashvili, Tea -- de Ruiter, Darryl J -- Tafforeau, Paul -- Carlson, Keely -- Berger, Lee R -- New York, N.Y. -- Science. 2011 Sep 9;333(6048):1402-7. doi: 10.1126/science.1203922. Epub 2011 Sep 8.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Institute for Human Evolution, University of the Witwatersrand, Palaeosciences Centre, Private Bag 3, Wits 2050, South Africa. kristian.carlson@wits.ac.za〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/21903804" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; *Biological Evolution ; Brain/*anatomy & histology/growth & development ; *Fossils ; Frontal Lobe/*anatomy & histology/growth & development ; Hominidae/*anatomy & histology/growth & development ; Humans ; Imaging, Three-Dimensional ; Male ; Olfactory Bulb/anatomy & histology ; Organ Size ; Principal Component Analysis ; Skull/anatomy & histology/growth & development ; South Africa ; Synchrotrons ; Temporal Lobe/anatomy & histology ; Tomography, X-Ray Computed
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 2
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    Sequence interpretation. Functional annotation of mouse genome sequences (2001)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2001-03-10
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Nadeau, J H -- Balling, R -- Barsh, G -- Beier, D -- Brown, S D -- Bucan, M -- Camper, S -- Carlson, G -- Copeland, N -- Eppig, J -- Fletcher, C -- Frankel, W N -- Ganten, D -- Goldowitz, D -- Goodnow, C -- Guenet, J L -- Hicks, G -- Hrabe de Angelis, M -- Jackson, I -- Jacob, H J -- Jenkins, N -- Johnson, D -- Justice, M -- Kay, S -- Kingsley, D -- Lehrach, H -- Magnuson, T -- Meisler, M -- Poustka, A -- Rinchik, E M -- Rossant, J -- Russell, L B -- Schimenti, J -- Shiroishi, T -- Skarnes, W C -- Soriano, P -- Stanford, W -- Takahashi, J S -- Wurst, W -- Zimmer, A -- International Mouse Mutagenesis Consortium -- New York, N.Y. -- Science. 2001 Feb 16;291(5507):1251-5.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Genetics, BRB 624, Case Western Reserve University School of Medicine, 10900 Euclid Avenue, Cleveland, OH 44106, USA. jhn4@po.cwru.edu〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/11233449" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Chromosome Mapping ; *Computational Biology ; Costs and Cost Analysis ; Genes/physiology ; Genetic Techniques ; *Genome ; *Genomics ; International Cooperation ; Mice/*genetics ; Mutagenesis ; Mutation ; Phenotype ; Private Sector ; Public Sector ; Research Support as Topic ; *Sequence Analysis, DNA
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 3
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    Candidate taste receptors in Drosophila (2000)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2000-03-10
    Description: Little is known about the molecular mechanisms of taste perception in animals, particularly the initial events of taste signaling. A large and diverse family of seven transmembrane domain proteins was identified from the Drosophila genome database with a computer algorithm that identifies proteins on the basis of structure. Eighteen of 19 genes examined were expressed in the Drosophila labellum, a gustatory organ of the proboscis. Expression was not detected in a variety of other tissues. The genes were not expressed in the labellum of a Drosophila mutant, pox-neuro70, in which taste neurons are eliminated. Tissue specificity of expression of these genes, along with their structural similarity, supports the possibility that the family encodes a large and divergent family of taste receptors.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Clyne, P J -- Warr, C G -- Carlson, J R -- DC-02174/DC/NIDCD NIH HHS/ -- New York, N.Y. -- Science. 2000 Mar 10;287(5459):1830-4.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Molecular, Cellular, and Developmental Biology, Yale University, Post Office Box 208103, New Haven, CT 06520-8103, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/10710312" target="_blank"〉PubMed〈/a〉
    Keywords: Algorithms ; Alternative Splicing ; Amino Acid Sequence ; Animals ; Chemoreceptor Cells/*metabolism ; *Drosophila Proteins ; Drosophila melanogaster/chemistry/*genetics/physiology ; Exons ; Gene Expression ; Genes, Insect ; In Situ Hybridization ; Insect Proteins/chemistry/*genetics/physiology ; Membrane Proteins/chemistry/*genetics/physiology ; Molecular Sequence Data ; Multigene Family ; Neurons, Afferent/*metabolism ; Organ Specificity ; Protein Structure, Tertiary ; Receptors, Cell Surface/chemistry/*genetics/physiology ; Reverse Transcriptase Polymerase Chain Reaction ; Sense Organs/chemistry/physiology ; Sequence Alignment ; Taste/physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 4
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    Redefining rats, mice, and birds. AAA (American Association of Anatomists) Public Affairs Committee (1999)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1999-04-24
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Besharse, J C -- Carlson, B M -- Jenkins, D P -- Lester, D S -- Olds, J L -- Satir, P -- New York, N.Y. -- Science. 1999 Apr 2;284(5411):49-50.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/10215528" target="_blank"〉PubMed〈/a〉
    Keywords: Animal Rights/*legislation & jurisprudence ; Animal Welfare/*legislation & jurisprudence ; Animals ; *Animals, Laboratory ; Birds ; Mice ; Rats ; Research ; Societies, Scientific ; United States ; United States Department of Agriculture/legislation & jurisprudence
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 5
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    Rapid evolution of a geographic cline in size in an introduced fly (2000)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2000-01-15
    Description: The introduction and rapid spread of Drosophila subobscura in the New World two decades ago provide an opportunity to determine the predictability and rate of evolution of a geographic cline. In ancestral Old World populations, wing length increases clinally with latitude. In North American populations, no wing length cline was detected one decade after the introduction. After two decades, however, a cline has evolved and largely converged on the ancestral cline. The rate of morphological evolution on a continental scale is very fast, relative even to rates measured within local populations. Nevertheless, different wing sections dominate the New versus Old World clines. Thus, the evolution of geographic variation in wing length has been predictable, but the means by which the cline is achieved is contingent.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Huey, R B -- Gilchrist, G W -- Carlson, M L -- Berrigan, D -- Serra, L -- New York, N.Y. -- Science. 2000 Jan 14;287(5451):308-9.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Zoology, University of Washington, Box 351800, Seattle, WA 98195-1800, USA. hueyrb@u.washington.edu〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/10634786" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; *Biological Evolution ; Drosophila/*anatomy & histology/*genetics ; Europe ; Female ; Geography ; Male ; North America ; Sex Characteristics ; Time Factors ; Wings, Animal/*anatomy & histology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 6
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    Distinct mechanism for antidepressant activity by blockade of central substance P receptors (1998)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1998-09-11
    Description: The localization of substance P in brain regions that coordinate stress responses and receive convergent monoaminergic innervation suggested that substance P antagonists might have psychotherapeutic properties. Like clinically used antidepressant and anxiolytic drugs, substance P antagonists suppressed isolation-induced vocalizations in guinea pigs. In a placebo-controlled trial in patients with moderate to severe major depression, robust antidepressant effects of the substance P antagonist MK-869 were consistently observed. In preclinical studies, substance P antagonists did not interact with monoamine systems in the manner seen with established antidepressant drugs. These findings suggest that substance P may play an important role in psychiatric disorders.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Kramer, M S -- Cutler, N -- Feighner, J -- Shrivastava, R -- Carman, J -- Sramek, J J -- Reines, S A -- Liu, G -- Snavely, D -- Wyatt-Knowles, E -- Hale, J J -- Mills, S G -- MacCoss, M -- Swain, C J -- Harrison, T -- Hill, R G -- Hefti, F -- Scolnick, E M -- Cascieri, M A -- Chicchi, G G -- Sadowski, S -- Williams, A R -- Hewson, L -- Smith, D -- Carlson, E J -- Hargreaves, R J -- Rupniak, N M -- New York, N.Y. -- Science. 1998 Sep 11;281(5383):1640-5.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Merck Research Laboratories, West Point, PA 19456, USA. Mark_Kramer@merck.com〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/9733503" target="_blank"〉PubMed〈/a〉
    Keywords: Adolescent ; Adult ; Aged ; Amygdala/drug effects/metabolism ; Animals ; Antidepressive Agents, Second-Generation/adverse ; effects/metabolism/pharmacology/*therapeutic use ; Behavior, Animal/drug effects ; Brain/drug effects/metabolism ; Depressive Disorder/*drug therapy/etiology/metabolism ; Female ; Gerbillinae ; Guinea Pigs ; Humans ; Male ; Middle Aged ; Morpholines/adverse effects/metabolism/pharmacology/*therapeutic use ; *Neurokinin-1 Receptor Antagonists ; Norepinephrine/physiology ; Paroxetine/therapeutic use ; Receptors, Neurokinin-1/metabolism ; Serotonin/physiology ; Stress, Psychological/drug therapy ; Substance P/*antagonists & inhibitors/metabolism ; Vocalization, Animal/drug effects
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 7
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    Autoinducer of virulence as a target for vaccine and therapy against Staphylococcus aureus (1998)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1998-05-09
    Description: Staphylococcus aureus causes pathologies ranging from minor skin infections to life-threatening diseases. Pathogenic effects are largely due to production of bacterial toxin, which is regulated by an RNA molecule, RNAIII. The S. aureus protein called RAP (RNAIII activating protein) activates RNAIII, and a peptide called RIP (RNAIII inhibiting peptide), produced by a nonpathogenic bacteria, inhibits RNAIII. Mice vaccinated with RAP or treated with purified or synthetic RIP were protected from S. aureus pathology. Thus, these two molecules may provide useful approaches for the prevention and treatment of diseases caused by S. aureus.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Balaban, N -- Goldkorn, T -- Nhan, R T -- Dang, L B -- Scott, S -- Ridgley, R M -- Rasooly, A -- Wright, S C -- Larrick, J W -- Rasooly, R -- Carlson, J R -- AI40830/AI/NIAID NIH HHS/ -- New York, N.Y. -- Science. 1998 Apr 17;280(5362):438-40.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Medical Pathology, University of California, Davis, CA 95616, USA. nbalaban@ucdavis.edu〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/9545222" target="_blank"〉PubMed〈/a〉
    Keywords: Amino Acid Sequence ; Animals ; Antibodies, Bacterial/biosynthesis ; Bacterial Proteins/antagonists & inhibitors/*immunology/isolation & purification ; Bacterial Toxins/biosynthesis ; *Bacterial Vaccines ; Male ; Mice ; Mice, Hairless ; Oligopeptides/isolation & purification/*therapeutic use ; RNA, Antisense/genetics ; RNA, Bacterial/genetics ; Signal Transduction ; Staphylococcal Skin Infections/*drug therapy/immunology/*prevention & control ; Staphylococcus aureus/metabolism/*pathogenicity ; Vaccination ; Virulence
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 8
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    Genetics of gene expression and its effect on disease (2008)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2008-03-18
    Description: Common human diseases result from the interplay of many genes and environmental factors. Therefore, a more integrative biology approach is needed to unravel the complexity and causes of such diseases. To elucidate the complexity of common human diseases such as obesity, we have analysed the expression of 23,720 transcripts in large population-based blood and adipose tissue cohorts comprehensively assessed for various phenotypes, including traits related to clinical obesity. In contrast to the blood expression profiles, we observed a marked correlation between gene expression in adipose tissue and obesity-related traits. Genome-wide linkage and association mapping revealed a highly significant genetic component to gene expression traits, including a strong genetic effect of proximal (cis) signals, with 50% of the cis signals overlapping between the two tissues profiled. Here we demonstrate an extensive transcriptional network constructed from the human adipose data that exhibits significant overlap with similar network modules constructed from mouse adipose data. A core network module in humans and mice was identified that is enriched for genes involved in the inflammatory and immune response and has been found to be causally associated to obesity-related traits.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Emilsson, Valur -- Thorleifsson, Gudmar -- Zhang, Bin -- Leonardson, Amy S -- Zink, Florian -- Zhu, Jun -- Carlson, Sonia -- Helgason, Agnar -- Walters, G Bragi -- Gunnarsdottir, Steinunn -- Mouy, Magali -- Steinthorsdottir, Valgerdur -- Eiriksdottir, Gudrun H -- Bjornsdottir, Gyda -- Reynisdottir, Inga -- Gudbjartsson, Daniel -- Helgadottir, Anna -- Jonasdottir, Aslaug -- Jonasdottir, Adalbjorg -- Styrkarsdottir, Unnur -- Gretarsdottir, Solveig -- Magnusson, Kristinn P -- Stefansson, Hreinn -- Fossdal, Ragnheidur -- Kristjansson, Kristleifur -- Gislason, Hjortur G -- Stefansson, Tryggvi -- Leifsson, Bjorn G -- Thorsteinsdottir, Unnur -- Lamb, John R -- Gulcher, Jeffrey R -- Reitman, Marc L -- Kong, Augustine -- Schadt, Eric E -- Stefansson, Kari -- England -- Nature. 2008 Mar 27;452(7186):423-8. doi: 10.1038/nature06758. Epub 2008 Mar 16.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉deCODE genetics, 101 Reykjavik, Iceland.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/18344981" target="_blank"〉PubMed〈/a〉
    Keywords: Adipose Tissue/metabolism ; Adolescent ; Adult ; Aged ; Aged, 80 and over ; Animals ; Blood/metabolism ; Body Mass Index ; Cohort Studies ; European Continental Ancestry Group/genetics ; Female ; *Gene Expression Profiling ; Gene Expression Regulation/*genetics ; Genome, Human ; Humans ; Iceland ; Lod Score ; Male ; Mice ; Middle Aged ; Obesity/*genetics ; Polymorphism, Single Nucleotide/genetics ; Quantitative Trait Loci/genetics ; Sample Size ; Waist-Hip Ratio
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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  • 9
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    Imbalance between pSmad3 and Notch induces CDK inhibitors in old muscle stem cells (2008)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2008-06-17
    Description: Adult skeletal muscle robustly regenerates throughout an organism's life, but as the muscle ages, its ability to repair diminishes and eventually fails. Previous work suggests that the regenerative potential of muscle stem cells (satellite cells) is not triggered in the old muscle because of a decline in Notch activation, and that it can be rejuvenated by forced local activation of Notch. Here we report that, in addition to the loss of Notch activation, old muscle produces excessive transforming growth factor (TGF)-beta (but not myostatin), which induces unusually high levels of TGF-beta pSmad3 in resident satellite cells and interferes with their regenerative capacity. Importantly, endogenous Notch and pSmad3 antagonize each other in the control of satellite-cell proliferation, such that activation of Notch blocks the TGF-beta-dependent upregulation of the cyclin-dependent kinase (CDK) inhibitors p15, p16, p21 and p27, whereas inhibition of Notch induces them. Furthermore, in muscle stem cells, Notch activity determines the binding of pSmad3 to the promoters of these negative regulators of cell-cycle progression. Attenuation of TGF-beta/pSmad3 in old, injured muscle restores regeneration to satellite cells in vivo. Thus a balance between endogenous pSmad3 and active Notch controls the regenerative competence of muscle stem cells, and deregulation of this balance in the old muscle microniche interferes with regeneration.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Carlson, Morgan E -- Hsu, Michael -- Conboy, Irina M -- R01 AG027252/AG/NIA NIH HHS/ -- R21 AG27892/AG/NIA NIH HHS/ -- England -- Nature. 2008 Jul 24;454(7203):528-32. doi: 10.1038/nature07034. Epub 2008 Jun 15.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Bioengineering, University of California, Berkeley, California 94720, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/18552838" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Cell Proliferation ; Coculture Techniques ; Cyclin-Dependent Kinase Inhibitor Proteins/genetics/*metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Myoblasts, Skeletal/cytology/drug effects/*metabolism ; Myostatin ; Promoter Regions, Genetic/genetics ; Protein Binding ; Receptors, Notch/antagonists & inhibitors/*metabolism ; Satellite Cells, Skeletal Muscle/cytology/drug effects/metabolism ; Smad3 Protein/genetics/*metabolism ; Transforming Growth Factor beta/metabolism/pharmacology
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
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  • 10
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    Odorant reception in the malaria mosquito Anopheles gambiae (2010)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2010-02-05
    Description: The mosquito Anopheles gambiae is the major vector of malaria in sub-Saharan Africa. It locates its human hosts primarily through olfaction, but little is known about the molecular basis of this process. Here we functionally characterize the Anopheles gambiae odorant receptor (AgOr) repertoire. We identify receptors that respond strongly to components of human odour and that may act in the process of human recognition. Some of these receptors are narrowly tuned, and some salient odorants elicit strong responses from only one or a few receptors, suggesting a central role for specific transmission channels in human host-seeking behaviour. This analysis of the Anopheles gambiae receptors permits a comparison with the corresponding Drosophila melanogaster odorant receptor repertoire. We find that odorants are differentially encoded by the two species in ways consistent with their ecological needs. Our analysis of the Anopheles gambiae repertoire identifies receptors that may be useful targets for controlling the transmission of malaria.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2833235/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2833235/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Carey, Allison F -- Wang, Guirong -- Su, Chih-Ying -- Zwiebel, Laurence J -- Carlson, John R -- 2T32GM07205/GM/NIGMS NIH HHS/ -- R01 AI056402/AI/NIAID NIH HHS/ -- R01 AI056402-06A2/AI/NIAID NIH HHS/ -- R01 AI056402-07/AI/NIAID NIH HHS/ -- R01 DC002174/DC/NIDCD NIH HHS/ -- R01 DC002174-24/DC/NIDCD NIH HHS/ -- R01 DC004729/DC/NIDCD NIH HHS/ -- R01 DC004729-10/DC/NIDCD NIH HHS/ -- R01 GM063364/GM/NIGMS NIH HHS/ -- R01 GM063364-08/GM/NIGMS NIH HHS/ -- England -- Nature. 2010 Mar 4;464(7285):66-71. doi: 10.1038/nature08834. Epub 2010 Feb 3.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Molecular, Cellular and Developmental Biology, Yale University, New Haven, Connecticut 06520, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/20130575" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Anopheles gambiae/anatomy & histology/genetics/*metabolism ; Drosophila melanogaster/cytology/genetics/metabolism ; Electrophysiology ; Humans ; Insect Bites and Stings/prevention & control ; Insect Vectors/*metabolism ; *Malaria/prevention & control/transmission ; Models, Biological ; Odors/*analysis ; Olfactory Pathways/*metabolism ; Olfactory Receptor Neurons/metabolism ; Receptors, Odorant/genetics/*metabolism ; Time Factors
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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