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  • Animals  (6)
  • fracture mechanics  (2)
  • *Morphogenesis  (1)
  • 0699  (1)
  • 2005-2009  (5)
  • 2000-2004  (4)
  • 1
    Electronic Resource
    Electronic Resource
    Effect of crack shape, inclination angle, and friction coefficient in crack surface contact problems (2000)
    Springer
    International journal of fracture 105 (2000), S. 367-389 
    Details
    ISSN: 1573-2673
    Keywords: Stress intensity factor ; tribology ; contact problem ; friction coefficient ; fracture mechanics ; rolling contact fatigue ; surface crack ; body force method.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: Abstract In rolling/sliding contact fatigue, it is known that the crack propagates at a characteristic angle θ=15–30 deg to the surface. To analyze the mechanism, however, the body force method has been widely used assuming 3D crack models for θ=45–90. In this study, therefore, the unknown body force densities are newly approximated by using fundamental density functions and polynomials. Then, a semi-elliptical crack model is analyzed for θ=15–90 under compressive residual stresses and Hertzian contact loads. The stress intensity factors K II, K III are calculated with varying the crack shape b/a, inclination crack angle θ, and crack face friction coefficient μ. The calculations show that the present method is useful for the analysis for θ=15–30 deg with high accuracy. It is seen that the K II-values when b/a→0 are larger than the ones when b/a=1 by 0–24% for both under compressive residual stress and Hertzian contact load. Regarding the maximum K II values under Hertzian contact load, the results of θ=15 deg are smaller than the ones of θ=45 deg by 23–34%. Regarding the amplitude of (K II max−K II min), the results of θ=15 deg are smaller than the ones of θ=45 deg by 4–24%. With increasing the value of friction coefficient μ for crack faces the value of K II decreases significantly. When the crack is short and the inclination angle θ is small, the value of friction coefficient f for Hertzian contact load largely affect the K II value.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/A:1007688027669
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    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Generalized stress intensity factors in the interaction between two fibers in matrix (2000)
    Springer
    International journal of fracture 103 (2000), S. 19-39 
    Details
    ISSN: 1573-2673
    Keywords: Elasticity ; composite material ; fracture mechanics ; fiber ; generalized stress intensity factor ; end effect ; interaction ; rectangular inclusions.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: Abstract To evaluate the mechanical strength of fiber reinforced composites it is necessary to consider singular stresses at the end of fibers because they cause crack initiation, propagation, and final failure. The singular stress is expressed by generalized stress intensity factors defined at the corner of fibers. As a 2D model an interaction between rectangular inclusions under longitudinal tension is treated in this paper. The body force method is used to formulate the problem as a system of singular integral equations with Cauchy-type or logarithmic-type singularities, where the unknown functions are the densities of body forces distributed in infinite plates having the same elastic constants as those of the matrix and inclusions. In order to analyze the problem accurately, the unknown functions are expressed as piecewize smooth functions using two types of fundamental densities and power series, where the fundamental densities are chosen to represent the symmetric stress singularity of 1/r 1−λ 1 and the skew-symmetric stress singularity of 1/r 1−λ 2. Then, generalized stress intensity factors at the end of inclusions are systematically calculated for various locations, spacings and elastic modulus of two rectangular inclusions in a plate subjected to longitudinal tension.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/A:1007696723382
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  • 3
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    Co-seismic geoelectric potential changes observed in Japan (2000)
    In:  Geophys. Res. Lett., Colorado Springs, US Air Force Academy, vol. 27, no. 10, pp. 1535-1538, pp. L12S08, (ISSN: 1340-4202)
    Details
    Publication Date: 2000
    Description: Nagao et al. monitor the geoelectric potential difference between pairs of electrodes (or dipoles) buried at about 2 m depth at several stations in Japan for the purpose of observing co-seismic signals. Studying several recent earthquakes, they report that the signals commence not with the origin time of the earthquakes but with the arrival time of seismic waves. They find two types of co-seismic changes: offset/decay type, which last long after the seismic vibrations cease, and oscillatory type, which decay with the seismic vibrations. They also find that the amplitude of the co-seismic signals does not scale with the dipole length. Also in this issue, Hayakawa et al. [1531] investigate ultra low frequency (ULF) electromagnetic emissions associated with a large earthquake that occurred at Biak Island, Indonesia, in February 1996. Examining data from Biak and Darwin, they find that the ULF emissions observed about 1.5 months before the quake serve as a precursory signature of the earthquake.
    Keywords: Earthquake ; Electromagnetic methods/phenomena ; 0699 ; Electromagnetics ; General ; or ; miscellaneous ; 7223 ; Seismology ; Seismic ; hazard ; assessment ; and ; prediction ; 9320 ; Information ; related ; to ; geographic ; region ; Asia ; 9810 ; General ; or ; miscellaneous ; New ; fields ; (not ; classifiable ; under ; other ; headings) ; GRL
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    BIBLIOGRAPHY SEISMOLOGY
  • 4
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    Ventroptin: a BMP-4 antagonist expressed in a double-gradient pattern in the retina (2001)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2001-07-07
    Description: In the visual system, the establishment of the anteroposterior and dorsoventral axes in the retina and tectum during development is important for topographic retinotectal projection. We identified chick Ventroptin, an antagonist of bone morphogenetic protein 4 (BMP-4), which is mainly expressed in the ventral retina, not only with a ventral high-dorsal low gradient but also with a nasal high-temporal low gradient at later stages. Misexpression of Ventroptin altered expression patterns of several topographic genes in the retina and projection of the retinal axons to the tectum along both axes. Thus, the topographic retinotectal projection appears to be specified by the double-gradient molecule Ventroptin along the two axes.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Sakuta, H -- Suzuki, R -- Takahashi, H -- Kato, A -- Shintani, T -- Iemura Si -- Yamamoto, T S -- Ueno, N -- Noda, M -- New York, N.Y. -- Science. 2001 Jul 6;293(5527):111-5.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Division of Molecular Neurobiology, National Institute for Basic Biology, The Graduate University for Advanced Studies, 38 Nishigonaka, Myodaiji-cho, Okazaki 444-8585, Japan.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/11441185" target="_blank"〉PubMed〈/a〉
    Keywords: Alternative Splicing ; Amino Acid Sequence ; Animals ; Bone Morphogenetic Protein 4 ; Bone Morphogenetic Proteins/*antagonists & inhibitors/genetics/metabolism ; Chick Embryo ; Cloning, Molecular ; Electroporation ; Embryo, Nonmammalian/cytology/metabolism ; Eye Proteins/chemistry/genetics/*metabolism ; *Gene Expression Regulation, Developmental ; Gene Library ; Humans ; In Situ Hybridization ; Mice ; Microinjections ; Molecular Sequence Data ; *Morphogenesis ; Nerve Tissue Proteins ; Precipitin Tests ; Protein Binding ; Protein Isoforms/chemistry/genetics/metabolism ; RNA, Messenger/analysis/genetics ; Retina/*embryology/*metabolism ; Sequence Alignment ; Surface Plasmon Resonance ; Xenopus Proteins ; Xenopus laevis/embryology/metabolism
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 5
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    Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1beta production (2008)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2008-10-14
    Description: Systems for protein degradation are essential for tight control of the inflammatory immune response. Autophagy, a bulk degradation system that delivers cytoplasmic constituents into autolysosomes, controls degradation of long-lived proteins, insoluble protein aggregates and invading microbes, and is suggested to be involved in the regulation of inflammation. However, the mechanism underlying the regulation of inflammatory response by autophagy is poorly understood. Here we show that Atg16L1 (autophagy-related 16-like 1), which is implicated in Crohn's disease, regulates endotoxin-induced inflammasome activation in mice. Atg16L1-deficiency disrupts the recruitment of the Atg12-Atg5 conjugate to the isolation membrane, resulting in a loss of microtubule-associated protein 1 light chain 3 (LC3) conjugation to phosphatidylethanolamine. Consequently, both autophagosome formation and degradation of long-lived proteins are severely impaired in Atg16L1-deficient cells. Following stimulation with lipopolysaccharide, a ligand for Toll-like receptor 4 (refs 8, 9), Atg16L1-deficient macrophages produce high amounts of the inflammatory cytokines IL-1beta and IL-18. In lipopolysaccharide-stimulated macrophages, Atg16L1-deficiency causes Toll/IL-1 receptor domain-containing adaptor inducing IFN-beta (TRIF)-dependent activation of caspase-1, leading to increased production of IL-1beta. Mice lacking Atg16L1 in haematopoietic cells are highly susceptible to dextran sulphate sodium-induced acute colitis, which is alleviated by injection of anti-IL-1beta and IL-18 antibodies, indicating the importance of Atg16L1 in the suppression of intestinal inflammation. These results demonstrate that Atg16L1 is an essential component of the autophagic machinery responsible for control of the endotoxin-induced inflammatory immune response.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Saitoh, Tatsuya -- Fujita, Naonobu -- Jang, Myoung Ho -- Uematsu, Satoshi -- Yang, Bo-Gie -- Satoh, Takashi -- Omori, Hiroko -- Noda, Takeshi -- Yamamoto, Naoki -- Komatsu, Masaaki -- Tanaka, Keiji -- Kawai, Taro -- Tsujimura, Tohru -- Takeuchi, Osamu -- Yoshimori, Tamotsu -- Akira, Shizuo -- AI070167/AI/NIAID NIH HHS/ -- England -- Nature. 2008 Nov 13;456(7219):264-8. doi: 10.1038/nature07383. Epub 2008 Oct 5.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Laboratory of Host Defense, WPI Immunology Frontier Research Center, Osaka University, 3-1 Yamada-oka, Suita, Osaka 565-0871, Japan.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/18849965" target="_blank"〉PubMed〈/a〉
    Keywords: Adenine/analogs & derivatives/pharmacology ; Animals ; Autophagy/*genetics ; Carrier Proteins/*genetics ; Chimera ; Colitis/chemically induced/immunology ; Dextran Sulfate/pharmacology ; Female ; Gene Expression Regulation/*drug effects ; Interleukin-1beta/*biosynthesis/metabolism ; Lipopolysaccharides/*pharmacology ; Macrophages/*drug effects/*metabolism ; Mice ; Mice, Inbred C57BL ; Mutation
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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  • 6
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    Emergence and pandemic potential of swine-origin H1N1 influenza virus (2009)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2009-06-16
    Description: Influenza viruses cause annual epidemics and occasional pandemics that have claimed the lives of millions. The emergence of new strains will continue to pose challenges to public health and the scientific communities. A prime example is the recent emergence of swine-origin H1N1 viruses that have transmitted to and spread among humans, resulting in outbreaks internationally. Efforts to control these outbreaks and real-time monitoring of the evolution of this virus should provide us with invaluable information to direct infectious disease control programmes and to improve understanding of the factors that determine viral pathogenicity and/or transmissibility.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2873852/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2873852/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Neumann, Gabriele -- Noda, Takeshi -- Kawaoka, Yoshihiro -- HHSN266200700010C/PHS HHS/ -- England -- Nature. 2009 Jun 18;459(7249):931-9. doi: 10.1038/nature08157.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Pathobiological Sciences, University of Wisconsin-Madison, Madison, Wisconsin 53711, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19525932" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; *Disease Outbreaks/prevention & control/veterinary ; Hemagglutinin Glycoproteins, Influenza Virus/metabolism ; Humans ; Influenza A Virus, H1N1 Subtype/pathogenicity/*physiology ; Influenza Vaccines/immunology ; Influenza, Human/*epidemiology/prevention & control/transmission/*virology ; Swine/*virology
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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  • 7
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    In vitro and in vivo characterization of new swine-origin H1N1 influenza viruses (2009)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2009-08-13
    Description: Influenza A viruses cause recurrent outbreaks at local or global scale with potentially severe consequences for human health and the global economy. Recently, a new strain of influenza A virus was detected that causes disease in and transmits among humans, probably owing to little or no pre-existing immunity to the new strain. On 11 June 2009 the World Health Organization declared that the infections caused by the new strain had reached pandemic proportion. Characterized as an influenza A virus of the H1N1 subtype, the genomic segments of the new strain were most closely related to swine viruses. Most human infections with swine-origin H1N1 influenza viruses (S-OIVs) seem to be mild; however, a substantial number of hospitalized individuals do not have underlying health issues, attesting to the pathogenic potential of S-OIVs. To achieve a better assessment of the risk posed by the new virus, we characterized one of the first US S-OIV isolates, A/California/04/09 (H1N1; hereafter referred to as CA04), as well as several other S-OIV isolates, in vitro and in vivo. In mice and ferrets, CA04 and other S-OIV isolates tested replicate more efficiently than a currently circulating human H1N1 virus. In addition, CA04 replicates efficiently in non-human primates, causes more severe pathological lesions in the lungs of infected mice, ferrets and non-human primates than a currently circulating human H1N1 virus, and transmits among ferrets. In specific-pathogen-free miniature pigs, CA04 replicates without clinical symptoms. The assessment of human sera from different age groups suggests that infection with human H1N1 viruses antigenically closely related to viruses circulating in 1918 confers neutralizing antibody activity to CA04. Finally, we show that CA04 is sensitive to approved and experimental antiviral drugs, suggesting that these compounds could function as a first line of defence against the recently declared S-OIV pandemic.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2748827/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2748827/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Itoh, Yasushi -- Shinya, Kyoko -- Kiso, Maki -- Watanabe, Tokiko -- Sakoda, Yoshihiro -- Hatta, Masato -- Muramoto, Yukiko -- Tamura, Daisuke -- Sakai-Tagawa, Yuko -- Noda, Takeshi -- Sakabe, Saori -- Imai, Masaki -- Hatta, Yasuko -- Watanabe, Shinji -- Li, Chengjun -- Yamada, Shinya -- Fujii, Ken -- Murakami, Shin -- Imai, Hirotaka -- Kakugawa, Satoshi -- Ito, Mutsumi -- Takano, Ryo -- Iwatsuki-Horimoto, Kiyoko -- Shimojima, Masayuki -- Horimoto, Taisuke -- Goto, Hideo -- Takahashi, Kei -- Makino, Akiko -- Ishigaki, Hirohito -- Nakayama, Misako -- Okamatsu, Masatoshi -- Takahashi, Kazuo -- Warshauer, David -- Shult, Peter A -- Saito, Reiko -- Suzuki, Hiroshi -- Furuta, Yousuke -- Yamashita, Makoto -- Mitamura, Keiko -- Nakano, Kunio -- Nakamura, Morio -- Brockman-Schneider, Rebecca -- Mitamura, Hiroshi -- Yamazaki, Masahiko -- Sugaya, Norio -- Suresh, M -- Ozawa, Makoto -- Neumann, Gabriele -- Gern, James -- Kida, Hiroshi -- Ogasawara, Kazumasa -- Kawaoka, Yoshihiro -- HHNSN266200700010C/NS/NINDS NIH HHS/ -- HHSN266200700010C/PHS HHS/ -- HHSN272200800060C/AI/NIAID NIH HHS/ -- R01 AI069274/AI/NIAID NIH HHS/ -- R01 AI069274-04/AI/NIAID NIH HHS/ -- U19 AI070503/AI/NIAID NIH HHS/ -- England -- Nature. 2009 Aug 20;460(7258):1021-5. doi: 10.1038/nature08260.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Pathology, Shiga University of Medical Science, Ohtsu, Shiga 520-2192, Japan.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19672242" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Antibodies, Viral/immunology ; Antiviral Agents/pharmacology ; Cell Line ; Dogs ; Female ; Ferrets/virology ; HN Protein/metabolism ; Humans ; Influenza A Virus, H1N1 Subtype/drug effects/enzymology/pathogenicity/*physiology ; Lung/immunology/pathology/virology ; Macaca fascicularis/immunology/virology ; Male ; Mice ; Mice, Inbred BALB C ; Neutralization Tests ; Orthomyxoviridae Infections/immunology/transmission/virology ; Primate Diseases/pathology/virology ; Swine/*virology ; Swine Diseases/pathology/virology ; Swine, Miniature/virology ; Virus Replication
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
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  • 8
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    Genome of an endosymbiont coupling N2 fixation to cellulolysis within protist cells in termite gut (2008)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2008-11-15
    Description: Termites harbor diverse symbiotic gut microorganisms, the majority of which are as yet uncultivable and their interrelationships unclear. Here, we present the complete genome sequence of the uncultured Bacteroidales endosymbiont of the cellulolytic protist Pseudotrichonympha grassii, which accounts for 70% of the bacterial cells in the gut of the termite Coptotermes formosanus. Functional annotation of the chromosome (1,114,206 base pairs) unveiled its ability to fix dinitrogen and recycle putative host nitrogen wastes for biosynthesis of diverse amino acids and cofactors, and import glucose and xylose as energy and carbon sources. Thus, nitrogen fixation and cellulolysis are coupled within the protist's cells. This highly evolved symbiotic system probably underlies the ability of the worldwide pest termites Coptotermes to use wood as their sole food.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Hongoh, Yuichi -- Sharma, Vineet K -- Prakash, Tulika -- Noda, Satoko -- Toh, Hidehiro -- Taylor, Todd D -- Kudo, Toshiaki -- Sakaki, Yoshiyuki -- Toyoda, Atsushi -- Hattori, Masahira -- Ohkuma, Moriya -- New York, N.Y. -- Science. 2008 Nov 14;322(5904):1108-9. doi: 10.1126/science.1165578.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Ecomolecular Biorecycling Science Research Team, RIKEN Advanced Science Institute, Saitama 351-0198, Japan. yhongo@riken.jp〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19008447" target="_blank"〉PubMed〈/a〉
    Keywords: Amino Acids/metabolism ; Animals ; Bacteroidetes/classification/*genetics/isolation & purification/metabolism ; Cellulose/*metabolism ; Chromosomes, Bacterial/genetics ; Digestive System/metabolism/microbiology/parasitology ; Eukaryota/isolation & purification/metabolism/*microbiology ; Fermentation ; Genes, Bacterial ; *Genome, Bacterial ; Glycolysis ; Isoptera/metabolism/*microbiology/parasitology ; Metabolic Networks and Pathways ; Molecular Sequence Data ; Monosaccharides/metabolism ; *Nitrogen Fixation/genetics ; Oxidoreductases/genetics ; Phylogeny ; *Symbiosis ; Wood/metabolism
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 9
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    IP3 receptor types 2 and 3 mediate exocrine secretion underlying energy metabolism (2005)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2005-10-01
    Description: Type 2 and type 3 inositol 1,4,5-trisphosphate receptors (IP3R2 and IP3R3) are intracellular calcium-release channels whose physiological roles are unknown. We show exocrine dysfunction in IP3R2 and IP3R3 double knock-out mice, which caused difficulties in nutrient digestion. Severely impaired calcium signaling in acinar cells of the salivary glands and the pancreas in the double mutants ascribed the secretion deficits to a lack of intracellular calcium release. Despite a normal caloric intake, the double mutants were hypoglycemic and lean. These results reveal IP3R2 and IP3R3 as key molecules in exocrine physiology underlying energy metabolism and animal growth.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Futatsugi, Akira -- Nakamura, Takeshi -- Yamada, Maki K -- Ebisui, Etsuko -- Nakamura, Kyoko -- Uchida, Keiko -- Kitaguchi, Tetsuya -- Takahashi-Iwanaga, Hiromi -- Noda, Tetsuo -- Aruga, Jun -- Mikoshiba, Katsuhiko -- New York, N.Y. -- Science. 2005 Sep 30;309(5744):2232-4.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Calcium Oscillation, International Cooperative Research Project, Japan Science and Technology Agency, Tokyo 108-0071, Japan. afutatsu@brain.riken.jp〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/16195467" target="_blank"〉PubMed〈/a〉
    Keywords: Amylases/secretion ; Animals ; Body Weight ; Calcium/metabolism ; Calcium Channels/genetics/*physiology ; Calcium Signaling ; Carbachol/pharmacology ; Digestion ; Eating ; Energy Intake ; *Energy Metabolism ; Inositol 1,4,5-Trisphosphate Receptors ; Lipase/secretion ; Mice ; Mice, Knockout ; Pancreas, Exocrine/cytology/*secretion ; Receptors, Cytoplasmic and Nuclear/genetics/*physiology ; Saliva/*secretion ; Salivation ; Submandibular Gland/metabolism/secretion ; Trypsinogen/secretion
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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