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  • 1
    Publication Date: 1981-09-18
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Grau, J W -- Hyson, R L -- Maier, S F -- Madden, J 4th -- Barchas, J D -- New York, N.Y. -- Science. 1981 Sep 18;213(4514):1409-11.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7268445" target="_blank"〉PubMed〈/a〉
    Keywords: Analgesia ; Animals ; Electroshock ; Endorphins/antagonists & inhibitors/*physiology ; Naltrexone/pharmacology ; Pain/*physiopathology ; Rats ; Stress, Physiological/*physiopathology ; Tail ; Time Factors
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 2
    Publication Date: 1982-03-19
    Description: The finding that some opioid-mediated forms of stress-induced analgesia are antagonized by hypophysectomy and dexamethasone has led to the suggestion that beta-endorphin, released from the pituitary, may mediate these analgesic reactions. "Long-term analgesia" (an opioid-mediated form of stress-induced analgesia), which is blocked by dexamethasone and hypophysectomy, was also blocked by adrenalectomy and reinstated with corticosterone therapy. Corticosterone is proposed to play a permissive role in long-term analgesia and to be a critical hormone mediating this phenomenon.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉MacLennan, A J -- Drugan, R C -- Hyson, R L -- Maier, S F -- Madden, J 4th -- Barchas, J D -- MH 23861/MH/NIMH NIH HHS/ -- New York, N.Y. -- Science. 1982 Mar 19;215(4539):1530-2.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7063862" target="_blank"〉PubMed〈/a〉
    Keywords: Adrenalectomy ; Analgesia ; Animals ; Corticosterone/*physiology ; Dexamethasone/pharmacology ; Endorphins/*physiology ; Hypophysectomy ; Pain/*physiopathology ; Rats ; Stress, Physiological/*physiopathology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 3
    Publication Date: 1983-08-05
    Description: Rats were given series of escapable shocks, identical inescapable shocks, or no shock. The subjects were reexposed to a small amount of shock 24 hours later, after which an in vitro measure of the cellular immune response was examined. Lymphocyte proliferation in response to the mitogens phytohemagglutinin and concanavalin A was suppressed in the inescapable shock group but not in the escapable shock group. This suggests that the controllability of stressors is critical in modulating immune functioning.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Laudenslager, M L -- Ryan, S M -- Drugan, R C -- Hyson, R L -- Maier, S F -- MH00314/MH/NIMH NIH HHS/ -- New York, N.Y. -- Science. 1983 Aug 5;221(4610):568-70.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6603018" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Concanavalin A/pharmacology ; Electroshock ; Humans ; Immune Tolerance ; *Immunity, Cellular ; Lymphocytes/drug effects/*physiology ; Phytohemagglutinins/pharmacology ; Rats ; Stress, Psychological/*immunology/physiopathology ; T-Lymphocytes/drug effects/physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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