Publication Date:
2016-12-08
Description:
Key Points Bcl-xL is a substrate for active caspase-3 in the exosomes. Molecular or chemical inhibition of exosomal Bcl-xL cleavage attenuates uptake of exosomes by hematological malignant cells.
Print ISSN:
0006-4971
Electronic ISSN:
1528-0020
Topics:
Biology
,
Medicine
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