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  • Chemistry  (379)
  • 550 - Earth sciences  (59)
  • Humans  (13)
  • Animals  (12)
  • 2000-2004  (53)
  • 1995-1999  (225)
  • 1975-1979  (182)
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  • 1
    Electronic Resource
    Electronic Resource
    Übergangsmetall-Carbin-Komplexe, XVII. Ringsubstituierte trans-Bromotetracarbonyl(phenylcarbin)-Komplexe von Chrom und Wolfram (1977)
    Weinheim : Wiley-Blackwell
    Berichte der deutschen chemischen Gesellschaft 110 (1977), S. 53-66 
    Details
    ISSN: 0009-2940
    Keywords: Chemistry ; Inorganic Chemistry
    Source: Wiley InterScience Backfile Collection 1832-2000
    Topics: Chemistry and Pharmacology
    Description / Table of Contents: Transition Metal Carbyne Complexes, XVII. Ring-substituted trans-Bromotetracarbonyl(phenylcarbyne) Complexes of Chromium and TungstenThe preparation and spectroscopic properties of a series of ring-substituted trans-bromotetra-carbonyl(phenylcarbyne) complexes Br(CO)4MCR [M = Cr, W; R = C6H4N(CH3)2-(p), C6H4CH3-(p), C6H4OCH3-(p), C6H2(CH3)3-(2,4,6), C6H4CF3-(p), C6H4C6H5-(o), C6H3Cl2-(2,6), C6Cl5] are described. The influence of the substituents on the vCO absorptions and on the 13C n. m. r. signals is discussed. The X-ray structure analysis of trans-Br(CO)4CrCC6H4CF3-(p) reveals the extremely short Cr—Ccarbyne bond length of 168 ± 2 pm.
    Notes: Die Darstellung und die spektroskopischen Eigenschaften einer Reihe von ringsubstituierten trans-Bromotetracarbonyl(phenylcarbin)-Komplexen Br(CO)4MCR [M = Cr, W; R = C6H4N(CH3)2-(p), C6H4CH3-(p), C6H4OCH3-(p), C6H2(CH3)3-(2,4,6), C6H4CF3-(p), C6H4C6H5-(o), C6H3Cl2-(2,6), C6Cl5] werden beschrieben und der Einfluß der Substituenten auf die vCO-Absorptionen und die 13C-NMR-Signale diskutiert. Die Röntgenstrukturanalyse von trans-Br(CO)4CrCC6H4CF3-(p) ergibt für die Cr—CCarbin-Bindung den extrem kurzen Abstand von 168 ± 2 pm.
    Additional Material: 5 Tab.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1002/cber.19771100107
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  • 2
    Electronic Resource
    Electronic Resource
    P/O Ligand Systems: Facile Synthesis, Structure, and Catalytic Tests of 2′-Phosphanyl-1,1′ -phenyl-2-ols and 2′-Phosphanyl-1,1′-binaphthyl-2-ols (1997)
    Weinheim : Wiley-Blackwell
    Berichte der deutschen chemischen Gesellschaft 130 (1997), S. 1663-1670 
    Details
    ISSN: 0009-2940
    Keywords: Lithiation ; P ligands ; NMR spectroscopy ; Molecular structure ; Hydrogenations ; Hydroformylations ; Chemistry ; Inorganic Chemistry
    Source: Wiley InterScience Backfile Collection 1832-2000
    Topics: Chemistry and Pharmacology
    Notes: A facile synthesis of 2′-phosphanyl-1,1′-biphenyl- and 2′-phosphanyl-1,1′ -binaphthyl-2-ols and their silyl ethers has been developed, consisting of electron-transfer-catalyzed ring-opening of dibenzofuran and dinaphthofuran, respectively, subsequent reaction with chlorophosphanes, and work-up with acetic acid or ClSiMe3. Studies of the molecular and crystal structures reveals the presence of P - H - O bridging bonds in the more basic tBuPhP derivative and a nearly perpendicular arrangement of the aryl planes in the biphenyl derivatives. The barrier to rotation of the aryl planes about the C-C axis was determined by NMR in the case of the P-asymmetric derivative 3d, using the appearence of diastereoisomers by atropisomerism and P-asymmetry. Comparative screening tests of the title compounds, phosphanyl phenols and phosphanylnaphthols in homogeneous Rh-catalyzed reactions demonstrate catalytic activity in hydroformylation reactions and superior properties of the biphenyl- and binaphthyl-2-ol derivatives in relation to other P-O ligands.
    Additional Material: 3 Ill.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1002/cber.19971301118
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  • 3
    Electronic Resource
    Electronic Resource
    Rubber toughening of polystyrene-acrylonitrile copolymers (1997)
    Weinheim : Wiley-Blackwell
    Acta Polymerica 48 (1997), S. 553-561 
    Details
    ISSN: 0323-7648
    Keywords: Chemistry ; Polymer and Materials Science
    Source: Wiley InterScience Backfile Collection 1832-2000
    Topics: Chemistry and Pharmacology , Physics
    Notes: The efficiency of rubber toughening of PSAN depends on the size of the rubber particles, their agglomeration, the deformation rate, the temperature, and the orientation of the polymer molecules. Large particles are more effective than small particles. By a suitable choice of processing, however, small particles can agglomerate, forming large soft units and improving in this way impact toughness. At high deformation rates crazing or/and stretching of the matrix wall between the rubber particles must be activated for ductility, otherwise the material is brittle. The temperature at impact must be above the glass transition temperature of the rubbery phase for toughening. Increasing the orientation of the material decreases the tendency for craze formation with the consequence of embrittlement, if stretching is not activated.
    Additional Material: 14 Ill.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1002/actp.1997.010481205
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  • 4
    Electronic Resource
    Electronic Resource
    Spontane Umlagerung Carben-/Carbin-Komplex: C, Cr-Wanderung einer Sn(C6H5)3-GruppeProfessor Kurt Issleib zum 60. Geburtstag gewidmetRoyal Society-Stipendiat. Ständige Adresse: Canol House, School Lane, Cutnall Green, Nr. Droitwich (England). (1979)
    Weinheim : Wiley-Blackwell
    Zeitschrift für die chemische Industrie 91 (1979), S. 929-930 
    Details
    ISSN: 0044-8249
    Keywords: Chemistry ; General Chemistry
    Source: Wiley InterScience Backfile Collection 1832-2000
    Topics: Chemistry and Pharmacology
    Additional Material: 1 Ill.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1002/ange.19790911117
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  • 5
    Electronic Resource
    Electronic Resource
    Spontaneous Rearrangement of a Carbene- to a Carbyne Complex: C,Cr Migration of a Sn(C6H5)3 GroupTransition Metal-Carbyne Complexes, Part 55.-Part 54: E. O. Fischer, F. J. Gammel, J. O. Besenhard, A. Frank, D. Neugebauer, J. Organomet. Chem., in press.Dedicated to Professor Kurt Issleib on the occasion of his 60th birthday (1979)
    Weinheim : Wiley-Blackwell
    Angewandte Chemie International Edition in English 18 (1979), S. 871-871 
    Details
    ISSN: 0570-0833
    Keywords: Chemistry ; General Chemistry
    Source: Wiley InterScience Backfile Collection 1832-2000
    Topics: Chemistry and Pharmacology
    Additional Material: 1 Ill.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1002/anie.197908711
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  • 6
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    Science over politics (1999)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1999-04-17
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Lanza, R P -- Arrow, K J -- Axelrod, J -- Baltimore, D -- Benacerraf, B -- Bloch, K E -- Bloembergen, N -- Brown, H C -- Brown, M S -- Cibelli, J B -- Cohen, S -- Cooper, L N -- Corey, E J -- Dulbecco, R -- Fischer, E H -- Fitch, V L -- Friedmen, M -- Friedman, M -- Furchgott, R F -- Gell-Mann, M -- Glaser, D A -- Glashow, S L -- Gilbert, W -- Goldstein, J L -- Wilson, R W -- New York, N.Y. -- Science. 1999 Mar 19;283(5409):1849-50.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/10206888" target="_blank"〉PubMed〈/a〉
    Keywords: *Bioethics ; Biomedical Research ; *Embryo Research ; Embryo, Mammalian/*cytology ; Federal Government ; Government Regulation ; Humans ; Politics ; Research/*legislation & jurisprudence ; Research Support as Topic/legislation & jurisprudence ; *Risk Assessment ; *Stem Cells ; United States ; United States Dept. of Health and Human Services
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 7
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    Perforin gene defects in familial hemophagocytic lymphohistiocytosis (1999)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1999-12-03
    Description: Familial hemophagocytic lymphohistiocytosis (FHL) is a rare, rapidly fatal, autosomal recessive immune disorder characterized by uncontrolled activation of T cells and macrophages and overproduction of inflammatory cytokines. Linkage analyses indicate that FHL is genetically heterogeneous and linked to 9q21.3-22, 10q21-22, or another as yet undefined locus. Sequencing of the coding regions of the perforin gene of eight unrelated 10q21-22-linked FHL patients revealed homozygous nonsense mutations in four patients and missense mutations in the other four patients. Cultured lymphocytes from patients had defective cytotoxic activity, and immunostaining revealed little or no perforin in the granules. Thus, defects in perforin are responsible for 10q21-22-linked FHL. Perforin-based effector systems are, therefore, involved not only in the lysis of abnormal cells but also in the down-regulation of cellular immune activation.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Stepp, S E -- Dufourcq-Lagelouse, R -- Le Deist, F -- Bhawan, S -- Certain, S -- Mathew, P A -- Henter, J I -- Bennett, M -- Fischer, A -- de Saint Basile, G -- Kumar, V -- New York, N.Y. -- Science. 1999 Dec 3;286(5446):1957-9.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Pathology and the Graduate Program in Immunology, University of Texas Southwestern Medical School, Dallas, TX 75235, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/10583959" target="_blank"〉PubMed〈/a〉
    Keywords: Antigen-Presenting Cells/immunology ; Cell Death ; Cell Line ; Cells, Cultured ; Chromosome Mapping ; Chromosomes, Human, Pair 10/*genetics ; Codon, Terminator ; Cytoplasmic Granules/chemistry ; Cytotoxicity, Immunologic ; Frameshift Mutation ; Genetic Linkage ; Granzymes ; Heterozygote ; Histiocytosis, Non-Langerhans-Cell/*genetics/immunology ; Humans ; Lymphocyte Activation ; Membrane Glycoproteins/analysis/*genetics/physiology ; Mutation, Missense ; Perforin ; Point Mutation ; Pore Forming Cytotoxic Proteins ; Serine Endopeptidases/analysis ; T-Lymphocytes, Cytotoxic/chemistry/immunology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 8
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    Translocation of Helicobacter pylori CagA into gastric epithelial cells by type IV secretion (2000)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2000-02-26
    Description: The Gram-negative bacterium Helicobacter pylori is a causative agent of gastritis and peptic ulcer disease in humans. Strains producing the CagA antigen (cagA(+)) induce strong gastric inflammation and are strongly associated with gastric adenocarcinoma and MALT lymphoma. We show here that such strains translocate the bacterial protein CagA into gastric epithelial cells by a type IV secretion system, encoded by the cag pathogenicity island. CagA is tyrosine-phosphorylated and induces changes in the tyrosine phosphorylation state of distinct cellular proteins. Modulation of host cells by bacterial protein translocation adds a new dimension to the chronic Helicobacter infection with yet unknown consequences.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Odenbreit, S -- Puls, J -- Sedlmaier, B -- Gerland, E -- Fischer, W -- Haas, R -- New York, N.Y. -- Science. 2000 Feb 25;287(5457):1497-500.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Max von Pettenkofer Institute for Hygiene and Medical Microbiology, Ludwig-Maximilians University Munich, D-80336 Munich, Germany.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/10688800" target="_blank"〉PubMed〈/a〉
    Keywords: Amino Acid Motifs ; *Antigens, Bacterial ; Bacterial Proteins/genetics/*metabolism ; Biological Transport ; Enzyme Inhibitors/pharmacology ; Epithelial Cells/metabolism/microbiology ; Fluorescent Antibody Technique ; Gastric Mucosa/*metabolism/*microbiology ; Genes, Bacterial ; Genetic Complementation Test ; Genistein/pharmacology ; Helicobacter pylori/genetics/*metabolism/pathogenicity ; Humans ; Mutation ; Phosphorylation ; Phosphotyrosine/metabolism ; Protein-Tyrosine Kinases/antagonists & inhibitors/metabolism ; Staurosporine/pharmacology ; Tumor Cells, Cultured ; Virulence
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 9
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    Impairment of mycobacterial immunity in human interleukin-12 receptor deficiency (1998)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1998-06-20
    Description: In humans, interferon gamma (IFN-gamma) receptor deficiency leads to a predisposition to mycobacterial infections and impairs the formation of mature granulomas. Interleukin-12 (IL-12) receptor deficiency was found in otherwise healthy individuals with mycobacterial infections. Mature granulomas were seen, surrounded by T cells and centered with epithelioid and multinucleated giant cells, yet reduced IFN-gamma concentrations were found to be secreted by activated natural killer and T cells. Thus, IL-12-dependent IFN-gamma secretion in humans seems essential in the control of mycobacterial infections, despite the formation of mature granulomas due to IL-12-independent IFN-gamma secretion.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Altare, F -- Durandy, A -- Lammas, D -- Emile, J F -- Lamhamedi, S -- Le Deist, F -- Drysdale, P -- Jouanguy, E -- Doffinger, R -- Bernaudin, F -- Jeppsson, O -- Gollob, J A -- Meinl, E -- Segal, A W -- Fischer, A -- Kumararatne, D -- Casanova, J L -- New York, N.Y. -- Science. 1998 May 29;280(5368):1432-5.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉INSERM U429, Hopital Necker-Enfants Malades, Paris 75015, France.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/9603732" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Cytotoxicity, Immunologic ; Female ; Granuloma/immunology ; Humans ; Hypersensitivity, Delayed ; Interferon-gamma/biosynthesis/immunology/secretion ; Interleukin-12/*immunology ; Killer Cells, Natural/immunology ; Lymphocyte Activation ; Male ; Mice ; Mice, Knockout ; Mutation ; Mycobacterium avium-intracellulare Infection/*immunology ; *Mycobacterium bovis ; Pedigree ; Receptors, Interferon/genetics/immunology ; Receptors, Interleukin/deficiency/*genetics ; Receptors, Interleukin-12 ; T-Lymphocytes/immunology ; Tuberculosis/*immunology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 10
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    Gene therapy of human severe combined immunodeficiency (SCID)-X1 disease (2000)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2000-04-28
    Description: Severe combined immunodeficiency-X1 (SCID-X1) is an X-linked inherited disorder characterized by an early block in T and natural killer (NK) lymphocyte differentiation. This block is caused by mutations of the gene encoding the gammac cytokine receptor subunit of interleukin-2, -4, -7, -9, and -15 receptors, which participates in the delivery of growth, survival, and differentiation signals to early lymphoid progenitors. After preclinical studies, a gene therapy trial for SCID-X1 was initiated, based on the use of complementary DNA containing a defective gammac Moloney retrovirus-derived vector and ex vivo infection of CD34+ cells. After a 10-month follow-up period, gammac transgene-expressing T and NK cells were detected in two patients. T, B, and NK cell counts and function, including antigen-specific responses, were comparable to those of age-matched controls. Thus, gene therapy was able to provide full correction of disease phenotype and, hence, clinical benefit.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Cavazzana-Calvo, M -- Hacein-Bey, S -- de Saint Basile, G -- Gross, F -- Yvon, E -- Nusbaum, P -- Selz, F -- Hue, C -- Certain, S -- Casanova, J L -- Bousso, P -- Deist, F L -- Fischer, A -- New York, N.Y. -- Science. 2000 Apr 28;288(5466):669-72.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉INSERM Unit 429, Gene Therapy Laboratory, Cell Therapy Laboratory, Unite d'Immunologie et d'Hematologie Pediatriques, Hopital Necker, 75743 Paris Cedex 15, France.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/10784449" target="_blank"〉PubMed〈/a〉
    Keywords: Antigens, CD34/analysis ; B-Lymphocytes/immunology ; Gene Transfer Techniques ; *Genetic Therapy ; Genetic Vectors ; Hematopoietic Stem Cell Transplantation ; *Hematopoietic Stem Cells/cytology ; Humans ; Immunoglobulins/blood ; Infant ; Killer Cells, Natural/immunology ; Lymphocyte Activation ; Lymphocyte Count ; Moloney murine leukemia virus/genetics ; Mutation ; Receptors, Antigen, T-Cell/analysis ; Receptors, Interleukin/biosynthesis/*genetics ; Severe Combined Immunodeficiency/genetics/*therapy ; T-Lymphocyte Subsets/immunology ; T-Lymphocytes/immunology ; Transgenes
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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