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  • Articles  (135)
  • lipid peroxidation  (135)
  • Springer  (135)
  • American Institute of Physics
  • Nature Publishing Group
  • 1990-1994  (135)
  • Medicine  (135)
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  • Articles  (135)
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  • Springer  (135)
  • American Institute of Physics
  • Nature Publishing Group
  • Wiley-Blackwell  (4)
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  • 1
    Electronic Resource
    Electronic Resource
    The effect of stress factors on the spontaneous photon emission from microorganisms (1992)
    Springer
    Cellular and molecular life sciences 48 (1992), S. 1030-1041 
    Details
    ISSN: 1420-9071
    Keywords: Ultraweak luminescence ; photon emission ; biophoton emission ; mitogenetic radiation ; lipid peroxidation ; yeast ; bacteria
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract The results of recent work on the photon emission from three yeasts and a bacterium is presented. Both visible region and ultraviolet photon emission is observed; however, no luminescence is observed in the absence of oxygen. The visible region emission is attributed to excited carbonyl groups and excited singlet oxygen dimers formed during the decomposition of lipid hydroperoxides. Possible sources of the ultraviolet photon emission are also examined. The use of microorganisms in the study of ultraweak photon emission and its relation to oxidative, temperature and chemical stress is reviewed and the applications and (or) functions of this photon emission are also discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01947991
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  • 2
    Electronic Resource
    Electronic Resource
    Protective effects of hsp70 in inflammation (1994)
    Springer
    Cellular and molecular life sciences 50 (1994), S. 1031-1038 
    Details
    ISSN: 1420-9071
    Keywords: Heat shock proteins ; inflammation ; reactive oxygen species ; nitric oxide ; lipid peroxidation ; tumor necrosis factor α ; interleukin 1 ; adult respiratory distress syndrome
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Inflammation results from the recruitement to a given tissue or organ and the activation of leucocytes, among which the monocytes-macrophages play a major role. These phagocytic cells produce high levels of reactive oxygen species (ROS) as well as cytokines. Whereas both ROS and cytokines have the potential to regulate the expression of heat shock (HS)/stress proteins (HSP), it appears that these proteins in turn have the ability to protect cells and tissues from the deleterious effects of inflammation. The mechanisms by which such protection occurs include prevention of ROS-induced DNA strand breaks and lipid peroxidation as well as protection from mitochondrial structure and function. In vivo, HS protects organs against a number of lesions associated with the increased production of ROS and/or cytokines. In an animal model for adult respiratory distress syndrome, an acute pulmonary inflammatory condition, HS completely prevented mortality. HSP (hsp70 in particular) may also exert protective effects in the immune system by contributing to the processing and presentation of bacterial and tumoral antigens. The analysis of the expression of hsp70 may prove of diagnostic and prognostic value in inflammatory conditions and therapeutical applications are being considered.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01923458
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  • 3
    Electronic Resource
    Electronic Resource
    Role of lipid peroxidation in ferric lactate-enhanced calcium uptake by Ehrlich carcinoma cells (1993)
    Springer
    Cellular and molecular life sciences 49 (1993), S. 335-336 
    Details
    ISSN: 1420-9071
    Keywords: Iron complex ; lipid peroxidation ; calcium uptake enhancement
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Comparison of Ca2+ uptake by Ehrlich carcinoma cells in presence of ferric lactate or aluminum lactate, and formation of thiobarbituric acid-reactive substances, suggests that lipid peroxidation is associated with but not the cause of calcium overload that can lead to cell injury and death.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01923415
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  • 4
    Electronic Resource
    Electronic Resource
    Lipid peroxidation, low-level chemiluminescence and regulation of secretion in the mammary gland (1993)
    Springer
    Cellular and molecular life sciences 49 (1993), S. 870-875 
    Details
    ISSN: 1420-9071
    Keywords: Mammary gland ; regulation of protein secretion ; chemiluminescence ; lipid peroxidation
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract In mammary explants of lactating mice, changes in the intensity of chemiluminescence (CL) were observed after the addition to the incubation medium of hormones and mediators that are involved in the regulation of secretion: oxytocin, acetylcholine, epinephrine and norepinephrine. A 15-min period of treatment with oxytocin, epinephrine or norepinephrine changed the level of thiobarbituric acid-reactive substances (TBARS). Two mammary explants, one of which was treated with oxytocin, acetylcholine, epinephrine or norepinephrine, were found to interact even when separated by a quartz glass wall. Analysis of the level of TBARS formation in these two explants showed that the observed interactions might be connected with light emission resulting from lipid peroxidation (LP) processes. The possible role of LP and low-level CL in the regulation of mammary gland secretion is discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01952600
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  • 5
    Electronic Resource
    Electronic Resource
    Short cerebral ischemia and subsequent reperfusion and treatment with stobadine (1992)
    Springer
    Cellular and molecular life sciences 48 (1992), S. 872-874 
    Details
    ISSN: 1420-9071
    Keywords: Cerebral reperfusion injury ; oxygen radicals ; lipid peroxidation ; antioxidative enzymes ; stobadine
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Lipid peroxidation and activities of antioxidative enzymes were studied in the brain cortex after short (15 min) cerebral ischemia and reperfusion (10 min) in rats. Conjugated dienes (CD) and thiobarbituric acid-reactive substances (TBARS) were significantly elevated in the group of rats with ischemia followed by reperfusion in comparison to the ischemic animals. Superoxide dismutase (SOD) activity significantly increased in the group of animals with ischemia and reperfusion. No significant changes in the activities of glutathione peroxidase (GP) were observed. Stobadine administered before ischemia or before reperfusion decreased the level of TBARS. Stobadine probably prevents malondialdehyde (MDA) formation from hydroperoxide or might elevate the activity of aldehyde dehydrogenase. In contradiction to the findings after long-lasting (4 h) ischemia and subsequent reperfusion1, no decrease in the concentration of CD or in the activity of SOD or GP was found.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02118422
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  • 6
    Electronic Resource
    Electronic Resource
    Age-related lipid peroxidation in the digestive gland of mussels: The role of the antioxidant defence systems (1991)
    Springer
    Cellular and molecular life sciences 47 (1991), S. 454-457 
    Details
    ISSN: 1420-9071
    Keywords: aging ; mussels ; free radicals ; antioxidant systems ; lipid peroxidation
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary The main cellular defence systems against free radical-mediated oxidative stress are significantly reduced in the dige+ive gland of aged (〉10 years old) compared to younger (2–4 years old) mussels (Mytilus edulis L.). Moreover, the concentration of lipid peroxidation products (malondialdehyde) is increased in the same age group with respect to younger animals. The obtained data indicate that an impairment of the antioxidant defence systems would render the older animals more susceptible to peroxidative stress, thus supporting the general significance of the free radical theory of aging.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01959942
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  • 7
    Electronic Resource
    Electronic Resource
    Antiatherosclerotic effect of probucol in WHHL rabbits: Are there plasma parameters to evaluate this effect? (1991)
    Springer
    European journal of clinical pharmacology 40 (1991), S. S77 
    Details
    ISSN: 1432-1041
    Keywords: Atherosclerosis ; lipid peroxidation ; antioxidants ; probucol ; WHHL rabbit
    Source: Springer Online Journal Archives 1860-2000
    Topics: Chemistry and Pharmacology , Medicine
    Notes: Summary Probucol has been used as a lipid-lowering agent for over 10 years. Lately it has been found that its antiatherogenic action is due mainly to its antioxidative capacity, in addition to its known lipid-lowering effect. To study the antioxidative capability of probucol and its influence on plaque developement we used the animal model of the LDL-receptor-defective Watanabe heritable hyperlipidemic (WHHL) rabbit. In this study we measured all lipid values before and after probucol feeding and compared them with corresponding values in untreated controls. Probucol levels were determined, as were the physiological antioxidants alpha and gamma tocopherol (vitamin E). Thiobarbituric reactive substances were measured in plasma as a parameter for lipid peroxidation. In addition to the biochemical measurements the plaque area was analyzed macroscopically and microscopically to check the antiatherosclerotic effect and correlate it with the biochemical parameters. In four experiments we showed that probucol treatment in WHHL rabbits decreases the progression of atherosclerotic plaques by way of a combined lipid-lowering and antioxidative effect.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01409414
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  • 8
    Electronic Resource
    Electronic Resource
    Effect of pentoxifylline on erythrocyte membrane and plasma lipids (1991)
    Springer
    European journal of clinical pharmacology 41 (1991), S. 623-624 
    Details
    ISSN: 1432-1041
    Keywords: Pentoxifylline ; erythrocytes ; membrane lipids ; plasma lipids ; lipid peroxidation ; erythrocyte deformability
    Source: Springer Online Journal Archives 1860-2000
    Topics: Chemistry and Pharmacology , Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00315000
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  • 9
    Electronic Resource
    Electronic Resource
    In vitro effects of reactive O2 species on the β-receptor-adenylyl cyclase system (1992)
    Springer
    Molecular and cellular biochemistry 110 (1992), S. 41-46 
    Details
    ISSN: 1573-4919
    Keywords: adenylyl cyclase ; antioxidants ; catecholamines ; lipid peroxidation ; reactive oxygen species ; β-receptor
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology , Medicine
    Notes: Abstract The irreversible loss of activity of the sarcolemma-localized β-receptor-adenylyl cyclase system (β-RAS) in myocardial ischemia is a well documented phenomenon. Alterations in the sarcolemma (SL) induced by reactive O2 species could be responsible for this loss. Therefore the influence of oxidation of SH-groups and lipid peroxidation induced by Fe2+/Vit. C on the β-RAS activity was studied. During incubation of SL with Fe2+/Vit. C a transient enhancement followed by a continuous loss of the β-RAS activity (isoprenaline-, NaF-, Gpp(NH)p-, forskolin-stimulated and basal activity) was observed. In contrast there occurred a continuous loss of SH-groups and lipid peroxidation, beginning immediately after the start of incubation. Loss of SH-groups and lipid peroxidation as well as changes in the β-RAS did not take place in the presence of the antioxidant t-Butyl-4-hydroxyanisole (BHA) or the Fe2+-chelator EGTA. In view of the known ischemia-induced formation of reactive O2 species our results show that these powerful oxidants could contribute to the modulation of the β-RAS during myocardial ischemia.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02385004
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  • 10
    Electronic Resource
    Electronic Resource
    Retardation of cell aging by lipid peroxidation (1992)
    Springer
    Molecular and cellular biochemistry 115 (1992), S. 59-62 
    Details
    ISSN: 1573-4919
    Keywords: lipid peroxidation ; cell aging ; life span ; population doubling ; subculture interval ; DNA synthesis
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology , Medicine
    Notes: Abstract Different concentrations of Fe e+/vitamin C mixtures were used as initiators of lipid peroxidation in diploid fibroblasts from cultured human embryonic lung. Malondialdehyde (MDA) formation in the cell cultures was correlated directly with the concentrations of Fe2+ and vitamine C. Lipid peroxidation was associated with an increase in life-span, decrease in the population doubling time and increase in cellular DNA synthesis. The effects of lipid peroxidation varied inversely with the MDA level. These data showed that low levels of lipid peroxidation retarded several biological properties of cultured cells that are associated with cell aging.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00229096
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