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  • 1
    Monograph available for loan
    Monograph available for loan
    Tectonics of Eastern Asia and Western Pacific continental margin : selected papers presented at the 1988 DELP Symposium in Tokyo, Japan, 13-16 December 1988 (1990)
    Amsterdam : Elsevier
    Associated volumes
    Details Availability
    Call number: ILP/M 06.0267
    In: Publication of the International Lithosphere Programme
    In: DELP Publication
    In: Tectonophysics
    Type of Medium: Monograph available for loan
    Pages: ix, 371 S. : Ill., graph. Darst.
    Series Statement: Publication of the International Lithosphere Programme 162
    Location: Reading room
    Branch Library: GFZ Library
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  • 2
    Monograph available for loan
    Monograph available for loan
    Solid earth sciences (1985)
    Tokyo
    Associated volumes
    Details Availability
    Call number: ILP/M 06.0433
    In: Publication of the International Lithosphere Programme
    In: DELP Publication
    Type of Medium: Monograph available for loan
    Pages: 99 S. : graph. Darst.
    Series Statement: [Publication of the International Lithosphere Programme] 3
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  • 3
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    SLAMF3 enhances CD4+ T-cell sensitivity to IL-2 [Immunology and Inflammation] (2016)
    National Academy of Sciences
    Details
    Publication Date: 2016-08-17
    Description: Signaling lymphocytic activation molecule family 3 (SLAMF3/Ly9) is a coregulatory molecule implicated in T-cell activation and differentiation. Systemic lupus erythematosus (SLE) is characterized by aberrant T-cell activation and compromised IL-2 production, leading to abnormal regulatory T-cell (Treg) development/function. Here we show that SLAMF3 functions as a costimulator on CD4+ T...
    Print ISSN: 0027-8424
    Electronic ISSN: 1091-6490
    Topics: Biology , Medicine , Natural Sciences in General
    Published by National Academy of Sciences
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  • 4
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    Gyro-viscosity and linear dispersion relations in pair-ion magnetized plasmas (2015)
    American Institute of Physics (AIP)
    Details
    Publication Date: 2015-11-03
    Description: A fluid theory has been developed by taking account of gyro-viscosity to study wave propagation characteristics in a homogeneous pair-ion magnetized plasma with a cylindrical symmetry. The exact dispersion relations derived by the Hankel-Fourier transformation are shown comparable with those observed in the experiment by Oohara and co-workers. The gyro-viscosity is responsible for the change in propagation characteristics of the ion cyclotron wave from forward to backward by suppressing the effect of the thermal pressure which normally causes the forward nature of dispersion. Although the experiment has been already explained by a kinetic theory by the present authors, the kinetic derivations are so involved because of exact particle orbits in phase space, finite Lamor radius effects, and higher order ion cyclotron resonances. The present fluid theory provides a simple and transparent structure to the dispersion relations since the gyro-viscosity is renormalized into the ion cyclotron frequency which itself indicates the backward nature of dispersion. The usual disadvantage of a fluid theory, which treats only fundamental modes of eigen-waves excited in a system and is not able to describe higher harmonics that a kinetic theory does, is compensated by simple derivations and clear picture based on the renormalization of the gyro-viscosity.
    Print ISSN: 1070-664X
    Electronic ISSN: 1089-7674
    Topics: Physics
    Published by American Institute of Physics (AIP)
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  • 5
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    Involvement of SLX4 in interstrand cross-link repair is regulated by the Fanconi anemia pathway [Cell Biology] (2011)
    National Academy of Sciences
    Details
    Publication Date: 2011-04-20
    Description: Interstrand cross-links (ICLs) block replication and transcription and thus are highly cytotoxic. In higher eukaryotes, ICLs processing involves the Fanconi anemia (FA) pathway and homologous recombination. Stalled replication forks activate the eight-subunit FA core complex, which ubiquitylates FANCD2-FANCI. Once it is posttranslationally modified, this heterodimer recruits downstream members of the ICL repairosome, including the FAN1 nuclease. However, ICL processing has been shown to also involve MUS81-EME1 and XPF-ERCC1, nucleases known to interact with SLX4, a docking protein that also can bind another nuclease, SLX1. To investigate the role of SLX4 more closely, we disrupted the SLX4 gene in avian DT40 cells. SLX4 deficiency caused cell death associated with extensive chromosomal aberrations, including a significant fraction of isochromatid-type breaks, with sister chromatids broken at the same site. SLX4 thus appears to play an essential role in cell proliferation, probably by promoting the resolution of interchromatid homologous recombination intermediates. Because ubiquitylation plays a key role in the FA pathway, and because the N-terminal region of SLX4 contains a ubiquitin-binding zinc finger (UBZ) domain, we asked whether this domain is required for ICL processing. We found that SLX4−/− cells expressing UBZ-deficient SLX4 were selectively sensitive to ICL-inducing agents, and that the UBZ domain was required for interaction of SLX4 with ubiquitylated FANCD2 and for its recruitment to DNA-damage foci generated by ICL-inducing agents. Our findings thus suggest that ubiquitylated FANCD2 recruits SLX4 to DNA damage sites, where it mediates the resolution of recombination intermediates generated during the processing of ICLs.
    Print ISSN: 0027-8424
    Electronic ISSN: 1091-6490
    Topics: Biology , Medicine , Natural Sciences in General
    Published by National Academy of Sciences
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  • 6
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    Electrostatic Ion Cyclotron and Ion Plasma Waves in a Symmetric Pair-Ion Plasma Cylinder (2014)
    American Physical Society (APS)
    Details
    Publication Date: 2014-03-13
    Description: Author(s): M. Kono, J. Vranjes, and N. Batool Complicated wave behavior observed in the cylindrical pair-ion (fullerene) experiments by Oohara and co-workers are now identified to be low harmonic ion cyclotron waves combined with ion plasma oscillations inherent to kinetic theory. The electrostatic dispersion equation derived is based on an app... [Phys. Rev. Lett. 112, 105001] Published Wed Mar 12, 2014
    Keywords: Plasma and Beam Physics
    Print ISSN: 0031-9007
    Electronic ISSN: 1079-7114
    Topics: Physics
    Published by American Physical Society (APS)
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  • 7
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    Energy in density gradient (2015)
    American Institute of Physics (AIP)
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    Publication Date: 2015-01-16
    Description: Inhomogeneous plasmas and fluids contain energy stored in inhomogeneity and they naturally tend to relax into lower energy states by developing instabilities or by diffusion. But the actual amount of energy in such inhomogeneities has remained unknown. In the present work, the amount of energy stored in a density gradient is calculated for several specific density profiles in a cylindrical configuration. This is of practical importance for drift wave instability in various plasmas, and, in particular, in its application in models dealing with the heating of solar corona because the instability is accompanied with stochastic heating, so the energy contained in inhomogeneity is effectively transformed into heat. It is shown that even for a rather moderate increase of the density at the axis in magnetic structures in the corona by a factor 1.5 or 3, the amount of excess energy per unit volume stored in such a density gradient becomes several orders of magnitude greater than the amount of total energy losses per unit volume (per second) in quiet regions in the corona. Consequently, within the life-time of a magnetic structure such energy losses can easily be compensated by the stochastic drift wave heating.
    Print ISSN: 1070-664X
    Electronic ISSN: 1089-7674
    Topics: Physics
    Published by American Institute of Physics (AIP)
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  • 8
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    Geophysics. Ships' logs and archeomagnetism (2006)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2006-05-13
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Kono, Masaru -- New York, N.Y. -- Science. 2006 May 12;312(5775):865-6.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Tokyo Institute of Technology, Tokyo, 152-8551, Japan. masarukono@nifty.com〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/16690853" target="_blank"〉PubMed〈/a〉
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 9
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    Compressed glassy carbon: An ultrastrong and elastic interpenetrating graphene network (2017)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2017-06-10
    Description: Carbon’s unique ability to have both sp 2 and sp 3 bonding states gives rise to a range of physical attributes, including excellent mechanical and electrical properties. We show that a series of lightweight, ultrastrong, hard, elastic, and conductive carbons are recovered after compressing sp 2 -hybridized glassy carbon at various temperatures. Compression induces the local buckling of graphene sheets through sp 3 nodes to form interpenetrating graphene networks with long-range disorder and short-range order on the nanometer scale. The compressed glassy carbons have extraordinary specific compressive strengths—more than two times that of commonly used ceramics—and simultaneously exhibit robust elastic recovery in response to local deformations. This type of carbon is an optimal ultralight, ultrastrong material for a wide range of multifunctional applications, and the synthesis methodology demonstrates potential to access entirely new metastable materials with exceptional properties.
    Electronic ISSN: 2375-2548
    Topics: Natural Sciences in General
    Published by American Association for the Advancement of Science (AAAS)
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  • 10
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    HDL-bound sphingosine-1-phosphate restrains lymphopoiesis and neuroinflammation (2015)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2015-06-09
    Description: Lipid mediators influence immunity in myriad ways. For example, circulating sphingosine-1-phosphate (S1P) is a key regulator of lymphocyte egress. Although the majority of plasma S1P is bound to apolipoprotein M (ApoM) in the high-density lipoprotein (HDL) particle, the immunological functions of the ApoM-S1P complex are unknown. Here we show that ApoM-S1P is dispensable for lymphocyte trafficking yet restrains lymphopoiesis by activating the S1P1 receptor on bone marrow lymphocyte progenitors. Mice that lacked ApoM (Apom(-/-)) had increased proliferation of Lin(-) Sca-1(+) cKit(+) haematopoietic progenitor cells (LSKs) and common lymphoid progenitors (CLPs) in bone marrow. Pharmacological activation or genetic overexpression of S1P1 suppressed LSK and CLP cell proliferation in vivo. ApoM was stably associated with bone marrow CLPs, which showed active S1P1 signalling in vivo. Moreover, ApoM-bound S1P, but not albumin-bound S1P, inhibited lymphopoiesis in vitro. Upon immune stimulation, Apom(-/-) mice developed more severe experimental autoimmune encephalomyelitis, characterized by increased lymphocytes in the central nervous system and breakdown of the blood-brain barrier. Thus, the ApoM-S1P-S1P1 signalling axis restrains the lymphocyte compartment and, subsequently, adaptive immune responses. Unique biological functions imparted by specific S1P chaperones could be exploited for novel therapeutic opportunities.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4506268/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4506268/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Blaho, Victoria A -- Galvani, Sylvain -- Engelbrecht, Eric -- Liu, Catherine -- Swendeman, Steven L -- Kono, Mari -- Proia, Richard L -- Steinman, Lawrence -- Han, May H -- Hla, Timothy -- F32 CA14211/CA/NCI NIH HHS/ -- F32 CA142117/CA/NCI NIH HHS/ -- HL67330/HL/NHLBI NIH HHS/ -- HL70694/HL/NHLBI NIH HHS/ -- HL89934/HL/NHLBI NIH HHS/ -- P01 HL070694/HL/NHLBI NIH HHS/ -- P20 RR017677/RR/NCRR NIH HHS/ -- P30 CA138313/CA/NCI NIH HHS/ -- R01 HL089934/HL/NHLBI NIH HHS/ -- R37 HL067330/HL/NHLBI NIH HHS/ -- Z01 DK056014-02/Intramural NIH HHS/ -- Z01 DK056015-01/Intramural NIH HHS/ -- England -- Nature. 2015 Jul 16;523(7560):342-6. doi: 10.1038/nature14462. Epub 2015 Jun 8.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉1] Center for Vascular Biology, Department of Pathology and Laboratory Medicine, Weill Medical College of Cornell University, New York, New York 10065, USA [2] Brain and Mind Research Institute, Weill Medical College of Cornell University, New York, New York 10065, USA. ; Center for Vascular Biology, Department of Pathology and Laboratory Medicine, Weill Medical College of Cornell University, New York, New York 10065, USA. ; Genetics of Development and Disease Branch, National Institute of Diabetes and Digestive and Kidney Diseases, NIH, Bethesda, Maryland 20892, USA. ; Department of Neurology and Neurological Sciences, Stanford University, Stanford, California 94305, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/26053123" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Apolipoproteins/deficiency/genetics/*metabolism ; Blood-Brain Barrier/pathology ; Cell Movement ; Cell Proliferation/genetics ; Central Nervous System/immunology/metabolism/*pathology ; Encephalomyelitis, Autoimmune, ; Experimental/genetics/immunology/metabolism/pathology ; Female ; Fingolimod Hydrochloride/pharmacology ; Hematopoietic Stem Cells/cytology/metabolism ; Inflammation/immunology/metabolism/pathology ; Lipoproteins, HDL/*metabolism ; Lymphocytes/*cytology/immunology/*metabolism ; Lymphoid Progenitor Cells/cytology/metabolism ; *Lymphopoiesis ; Lysophospholipids/agonists/blood/genetics/*metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Protein Binding ; Receptors, Lysosphingolipid/metabolism ; Signal Transduction ; Sphingosine/agonists/*analogs & derivatives/blood/genetics/metabolism
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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