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  • 1
    Electronic Resource
    Electronic Resource
    Kinetics of N-Nitrosation in Oxygenated Nitric Oxide Solutions at Physiological pH: Role of Nitrous Anhydride and Effects of Phosphate and Chloride (1995)
    s.l. : American Chemical Society
    Journal of the American Chemical Society 117 (1995), S. 3933-3939 
    Details
    ISSN: 1520-5126
    Source: ACS Legacy Archives
    Topics: Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1021/ja00119a006
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  • 2
    Electronic Resource
    Electronic Resource
    Co-ordination of protocol and construction: a team approach (1995)
    Bradford : Emerald
    The @TQM magazine 7 (1995), S. 23-34 
    Details
    ISSN: 0954-478X
    Source: Emerald Fulltext Archive Database 1994-2005
    Topics: Economics
    Notes: Offers an overview of the unique situations arising when installinga wafer fabrication facility into a pre-existing structure. Discussionincludes management techniques used to implement a "cleanbuild", while at the same time completing a state-of-the-artfacility in record time. Outlines suggestions for co-ordinatingconstruction phases with upgrades in protocol cleanliness and theorganizational development of a safety and protocol team, composedprimarily of manufacturing personnel. Draws final conclusions and makesrecommendations to provide assistance for those contemplating a similarapproach in constructing a sub-micron, wafer fabrication facility.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1108/09544789510092827
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  • 3
    Electronic Resource
    Electronic Resource
    Hadronic contribution to the photon vacuum polarization: a theoretical estimate (1994)
    Springer
    The European physical journal 63 (1994), S. 71-76 
    Details
    ISSN: 1434-6052
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract A simple model for the hadronic contribution to the photon vacuum polarization function ∏had(q 2), for spacelike momenta, is presented. For small momenta, the two loop contribution from the pseudoscalar meson octet is computed from the chiral Lagrangian. The light quark contribution (which at low momentum gives the $$\mathcal{O}(q^6 )$$ counterterm in the chiral Lagrangian) is calculated within a relativistic constituent quark model incorporating the momentum dependence of the quark mass. The perturbative gluons of QCD are included in a standard fashion. The total result is close to an estimate of ∏had(q 2) that is obtained directly frome + e→hadrons data. We further use our results for ∏had(q 2) to calculate the $$\mathcal{O}(e^4 )$$ hadronic contribution to lepton magnetic moments and to calculate αQED(M Z 2 ). A simpler model of constituent quarks with momentum independent masses gives less favourable results.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01577545
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  • 4
    Electronic Resource
    Electronic Resource
    Erratum: “Design of a Novel Apparatus to Study Nitric Oxide (NO) Inhibition of Platelet Adhesion,” Ann. Biomed. Eng. 26(6):1036–1043, 1998 (1999)
    Springer
    Annals of biomedical engineering 27 (1999), S. 411-411 
    Details
    ISSN: 1573-9686
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine , Technology
    Notes: Abstract PAC99: 8719Uv, 8780-y, 9910+g
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1114/1.184
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  • 5
    Electronic Resource
    Electronic Resource
    Nitric Oxide, Superoxide, and Peroxynitrite Effects on the Insulin Secretion and Viability of βTC3 Cells (2000)
    Springer
    Annals of biomedical engineering 28 (2000), S. 102-109 
    Details
    ISSN: 1573-9686
    Keywords: Nitric oxide ; Superoxide ; Pancreatic cells ; Insulin
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine , Technology
    Notes: Abstract The onset of insulin-dependent diabetes mellitus (IDDM) is often associated with the infiltration of pancreatic cells by macrophages. Upon activation, macrophages release nitric oxide (NO) and superoxide (O 2 - ). These species or their reactive intermediates can be cytoxic, mutagenic, or carcinogenic. Previous studies have reported both positive and negative effects of extracellularly generated NO on insulin secretion and viability of pancreatic cells. Inherent problems of several previous studies assessing the effects of NO on insulin secretion include unsteady state NO concentration exposures and the generation of other potentially damaging species. In this study, these problems were eliminated by using a modified experimental system in which NO delivery was achieved via diffusion across a gas-permeable tube and O 2 - delivery was maintained using an enzymatic reaction. The delivery rates were constant, leading to steady state concentrations of NO and O 2 - in the experimental system. Based on reaction kinetics, a model was developed to predict NO, O 2 - and peroxynitrite ONOO- concentrations during the experiment. This study showed that NO, O 2 - and ONOO- at predicted concentrations as high as 2.8 μM, 0.25 μM, and 0.1 nM, respectively, do not affect the insulin secretion rates of βTC3 pancreatic cells over short times. © 2000 Biomedical Engineering Society. PAC00: 8717-d
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1114/1.258
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  • 6
    Electronic Resource
    Electronic Resource
    Influence of Agonist, Shear Rate, and Perfusion Time on Nitric Oxide Inhibition of Platelet Deposition (2000)
    Springer
    Annals of biomedical engineering 28 (2000), S. 174-181 
    Details
    ISSN: 1573-9686
    Keywords: Thrombosis ; Emboli ; Antagonist
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine , Technology
    Notes: Abstract Nitric oxide (NO) is a physiological species involved in inhibition of platelet adhesion and aggregation. A novel NO delivery device was utilized to quantitatively assess the effects of gaseous NO on platelet deposition to agonist-coated biomaterials in the presence of a platelet suspension. Platelet deposition was evaluated as a function of agonist (collagen, fibrinogen, or IgG), shear rate (250, 500, and 750 s−1), and perfusion time (5, 7.5, and 15 min). The minimal aqueous surface NO concentrations and fluxes necessary for significant inhibition of platelet deposition were quantified. Platelet deposition was completely inhibited at a gaseous NO exposure of 0.1 ppm, irrespective of the platelet agonist, shear rate, and perfusion time. The corresponding aqueous surface NO concentration was 0.09 nM at 250 s−1 as predicted by a validated model. Surface fluxes ranged between 0.3 and 0.6 femtomoles cm−2 s−1. The results of this study are useful for establishing generalized guidelines (i.e., NO flux requirements in the presence of agonists, shear rate, and perfusion time) for the design and development of suitable biomaterials incorporating NO to reduce platelet deposition. Further studies incorporating blood, rather than platelet suspensions, are required to provide a more complete assessment of the required NO flux necessary to inhibit platelet deposition. © 2000 Biomedical Engineering Society. PAC00: 8717-d, 8719Tt
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1114/1.242
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  • 7
    Electronic Resource
    Electronic Resource
    Design of a Novel Apparatus to Study Nitric Oxide (NO) Inhibition of Platelet Adhesion (1998)
    Springer
    Annals of biomedical engineering 26 (1998), S. 1036-1043 
    Details
    ISSN: 1573-9686
    Keywords: Mathematical model ; Perfusion ; Diffusion ; Collagen ; Nitric oxide (NO) inhibition ; Platelet adhesion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine , Technology
    Notes: Abstract Nitric oxide (NO) is a simple biological molecule which inhibits adhesion and aggregation of platelets. A novel NO delivery device has been developed to quantitatively study the effects of NO concentration and flux on the adhesion of platelets to a surface. The slit-flow device is lined with a protein-coated membrane through which NO gas permeates into a perfusing platelet suspension. A model predicting spatial NO concentrations and fluxes within the flow slit was validated. At a wall shear rate of 250s-1, platelet adhesion was inhibited 87% relative to controls for exposures as low as 0.1 ppm NO. Corresponding model predictions of the aqueous NO concentration and fluxes at the surface were 0.15 nM, and between 0.5 and 1.1 nanomoles cm-2 s-1, respectively. Endo-thelial cells, which release NO to inhibit platelet adhesion in vivo, generate NO at an estimated flux similar to the above values. At a NO exposure of 0.02 ppm, platelet inhibition was only 10%. The delivery device is useful for other studies in which a knowledge of the spatial NO fluxes or concentrations is desired. Knowledge of these fluxes or concentrations is beneficial towards the design of biomaterials incorporating NO to inhibit platelet adhesion. © 1998 Biomedical Engineering Society. PAC98: 8745Hw, 8722Fy, 8780+s, 8710+e, 8380Lz
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1114/1.34
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  • 8
    Electronic Resource
    Electronic Resource
    Membrane mass spectrometer inlet for quantitation of nitric oxide (1993)
    Chichester : Wiley-Blackwell
    Biological Mass Spectrometry 22 (1993), S. 45-52 
    Details
    ISSN: 1052-9306
    Keywords: Chemistry ; Analytical Chemistry and Spectroscopy
    Source: Wiley InterScience Backfile Collection 1832-2000
    Topics: Chemistry and Pharmacology
    Notes: Nitric oxide (NO) is an important physiological and biochemical messenger that may be involved in endogenous carcinogenesis and cell toxicity via formation of N-nitroso compounds or direct DNA damage by nitrosating agents arising from the reaction of NO with O2. To study the reaction of NO with O2 in model systems and the formation and disappearance of NO in more physiological systems such as cell cultures, we adapted and optimized a membrane mass spectrometer inlet specifically for such analyses. The inlet consisted of Silastic tubing inserted into a Swagelok ‘tee’, which was attached to the mass spectrometer via the tuning probe. Kinetics of NO disappearance can be followed under electron impact conditions until NO2 interferes via the formation of NO+ during fragmentation of NO2+. The aqueous NO concentration for minimum detection was determined to be 1.4 μM. The inlet response time to step changes in aqueous NO concentrations was 7.0 s, fast enough to permit real-time measurements of aqueous NO changes upon addition of O2. Finally, the depletion of aqueous NO was observed in the presence of O2. The relative steady state responses of inlets designed for gas or aqueous samples, and their relative response times, are explained by an analysis based on mass transfer theory.
    Additional Material: 5 Ill.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1002/bms.1200220106
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  • 9
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    Low-energy constants and condensates from ALEPH hadronicτdecay data (2015)
    American Physical Society
    Details
    Publication Date: 2015-12-01
    Print ISSN: 1550-7998
    Electronic ISSN: 1550-2368
    Topics: Physics
    Published by American Physical Society
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  • 10
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    More about excited bottomonium radiative decays (2012)
    American Physical Society
    Details
    Publication Date: 2012-09-17
    Print ISSN: 1550-7998
    Electronic ISSN: 1550-2368
    Topics: Physics
    Published by American Physical Society
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