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  • 1
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    Activity-dependent transfer of brain-derived neurotrophic factor to postsynaptic neurons (2001)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2001-03-27
    Description: Neurotrophins such as brain-derived neurotrophic factor (BDNF) are thought to be transferred from post- to presynaptic neurons and to be involved in the formation and plasticity of neural circuits. However, direct evidence for a transneuronal transfer of BDNF and its relation to neuronal activity remains elusive. We simultaneously injected complementary DNAs of green fluorescent protein (GFP)-tagged BDNF and red fluorescence protein into the nucleus of single neurons and visualized expression, localization, and transport of BDNF in living neurons. Fluorescent puncta representing BDNF moved in axons in the anterograde direction, though some moved retrogradely, and transferred to postsynaptic neurons in an activity-dependent manner.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Kohara, K -- Kitamura, A -- Morishima, M -- Tsumoto, T -- New York, N.Y. -- Science. 2001 Mar 23;291(5512):2419-23.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Division of Neurophysiology, Biomedical Research Center, Osaka University Graduate School of Medicine, 2-2 Yamadaoka, Suita, Osaka 565-0871 Japan.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/11264540" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Animals, Newborn ; Antibodies ; *Axonal Transport ; Axons/*metabolism ; Brain-Derived Neurotrophic Factor/genetics/*metabolism ; Cell Nucleus/metabolism ; Cells, Cultured ; DNA, Complementary ; Dendrites/metabolism ; Immunohistochemistry ; Luminescent Proteins/metabolism ; Mice ; Mice, Inbred C57BL ; Microscopy, Fluorescence ; Microtubule-Associated Proteins/analysis/immunology ; Neurites/metabolism ; Neuronal Plasticity ; Neurons/drug effects/*metabolism ; Plasmids ; Protein Transport ; Recombinant Fusion Proteins/metabolism ; Synapses/*metabolism ; Tetrodotoxin/pharmacology ; Visual Cortex/cytology ; tau Proteins/analysis/immunology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 2
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    Island cells control temporal association memory (2014)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2014-01-25
    Description: Episodic memory requires associations of temporally discontiguous events. In the entorhinal-hippocampal network, temporal associations are driven by a direct pathway from layer III of the medial entorhinal cortex (MECIII) to the hippocampal CA1 region. However, the identification of neural circuits that regulate this association has remained unknown. In layer II of entorhinal cortex (ECII), we report clusters of excitatory neurons called island cells, which appear in a curvilinear matrix of bulblike structures, directly project to CA1, and activate interneurons that target the distal dendrites of CA1 pyramidal neurons. Island cells suppress the excitatory MECIII input through the feed-forward inhibition to control the strength and duration of temporal association in trace fear memory. Together, the two EC inputs compose a control circuit for temporal association memory.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Kitamura, Takashi -- Pignatelli, Michele -- Suh, Junghyup -- Kohara, Keigo -- Yoshiki, Atsushi -- Abe, Kuniya -- Tonegawa, Susumu -- Howard Hughes Medical Institute/ -- New York, N.Y. -- Science. 2014 Feb 21;343(6173):896-901. doi: 10.1126/science.1244634. Epub 2014 Jan 23.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉RIKEN-MIT Center for Neural Circuit Genetics at the Picower Institute for Learning and Memory, Department of Biology and Department of Brain and Cognitive Sciences, Massachusetts Institute of Technology (MIT), Cambridge, MA 02139, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/24457215" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; *Association ; CA1 Region, Hippocampal/cytology/*physiology ; Entorhinal Cortex/cytology/*physiology ; GABAergic Neurons/physiology ; Interneurons/physiology ; Membrane Proteins/genetics ; *Memory, Episodic ; Mice ; Mice, Inbred C57BL ; Mice, Transgenic ; Nerve Net ; Neurons/*physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 3
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    Structure of forward $pp$ and $p\overline{p}$ elastic amplitudes at low energies (2018)
    American Physical Society (APS)
    Details
    Publication Date: 2018-11-30
    Description: Author(s): E. Ferreira, A. K. Kohara, and J. Sesma Exact analytical forms of solutions for dispersion relations for amplitudes and dispersion relations for slopes are applied in the analysis of p p and p p ¯ scattering data in the forward range at energies below s ≈ 30     GeV . As inputs for the energy dependence of the imaginary part, use is made of analyt... [Phys. Rev. D 98, 094029] Published Thu Nov 29, 2018
    Keywords: Strong Interactions
    Print ISSN: 0556-2821
    Electronic ISSN: 1089-4918
    Topics: Physics
    Published by American Physical Society (APS)
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  • 4
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    Elastic pp[over ¯] scattering amplitude at 1.8 TeV and determination of total cross section (2013)
    American Physical Society (APS)
    Details
    Publication Date: 2013-03-21
    Description: Author(s): A. K. Kohara, E. Ferreira, and T. Kodama The data on pp̅ elastic scattering at 1.8 and 1.96 TeV are analyzed in terms of real and imaginary amplitudes, in a treatment with high accuracy, covering the whole t range, and satisfying the expectation of dispersion relation for amplitudes and for slopes. A method is introduced for the de... [Phys. Rev. D 87, 054024] Published Wed Mar 20, 2013
    Keywords: Strong interactions & Lattice methods
    Print ISSN: 0556-2821
    Electronic ISSN: 1089-4918
    Topics: Physics
    Published by American Physical Society (APS)
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  • 5
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    Exact treatment of dispersion relations in $pp$ and $p\overline{p}$ elastic scattering (2018)
    American Physical Society (APS)
    Details
    Publication Date: 2018-01-27
    Description: Author(s): E. Ferreira, A. K. Kohara, and J. Sesma Proofs are given of exact analytical solutions for general principal value integrations of the dispersion relations of p p and p p ¯ scattering amplitudes. The proofs are based on recent developments in the study of properties of the Lerch's transcendent of the mathematical literature. Dispersion relat... [Phys. Rev. C 97, 014003] Published Fri Jan 26, 2018
    Keywords: Nucleon-Nucleon Interaction, Few-Body Systems
    Print ISSN: 0556-2813
    Electronic ISSN: 1089-490X
    Topics: Physics
    Published by American Physical Society (APS)
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  • 6
    Electronic Resource
    Electronic Resource
    Hypotensive effect associated with a phospholipase C-δ1 gene mutation in the spontaneously hypertensive rat
    Amsterdam : Elsevier
    Biochemical and Biophysical Research Communications 187 (1992), S. 1359-1366 
    Details
    ISSN: 0006-291X
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology , Physics
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1016/0006-291X(92)90452-Q
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  • 7
    Electronic Resource
    Electronic Resource
    Angiotensin(1-7) in the spontaneously hypertensive rat
    Amsterdam : Elsevier
    Peptides 14 (1993), S. 883-891 
    Details
    ISSN: 0196-9781
    Keywords: Angiotensin II ; Angiotensin converting enzyme ; Angiotensin(1-7) ; Ceranapril ; Hypertensive mechanisms ; Lisinopril ; Vasopressin
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0196-9781(93)90063-M
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  • 8
    Electronic Resource
    Electronic Resource
    Reassessment of plasma angiotensins measurement: Effects of protease inhibitors and sample handling procedures
    Amsterdam : Elsevier
    Peptides 12 (1991), S. 1135-1141 
    Details
    ISSN: 0196-9781
    Keywords: Aminopeptidases ; Angiotensin I ; Angiotensin II ; Angiotensin(1-7) ; Angiotensinases ; Converting enzyme ; HPLC ; Prolyl endopeptidase ; Protease inhibitors ; Radioimmunoassays
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0196-9781(91)90070-6
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  • 9
    Electronic Resource
    Electronic Resource
    Association of ACE I/D polymorphism with cardiovascular risk factors (2000)
    Springer
    Human genetics 〈Berlin〉 107 (2000), S. 239-242 
    Details
    ISSN: 1432-1203
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract. Since the identification of an insertion/deletion (I/D) polymorphism in the angiotensin-converting enzyme (ACE) gene, the D allele has been recognized to be associated with cardiovascular disease. Moreover, significant associations of this polymorphism with multiple cardiovascular risk factors have been reported, although some studies failed to detect such associations. In the present study, we investigated the association of the ACE gene polymorphism with the parameters of multiple risk factors in 300 Japanese men who participated in a medical check-up. This investigation detected a significant association of the polymorphism with systolic blood pressure (P=0.007) and diastolic blood pressure (P=0.026), with their highest values in DD subjects and lowest values in II subjects. This significant association is consistent with the proposition that the polymorphism influences blood-pressure variability in men. Furthermore, we investigated the association of the polymorphism with four major disorders (obesity, hyperlipidemia, hypertension, diabetes mellitus) correlated with the risk for cardiovascular disease in the same 300 subjects. This investigation failed to detect any significant association of the polymorphism with each disorder. However, there was a trend that all four disorders were more frequent in ID and DD subjects than in II subjects. We therefore analyzed the association between the ACE gene polymorphism and having at least one of the four disorders in the same population. This analysis detected a significant difference: that ID and DD subjects had at least one of the four disorders more frequently than II subjects (P=0.008; odds ratio=1.89, 95% confidence interval=1.19–2.99). Taken together, the results of this study are compatible with the proposition that the ACE polymorphism is associated with cardiovascular disease partially mediated through the four disorders in our population.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s004390000358
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  • 10
    Electronic Resource
    Electronic Resource
    Helicases and aging (2000)
    Springer
    Cellular and molecular life sciences 57 (2000), S. 716-730 
    Details
    ISSN: 1420-9071
    Keywords: Key words. Helicase disorder; aging; Werner syndrome; Cockayne syndrome; Bloom syndrome; transcription; segmental progeroid syndrome.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract. Studying monogenic hereditary disorders that manifest age-related phenotypes in cells, tissues, and the total organism would be helpful for clarifying the mechanisms of aging. In this context, seven human disorders that manifest age-related phenotypes have been found to be caused by aberrations of five proteins with seven helicase motifs conserved in most of the helicases. These disorders are xeroderma pigmentosum, Cockayne syndrome, trichothiodystrophy, Bloom syndrome, Werner syndrome, X-linked α -thalassemia/mental retardation syndrome, and Juberg-Marsidi syndrome. A decline of probably pleiotropic and fundamental function of helicases in these disorders is, therefore, implied to underlie not only the various age-related phenotypes of the disorders but also the pleiotropic and universal nature of ordinary aging. Consistent with this implication, studies of these seven disorders suggest that their various age-related phenotypes are caused by aberrations in multiple processes, especially transcription. Furthermore, a few studies imply some association between aberration of the helicases and phenotypes in ordinary aging.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s000180050036
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