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  • 1
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    Severe fibronectin-deposit renal glomerular disease in mice lacking uteroglobin (1997)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1997-05-30
    Description: Despite myriads of biological activities ascribed to uteroglobin (UG), a steroid-inducible secreted protein, its physiological functions are unknown. Mice in which the uteroglobin gene was disrupted had severe renal disease that was associated with massive glomerular deposition of predominantly multimeric fibronectin (Fn). The molecular mechanism that normally prevents Fn deposition appears to involve high-affinity binding of UG with Fn to form Fn-UG heteromers that counteract Fn self-aggregation, which is required for abnormal tissue deposition. Thus, UG is essential for maintaining normal renal function in mice, which raises the possibility that an analogous pathogenic mechanism may underlie genetic Fn-deposit human glomerular disease.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Zhang, Z -- Kundu, G C -- Yuan, C J -- Ward, J M -- Lee, E J -- DeMayo, F -- Westphal, H -- Mukherjee, A B -- HL47620/HL/NHLBI NIH HHS/ -- New York, N.Y. -- Science. 1997 May 30;276(5317):1408-12.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Section on Developmental Genetics, Heritable Disorders Branch, National Institute of Child Health and Human Development (NICHD), National Insitutes of Health (NIH), Bethesda, MD 20892-1830, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/9162006" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Cells, Cultured ; Crosses, Genetic ; Fibronectins/*metabolism ; Gene Targeting ; Humans ; Kidney Diseases/embryology/genetics/pathology ; *Kidney Glomerulus/embryology/metabolism/ultrastructure ; Mice ; Mice, Inbred C57BL ; Uteroglobin/deficiency/genetics/*physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 2
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    Maternal ethanol exposure induces transient impairment of umbilical circulation and fetal hypoxia in monkeys (1982)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1982-11-12
    Description: When ethanol was administered intravenously to pregnant monkeys, a transient but marked collapse of umbilical vasculature was observed uniformly within about 15 minutes. The ethanol-induced impairment of umbilical circulation produced severe hypoxia and acidosis in the fetus; recovery occurred during the succeeding hour. This striking interruption of feto-placental circulation may explain one of the mechanisms of mental retardation, a frequent manifestation in children afflicted with fetal alcohol syndrome.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Mukherjee, A B -- Hodgen, G D -- New York, N.Y. -- Science. 1982 Nov 12;218(4573):700-2.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6890235" target="_blank"〉PubMed〈/a〉
    Keywords: Acetaldehyde/blood ; Animals ; Disease Models, Animal ; Ethanol/blood/*pharmacology ; Female ; Fetal Alcohol Spectrum Disorders/*etiology ; Fetal Distress/*chemically induced ; Macaca fascicularis ; Macaca mulatta ; Pregnancy ; Pregnancy, Animal/*drug effects ; Umbilical Cord/*drug effects
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 3
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    In reply: alcohol and pregnancy (1983)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1983-09-23
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Mukherjee, A B -- Hodgen, G D -- New York, N.Y. -- Science. 1983 Sep 23;221(4617):1246.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/17776305" target="_blank"〉PubMed〈/a〉
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 4
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    Suppression of epididymal sperm antigenicity in the rabbit by uteroglobin and transglutaminase in vitro (1983)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1983-02-25
    Description: There is evidence that the mammalian female genital tract is capable of responding immunologically when challenged with alloantigens. The antigenic properties of male gametes have been well delineated. However, it is only rarely that a female mammal ever responds immunologically to the male gametic antigens as a result of coitus. When a proposed mechanism of suppression of antigenicity of epididymal spermatozoa was tested experimentally, the results indicated that two proteins (uteroglobin and transglutaminase) present in the prostate may be responsible for suppressing sperm antigenicity in the rabbit.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Mukherjee, D C -- Agrawal, A K -- Manjunath, R -- Mukherjee, A B -- New York, N.Y. -- Science. 1983 Feb 25;219(4587):989-91.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6130601" target="_blank"〉PubMed〈/a〉
    Keywords: Acyltransferases/*immunology/metabolism ; Animals ; Epididymis/immunology ; Female ; Glycoproteins/*immunology ; *Immune Tolerance ; Lymphocyte Activation ; Male ; Rabbits ; Semen/enzymology/*immunology ; Spermatozoa/*immunology ; Transglutaminases ; Uteroglobin/*immunology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 5
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    Response to ethanol reduced by past thiamine deficiency (1985)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1985-03-15
    Description: Ethanol-induced intoxication and hypothermia were studied in rats approximately 7 months after severe thiamine deficiency, when treated rats appeared to have recovered their physical health. Previously induced thiamine deficiency without prior ethanol exposure significantly decreased the area under the curve plotted for the concentration of ethanol in blood and also decreased behavioral impairment and hypothermia due to ethanol exposure. Pathophysiologic changes resulting from thiamine deficiency may contribute to both the pharmacodynamic and pharmacokinetic tolerance to ethanol in chronic alcoholics.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Martin, P R -- Majchrowicz, E -- Tamborska, E -- Marietta, C -- Mukherjee, A B -- Eckardt, M J -- New York, N.Y. -- Science. 1985 Mar 15;227(4692):1365-8.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/3975622" target="_blank"〉PubMed〈/a〉
    Keywords: Alcohol Amnestic Disorder/pathology ; Alcoholic Intoxication/physiopathology ; Animals ; Behavior, Animal/drug effects ; Body Temperature/drug effects ; Brain/pathology ; Ethanol/*pharmacology ; Female ; Humans ; Hypothermia/chemically induced ; Male ; Rats ; Rats, Inbred Strains ; Thiamine Deficiency/*physiopathology ; Wernicke Encephalopathy/pathology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 6
    Electronic Resource
    Electronic Resource
    Transitional cryptoperthites (1961)
    Copenhagen : International Union of Crystallography (IUCr)
    Acta crystallographica 14 (1961), S. 1098-1099 
    Details
    ISSN: 0001-5520
    Source: Crystallography Journals Online : IUCR Backfile Archive 1948-2001
    Topics: Geosciences
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1107/S0365110X61003235
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    Paper (German National Licenses)
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  • 7
    Electronic Resource
    Electronic Resource
    Replication pattern of double minutes derived from an insect cell line (1985)
    Springer
    Cellular and molecular life sciences 41 (1985), S. 85-86 
    Details
    ISSN: 1420-9071
    Keywords: Aedes albopictus ; DNA replication ; mosquito ; double minutes ; chromosomal staining ; cell line, insect
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary The DNA replication pattern of double minutes derived from an established cell line ofAedes albopictus is described. Although the vast majority of double minutes replicate semiconservatively once during the S phase, some double minutes appear to exhibit different pattern(s). Two theories are suggested as possible explanations of our findings.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02005888
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  • 8
    Electronic Resource
    Electronic Resource
    Uteroglobin: a novel cytokine? (1999)
    Springer
    Cellular and molecular life sciences 55 (1999), S. 771-787 
    Details
    ISSN: 1420-9071
    Keywords: Key words. Blastokinin; uteroglobin; CC10; ECM; fibronectin; PLA2; receptor; cytokine.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract. Blastokinin or uteroglobin (UG) is a steroid-inducible, evolutionarily conserved, multifunctional protein secreted by the mucosal epithelia of virtually all mammals. It is present in the blood and in other body fluids including urine. An antigen immunoreactive to UG antibody is also detectable in the mucosal epithelia of all vertebrates. UG-binding proteins (putative receptor), expressed on several normal and cancer cell types, have been characterized. The human UG gene is mapped to chromosome 11q12.2 – 13.1, a region that is frequently rearranged or deleted in many cancers. The generation of UG knockout mice revealed that disruption of this gene causes: (i) severe renal disease due to an abnormal deposition of fibronectin and collagen in the glomeruli; (ii) predisposition to a high incidence of malignancies; and (iii) a lack of polychlorinated biphenyl binding and increased oxygen toxicity in the lungs. The mechanism(s) of UG action is likely to be even more complex as it also functions via a putative receptor-mediated pathway that has not yet been clearly defined. Molecular characterization of the UG receptor and signal transduction via this receptor pathway may show that this protein belongs to a novel cytokine/chemokine family.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s000180050331
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  • 9
    Electronic Resource
    Electronic Resource
    Industrial emissions of mercury in Finland between 1967 and 1987 (1991)
    Springer
    Water, air & soil pollution 56 (1991), S. 35-49 
    Details
    ISSN: 1573-2932
    Source: Springer Online Journal Archives 1860-2000
    Topics: Energy, Environment Protection, Nuclear Power Engineering
    Notes: Abstract Mercury pollution has been studied in Finland since 1960. In the 1960 's, industrial use of Hg was about 50 t yr−1, 80% of which was consumed by chlor-alkali plants. In the 1980's, about 19, to 25 t yr−1 Hg was used and compared to past consumption, its use has decreased by 40 to 50%. In 1987, the import of Hg through raw materials and other sources in Finland was, however about 170 t yr−1 which is rather high. There are no detailed statistics about Hg emissions in the 1960's. In 1967, air and water emissions of Hg were 2.8 and 8.3 t yr−1 respectively. In 1987, estimated air emission of Hg was 3.48 t yr−1 whereas water emission was 0.15 t yr−1 Aquatic emission has been lowered by 50% which is due to the fact that pulp and paper industries stopped using Hg as slimicide. On the other hand, the air emission of Hg has not decreased. This is due to the high Hg content in raw materials used by metallurgical and power plants.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00342259
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  • 10
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    Recombinant human uteroglobin suppresses cellular invasiveness via a novel class of high-affinity cell surface binding site. (1996)
    National Academy of Sciences
    Details
    Publication Date: 1996-04-02
    Print ISSN: 0027-8424
    Electronic ISSN: 1091-6490
    Topics: Biology , Medicine , Natural Sciences in General
    Published by National Academy of Sciences
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