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  • Male  (5)
  • Female  (4)
  • Mice  (4)
  • Statistical physics  (4)
  • Letters  (3)
  • 1
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    Alzheimer random walk model: Two previously overlooked diffusion regimes (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-10-03
    Description: Author(s): J. C. Cressoni, G. M. Viswanathan, A. S. Ferreira, and M. A. A. da Silva A non-Markovian one-dimensional random walk model is studied with emphasis on the phase-diagram, showing all the diffusion regimes, along with the exactly determined critical lines. The model, known as the Alzheimer walk, is endowed with memory-controlled diffusion, responsible for the model's long-... [Phys. Rev. E 86, 042101] Published Tue Oct 02, 2012
    Keywords: Statistical physics
    Print ISSN: 1539-3755
    Electronic ISSN: 1550-2376
    Topics: Physics
    Published by American Physical Society (APS)
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  • 2
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    Conditions under which a superdiffusive random-search strategy is necessary (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-09-25
    Description: Author(s): S. A. Sotelo-López, M. C. Santos, E. P. Raposo, G. M. Viswanathan, and M. G. E. da Luz Intuitively, lower target densities and lower detection capabilities should demand more sophisticated search strategies for a random search reasonable outcome. In contrast, when targets are easily found, a simple Brownian random walk strategy is enough. But where is the threshold between these two s... [Phys. Rev. E 86, 031133] Published Mon Sep 24, 2012
    Keywords: Statistical physics
    Print ISSN: 1539-3755
    Electronic ISSN: 1550-2376
    Topics: Physics
    Published by American Physical Society (APS)
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  • 3
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    Dissipative Lévy random searches: Universal behavior at low target density (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-12-05
    Description: Author(s): P. J. Ribeiro-Neto, E. P. Raposo, H. A. Araújo, C. L. Faustino, M. G. E. da Luz, and G. M. Viswanathan We investigate the problem of survival at the very low target-density limit and report on a second-order phase transition for one-dimensional random searches in which the energy cost of locomotion is a function of the distance traveled by the searcher. Surprisingly, from analytical calculations (als... [Phys. Rev. E 86, 061102] Published Tue Dec 04, 2012
    Keywords: Statistical physics
    Print ISSN: 1539-3755
    Electronic ISSN: 1550-2376
    Topics: Physics
    Published by American Physical Society (APS)
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  • 4
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    Weakly anomalous diffusion with non-Gaussian propagators (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-08-28
    Description: Author(s): J. C. Cressoni, G. M. Viswanathan, A. S. Ferreira, and M. A. A. da Silva A poorly understood phenomenon seen in complex systems is diffusion characterized by Hurst exponent H ≈1/2 but with non-Gaussian statistics. Motivated by such empirical findings, we report an exact analytical solution for a non-Markovian random walk model that gives rise to weakly anomalous diffusion... [Phys. Rev. E 86, 022103] Published Mon Aug 27, 2012
    Keywords: Statistical physics
    Print ISSN: 1539-3755
    Electronic ISSN: 1550-2376
    Topics: Physics
    Published by American Physical Society (APS)
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  • 5
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    Correct modeling confirms female storms deadlier [Social Sciences] (2014)
    National Academy of Sciences
    Details
    Publication Date: 2014-09-17
    Description: We report that, for severe hurricanes, name femininity predicts more fatalities (1). Maley (2) argues that outliers drive this effect and that their inland fatalities are not relevant. These arguments reflect misunderstandings about hurricane impacts and their analysis. Maley examines main effects of male/female names. However, our hypothesis addresses the...
    Keywords: Letters
    Print ISSN: 0027-8424
    Electronic ISSN: 1091-6490
    Topics: Biology , Medicine , Natural Sciences in General
    Published by National Academy of Sciences
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  • 6
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    Correct analyses show female storms are deadlier [Social Sciences] (2014)
    National Academy of Sciences
    Details
    Publication Date: 2014-08-27
    Description: Our article (1) reports evidence for a relationship between femininity of hurricane names and fatality rates, along with experimental evidence that female-named storms elicit lower risk perceptions and preparedness intentions. In response, Malter (2) cites some bloggers’ critiques about inclusion of hurricanes from the era of female-only names, interpretation of...
    Keywords: Letters
    Print ISSN: 0027-8424
    Electronic ISSN: 1091-6490
    Topics: Biology , Medicine , Natural Sciences in General
    Published by National Academy of Sciences
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  • 7
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    Population may matter but conclusions unaltered [Social Sciences] (2014)
    National Academy of Sciences
    Details
    Publication Date: 2014-12-17
    Description: We report that for highly damaging hurricanes, not for less damaging hurricanes, name femininity predicts more fatalities (1). We suggest this may be because, for damaging storms, factors such as storm names that motivate protective action are more predictive of survival. Bakkensen and Larson (2) assert that our modeling suffers...
    Keywords: Letters
    Print ISSN: 0027-8424
    Electronic ISSN: 1091-6490
    Topics: Biology , Medicine , Natural Sciences in General
    Published by National Academy of Sciences
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  • 8
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    A role for Lin28 in primordial germ-cell development and germ-cell malignancy (2009)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2009-07-07
    Description: The rarity and inaccessibility of the earliest primordial germ cells (PGCs) in the mouse embryo thwart efforts to investigate molecular mechanisms of germ-cell specification. stella (also called Dppa3) marks the rare founder population of the germ lineage. Here we differentiate mouse embryonic stem cells carrying a stella transgenic reporter into putative PGCs in vitro. The Stella(+) cells possess a transcriptional profile similar to embryo-derived PGCs, and like their counterparts in vivo, lose imprints in a time-dependent manner. Using inhibitory RNAs to screen candidate genes for effects on the development of Stella(+) cells in vitro, we discovered that Lin28, a negative regulator of let-7 microRNA processing, is essential for proper PGC development. Furthermore, we show that Blimp1 (also called Prdm1), a let-7 target and a master regulator of PGC specification, can rescue the effect of Lin28 deficiency during PGC development, thereby establishing a mechanism of action for Lin28 during PGC specification. Overexpression of Lin28 promotes formation of Stella(+) cells in vitro and PGCs in chimaeric embryos, and is associated with human germ-cell tumours. The differentiation of putative PGCs from embryonic stem cells in vitro recapitulates the early stages of gamete development in vivo, and provides an accessible system for discovering novel genes involved in germ-cell development and malignancy.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2729657/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2729657/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉West, Jason A -- Viswanathan, Srinivas R -- Yabuuchi, Akiko -- Cunniff, Kerianne -- Takeuchi, Ayumu -- Park, In-Hyun -- Sero, Julia E -- Zhu, Hao -- Perez-Atayde, Antonio -- Frazier, A Lindsay -- Surani, M Azim -- Daley, George Q -- DP1 OD000256/OD/NIH HHS/ -- DP1 OD000256-01/OD/NIH HHS/ -- G0300723/Medical Research Council/United Kingdom -- G0800784/Medical Research Council/United Kingdom -- T32 CA009172/CA/NCI NIH HHS/ -- Howard Hughes Medical Institute/ -- England -- Nature. 2009 Aug 13;460(7257):909-13. doi: 10.1038/nature08210. Epub 2009 Jul 5.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Division of Pediatric Hematology/Oncology, Children's Hospital Boston and the Dana-Farber Cancer Institute, MA 02115, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19578360" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; *Cell Differentiation ; Cell Line ; Embryonic Stem Cells/cytology/metabolism ; Female ; Gene Expression Regulation, Neoplastic ; Germ Cells/*cytology/*metabolism/pathology ; Humans ; Mice ; Mice, Inbred C57BL ; Neoplasms, Germ Cell and Embryonal/genetics/*metabolism/*pathology ; RNA-Binding Proteins/genetics/*metabolism ; Repressor Proteins/genetics/metabolism ; Transcription Factors/metabolism ; Transgenes
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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  • 9
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    Hemagglutinin receptor binding avidity drives influenza A virus antigenic drift (2009)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2009-11-11
    Description: Rapid antigenic evolution in the influenza A virus hemagglutinin precludes effective vaccination with existing vaccines. To understand this phenomenon, we passaged virus in mice immunized with influenza vaccine. Neutralizing antibodies selected mutants with single-amino acid hemagglutinin substitutions that increased virus binding to cell surface glycan receptors. Passaging these high-avidity binding mutants in naive mice, but not immune mice, selected for additional hemagglutinin substitutions that decreased cellular receptor binding avidity. Analyzing a panel of monoclonal antibody hemagglutinin escape mutants revealed a positive correlation between receptor binding avidity and escape from polyclonal antibodies. We propose that in response to variation in neutralizing antibody pressure between individuals, influenza A virus evolves by adjusting receptor binding avidity via amino acid substitutions throughout the hemagglutinin globular domain, many of which simultaneously alter antigenicity.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2784927/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2784927/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Hensley, Scott E -- Das, Suman R -- Bailey, Adam L -- Schmidt, Loren M -- Hickman, Heather D -- Jayaraman, Akila -- Viswanathan, Karthik -- Raman, Rahul -- Sasisekharan, Ram -- Bennink, Jack R -- Yewdell, Jonathan W -- GM 57073/GM/NIGMS NIH HHS/ -- U54 GM62116/GM/NIGMS NIH HHS/ -- Z01 AI001014-01/Intramural NIH HHS/ -- New York, N.Y. -- Science. 2009 Oct 30;326(5953):734-6. doi: 10.1126/science.1178258.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Laboratory of Viral Diseases, National Institute of Allergy and Infectious Diseases, Bethesda, MD 20892, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/19900932" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Antibodies, Neutralizing/immunology ; Antibodies, Viral/immunology ; Antigenic Variation/genetics/*immunology ; Cell Line ; Hemagglutinin Glycoproteins, Influenza Virus/genetics/immunology/*metabolism ; Influenza A Virus, H1N1 Subtype/genetics/*immunology ; Influenza Vaccines/immunology ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; Models, Immunological ; Mutation ; Receptors, Virus/*metabolism ; Serial Passage
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 10
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    A screen for genes induced in the suprachiasmatic nucleus by light (1998)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1998-03-21
    Description: The mechanism by which mammalian circadian clocks are entrained to light-dark cycles is unknown. The clock that drives behavioral rhythms is located in the suprachiasmatic nucleus (SCN) of the brain, and entrainment is thought to require induction of genes in the SCN by light. A complementary DNA subtraction method based on genomic representational difference analysis was developed to identify such genes without making assumptions about their nature. Four clones corresponded to genes induced specifically in the SCN by light, all of which showed gating of induction by the circadian clock. Among these genes are c-fos and nur77, two of the five early-response genes known to be induced in the SCN by light, and egr-3, a zinc finger transcription factor not previously identified in the SCN. In contrast to known examples, egr-3 induction by light is restricted to the ventral SCN, a structure implicated in entrainment.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Morris, M E -- Viswanathan, N -- Kuhlman, S -- Davis, F C -- Weitz, C J -- New York, N.Y. -- Science. 1998 Mar 6;279(5356):1544-7.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Neurobiology, Harvard Medical School, Boston, MA 02115, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/9488654" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Antisense Elements (Genetics) ; Blotting, Southern ; Circadian Rhythm ; Cloning, Molecular ; Cricetinae ; DNA, Complementary ; DNA-Binding Proteins/*genetics ; Early Growth Response Protein 3 ; *Gene Expression Regulation ; *Genes, fos ; *Light ; Male ; Mesocricetus ; Nuclear Receptor Subfamily 4, Group A, Member 1 ; Polymerase Chain Reaction ; RNA, Messenger/analysis/genetics ; Receptors, Cytoplasmic and Nuclear ; Receptors, Steroid ; Suprachiasmatic Nucleus/*physiology ; Transcription Factors/*genetics
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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