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  • Articles  (7)
  • Endorphins/*physiology  (6)
  • Binding Sites
  • American Association for the Advancement of Science (AAAS)  (7)
  • American Institute of Physics
  • 2020-2022
  • 1995-1999
  • 1980-1984  (7)
  • 1940-1944
  • 1982  (7)
Collection
  • Articles  (7)
Source
Keywords
Publisher
  • American Association for the Advancement of Science (AAAS)  (7)
  • American Institute of Physics
Years
  • 2020-2022
  • 1995-1999
  • 1980-1984  (7)
  • 1940-1944
Year
Journal
Topic
  • 1
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    Biospecific labeling with undecagold: visualization of the biotin-binding site on avidin (1982)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1982-10-15
    Description: The biotin-binding site on avidin has been labeled with biotin conjugated to undecagold, an organometallic cluster compound containing 11 gold atoms in a core angestroms in diameter. Examination of unstained specimens by scanning transmission electron microscopy reveals the labeled sites directly, without computational averaging or filtering of the images. This approach should be widely applicable for determining the locations of subunits and functional site in biological macromolecules at a resolution at a resolution in range of 15 angstroms.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Safer, D -- Hainfeld, J -- Wall, J S -- Reardon, J E -- AM 28607/AM/NIADDK NIH HHS/ -- GM 12202/GM/NIGMS NIH HHS/ -- GM 28750/GM/NIGMS NIH HHS/ -- New York, N.Y. -- Science. 1982 Oct 15;218(4569):290-1.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7123234" target="_blank"〉PubMed〈/a〉
    Keywords: Avidin/*metabolism ; Binding Sites ; Biotin/*metabolism ; Gold/*metabolism ; Organogold Compounds ; Organometallic Compounds/*metabolism ; Ovalbumin/*analogs & derivatives
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 2
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    Hypotensive effect of fasting: possible involvement of the sympathetic nervous system and endogenous opiates (1982)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1982-08-20
    Description: Fasting lowers blood pressure to a greater extent in spontaneously hypertensive rats than in normotensive rats. While fasting reduced cardiac sympathetic activity to an equivalent extent in both groups of animals, only in the hypertensive rats did fasting elicit an opiate-mediated vasodepressor response that was independent of sympathetic withdrawal. Both sympathetic nervous system suppression and endogenous opiate activation, therefore, may contribute to the hypotensive effect of fasting in the spontaneously hypertensive rat.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Einhorn, D -- Young, J B -- Landberg, L -- AM 20378/AM/NIADDK NIH HHS/ -- HL 24084/HL/NHLBI NIH HHS/ -- RR 76/RR/NCRR NIH HHS/ -- New York, N.Y. -- Science. 1982 Aug 20;217(4561):727-9.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7100917" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; *Blood Pressure/drug effects ; Endorphins/*physiology ; *Fasting ; Hypertension/physiopathology ; Male ; Myocardium/metabolism ; Naltrexone/pharmacology ; Norepinephrine/metabolism ; Rats ; Rats, Inbred Strains ; Sympathetic Nervous System/*physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 3
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    Nonopiate effects of dynorphin and des-Tyr-dynorphin (1982)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1982-12-10
    Description: Intracerebroventricular administration of dynorphin produced potent and long-lasting effects on motor function and the electroencephalogram in rats. In addition, local iontophoretic or pressure ejection of dynorphin consistently inhibited hippocampal unit activity. None of these effects were significantly affected by naloxone even at high doses. Moreover, a fragment of dynorphin that failed to displace any of a number of tritiated narcotics from rat brain homogenates produced similar effects on these physiological measures in vivo. On the basis of a variety of criteria for "opiate action," the results suggest that a second biologically active site within the dynorphin sequence is capable of quite potent but nonopiate effects.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Walker, J M -- Moises, H C -- Coy, D H -- Baldrighi, G -- Akil, H -- 1F32DA04183/DA/NIDA NIH HHS/ -- DA02265/DA/NIDA NIH HHS/ -- New York, N.Y. -- Science. 1982 Dec 10;218(4577):1136-8.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6128791" target="_blank"〉PubMed〈/a〉
    Keywords: Action Potentials ; Amino Acid Sequence ; Animals ; Dynorphins ; Endorphins/*physiology ; Hippocampus/*physiology ; Male ; Pain/*physiopathology ; Rats ; Structure-Activity Relationship
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 4
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    Comparison of the distribution of dynorphin systems and enkephalin systems in brain (1982)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1982-12-10
    Description: A study of the anatomical distribution of the endogenous opioid dynorphin in rat brain showed that the peptide is localized in a widespread system with multiple cell groups and projections. This network is revealed by the use of multiple antiserums against dynorphin and can be distinguished from the system containing methionine-enkephalin and leucine-enkephalin, which is mapped by the use of antiserums against the enkephalins and biosynthetically related peptides in the adrenal. It thus appears that the brain contains at least three separate opioid neuronal networks: an enkephalin family with components similar to those found in the adrenal, a beta-endorphin family, and a dynorphin family.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Watson, S J -- Khachaturian, H -- Akil, H -- Coy, D H -- Goldstein, A -- DA00154/DA/NIDA NIH HHS/ -- DA02265/DA/NIDA NIH HHS/ -- MH15794/MH/NIMH NIH HHS/ -- New York, N.Y. -- Science. 1982 Dec 10;218(4577):1134-6.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6128790" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Brain/*physiology ; Brain Mapping ; Dynorphins ; Endorphins/*physiology ; Enkephalins/*physiology ; Immunologic Techniques ; Rats
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 5
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    Organization of endogenous opiate and nonopiate pain control systems (1982)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1982-06-11
    Description: Research during the past decade has revealed the existence of neural systems that modulate pain transmission. Much of this work has focused on the role of endogenous opiate systems, but recent research indicates the involvement of nonopiate mechanisms as well. In this article, we present data demonstrating that opiate and nonopiate analgesia systems can be selectively activated by different environmental manipulations and describe the neural circuitry involved. Both neural and hormonal pathways and both opiate and nonopiate substances play roles in the complex modulation of pain transmission. The existence and description of these modulatory mechanisms have important clinical implications for the treatment of pain.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Watkins, L R -- Mayer, D J -- DA 00576/DA/NIDA NIH HHS/ -- New York, N.Y. -- Science. 1982 Jun 11;216(4551):1185-92.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6281891" target="_blank"〉PubMed〈/a〉
    Keywords: Acupuncture Therapy ; Brain/physiology ; Electric Stimulation ; Endorphins/*physiology ; Foot ; Hypnosis ; Morphine/pharmacology ; Pain/*physiopathology ; Placebos ; Receptors, Opioid/*physiology ; Spinal Cord/physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 6
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    Corticosterone: a critical factor in an opioid form of stress-induced analgesia (1982)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1982-03-19
    Description: The finding that some opioid-mediated forms of stress-induced analgesia are antagonized by hypophysectomy and dexamethasone has led to the suggestion that beta-endorphin, released from the pituitary, may mediate these analgesic reactions. "Long-term analgesia" (an opioid-mediated form of stress-induced analgesia), which is blocked by dexamethasone and hypophysectomy, was also blocked by adrenalectomy and reinstated with corticosterone therapy. Corticosterone is proposed to play a permissive role in long-term analgesia and to be a critical hormone mediating this phenomenon.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉MacLennan, A J -- Drugan, R C -- Hyson, R L -- Maier, S F -- Madden, J 4th -- Barchas, J D -- MH 23861/MH/NIMH NIH HHS/ -- New York, N.Y. -- Science. 1982 Mar 19;215(4539):1530-2.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7063862" target="_blank"〉PubMed〈/a〉
    Keywords: Adrenalectomy ; Analgesia ; Animals ; Corticosterone/*physiology ; Dexamethasone/pharmacology ; Endorphins/*physiology ; Hypophysectomy ; Pain/*physiopathology ; Rats ; Stress, Physiological/*physiopathology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 7
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    Endogenous opiates and energy balance (1982)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1982-03-19
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Mandenoff, A -- Fumeron, F -- Apfelbaum, M -- Margules, D L -- New York, N.Y. -- Science. 1982 Mar 19;215(4539):1536-8.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7063865" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Body Weight ; Endorphins/*physiology ; Energy Intake ; *Energy Metabolism ; Feeding Behavior/*physiology ; Naloxone/pharmacology ; Oxygen Consumption ; Rats
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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