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Hepatic cyclophorase activity in rats treated with 1,2,3,4-tetrahydro-β-naphtylamine (THNA)

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Gli omogenati di fegato dei ratti sacrificati dopo 4 h dalla somministrazione diβ-tetraidronaftilammina (30 mg pro kg per via intraperitoneale) presentano una aumentata capacità di ossidare alcuni composti del Ciclo di Krebs, in rapporto ai controlli. Viene avanzata l'ipotesi che l'intensa glicogenolisi epatica e muscolare e l'aumentata acetilazionein vivo dell'acido para-aminobenzoico provocate nel ratto dalla somministrazione diβ-tetraidronaftilammina, siano causate da un esaltato metabolismo intermedio ossidativo.

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References

  1. V. Martini andM. Orunesu, Boll. Soc. Ital. Biol. sperim.33, 1682 (1957).

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  2. V. Martini andM. Orunesu, Boll. Soc. Ital. Biol. sperim.33, 1684 (1957).

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  3. F. J. Moore, F. B. Cramer, andR. G. Knowles,Statistics for Medical Students (The Blakiston Company, Toronto 1951).

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Authors and Affiliations

  1. Institute of General Physiology, University of Bari, Italy

    V. Martini & M. Orunesu

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  1. V. Martini
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  2. M. Orunesu
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Martini, V., Orunesu, M. Hepatic cyclophorase activity in rats treated with 1,2,3,4-tetrahydro-β-naphtylamine (THNA). Experientia 15, 331–332 (1959). https://doi.org/10.1007/BF02159813

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  • DOI: https://doi.org/10.1007/BF02159813

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