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Title:	A Genome-Wide Association Study Identifies IL23R as an Inflammatory Bowel Disease Gene
Source:	Science [0036-8075] Duerr, Richard yr:2006

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description	1.	Rioux, John D D. "Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis." Nature genetics 39.5 (2007): 596-604.
description	2.	Hampe, J. "A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1." Nature genetics 39.2 (2007): 207-211.
description	3.	Neurath, Markus F F. "IL-23: a master regulator in Crohn disease." Nature medicine 13.1 (2006): 26-8.
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description	5.	Tremelling, M. "IL23R variation determines susceptibility but not disease phenotype in inflammatory bowel disease." Gastroenterology 132.5 (2007): 1657-1664.
description	6.	Daly, Jeffrey J. "Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease." Nature genetics 40.8 (2008): 955-62.
description	7.	Gross, S. "Clinical practice. Prenatal screening for aneuploidy." New England Journal of Medicine, The 360.24 (2009): 2556-62.
description	8.	Silverberg, Mark S S. "Refined genomic localization and ethnic differences observed for the IBD5 association with Crohn's disease." European journal of human genetics 15.3 (2007): 328-35.
description	9.	Mathew, W. "Sequence variants in the autophagy gene IRGM and multiple other replicating loci contribute to Crohn's disease susceptibility." Nature genetics 39.7 (2007): 830-2.
description	10.	Todd, John A A. "Robust associations of four new chromosome regions from genome-wide analyses of type 1 diabetes." Nature genetics 39.7 (2007): 857-64.
description	11.	Sladek, R. "A genome-wide association study identifies novel risk loci for type 2 diabetes." Nature 445.7130 (2007): 881-5.
description	12.	Delgado, V. "Toll-like receptors control autophagy." The EMBO journal 27.7 (2008): 1110-21.
description	13.	Hue, S. "Interleukin-23 drives innate and T cell-mediated intestinal inflammation." The Journal of experimental medicine 203.11 (2006): 2473-83.
description	14.	Marchini, Jonathan L. "Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls." Nature 447.7145 (2007): 661-78.
description	15.	Yamazaki, K. "Single nucleotide polymorphisms in TNFSF15 confer susceptibility to Crohn's disease." Human Molecular Genetics 14.22 (2005): 3499-3506.
description	16.	Smyth, Deborah C. "Analysis of polymorphisms in 16 genes in type 1 diabetes that have been associated with other immune-mediated diseases." BMC medical genetics 7.1: 20-20.
description	17.	Klein, Robert J J. "Complement factor H polymorphism in age-related macular degeneration." Science 308.5720: 385-389.
description	18.	Burakoff, R. "A phase 1/2A trial of STA 5326, an oral interleukin-12/23 inhibitor, in patients with active moderate to severe Crohn's disease." Inflammatory bowel diseases 12.7 (2006): 558-65.
description	19.	Duerr, Richard H H. "Genome-wide association studies herald a new era of rapid discoveries in inflammatory bowel disease research." Gastroenterology 132.5 (2007): 2045-9.
description	20.	Eckburg, Paul B. "The role of microbes in Crohn's disease." Clinical infectious diseases 44.2 (2007): 256-62.