Amiloride and its analogs as tools to inhibit Na+ transport via the Na+ channel, the Na+/H+ antiport and the Na+/Ca2+ exchanger
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CNS sites activated by renal pelvic epithelial sodium channels (ENaCs) in response to hypertonic saline in awake rats
2017, Autonomic Neuroscience: Basic and ClinicalCitation Excerpt :Mechanoreceptor transduction in the heart and kidney has been reported to be dependent on activation of ENaCs since afferent nerve activity is prevented by low concentrations of amiloride (Kopp et al., 1998; Ditting et al., 2003). Frelin et al. (1988) determined that ENaCs can be inhibited by lower doses of amiloride or benzamil without interfering with other Na dependent channels. Therefore, ENaCs play a role both in regulating sodium excretion and reabsorption and in pressure sensitive renal afferent pelvic nerves.
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