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  • 1
    Electronic Resource
    Electronic Resource
    Spectra of Hamiltonians with generalized single-site dynamical disorder (1994)
    Springer
    The European physical journal 95 (1994), S. 101-111 
    Details
    ISSN: 1434-6036
    Keywords: 05.40.+j ; 02.50.+s
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract Starting from the deformed commutation relationsa q (t) a † q (s)−q a † q (s) a q (t)=Γ(t−s)1, −1≦q≦1 with a covariance Γ(t−s) and a parameterq varying between −1 and 1, a stochastic process is constructed which continuously deforms the classical Gaussian and classical compound Poisson process. The moments of these distinguished stochastic processes are identified with the Hilbert space vacuum expectation values of products of $$\hat \omega _q (t) = \gamma (a_q (t) + a_q^\dag (t)) + \xi a_q^\dag (t)a_q (t)$$ with fixed parametersq, γ and ξ. Thereby we can interpolate between dichotomic, random matrix and classical Gaussian and compound Poisson processes. The spectra of Hamiltonians with single-site dynamical disorder are calculated for an exponential covariance (coloured noise) by means of the time convolution generalized master equation formalism (TC-GME) and the partial cumulants technique. The final result for the spectral function is given as aq-dependent infinite continued fraction. In the case of the random matrix processes the infinite continued fraction can be summed up yielding a self-consistent equation for the one-particle Green function.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01316850
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  • 2
    Electronic Resource
    Electronic Resource
    A self-consistent master equation and a new kind of cumulants (1993)
    Springer
    The European physical journal 92 (1993), S. 399-407 
    Details
    ISSN: 1434-6036
    Keywords: 05.40.+j ; 02.50.+s
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract Generalized master equations correspond to different kinds of cumulants. Here, we discuss the recently introduced non-crossing cumulants from a physical point of view and propose the corresponding integro-differential master equation as a new type of equation for a self-consistent treatment of memory effects. We prove the cluster property of the non-crossing cumulants, and show that the Gaussian approximation of our equation is given by a random matrix process. As an instructive example for our expansion formula we treat the random frequency-modulated oscillator.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01308760
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  • 3
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    Chlamydia infections and heart disease linked through antigenic mimicry (1999)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1999-02-26
    Description: Chlamydia infections are epidemiologically linked to human heart disease. A peptide from the murine heart muscle-specific alpha myosin heavy chain that has sequence homology to the 60-kilodalton cysteine-rich outer membrane proteins of Chlamydia pneumoniae, C. psittaci, and C. trachomatis was shown to induce autoimmune inflammatory heart disease in mice. Injection of the homologous Chlamydia peptides into mice also induced perivascular inflammation, fibrotic changes, and blood vessel occlusion in the heart, as well as triggering T and B cell reactivity to the homologous endogenous heart muscle-specific peptide. Chlamydia DNA functioned as an adjuvant in the triggering of peptide-induced inflammatory heart disease. Infection with C. trachomatis led to the production of autoantibodies to heart muscle-specific epitopes. Thus, Chlamydia-mediated heart disease is induced by antigenic mimicry of a heart muscle-specific protein.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Bachmaier, K -- Neu, N -- de la Maza, L M -- Pal, S -- Hessel, A -- Penninger, J M -- New York, N.Y. -- Science. 1999 Feb 26;283(5406):1335-9.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Amgen Institute, Ontario Cancer Institute, Departments of Medical Biophysics and Immunology, University of Toronto, Toronto, Ontario M5G 2C1, Canada.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/10037605" target="_blank"〉PubMed〈/a〉
    Keywords: Adoptive Transfer ; Amino Acid Sequence ; Animals ; Antigens, Bacterial/chemistry/immunology ; Autoantibodies/biosynthesis ; Autoimmune Diseases/immunology/*microbiology/pathology ; B-Lymphocytes/immunology ; Bacterial Outer Membrane Proteins/chemistry/*immunology ; CD4-Positive T-Lymphocytes/immunology ; Chlamydia/*immunology ; Chlamydia Infections/complications/*immunology ; Chlamydia trachomatis/immunology ; CpG Islands ; Humans ; Immunization ; Lymphocyte Activation ; Mice ; Mice, Inbred BALB C ; *Molecular Mimicry ; Molecular Sequence Data ; Myocarditis/immunology/*microbiology/pathology ; Myocardium/immunology/pathology ; Myosin Heavy Chains/chemistry/*immunology ; Oligodeoxyribonucleotides/immunology ; Sequence Homology, Amino Acid
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 4
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    The crisis in antibiotic resistance (1992)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1992-08-21
    Description: The synthesis of large numbers of antibiotics over the past three decades has caused complacency about the threat of bacterial resistance. Bacteria have become resistant to antimicrobial agents as a result of chromosomal changes or the exchange of the exchange of genetic material via plasmids and transposons. Streptococcus pneumoniae, Streptococcus pyogenes, and staphylococci, organisms that cause respiratory and cutaneous infections, and members of the Enterobacteriaceae and Pseudomonas families, organisms that cause diarrhea, urinary infection, and sepsis, are now resistant to virtually all of the older antibiotics. The extensive use of antibiotics in the community and hospitals has fueled this crisis. Mechanisms such as antibiotic control programs, better hygiene, and synthesis of agents with improved antimicrobial activity need to be adopted in order to limit bacterial resistance.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Neu, H C -- New York, N.Y. -- Science. 1992 Aug 21;257(5073):1064-73.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉College of Physicians & Surgeons, Columbia University, New York, NY 10032.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/1509257" target="_blank"〉PubMed〈/a〉
    Keywords: Anti-Bacterial Agents/pharmacology ; Bacterial Infections/*drug therapy ; Cross Infection/microbiology ; *Drug Resistance, Microbial ; Enterobacteriaceae/drug effects ; Enterococcus/drug effects ; Haemophilus influenzae/drug effects ; Humans ; Staphylococcus/drug effects ; Streptococcus pneumoniae/drug effects ; Streptococcus pyogenes/drug effects
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 5
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    Digoxin-inactivating bacteria: identification in human gut flora (1983)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1983-04-15
    Description: Digoxin, the most widely used cardiac glycoside, undergoes significant metabolic conversion in many patients to cardioinactive metabolites in which the lactone ring is reduced. This appears to occur within the gastrointestinal tract. An attempt was made to isolate and identify the organisms capable of reducing digoxin from stool cultures obtained from human volunteers. Of hundreds of isolates studied, only Eubacterium lentum, a common anaerobe of the human colonic flora, converted digoxin to reduced derivatives. Such organisms were also isolated in high concentrations from the stools of individuals who did not excrete these metabolites when given digoxin in vivo. When the growth of E. lentum was stimulated by arginine, inactivation of digoxin was inhibited. Neither the presence of these organisms alone nor their concentration within the gut flora appeared to determine whether digoxin would be inactivated by this pathway in vivo.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Saha, J R -- Butler, V P Jr -- Neu, H C -- Lindenbaum, J -- AA 00249/AA/NIAAA NIH HHS/ -- HL 10608/HL/NHLBI NIH HHS/ -- New York, N.Y. -- Science. 1983 Apr 15;220(4594):325-7.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6836275" target="_blank"〉PubMed〈/a〉
    Keywords: Arginine/pharmacology ; Colon/microbiology ; Digoxin/*metabolism ; Eubacterium/drug effects/*metabolism ; Feces/microbiology ; Humans ; Oxidation-Reduction
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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