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  • 1
    Publication Date: 1984-01-20
    Description: Low levels of cerebral concussion in the cat produce reversible behavioral suppression presumably associated with unconsciousness. This injury is also associated with increased rates of glucose utilization in regions within the dorsomedial pontine tegmentum. Microinjection of carbachol into these regions produced behavioral suppression resembling that following concussion. These data, together with previously published observations on cholinergic responses to brain injury, suggest that concussive unconsciousness may be attributable in part to activation of cholinergic pontine sites.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Hayes, R L -- Pechura, C M -- Katayama, Y -- Povlishock, J T -- Giebel, M L -- Becker, D P -- NS 12587/NS/NINDS NIH HHS/ -- New York, N.Y. -- Science. 1984 Jan 20;223(4633):301-3.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6701514" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Atropine/pharmacology ; Brain Concussion/*physiopathology ; Carbachol/pharmacology ; Cats ; Cholinergic Fibers/*physiopathology ; Deoxyglucose/metabolism ; Pons/metabolism/*physiopathology ; Tetracaine/pharmacology ; Unconsciousness/*physiopathology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 2
    Publication Date: 1980-09-12
    Description: Application of arachidonic acid or prostaglandin G(2) to the brain surface of anesthetized cats induced cerebral arteriolar damage. Scavengers of free oxygen radicals inhibited this damage. Prostaglandin H(2), prostaglandin E(2), and 11,14,17-eicosatrienoic acid did not produce arteriolar damage. It appears that increased prostaglandin synthesis produces cerebral vascular damage by generating free oxygen radicals.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Kontos, H A -- Wei, E P -- Povlishock, J T -- Dietrich, W D -- Magiera, C J -- Ellis, E F -- New York, N.Y. -- Science. 1980 Sep 12;209(4462):1242-5.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7403881" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Arachidonic Acids/*pharmacology ; Arterioles/drug effects/pathology ; Cats ; Cerebral Arteries/*drug effects/pathology ; Endothelium/drug effects/pathology ; Hypertension/*pathology ; Prostaglandin Endoperoxides/*pharmacology ; Prostaglandins E/pharmacology ; Prostaglandins G/*pharmacology ; Prostaglandins H/pharmacology ; Vasodilation/drug effects
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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