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  • 1
    Electronic Resource
    Electronic Resource
    Changes in myoblast membrane electrical properties during cell-cell adhesion and fusion in vitro
    Amsterdam : Elsevier
    Biochimica et Biophysica Acta (BBA)/Biomembranes 903 (1987), S. 89-95 
    Details
    ISSN: 0005-2736
    Keywords: (Chicken myoblast) ; Cell adhesion ; Cell-cell recognition ; Conductivity ; Differentiation ; Myoblast fusion ; Myogenesis ; Radiowave frequencies
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology , Medicine , Physics
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0005-2736(87)90158-1
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Changes in myoblast membrane order during differentiation as measured by EPR
    Amsterdam : Elsevier
    Biochimica et Biophysica Acta (BBA)/Biomembranes 896 (1987), S. 19-25 
    Details
    ISSN: 0005-2736
    Keywords: (Chicken myoblast) ; Cell-cell recognition ; Differentiation ; ESR ; Membrane fusion ; Membrane order ; Myogenesis
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology , Medicine , Physics
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0005-2736(87)90351-8
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  • 3
    Electronic Resource
    Electronic Resource
    Rescue of the Li^+-induced delay of embryonic myogenesis in vitro by added inositol
    Amsterdam : Elsevier
    Biochimica et Biophysica Acta (BBA)/Molecular Cell Research 1013 (1989), S. 92-96 
    Details
    ISSN: 0167-4889
    Keywords: Cell-cell recognition ; Differentiation ; Inositol ; Lithium ion ; Myogenesis ; polyphosphatidylinositol
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Biology , Chemistry and Pharmacology , Medicine , Physics
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1016/0167-4889(89)90133-X
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  • 4
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    Identification of vaccine candidates against serogroup B meningococcus by whole-genome sequencing (2000)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2000-03-10
    Description: Neisseria meningitidis is a major cause of bacterial septicemia and meningitis. Sequence variation of surface-exposed proteins and cross-reactivity of the serogroup B capsular polysaccharide with human tissues have hampered efforts to develop a successful vaccine. To overcome these obstacles, the entire genome sequence of a virulent serogroup B strain (MC58) was used to identify vaccine candidates. A total of 350 candidate antigens were expressed in Escherichia coli, purified, and used to immunize mice. The sera allowed the identification of proteins that are surface exposed, that are conserved in sequence across a range of strains, and that induce a bactericidal antibody response, a property known to correlate with vaccine efficacy in humans.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Pizza, M -- Scarlato, V -- Masignani, V -- Giuliani, M M -- Arico, B -- Comanducci, M -- Jennings, G T -- Baldi, L -- Bartolini, E -- Capecchi, B -- Galeotti, C L -- Luzzi, E -- Manetti, R -- Marchetti, E -- Mora, M -- Nuti, S -- Ratti, G -- Santini, L -- Savino, S -- Scarselli, M -- Storni, E -- Zuo, P -- Broeker, M -- Hundt, E -- Knapp, B -- Blair, E -- Mason, T -- Tettelin, H -- Hood, D W -- Jeffries, A C -- Saunders, N J -- Granoff, D M -- Venter, J C -- Moxon, E R -- Grandi, G -- Rappuoli, R -- New York, N.Y. -- Science. 2000 Mar 10;287(5459):1816-20.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉IRIS, Chiron S.p.A., Via Fiorentina 1, 53100 Siena, Italy.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/10710308" target="_blank"〉PubMed〈/a〉
    Keywords: Amino Acid Sequence ; Animals ; Antibodies, Bacterial/biosynthesis/blood ; Antigens, Bacterial/chemistry/genetics/*immunology ; Antigens, Surface/chemistry/genetics/immunology ; Bacterial Capsules ; Bacterial Proteins/chemistry/genetics/*immunology ; *Bacterial Vaccines/genetics/immunology ; Conserved Sequence ; Escherichia coli/genetics ; *Genome, Bacterial ; Humans ; Immune Sera/immunology ; Mice ; Neisseria meningitidis/classification/*genetics/*immunology/pathogenicity ; Open Reading Frames ; Recombinant Fusion Proteins/chemistry/immunology/isolation & purification ; Recombination, Genetic ; Sequence Analysis, DNA ; Serotyping ; Vaccination ; Virulence
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 5
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    Evolutionary biology: A flourishing of fish forms (2010)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2010-04-09
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Alfaro, Michael -- Santini, Francesco -- England -- Nature. 2010 Apr 8;464(7290):840-2. doi: 10.1038/464840a.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/20376139" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; *Biodiversity ; *Biological Evolution ; Body Size ; Extinction, Biological ; Fishes/*anatomy & histology ; Fossils
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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  • 6
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    Comment on "Origin of the superflock of cichlid fishes from Lake Victoria, East Africa" (2004)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2004-05-15
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Stager, J C -- Day, J J -- Santini, S -- New York, N.Y. -- Science. 2004 May 14;304(5673):963; author reply 963.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Natural Resources Division, Paul Smith's College, Paul Smiths, NY 12970, USA. stagerj@pscmail.paulsmiths.edu〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/15143263" target="_blank"〉PubMed〈/a〉
    Keywords: Africa, Eastern ; Animals ; Biodiversity ; Biological Evolution ; *Cichlids/genetics/physiology ; Ecosystem ; *Fresh Water ; Genetic Variation ; Geography ; *Geologic Sediments ; *Haplotypes ; Time
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 7
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    EFF-1-mediated regenerative axonal fusion requires components of the apoptotic pathway (2015)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2015-01-09
    Description: Functional regeneration after nervous system injury requires transected axons to reconnect with their original target tissue. Axonal fusion, a spontaneous regenerative mechanism identified in several species, provides an efficient means of achieving target reconnection as a regrowing axon is able to contact and fuse with its own separated axon fragment, thereby re-establishing the original axonal tract. Here we report a molecular characterization of this process in Caenorhabditis elegans, revealing dynamic changes in the subcellular localization of the EFF-1 fusogen after axotomy, and establishing phosphatidylserine (PS) and the PS receptor (PSR-1) as critical components for axonal fusion. PSR-1 functions cell-autonomously in the regrowing neuron and, instead of acting in its canonical signalling pathway, acts in a parallel phagocytic pathway that includes the transthyretin protein TTR-52, as well as CED-7, NRF-5 and CED-6 (refs 9, 10, 11, 12). We show that TTR-52 binds to PS exposed on the injured axon, and can restore fusion several hours after injury. We propose that PS functions as a 'save-me' signal for the distal fragment, allowing conserved apoptotic cell clearance molecules to function in re-establishing axonal integrity during regeneration of the nervous system.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Neumann, Brent -- Coakley, Sean -- Giordano-Santini, Rosina -- Linton, Casey -- Lee, Eui Seung -- Nakagawa, Akihisa -- Xue, Ding -- Hilliard, Massimo A -- GM059083/GM/NIGMS NIH HHS/ -- GM079097/GM/NIGMS NIH HHS/ -- GM088241/GM/NIGMS NIH HHS/ -- P40 OD010440/OD/NIH HHS/ -- R01 NS060129/NS/NINDS NIH HHS/ -- England -- Nature. 2015 Jan 8;517(7533):219-22. doi: 10.1038/nature14102.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉CJCADR, Queensland Brain Institute, The University of Queensland, Brisbane QLD 4072, Australia. ; Department of Molecular, Cellular, and Developmental Biology, University of Colorado, Boulder, Colorado 80309, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/25567286" target="_blank"〉PubMed〈/a〉
    Keywords: ATP-Binding Cassette Transporters/metabolism ; Animals ; Apoptosis/*physiology ; Axons/*metabolism/pathology ; Caenorhabditis elegans/*cytology/*metabolism ; Caenorhabditis elegans Proteins/genetics/*metabolism ; Carrier Proteins/metabolism ; Growth Cones/metabolism ; Membrane Glycoproteins/*metabolism ; Mutation ; Nerve Regeneration/*physiology ; Phagocytes/metabolism ; Phagocytosis ; Phosphatidylserines/metabolism ; Phosphoproteins/metabolism ; Receptors, Cell Surface/metabolism ; Signal Transduction ; Spectrin/genetics/metabolism
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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  • 8
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    Exaggerated translation causes synaptic and behavioural aberrations associated with autism (2012)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2012-12-25
    Description: Autism spectrum disorders (ASDs) are an early onset, heterogeneous group of heritable neuropsychiatric disorders with symptoms that include deficits in social interaction skills, impaired communication abilities, and ritualistic-like repetitive behaviours. One of the hypotheses for a common molecular mechanism underlying ASDs is altered translational control resulting in exaggerated protein synthesis. Genetic variants in chromosome 4q, which contains the EIF4E locus, have been described in patients with autism. Importantly, a rare single nucleotide polymorphism has been identified in autism that is associated with increased promoter activity in the EIF4E gene. Here we show that genetically increasing the levels of eukaryotic translation initiation factor 4E (eIF4E) in mice results in exaggerated cap-dependent translation and aberrant behaviours reminiscent of autism, including repetitive and perseverative behaviours and social interaction deficits. Moreover, these autistic-like behaviours are accompanied by synaptic pathophysiology in the medial prefrontal cortex, striatum and hippocampus. The autistic-like behaviours displayed by the eIF4E-transgenic mice are corrected by intracerebroventricular infusions of the cap-dependent translation inhibitor 4EGI-1. Our findings demonstrate a causal relationship between exaggerated cap-dependent translation, synaptic dysfunction and aberrant behaviours associated with autism.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3548017/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3548017/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Santini, Emanuela -- Huynh, Thu N -- MacAskill, Andrew F -- Carter, Adam G -- Pierre, Philippe -- Ruggero, Davide -- Kaphzan, Hanoch -- Klann, Eric -- CA154916/CA/NCI NIH HHS/ -- NS034007/NS/NINDS NIH HHS/ -- NS047384/NS/NINDS NIH HHS/ -- NS078718/NS/NINDS NIH HHS/ -- R01 CA154916/CA/NCI NIH HHS/ -- R01 NS034007/NS/NINDS NIH HHS/ -- R01 NS047384/NS/NINDS NIH HHS/ -- R21 NS078718/NS/NINDS NIH HHS/ -- Wellcome Trust/United Kingdom -- England -- Nature. 2013 Jan 17;493(7432):411-5. doi: 10.1038/nature11782. Epub 2012 Dec 23.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Center for Neural Science, New York University, New York, New York 10003, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/23263185" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Autistic Disorder/drug therapy/*genetics/pathology/*physiopathology ; Behavior, Animal/drug effects ; Dendrites/metabolism/pathology ; Eukaryotic Initiation Factor-4E/genetics/*metabolism ; Eukaryotic Initiation Factor-4G/metabolism ; Female ; Hippocampus/metabolism ; Hydrazones ; Infusions, Intraventricular ; Male ; Mice ; Mice, Transgenic ; Neostriatum/metabolism ; Neuronal Plasticity ; Nitro Compounds/administration & dosage/pharmacology/therapeutic use ; Prefrontal Cortex/metabolism ; *Protein Biosynthesis/drug effects/genetics ; RNA Caps/metabolism ; Synapses/*metabolism/*pathology ; Thiazoles/administration & dosage/pharmacology/therapeutic use
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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