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  • 1
    Electronic Resource
    Electronic Resource
    Mass Soldering (1996)
    Bradford : Emerald
    Soldering & surface mount technology 8 (1996), S. 42-48 
    Details
    ISSN: 0954-0911
    Source: Emerald Fulltext Archive Database 1994-2005
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: This issue of the journal features the first part of a two-part serieswhich comprises Chapter 15 from Volume 1 of a recently published book 'AComprehensive Guide to the Manufacture of Printed Board Assemblies'*edited byW. MacLeod Ross. Volume 1, containing 800 pages, and Volume 2, scheduled to bepublished in the Spring of 1997 and estimated to contain around 900 pages, will, as far as thepublishers are aware, be the most comprehensive book ever published on the subject of printed boardsand printed board assemblies.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1108/09540919610800585
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    Paper (German National Licenses)
    Fulltext
  • 2
    Electronic Resource
    Electronic Resource
    Mass Soldering (1997)
    Bradford : Emerald
    Soldering & surface mount technology 9 (1997), S. 23-29 
    Details
    ISSN: 0954-0911
    Source: Emerald Fulltext Archive Database 1994-2005
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: This issue of thejournal features the final part of a two-part series which comprises Chapter 15 fromVolume 1 of a recently published book 'A Comprehensive Guide to the Manufacture ofPrinted Board Assemblies'* edited by W.MacLeod Ross.Volume 1, containing 800 pages, and Volume 2, scheduled to be published in theSpring of 1997 and estimated to contain around 900 pages, will, as far as the publishers areaware, be the most comprehensive book ever published on the subject of printed boardassemblies.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1108/09540919710800593
    Location Call Number Expected Availability
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    Paper (German National Licenses)
    Fulltext
  • 3
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    Unknown
    Potentiation of leukotriene production following sequestration of neutrophils in isolated lungs: indirect evidence for intercellular leukotriene A4 transfer (1988)
    American Society of Hematology
    Details
    Publication Date: 1988-11-01
    Description: Granulocyte (polymorphonuclear leukocyte, PMN) sequestration in the microvascular bed with release of different mediators has been implicated in the pathogenesis of inflammatory and allergic disorders in many organs including the lung. In the present study, we investigated the profile and quantity of leukotriene (LT) generation in isolated blood-free perfused rabbit lungs, in isolated PMNs in vitro and in rabbit lungs, following administration of PMNs, mimicking pulmonary leukostasis. Following stimulation with increasing concentrations of the calcium ionophore A 23137 (0.1 to 2 mumol/L), LTs were detected in the buffer fluid by their chromatographic mobility in different high-performance liquid chromatography (HPLC) systems, by on- line peak spectrum analysis, and by post-HPLC radioimmunoassay (RIA). In isolated lungs, a dose-dependent generation of cysteinyl LTs greater than LTB4, in the complete absence of omega-oxidation products of LTB4 as well as nonenzymatic hydrolysis products of LTA4, was evoked. PMNs in vitro showed a typical profile of LT liberation (LTB4, 20-OH-, and COOH-LTB4, nonenzymatic LTA4 metabolites). In the model of pulmonary leukostasis, the presence of omega-oxidation products of LTB4 indicated metabolic integrity of the trapped PMNs. Nonenzymatic hydrolysis products of LTA4 were, however, not detected in the combined system, whereas the cysteinyl LTs increased markedly. This profile suggests intercellular transfer of PMN-derived LTA4 to lung cells in the microenvironment. In addition, at 2 mumol/L A23187, the sum of all LTA4- derived products surpassed the arithmetic sum of the isolated preparations more than threefold. This potentiation of an LT generation under conditions of pulmonary leukostasis may be of biologic significance for amplification of inflammatory events.
    Print ISSN: 0006-4971
    Electronic ISSN: 1528-0020
    Topics: Biology , Medicine
    Published by American Society of Hematology
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    PAPER CURRENT
  • 4
    facet.materialart.
    Unknown
    Potentiation of leukotriene production following sequestration of neutrophils in isolated lungs: indirect evidence for intercellular leukotriene A4 transfer (1988)
    American Society of Hematology
    Details
    Publication Date: 1988-11-01
    Description: Granulocyte (polymorphonuclear leukocyte, PMN) sequestration in the microvascular bed with release of different mediators has been implicated in the pathogenesis of inflammatory and allergic disorders in many organs including the lung. In the present study, we investigated the profile and quantity of leukotriene (LT) generation in isolated blood-free perfused rabbit lungs, in isolated PMNs in vitro and in rabbit lungs, following administration of PMNs, mimicking pulmonary leukostasis. Following stimulation with increasing concentrations of the calcium ionophore A 23137 (0.1 to 2 mumol/L), LTs were detected in the buffer fluid by their chromatographic mobility in different high-performance liquid chromatography (HPLC) systems, by on- line peak spectrum analysis, and by post-HPLC radioimmunoassay (RIA). In isolated lungs, a dose-dependent generation of cysteinyl LTs greater than LTB4, in the complete absence of omega-oxidation products of LTB4 as well as nonenzymatic hydrolysis products of LTA4, was evoked. PMNs in vitro showed a typical profile of LT liberation (LTB4, 20-OH-, and COOH-LTB4, nonenzymatic LTA4 metabolites). In the model of pulmonary leukostasis, the presence of omega-oxidation products of LTB4 indicated metabolic integrity of the trapped PMNs. Nonenzymatic hydrolysis products of LTA4 were, however, not detected in the combined system, whereas the cysteinyl LTs increased markedly. This profile suggests intercellular transfer of PMN-derived LTA4 to lung cells in the microenvironment. In addition, at 2 mumol/L A23187, the sum of all LTA4- derived products surpassed the arithmetic sum of the isolated preparations more than threefold. This potentiation of an LT generation under conditions of pulmonary leukostasis may be of biologic significance for amplification of inflammatory events.
    Print ISSN: 0006-4971
    Electronic ISSN: 1528-0020
    Topics: Biology , Medicine
    Published by American Society of Hematology
    Location Call Number Expected Availability
    BibTip Others were also interested in ...
    PAPER CURRENT
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