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  • 1
    Series available for loan
    Series available for loan
    Washington, DC : United States Gov. Print. Off.
    Associated volumes
    Call number: SR 90.0002(1500-A-J)
    In: Professional paper
    Type of Medium: Series available for loan
    Pages: Getr. Zählung
    Series Statement: U.S. Geological Survey professional paper 1500-A-J
    Language: English
    Location: Lower compact magazine
    Branch Library: GFZ Library
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  • 2
    Call number: SR 90.0002(1519)
    In: Professional paper
    Type of Medium: Series available for loan
    Pages: VIII, 167 S.
    Series Statement: U.S. Geological Survey professional paper 1519
    Language: English
    Location: Lower compact magazine
    Branch Library: GFZ Library
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  • 3
    Electronic Resource
    Electronic Resource
    s.l. : American Chemical Society
    Biochemistry 33 (1994), S. 12356-12366 
    ISSN: 1520-4995
    Source: ACS Legacy Archives
    Topics: Biology , Chemistry and Pharmacology
    Type of Medium: Electronic Resource
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  • 4
    ISSN: 1520-4995
    Source: ACS Legacy Archives
    Topics: Biology , Chemistry and Pharmacology
    Type of Medium: Electronic Resource
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  • 5
    Electronic Resource
    Electronic Resource
    Springer
    Decisions in economics and finance 13 (1990), S. 111-131 
    ISSN: 1129-6569
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mathematics , Economics
    Description / Table of Contents: Riassunto Si considera una versione modificata del modello di crescita non-lineare di Goodwin, in cui il reddito,Y, è una funzione quadratica dello stock di capitale,k, così da permettere la possibilità di rendimenti crescenti o decrescenti. La dinamica del modello viene allora rappresentata da un sistema non autonomo di due equazioni differenziali. Con l'ipotesi che l'offerta di lavoro,N, e la produttività,a, siano costanti nel tempo, il modello diventa autonomo e può essere immerso in una famiglia stabile di sistemi dinamici piani, di cui vengono descritti i flussi globali e le biforcazioni.
    Notes: Abstract We consider a modified version of Goodwin's celebrated non-linear model of fluctuating growth, where the incomeY is a quadratic function of the capital stockk, in order to take into account the possibility of increasing or decreasing returns. The dynamics of the model is then defined by a non-autonomous system of two differential equations. Assuming the labour supply,N, and the productivity,a, to be constant in the time, the model becomes autonomous and can be embedded in a stable family of planar dynamical systems whose flows and bifurcations are globally described
    Type of Medium: Electronic Resource
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  • 6
    Electronic Resource
    Electronic Resource
    Springer
    Decisions in economics and finance 13 (1990), S. 133-145 
    ISSN: 1129-6569
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mathematics , Economics
    Description / Table of Contents: Riassunto Un modello non-lineare di crescita alla Goodwin, in cui i rendimenti possono essere non costanti, è rappresentato da un sistema dinamico piano non autonomo, che può essere trasformato in un sistema autonomo tridimensionale. Viene descritto il comportamento globale di quest'ultimo nei due casi di rendimenti crescenti o decrescenti. Si dimostra, in particolare, l'assenza di attrattori economicamente significativi ed il diverso carattere delle orbite: mentre, se i rendimenti sono crescenti, le traiettorie convergono asintoticamente ad un punto singolare dove “l'economia muore”, nel caso opposto le traiettorie sono illimitate e spiraleggiano intomo ad una retta che ha la direzione dell'assek (la variabile di stato che rappresenta lo stock di capitale).
    Notes: Abstract The dynamics of a model of fluctuating growth, where non-constant returns are allowed, is represented, under Goodwin's classical assumptions, by a non-autonomous two-dimensional system, which can be transformed into an autonomous three-dimensional one. We describe the global phase portrait of the latter, in the two cases of increasing and decreasing returns, proving, in particular, the absence of economically meaningful “attractors”. However the orbits exhibit different features in the two cases: namely they asymptotically converge to a singular point, where “the economy dies”, if the returns are decreasing, and diverge, spiralling around a certain line, if the returns are increasing.
    Type of Medium: Electronic Resource
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  • 7
    Electronic Resource
    Electronic Resource
    Springer
    Decisions in economics and finance 16 (1993), S. 39-58 
    ISSN: 1129-6569
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mathematics , Economics
    Notes: Abstract We study a non-linear model of the interactions between stock market prices and the level of assets owned by investment funds. The model dynamics is described, in continuous time, by a smooth vector field in the plane, which presents, under suitable hypotheses, a unique equilibrium point. Our analisis of the system flow is qualitative and focuses on detecting endogenous fluctuations of the state variables, i.e. on checking existence and number of limit cycles. We prove that several, and quite different, dynamical patterns can occur, even in cases where the system isoclines assume that “most simple” geometrical forms. It is shown, in particular, that the equilibrium point can undergo either a sub-critical or a super-critical Hopf-bifurcation whenever two economically meaningful exogenous parameters are made to cross a given set of critical values. Hence, in the subcritical case, as a trapping region exists, at least two limit cycles appear. Next, we give analytical examples of model-consistent vector fields which present a multiplicity of fluctuating trends, and prove the apparently surprising result that the number of limit cycles can be as large as one wants, provided a specific isocline assumes a “cubic” shape.
    Type of Medium: Electronic Resource
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  • 8
    Electronic Resource
    Electronic Resource
    Oxford, UK : Blackwell Publishing Ltd
    Annals of the New York Academy of Sciences 717 (1994), S. 0 
    ISSN: 1749-6632
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Natural Sciences in General
    Type of Medium: Electronic Resource
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  • 9
    Electronic Resource
    Electronic Resource
    [s.l.] : Nature Publishing Group
    Nature 344 (1990), S. 821-821 
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Type of Medium: Electronic Resource
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  • 10
    ISSN: 0021-9541
    Keywords: Life and Medical Sciences ; Cell & Developmental Biology
    Source: Wiley InterScience Backfile Collection 1832-2000
    Topics: Biology , Medicine
    Notes: Increasing evidence suggests that transforming growth factor-β (TGF-β) is involved in bone formation during remodeling. Using a recently cloned human leukemic cell line (FLG 29.1 cells) we demonstrate that these cells synthesize and secrete TGF-β1 and that exogenous or autocrine TGF-β1 can induce the same features of osteoclastic-like cells, exerting its effects through the binding to TGF-β specific receptors. Scatchard analysis of 125I-labeled TGF-β1 to FLG 29.1 cells revealed the presence of a single high affinity binding site with a Kd value of ∼25 pM and a binding capacity of ∼900 sites/cell. Affinity labeling experiments showed that FLG 29.1 cells express type I and type II TGF-β receptors. Stimulation of FLG 29.1 cells with low TGF-β1 doses reduced cell proliferation and increased cell adhesion and tartrate resistant acid phosphatase (TRAcP) activity. Pretreatment of FLG 29.1 cells with TGF-β1 caused a significant and dose-dependent response to calcitonin. Northern blot of total mRNA and analysis of the conditioned media (CM) showed that TGF-β1 was synthesized by FLG 29.1 cells. TPA treatment, which induces partial differentiation of these cells, markedly increased TGF-β1 mRNA expression and growth factor release. The majority of TGF-β1 secreted by TPA-treated cells was in its latent form. However, anti-TGF-β antibodies inhibited TGF-β1 and TPA-induced growth inhibition, calcitonin responsiveness, and TRAcP activity, suggesting that the TPA effect is mediated in part by autocrine TGF-β1 and indicating that the cells can activate and respond to the TGF-β that they secrete. These findings support a potential autocrine role for TGF-β1 in osteoclast differentiation. © 1994 Wiley-Liss, Inc.
    Additional Material: 11 Ill.
    Type of Medium: Electronic Resource
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