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  • 2005-2009  (94)
  • 2000-2004  (80)
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  • 1
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    Stable isotopes of sediment core MD84-658 (2006)
    PANGAEA
    Details
    Publication Date: 2023-02-25
    Keywords: Climatic Variability of the Mediterranean Paleo-circulation; CLIVAMP; DEPTH, sediment/rock; Globigerinoides ruber white, δ18O; Marion Dufresne (1972); Mass spectrometer VG Micromass 602; MD84-658; Mediterranean Sea; NOE; PC; Piston corer
    Type: Dataset
    Format: text/tab-separated-values, 9 data points
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  • 2
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    Stable isotopes of sediment core KET82-16 (2006)
    PANGAEA
    Details
    Publication Date: 2023-06-27
    Keywords: Climatic Variability of the Mediterranean Paleo-circulation; CLIVAMP; DEPTH, sediment/rock; ETNA82; Globigerina bulloides, δ18O; Globigerinoides ruber white, δ18O; KET82-16; Mass spectrometer VG Micromass 602; Mediterranean Sea; PC; Piston corer
    Type: Dataset
    Format: text/tab-separated-values, 47 data points
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  • 3
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    Stable isotopes of sediment core KET82-22 (2006)
    PANGAEA
    Details
    Publication Date: 2023-06-27
    Keywords: Climatic Variability of the Mediterranean Paleo-circulation; CLIVAMP; DEPTH, sediment/rock; ETNA82; Globigerina bulloides, δ18O; Globigerinoides ruber white, δ18O; KET82-22; Mass spectrometer VG Micromass 602; Mediterranean Sea; PC; Piston corer
    Type: Dataset
    Format: text/tab-separated-values, 38 data points
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  • 4
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    Stable isotopes of sediment core RC09-191 (2006)
    PANGAEA
    Details
    Publication Date: 2023-06-27
    Keywords: Climatic Variability of the Mediterranean Paleo-circulation; CLIVAMP; DEPTH, sediment/rock; Globigerinoides ruber white, δ18O; Mass spectrometer VG Micromass 602; PC; Piston corer; RC09; RC09-191; Robert Conrad
    Type: Dataset
    Format: text/tab-separated-values, 20 data points
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  • 5
    Electronic Resource
    Electronic Resource
    Basal body dysfunction is a likely cause of pleiotropic Bardet–Biedl syndrome (2003)
    [s.l.] : Macmillian Magazines Ltd.
    Nature 425 (2003), S. 628-633 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] Bardet–Biedl syndrome (BBS) is a genetically heterogeneous disorder characterized primarily by retinal dystrophy, obesity, polydactyly, renal malformations and learning disabilities. Although five BBS genes have been cloned, the molecular basis of this syndrome remains elusive. Here we ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/nature02030
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  • 6
    Electronic Resource
    Electronic Resource
    Real Business Cycle Theory and the Great Depression: The Abandonment of the Abstentionist Viewpoint (2006)
    Berkeley, Calif. : Berkeley Electronic Press (now: De Gruyter)
    Contributions to macroeconomics 6.2006, 1, art13 
    Details
    ISSN: 1534-6005
    Source: Berkeley Electronic Press Academic Journals
    Topics: Economics
    Notes: Is the Great Depression amenable to real business cycle theory? In the 1970s and 1980s Lucas and Prescott took an abstentionist stance. They maintained that, because of its exceptional character, an explanation of the Great Depression was beyond the grasp of the equilibrium approach to the business cycle. However, while Lucas stuck to this view, Prescott changed his mind at the end of the 1990s, breaking his earlier self-imposed restraint. In this paper we document this evolution of opinion and produce a first assessment of real business cycle models of the Great Depression. We claim that the fact of having constructed an equilibrium model of the Great Depression constitutes a methodological breakthrough. However, as far as substance is concerned, we argue that the contribution of real business cycle literature on the Great Depression is slim, and does not gain the upper hand over the work of economic historians.
    Type of Medium: Electronic Resource
    URL: http://www.bepress.com/bejm/contributions/vol6/iss1/art13
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  • 7
    Electronic Resource
    Electronic Resource
    Antibiotic-induced expression of a cryptic cpb2 gene in equine β2-toxigenic Clostridium perfringens (2005)
    Oxford, UK : Blackwell Science Ltd
    Molecular microbiology 57 (2005), S. 0 
    Details
    ISSN: 1365-2958
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology , Medicine
    Notes: The cpb2 gene of β2-toxigenic Clostridium perfringens isolated from horses, cattle, sheep, human and pigs was sequenced. The cpb2 gene of equine and other non-porcine isolates differed from porcine isolates by the absence of an adenine in a poly A tract immediately downstream of the start codon in all non-porcine C. perfringens strains. This deletion involved formation of a cryptic gene harbouring a premature stop codon after only nine amino acid codons, while the full β2-toxin protein consists of 265 amino acids. Immunoblots carried out with antibodies directed against a recombinant β2-toxin showed the absence of expression of the β2-toxin in equine and the other non-porcine strains under standard culture conditions. However, treatment of C. perfringens with the aminoglycosides gentamicin or streptomycin was able to induce expression of the cpb2 gene in a representative equine strain of this group, presumably by frameshifting. The presence of the β2-toxin was revealed by immunohistology in tissue samples of small and large intestine from horses with severe typhlocolitis that had been treated before with gentamicin. This result may explain the finding that antibiotic treatment of horses affected by β2-toxigenic C. perfringens leads to a more accentuated and fatal progression of equine typhlocolitis. Clinical observations show a reduced appearance of strong typhlocolitis in horses with intestinal complications admitted to hospital care since the standard use of gentamicin has been abandoned. This is the first report on expression of a bacterial toxin gene by antibiotic-induced ribosomal frameshifting.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2958.2005.04789.x
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  • 8
    Electronic Resource
    Electronic Resource
    Expression of cnf1 by Escherichia coli J96 involves a large upstream DNA region including the hlyCABD operon, and is regulated by the RfaH protein (2003)
    Oxford, UK : Blackwell Science, Ltd
    Molecular microbiology 47 (2003), S. 0 
    Details
    ISSN: 1365-2958
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology , Medicine
    Notes: Examination of 55 clinical isolates of uropathogenic Escherichia coli producing the CNF1 toxin demonstrated that the cnf1 gene is systematically associated with a hly operon via a highly conserved hlyD-cnf1 intergenic region (igs, 943 bp) as shown in the J96 UPEC strain. We examined if this association could reflect a co-regulation of the production of these toxins. Translation of cnf1 from an immediately upstream promoter has been shown to be controlled by means of an anti-Shine–Dalgarno sequence present in the cnf1 coding sequence [fold-back inhibition (cnf1 fbi)]. The cnf1 fbi was not regulated by elements present in the igs. An RNA covering the full hlyD sequence, the igs and extending on the cnf1 gene, was then detected in the J96 strain. This RNA could be part of a HlyCABD mRNA. Transcription of the haemolysin operon requires RfaH antitermination activity. Inactivation of rfaH in J96 resulted in a 100-fold reduction of the CNF1 content of bacteria. The production of CNF1 from a plasmidic igscnf1 DNA was not sensitive to RfaH, indicating that this factor acted on cnf1 transcription via the hly promoter. This way the cnf1 fbi mechanism might be overcome by transcription of cnf1 from the haemolysin promoter and antitermination by RfaH. This constitutes a novel system of bacterial virulence factors co-regulation.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1046/j.1365-2958.2003.03391.x
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  • 9
    Electronic Resource
    Electronic Resource
    BotR/A and TetR are alternative RNA polymerase sigma factors controlling the expression of the neurotoxin and associated protein genes in Clostridium botulinum type A and Clostridium tetani (2005)
    Oxford, UK : Blackwell Publishing Ltd.
    Molecular microbiology 55 (2005), S. 0 
    Details
    ISSN: 1365-2958
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology , Medicine
    Notes: Clostridium botulinum and Clostridium tetani, respectively, produce potent toxins, botulinum neurotoxin (BoNT) and tetanus neurotoxin (TeTx), which are responsible for severe diseases, botulism and tetanus. Neurotoxin synthesis is a regulated process in Clostridium. The genes botR/A in C. botulinum A and tetR in C. tetani positively regulate expression of BoNT/A and associated non-toxic proteins (ANTPs), as well as TeTx respectively. The botR/A gene lies in close vicinity of the two operons which contain bont/A and antps genes in C. botulinum A, and tetR immediately precedes the tetX gene in C. tetani. We show that BotR/A and TetR function as specific alternative sigma factors rather than positive regulators based on the following results: (i) BotR/A and TetR associated with target DNAs only in the presence of the RNA polymerase core enzyme (Core), (ii) BotR/A and TetR directly bound with the core enzyme, (iii) BotR/A-Core recognized −35 and −10 regions of ntnh-bont/A promoter and (iv) BotR/A and TetR triggered in vitro transcription from the target promoters. In C. botulinum A, bont/A and antps genes are transcribed as bi- and tricistronic operons controlled by BotR/A. BotR/A and TetR are seemingly related to a new subgroup of the σ70 family that includes TcdR and UviA, which, respectively, regulate production of toxins A and B in C. difficile and bacteriocin in C. perfringens. Sequences of −35 region are highly conserved in the promoter of target toxin genes in C. botulinum, C. tetani, C. difficile and C. perfringens. Overall, a common regulation mechanism probably controls toxin gene expression in these four toxigenic clostridial species.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1365-2958.2004.04377.x
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  • 10
    Electronic Resource
    Electronic Resource
    Transcytosis of iota-toxin across polarized CaCo-2 cells (2002)
    Oxford, UK : Blackwell Science Ltd
    Molecular microbiology 43 (2002), S. 0 
    Details
    ISSN: 1365-2958
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Biology , Medicine
    Notes: Iota-toxin from Clostridium perfringens type E is a binary toxin consisting of two independent proteins, an enzymatic Ia and binding Ib component. Ia catalyses ADP-ribosylation of actin monomers, thus disrupting the actin cytoskeleton. In this report, we show that Ia plus Ib applied apically or basolaterally induce a rapid decrease in the transepithelial resistance (TER) of CaCo-2 cell monolayers and disorganization of actin filaments as well as the tight and adherens junctions. Ib alone, on the apical or basolateral side, slowly decreased the TER without affecting the actin cytoskeleton, possibly via pore formation. Interestingly, the two iota-toxin components inoculated separately on each cell surface induced cytopathic effects and a TER decrease. Anti-Ib sera, raised against the whole molecule or the Ia docking domain and applied to the opposite cell side versus Ib, neutralized the TER decrease. In addition, radioactive Ib incubated in the basolateral compartment was detected on the apical side by selective cell surface biotinylation. This argues for a transcytotic routing of Ib to mediate internalization of Ia from the opposite cell surface. Bafilomycin A1 also prevented the cytopathic effects of Ia and Ib applied separately to each cell side, possibly by blocking translocation of Ia into the cytosol and/or the intracellular transport of Ib. Ib is either routed into the cell independently of Ia, trans-cytosed and permanently exposed on the opposite cell surface or continuously recycled between an endosomal compartment and the cell surface.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1046/j.1365-2958.2002.02806.x
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