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  • 1
    ISSN: 1588-2837
    Keywords: Nitrogen oxide reduction ; catalyst selectivity ; ammonia
    Source: Springer Online Journal Archives 1860-2000
    Topics: Chemistry and Pharmacology
    Notes: Abstract The activity and selectivity of V2O5/γ-Al2O3 catalyst were studied in the catalytic reduction of nitrogen oxide by ammonia. The activity of the catalyst monotonically increases as a function of temperature, however, its selectivity decreases. The DeNOx reaction of nitrogen oxides with ammonia can be described well by a mathematical model, which considers selectivity-decreasing side reactions as well in a wide temperature range (220–420°C).
    Type of Medium: Electronic Resource
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  • 2
    Publication Date: 2015-10-05
    Description: Three spatial structure groups of radionuclides in U and Th series, 210Pb-excess and 137Cs, and 40K were found based on analyzing temporal and spatial datum of their content by factor analysis with oblique rotation in Nhatrang bay. U and Th spatial structure with their contours decreased toward the offshore, ran longshore and divided seawater of bay into two parts with strong gradient on both sides. Inside part located from center of Nhatrang bay toward the seashore with three main deposit centers of their contents higher than 23 Bq/kg.dry for 238U and 40 Bq/kg.dry for 232Th, indicated unstability of shoreline. Almost sediments coming from river extended toward the offshore, were stopped and transported toward southeastern. The outside part was less than above mentioned content. The boundary line between two parts superposed with the constantly limit line of turbid plume in the rainy season. Direct influence of the continental runoff was limited by the 9 Bq/kg.dry contour of 238U, 19 Bq/kg.dry contour of 232Th. Longshore current was a predominant process whereas lateral transport as sifting and winnowing process of finer grains in sediments of Nhatrang bay. Areas that had very low content of 137Cs and 210 Pb-excess adjoining shoreline showed areas being eroded. Accumulation of 137Cs and 210 Pbexcess nearby river mouth characterized for fine compositions of sediments controlled by seasonal plumes and sites further toward the south indicated finer materials transported from river and accumulated in lack of hydrodynamic process. Near shore accumulation of 40K revealed the sediments there originated from bed erosion.
    Keywords: Oceanography
    Repository Name: Aquatic Commons
    Type: Article , NonPeerReviewed
    Format: application/pdf
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  • 3
    Publication Date: 2019-04-05
    Description: Distribution of phytoplankton pigments was investigated in the relation to Chlorophyll a (Chl-a) and light intensity in Vietnamese waters located at longitude 102E - 112W, latitute 23N - 7N. Over 200 samples collected at 58 stations were analyzed for pigments (Chlorophyll a, b, c and carotenoids) and degradation products (Phaeophytill). Chlorophyll a was measured by fluorescence. Results show that average values in the seawater were 0.18 ± 0.04 mg.m-3 for Chl-a; 0.05 ± 0.01 mg.m-3 for Chl-b; 0.062 mg.m-3 for Phaeophytill. Higher value of Chl-a occurred at the thermocline but maxima were found at 75 or 50m depths. Average value of Carotenoids concentration was very low about 0.052 ± 0.12 mg.m-3. The report used a model for the relationship between Chlorophyll a content and light intensity to estimate the primary production. Average value of primary production was about 9.04 mgC.m3.day-1 at the surface and 2.63 mgC.m3.day-1 at the bottom. The relationship between Chlorophyll and some environmental parameters such as temperature, salinity was examined. The effects of thermocline and halocline to the primary production were analyzed.
    Keywords: Biology ; Oceanography
    Repository Name: Aquatic Commons
    Type: Book Section , NonPeerReviewed
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  • 4
    Publication Date: 2014-04-16
    Description: MicroRNAs (miRNAs), including miR-1, miR-133, and miR-206, play a crucial role in muscle development by regulating muscle cell proliferation and differentiation. The aim of the present study was to define the effect of miR-124 on myogenic differentiation of mesenchymal stem cells (MSCs). The expression level of miR-124 in skeletal muscles was much lower than those in primary cultured bone marrow-derived MSCs and the bone, fat and brain tissues obtained from C57BL/6 mice. Myogenic stimuli significantly decreased the expression levels of miR-124 in mouse bone marrow-derived MSCs and C2C12 cells. Forced expression of miR-124 suppressed the expression of myogenic marker genes such as Myf5, Myod1, myogenin and myosin heavy chain and multinucleated myotube formation. Blockade of endogenous miR-124 with a hairpin inhibitor enhanced myogenic marker gene expression and myotube formation. During myogenic differentiation of MSCs and C2C12 cells, the levels of Dlx5, a known target of miR-124, were inversely regulated with those of miR-124. Furthermore, overexpression of Dlx5 increased myogenic differentiation, whereas knockdown of Dlx5 using siRNA inhibited myogenesis in C2C12 cells. These results suggest that miR-124 is a negative regulator of myogenic differentiation of MSCs and that upregulation of Dlx5 accompanied with downregulation of miR-124 by myogenic stimuli is necessary for the proper progression of myogenic differentiation. J. Cell. Biochem. © 2014 Wiley Periodicals, Inc.
    Electronic ISSN: 0091-7419
    Topics: Biology , Chemistry and Pharmacology , Medicine
    Published by Wiley
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  • 5
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    American Association for the Advancement of Science (AAAS)
    Publication Date: 1998-08-28
    Description: Comparisons of gyre-scale acoustic and direct thermal measurements of heat content in the Pacific Ocean, satellite altimeter measurements of sea surface height, and results from a general circulation model show that only about half of the seasonal and year-to-year changes in sea level are attributable to thermal expansion. Interpreting climate change signals from fluctuations in sea level is therefore complicated. The annual cycle of heat flux is 150 +/- 25 watts per square meter (peak-to-peak, corresponding to a 0.2 degreesC vertically averaged temperature cycle); an interannual change of similar magnitude is also detected. Meteorological estimates of surface heat flux, if accurate, require a large seasonal cycle in the advective heat flux.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉TAC -- New York, N.Y. -- Science. 1998 Aug 28;281(5381):1327-32.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉The ATOC Consortium: A. B. Baggeroer, T. G. Birdsall, C. Clark, J. A. Colosi, B. D. Cornuelle, D. Costa, B. D. Dushaw, M. Dzieciuch, A. M. G. Forbes, C. Hill, B. M. Howe, J. Marshall, D. Menemenlis, J. A. Mercer, K. Metzger, W. Munk, R. C. Spindel, D.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/9721093" target="_blank"〉PubMed〈/a〉
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 6
    Publication Date: 2014-10-10
    Description: A non-invasive method to characterize human mesenchymal stromal cells during adipogenic differentiation was developed for the first time. Seven fatty acid methyl esters (FAMEs), including methyl laurate, methyl myristate, methyl palmitate, methyl linoleate, methyl oleate, methyl elaidate and methyl stearate, were used for characterizing adipogenic differentiation using headspace solid-phase microextraction (HS-SPME) which is a very simple and non-invasive method for the extraction of volatile compounds. Glassware was used for culturing mesenchymal stromal cells rather than the common plasticware to minimize contamination by volatile impurities. The optimal SPME fiber was selected by comparing diverse fibers containing two pure liquid polymers (PDMS and PA) and two porous solids (PDMS/DVB and CAR/PDMS). Using optimized procedures, we discovered that seven FAMEs were only detected in adipogenic differentiated mesenchymal stromal cells and not in the mesenchymal stromal cells before differentiation. These data could support the quality control of clinical mesenchymal stromal cell culture in the pharmaceutical industry in addition to the development of many clinical applications using mesenchymal stromal cells. Scientific Reports 4 doi: 10.1038/srep06550
    Electronic ISSN: 2045-2322
    Topics: Natural Sciences in General
    Published by Springer Nature
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  • 7
    Publication Date: 2014-02-15
    Description: Journal of Proteome Research DOI: 10.1021/pr400990k
    Print ISSN: 1535-3893
    Electronic ISSN: 1535-3907
    Topics: Chemistry and Pharmacology
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  • 8
    Publication Date: 2017-02-24
    Description: Mesenchymal stromal cells inhibit CD25 expression via the mTOR pathway to potentiate T-cell suppression Cell Death and Disease 8, e2632 (February 2017). doi:10.1038/cddis.2017.45 Authors: Hyun Seung Yoo, Kyuheon Lee, Kwangmin Na, Yong Xu Zhang, Hyun-Ja Lim, TacGhee Yi, Sun U Song & Myung-Shin Jeon
    Electronic ISSN: 2041-4889
    Topics: Biology , Medicine
    Published by Springer Nature
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  • 9
    Publication Date: 2012-10-06
    Description: Smad Ubiquitination Regulatory Factor 1 (Smurf1) is an E3 ubiquitin ligase that negatively regulates osteoblast differentiation. Although tumor necrosis factor-α (TNF-α) has been shown to increase Smurf1 expression, the details of the regulatory mechanisms remain unclear. Here, we investigated the molecular mechanism by which TNF-α stimulates Smurf1 expression in C2C12 and primary cultured mouse calvarial cells. TNF-α treatment rapidly induced the activation of NF-κB and MAPKs. Smurf1 induction by TNF-α was blocked by the inhibition of JNK or ERK, while the inhibition of NF-κB and p38 MAPK had no effect on Smurf1 induction. TNF-α treatment or c-Jun overexpression enhanced the activity of a luciferase reporter that contained a 2.7 kb mouse Smurf1 promoter sequence. Site-directed mutagenesis of the Smurf1 reporter and chromatin immunoprecipitation analysis demonstrated that the activating protein-1 (AP-1) binding motif at -922 bp on the mouse Smurf1 promoter mediated TNF-α/JNK/AP-1-stimulated Smurf1 transcription. Interestingly, Smurf1 expression was not observed in Runx2-null mouse calvarial cells. When Runx2 was ectopically expressed in these cells, the basal and TNF-α-induced expression of Smurf1 was restored. Overexpression of Runx2 transactivated the Smurf1 promoter in a dose-dependent manner. Reporter and chromatin immunoprecipitation assays demonstrated that the Runx2-binding motif at -202 bp functioned in Runx2-mediated Smurf1 expression. ERK activation by TNF-α treatment or constitutively active MEK1 overexpression increased Smurf1 expression in a Runx2-dependent manner. These results suggest that the JNK/AP-1 and ERK/Runx2 signaling pathways mediate TNF-α-dependent Smurf1 transcription. J. Cell. Physiol. © 2012 Wiley Periodicals, Inc.
    Electronic ISSN: 1097-4652
    Topics: Biology , Medicine
    Published by Wiley
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  • 10
    Publication Date: 1995-10-15
    Print ISSN: 0094-8276
    Electronic ISSN: 1944-8007
    Topics: Geosciences , Physics
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