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  • 1
    Electronic Resource
    Electronic Resource
    REGULATION OF SALIVARY GLAND AMYLASE ACTIVITY (1960)
    Oxford, UK : Blackwell Publishing Ltd
    Annals of the New York Academy of Sciences 85 (1960), S. 0 
    Details
    ISSN: 1749-6632
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Natural Sciences in General
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1749-6632.1960.tb49957.x
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  • 2
    Electronic Resource
    Electronic Resource
    Alteration of antigenic components of rat parotid gland following denervation (1974)
    Springer
    Cellular and molecular life sciences 30 (1974), S. 950-951 
    Details
    ISSN: 1420-9071
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Résumé Altération des caractères de la glande parotide du rat par dénervation séléctive ou après diète liquide. A la suite de l'ablation unilatérale du ganglion cervical supérieur, une augmentation marquante de la cocentration des composants antigéniques solubles dans le salin se produisit aussi bien dans la glande parotide denervée que dans la glande intacte. Par ailleurs, après l'ablation partielle du nerf auriculotemporal, les composants antigéniques de la glande parotide denervée diminuèrent. La corrélation possible entre ces changements et le développement de la glande parotide est discutée.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01938380
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  • 3
    Electronic Resource
    Electronic Resource
    Prostaglandin E1 induced salivary secretion (1982)
    Springer
    Cellular and molecular life sciences 38 (1982), S. 1077-1078 
    Details
    ISSN: 1420-9071
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary PGE1 but not PGF2α at 500–1000 μg/kg induced a slow and sparse flow from the parotid and no flow at all from submaxillary glands. Composition of PGE1-induced parotid saliva was quite different from that evoked by any autonomic agonists. The present study suggests that PGE1 might act directly on parotid acinar cells.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01955376
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  • 4
    Electronic Resource
    Electronic Resource
    Cell surface galactosyltransferase acts as a general modulator of rat acinar cell proliferation (1990)
    Springer
    Molecular and cellular biochemistry 95 (1990), S. 1-11 
    Details
    ISSN: 1573-4919
    Keywords: parotid gland ; galactosyltransferase ; cell proliferation ; diet ; growth factors
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology , Medicine
    Notes: Summary Several physiological parameters were examined for inducing acinar cell proliferation and corresponding increased expression of β1–4 galactosyltransferase. In this study, dietary changes causing acinar cell proliferation included the following: the introduction of animals to a liquid diet (causing gland atrophy) followed by reintroduction of solid chow, gustatory stimulation provided by the introduction of 0.5% citric acid to animal drinking water, and removal of the submandibular gland with subsequent reliance on the parotid gland for salivary protein. Alterations in growth factor levels were produced by injecting animals with a chronic (3 day) regimen of either nerve growth factor or epidermal growth factor. Parotid gland proliferation could be blocked in all cases except EGF by the injection of propranolol, a β-adrenoceptor antagonist, or the galactosyltransferase specific modifier protein, α-lactalbumin. EGF-induced proliferation could, however, be prevented by treating the animals with monoclonal antibody to EGF receptor or galactosyltransferase modifier protein a-lactalbumin. These results for normal acinar cell proliferation suggest a direct role for cell surface β1–4 galactosyltransferase in signalling and maintaining active cell growth.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00219524
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  • 5
    Electronic Resource
    Electronic Resource
    Neural regulation of compensatory enlargement of the parotid gland of the rat (1978)
    Springer
    Cell & tissue research 187 (1978), S. 147-151 
    Details
    ISSN: 1432-0878
    Keywords: Parotid gland ; Compensatory enlargement ; Neural regulation
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary The role of the innervation in mediating compensatory enlargement of the parotid gland of the rat after partial desalivation was examined. The results of denervation experiments show that full compensatory growth requires both parasympathetic and sympathetic innervation. The presence of the parasympathetic innervation alone results in an increase in the number of cells, but not the size of the cells. The sympathetic innervation alone does not mediate either response. We, therefore, conclude that the two types of innervation have a synergistic action on the parotid to produce the maximal compensatory response, which includes an increase in both number and size of acinar cells.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00220626
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  • 6
    Electronic Resource
    Electronic Resource
    Functional mediation of compensatory enlargement of the parotid gland (1977)
    Springer
    Cell & tissue research 184 (1977), S. 249-254 
    Details
    ISSN: 1432-0878
    Keywords: Parotid gland ; Compensatory enlargement ; Function
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary The role of functional activity in mediating compensatory enlargement of the parotid gland after removal of the other major salivary glands was investigated. Increased levels of activity were achieved by feeding rats a bulk diet. Conversely, a liquid diet was used to reduce the functional demands on the parotid. It was found that the liquid diet completely prevented the compensatory response from occurring. Bulk diet, on the other hand, caused an even greater compensatory response than did the standard chow diet. Compensatory enlargement of the parotid, therefore, depends on its functional activity and not on other, e.g., humoral factors dissociated from function. The character of the cellular response in compensatory enlargement was also examined. The chow diet caused compensatory enlargement by an increase in cell size with little, if any, increase in cell number.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00223072
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  • 7
    Electronic Resource
    Electronic Resource
    Ultrastructural changes of rat parotid glands induced by a diet of liquid Metrecal (1970)
    Springer
    Cell & tissue research 103 (1970), S. 1-11 
    Details
    ISSN: 1432-0878
    Keywords: Parotid glands ; Cytology ; Liquid diet ; Atrophy ; Secretory rate
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary The ultrastructure of parotid glands was studied in rats fed a diet of liquid Metrecal for two weeks and compared with that of parotid glands of control rats which received a diet of Purina lab chow. The liquid diet induced major alterations of acinar cells, but other parenchymal components were apparently unaffected. Most acinar cells of experimental rats were atrophic and some of these were undergoing necrosis. Lipid droplets and dense bodies (believed to be lysosomes) were numerous in atrophic cells. The Golgi apparatus, quantity of secretory granules, and intercellular canaliculi were smaller than in acinar cells of control rats. Such findings suggest that the secretory process was impaired and support the conclusion that parotid glands of rats maintained on a liquid diet are physiologically less active than those of chow fed rats. The decreased activity, as previously reported, may result from reduced masticatory activity.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00335396
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  • 8
    Electronic Resource
    Electronic Resource
    Inhibition of isoproterenol-induced DNA and RNA synthesis in rat parotid and pancreas following removal of submandibular-sublingual glands (1989)
    Springer
    Cell & tissue research 256 (1989), S. 361-363 
    Details
    ISSN: 1432-0878
    Keywords: [3H] thymidine ; [3H] uridine ; Parotid ; Pancreas ; Isoproterenol ; Rat (Long-Evans)
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary [3H] thymidine incorporation into DNA of the parotid (PA) gland of adult and 20-day-old rats and into DNA of the pancreas (PANC) of 20-day-old rats was increased markedly following a 2-day regimen of isoproterenol (ISO) administration. However, when the submandibular-sublingual (SM-SL) glands had been removed just prior to initiation of the ISO injections, the [3H] thymidine incorporation into PA and PANC was inhibited, and cpm/mg protein of these organs was even lower than that of organs of untreated rats with SM-SL glands present. Removal of the PA glands just prior to initiation of the ISO regimen had no effect on the ISO-induced [3H] thymidine incorporation into DNA of PANC but partially inhibited that of the submandibular (SM) gland. It is suggested that the inhibitory effects on DNA and RNA synthesis that follow removal of SM-SL glands are attributable to the growth factors (epidermal growth factor and nerve growth factor) found in the rat SM gland. These factors appear to regulate normal DNA synthetic activity of exocrine glands as well as β 1-adrenoceptor mediated DNA synthesis. Cellular hypertrophy induced by the ISO was less markedly affected by absence of the SM glands, but a partial inhibition of [3H] uridine incorporation into RNA of PA of adult rats also occurred when SM-SL glands were removed prior to initiation of the ISO-regimen.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00218893
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  • 9
    Electronic Resource
    Electronic Resource
    Effect of postganglionic sympathectomy on the ultrastructure of the rat parotid gland (1972)
    Springer
    Cell & tissue research 130 (1972), S. 471-480 
    Details
    ISSN: 1432-0878
    Keywords: Parotid glands ; Cytology ; Sympathectomy ; Secretion
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Following two weeks of superior cervical ganglionectomy, the parotid glands of adult rats were removed and studied by electron microscopy. Sympathectomy induced striking alterations of acini, resulting in a heterogeneous population of acinar cells, but it had no obvious effect on the duct system. Most of the altered cells could be classified on a cytological basis as “dark cells” or “light cells.” Dark cells predominated and contained more secretory granules, less granular endoplasmic reticulum, fewer Golgi membranes, and smaller lumina and intercellular canaliculi than normal acinar cells. The synthesis and extrusion of secretory products appeared to be minimal in these cells. Light cells possessed ultrastructural features, such as dilated cisternae of granular endoplasmic reticulum and prominent Golgi membranes, which were opposite to those of dark cells and indicative of a high degree of secretory activity. The heterogeneous population of cells following sympathectomy indicates that the sympathetic nervous system may play an important role in regulating the secretory synchrony of acinar cells.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00307001
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  • 10
    Electronic Resource
    Electronic Resource
    Effects of cystic fibrosis serum on the rat parotid gland (1976)
    Springer
    Cell & tissue research 169 (1976), S. 111-121 
    Details
    ISSN: 1432-0878
    Keywords: Cystic fibrosis ; Bioassay, cystic fibrosis ; Mitotic rate ; Rat parotid
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Injections of serum from human patients with cystic fibrosis into adult rats caused pronounced structural modifications and increased mitotic rate in the parotid gland. Mitotic rate was increased from a low level of 0.02/1,000 acinar cells in parotid glands of adult rats to 6.5/1,000 acinar cells after 2 or 3 days of serum injection. At the light and electron microscopic levels, significant acinar cell atrophy and degranulation were observed. Cellular necrosis, and increases in quantity of lysosome-like dense bodies, mast cells, and macrophages were also detected. These changes are suggestive of tissue response to injurious foreign protein. Furthermore, the fact that normal sera pronounced the same kind of effects (but greatly reduced in extent) strengthens the view that these effects result from the immunologic response of the host organ to foreign antigen. Since, however, the responses of the rat parotid to cystic fibrosis serum were considerably more marked than those elicited by normal serum, the rat parotid may thus have potential usefulness in assaying for the presence of human cystic fibrosis factor.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00219311
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