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  • 1
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    Seismological constraints on the gabbro-eclogite transition in subducted oceanic crust (1994)
    In:  Earth planet. Sci. Lett., Kleinmachnow, Dt. Geophys. Ges. e. V., vol. 122, no. 6, pp. 89-101, pp. B05401, (ISBN: 0534351875, 2nd edition)
    Details
    Publication Date: 1994
    Keywords: Seismology ; Earth model, also for more shallow analyses ! ; Subduction zone ; phase ; transition
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    BIBLIOGRAPHY SEISMOLOGY
  • 2
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    Soluble adenylyl cyclase as an evolutionarily conserved bicarbonate sensor (2000)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2000-08-01
    Description: Spermatozoa undergo a poorly understood activation process induced by bicarbonate and mediated by cyclic adenosine 3',5'-monophosphate (cAMP). It has been assumed that bicarbonate mediates its effects through changes in intracellular pH or membrane potential; however, we demonstrate here that bicarbonate directly stimulates mammalian soluble adenylyl cyclase (sAC) activity in vivo and in vitro in a pH-independent manner. sAC is most similar to adenylyl cyclases from cyanobacteria, and bicarbonate regulation of cyclase activity is conserved in these early forms of life. sAC is also expressed in other bicarbonate-responsive tissues, which suggests that bicarbonate regulation of cAMP signaling plays a fundamental role in many biological systems.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Chen, Y -- Cann, M J -- Litvin, T N -- Iourgenko, V -- Sinclair, M L -- Levin, L R -- Buck, J -- New York, N.Y. -- Science. 2000 Jul 28;289(5479):625-8.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Pharmacology, Joan and Sanford I. Weill Medical College of Cornell University, 1300 York Avenue, New York, NY 10021, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/10915626" target="_blank"〉PubMed〈/a〉
    Keywords: Adenylyl Cyclases/chemistry/genetics/isolation & purification/*metabolism ; Animals ; Bicarbonates/*metabolism/pharmacology ; Catalytic Domain ; Cell Line ; Cyanobacteria/enzymology ; Cyclic AMP/metabolism ; Enzyme Activation ; Evolution, Molecular ; Humans ; Hydrogen-Ion Concentration ; Male ; Phylogeny ; Rats ; Recombinant Proteins/isolation & purification/metabolism ; Second Messenger Systems ; Signal Transduction ; Solubility ; Sperm Capacitation ; Spermatozoa/enzymology/*metabolism/physiology ; Testis/metabolism
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 3
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    The identification of carbon dioxide mediated protein post-translational modifications (2018)
    Springer Nature
    Details
    Publication Date: 2018-08-07
    Description: The identification of carbon dioxide mediated protein post-translational modifications The identification of carbon dioxide mediated protein post-translational modifications, Published online: 06 August 2018; doi:10.1038/s41467-018-05475-z Carbon dioxide can interact with proteins to form carbamate post-translational modifications. Here, the authors developed a strategy to identify carbamate post-translational modifications by trapping carbon dioxide and subsequently identifying the carbamylated proteins.
    Electronic ISSN: 2041-1723
    Topics: Biology , Chemistry and Pharmacology , Natural Sciences in General , Physics
    Published by Springer Nature
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  • 4
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    Central role of detachment faults in accretion of slow-spreading oceanic lithosphere (2008)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2008-10-10
    Description: The formation of oceanic detachment faults is well established from inactive, corrugated fault planes exposed on sea floor formed along ridges spreading at less than 80 km Myr(-1) (refs 1-4). These faults can accommodate extension for up to 1-3 Myr (ref. 5), and are associated with one of the two contrasting modes of accretion operating along the northern Mid-Atlantic Ridge. The first mode is asymmetrical accretion involving an active detachment fault along one ridge flank. The second mode is the well-known symmetrical accretion, dominated by magmatic processes with subsidiary high-angle faulting and the formation of abyssal hills on both flanks. Here we present an examination of approximately 2,500 km of the Mid-Atlantic Ridge between 12.5 and 35 degrees N, which reveals asymmetrical accretion along almost half of the ridge. Hydrothermal activity identified so far in the study region is closely associated with asymmetrical accretion, which also shows high levels of near-continuous hydroacoustically and teleseismically recorded seismicity. Increased seismicity is probably generated along detachment faults that accommodate a sizeable proportion of the total plate separation. In contrast, symmetrical segments have lower levels of seismicity, which occurs primarily at segment ends. Basalts erupted along asymmetrical segments have compositions that are consistent with crystallization at higher pressures than basalts from symmetrical segments, and with lower extents of partial melting of the mantle. Both seismic evidence and geochemical evidence indicate that the axial lithosphere is thicker and colder at asymmetrical sections of the ridge, either because associated hydrothermal circulation efficiently penetrates to greater depths or because the rising mantle is cooler. We suggest that much of the variability in sea-floor morphology, seismicity and basalt chemistry found along slow-spreading ridges can be thus attributed to the frequent involvement of detachment faults in oceanic lithospheric accretion.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Escartin, J -- Smith, D K -- Cann, J -- Schouten, H -- Langmuir, C H -- Escrig, S -- England -- Nature. 2008 Oct 9;455(7214):790-4. doi: 10.1038/nature07333.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Marine Geosciences Group, CNRS Institut de Physique du Globe de Paris, 4 Place Jussieu, 75252 Paris Cedex 05, France. escartin@ipgp.jussieu.fr〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/18843367" target="_blank"〉PubMed〈/a〉
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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  • 5
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    HIV-1 production from infected peripheral blood T cells after HTLV-I induced mitogenic stimulation (1988)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1988-05-20
    Description: The human immunodeficiency virus type 1 (HIV-1) and human T-cell leukemia virus type I (HTLV-I) are two distinct human retroviruses that infect T cells. Recent epidemiologic studies have identified a cohort of individuals that are coinfected with both viruses. It is reported here that human peripheral blood leukocytes infected with HIV-1 in vitro can be induced to produce large quantities of HIV-1 after mitogenic stimulation by noninfectious HTLV-I virions. It is also shown that HTLV-I virions may exert this effect prior to, immediately following, or well after the cells are infected with HIV-1. These results provide further impetus for epidemiologic studies of dually infected individuals to determine whether HTLV-I may act as a cofactor for acquired immunodeficiency syndrome (AIDS).〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Zack, J A -- Cann, A J -- Lugo, J P -- Chen, I S -- CA 32737/CA/NCI NIH HHS/ -- CA 38597/CA/NCI NIH HHS/ -- CA 43370/CA/NCI NIH HHS/ -- etc. -- New York, N.Y. -- Science. 1988 May 20;240(4855):1026-9.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Division of Hematology-Oncology, UCLA School of Medicine 90024.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/2835813" target="_blank"〉PubMed〈/a〉
    Keywords: Acquired Immunodeficiency Syndrome/microbiology ; Deltaretrovirus/*immunology ; HIV/*growth & development/isolation & purification ; Humans ; *Lymphocyte Activation ; T-Lymphocytes/immunology/*microbiology ; Virus Activation
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 6
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    HTLV-II transactivation is regulated by the overlapping tax/rex nonstructural genes (1988)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1988-05-13
    Description: The human T-cell leukemia virus (HTLV) types I and II have two nonstructural genes that are encoded in overlapping reading frames. One of these genes, known as tax, has been shown to encode a protein responsible for enhanced transcription (transactivation) from the viral long terminal repeats (LTRs). Genetic evidence indicates that the second nonstructural gene of HTLV-II, here designated rex, acts in trans to modulate tax gene-mediated transactivation in a concentration-dependent fashion. The rex gene may regulate the process of transactivation during the viral life cycle.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Rosenblatt, J D -- Cann, A J -- Slamon, D J -- Smalberg, I S -- Shah, N P -- Fujii, J -- Wachsman, W -- Chen, I S -- 1 R01 CA 43370/CA/NCI NIH HHS/ -- 1K11 CA 01314/CA/NCI NIH HHS/ -- CA 32737/CA/NCI NIH HHS/ -- New York, N.Y. -- Science. 1988 May 13;240(4854):916-9.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Medicine, UCLA School of Medicine.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/2834826" target="_blank"〉PubMed〈/a〉
    Keywords: Base Sequence ; DNA, Recombinant ; DNA, Viral/genetics ; Deltaretrovirus/*genetics ; *Genes, Regulator ; *Genes, Viral ; Mutation ; Promoter Regions, Genetic ; RNA, Messenger/genetics/metabolism ; RNA, Viral/genetics/metabolism ; Simian virus 40/genetics ; *Transcription, Genetic ; Transfection
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 7
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    U3 sequences from HTLV-I and -II LTRs confer pX protein response to a murine leukemia virus LTR (1987)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1987-02-20
    Description: Human T-cell leukemia virus (HTLV) types I and II are unusual among replication-competent retroviruses in that they contain a fourth gene (chi) necessary for replication. The chi gene product, p chi, transcriptionally transactivates the viral long repeat (LTR), and is thus a positive regulator. To investigate p chi transactivation, sequences from the U3 regions of the LTRs of HTLV-I and -II were inserted into the Moloney murine leukemia virus (M-MuLV) LTR by recombinant DNA techniques. Transient expression assays of the chimeric LTRs indicated that the HTLV sequences conferred to the M-MuLV LTR responsiveness to HTLV p chi protein. M-MuLV enhancers were not required for function of the chimeric LTRs. Infectious recombinant M-MuLVs containing chimeric LTRs were also generated. These viruses showed higher infectivity when assayed in mouse cells expressing HTLV-II p chi protein compared to normal mouse cells. Thus the HTLV sequences were able to confer p chi responsiveness to infectious M-MuLV. The generation of a virus dependent on a transactivating protein for its replication has implications for the evolution of the human T-cell leukemia viruses.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Kitado, H -- Chen, I S -- Shah, N P -- Cann, A J -- Shimotohno, K -- Fan, H -- CA32454/CA/NCI NIH HHS/ -- CA32455/CA/NCI NIH HHS/ -- CA38597/CA/NCI NIH HHS/ -- New York, N.Y. -- Science. 1987 Feb 20;235(4791):901-4.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/3027896" target="_blank"〉PubMed〈/a〉
    Keywords: DNA, Viral/*genetics ; Deltaretrovirus/*genetics ; Enhancer Elements, Genetic ; Gene Expression Regulation ; Moloney murine leukemia virus/*genetics ; Promoter Regions, Genetic ; Repetitive Sequences, Nucleic Acid ; Retroviridae Proteins/*genetics ; Trans-Activators ; Transcription Factors/*genetics ; Transcription, Genetic ; *Virus Replication
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 8
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    HTLV x gene mutants exhibit novel transcriptional regulatory phenotypes (1987)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1987-02-06
    Description: The human T-cell leukemia viruses, HTLV-I and HTLV-II, contain a gene, termed x, with transcriptional regulatory function. The properties of the x proteins were analyzed by constructing mutant genes containing site-directed deletions and point mutations. The results demonstrate that the amino terminal 17 amino acids of the x protein constitute part of a functional domain that is critical for the transcriptional activating properties of the protein. Within this region, substitution of a leucine residue for a proline residue results in major changes in the trans-activation phenotype of the protein. The mutant HTLV-II x protein, though incapable of activating the HTLV-II long terminal repeat, will block trans-activation of the HTLV-II long terminal repeat by the wild-type protein. The altered phenotype of this mutant suggests a potential negative regulatory function of the x protein.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Wachsman, W -- Cann, A J -- Williams, J L -- Slamon, D J -- Souza, L -- Shah, N P -- Chen, I S -- CA 30388/CA/NCI NIH HHS/ -- CA 32727/CA/NCI NIH HHS/ -- CA 38597/CA/NCI NIH HHS/ -- etc. -- New York, N.Y. -- Science. 1987 Feb 6;235(4789):674-7.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/3027894" target="_blank"〉PubMed〈/a〉
    Keywords: Deltaretrovirus/*genetics ; Gene Expression Regulation ; *Genes, Viral ; Mutation ; Transcription Factors/*genetics ; Transcription, Genetic
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 9
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    Functional relation between HTLV-II x and adenovirus E1A proteins in transcriptional activation (1985)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1985-11-01
    Description: The mechanism of cellular transformation by the human T-cell leukemia viruses (HTLV) is thought to involve a novel gene known as the x gene. This gene is essential for HTLV replication and acts by enhancing transcription from the HTLV long terminal repeat. The HTLV x gene product may also cause aberrant transcription of normal cellular genes, resulting in transformation of the infected cells. Although there is no evidence as yet for such a mechanism, it was shown that the HTLV-II x gene product can activate transcription from adenovirus E1A-dependent early promoters and therefore has the potential to activate cellular genes. It was also shown that the adenovirus and herpes pseudorabies immediate early proteins activate expression from the HTLV-I and HTLV-II long terminal repeats, though at lower levels than with the x gene product. These findings indicate possible common mechanisms of action for transcription-regulatory genes of distinct viruses.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Chen, I S -- Cann, A J -- Shah, N P -- Gaynor, R B -- CA 16042/CA/NCI NIH HHS/ -- CA 32737/CA/NCI NIH HHS/ -- CA 38597/CA/NCI NIH HHS/ -- etc. -- New York, N.Y. -- Science. 1985 Nov 1;230(4725):570-3.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/2996140" target="_blank"〉PubMed〈/a〉
    Keywords: Adenoviridae/*genetics ; Cell Transformation, Viral ; Deltaretrovirus/*genetics ; Endonucleases/metabolism ; HeLa Cells ; Herpesvirus 4, Human ; Humans ; Operon ; Repetitive Sequences, Nucleic Acid ; Single-Strand Specific DNA and RNA Endonucleases ; Transcription, Genetic/*drug effects ; Transfection ; Viral Proteins/*pharmacology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 10
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    Author Correction: The identification of carbon dioxide mediated protein post-translational modifications (2018)
    Springer Nature
    Details
    Publication Date: 2018-10-04
    Description: Author Correction: The identification of carbon dioxide mediated protein post-translational modifications Author Correction: The identification of carbon dioxide mediated protein post-translational modifications, Published online: 03 October 2018; doi:10.1038/s41467-018-06711-2 Author Correction: The identification of carbon dioxide mediated protein post-translational modifications
    Electronic ISSN: 2041-1723
    Topics: Biology , Chemistry and Pharmacology , Natural Sciences in General , Physics
    Published by Springer Nature
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