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  • 1
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    American Association for the Advancement of Science (AAAS)
    Publication Date: 1978-06-16
    Description: Children with neoplastic diseases were offered an unusual ice cream before their drug treatments. Patients experiencing gastrointestinal toxicity due to the drugs were subsequently less likely to choose that ice cream again than controls. This suggests that taste aversions induced by drug-associated symptoms may contribute to the appetite loss experienced by cancer patients.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Bernstein, I L -- New York, N.Y. -- Science. 1978 Jun 16;200(4347):1302-3.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/663613" target="_blank"〉PubMed〈/a〉
    Keywords: Adolescent ; Anorexia/chemically induced ; *Antineoplastic Agents/adverse effects/therapeutic use ; Avoidance Learning/*physiology ; Child ; Child, Preschool ; Cyclophosphamide/adverse effects ; Cytarabine/adverse effects ; Digestive System/drug effects ; Doxorubicin/adverse effects ; Feeding Behavior/physiology ; Humans ; Neoplasms/drug therapy ; Taste/physiology ; Vincristine/adverse effects
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 2
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    American Association for the Advancement of Science (AAAS)
    Publication Date: 1980-07-18
    Description: Anorexia can occur when a specific diet is associated with a developing illness. The studies reported here show that the decline in food intake which accompanies tumor growth is accompanied by the development of aversions to the specific diet consumed during tumor growth. An immediate elevation in food consumption occurred when a novel diet was introduced. Therefore, the development of learned aversions to the specific diet eaten during tumor growth may be a causal factor in the development of tumor anorexia.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Bernstein, I L -- Sigmundi, R A -- New York, N.Y. -- Science. 1980 Jul 18;209(4454):416-8.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6930106" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Anorexia/*etiology ; Feeding and Eating Disorders/*etiology ; Food Preferences ; Humans ; *Learning ; Male ; Rats ; Sarcoma, Experimental/complications/*physiopathology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 3
    Electronic Resource
    Electronic Resource
    [s.l.] : Nature Publishing Group
    Nature 385 (1997), S. 214-214 
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] On the basis of their observation that intracerebroventricular administration of glucagon-like peptide-1 (residues 7-36) amide (GLP-1) reduced food intake in rats, Turton et aV suggest that GLP-1 is a physiological mediator of satiety. Using c-Fos immunohistochemistry as a marker of neuronal ...
    Type of Medium: Electronic Resource
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  • 4
    Publication Date: 2011-03-30
    Description: Several emerging theories of addiction have described how abused substances exploit vulnerabilities in decision-making processes. These vulnerabilities have been proposed to result from pharmacologically corrupted neural mechanisms of normal brain valuation systems. High alcohol intake in rats during adolescence has been shown to increase risk preference, leading to suboptimal performance on a decision-making task when tested in adulthood. Understanding how alcohol use corrupts decision making in this way has significant clinical implications. However, the underlying mechanism by which alcohol use increases risk preference remains unclear. To address this central issue, we assessed dopamine neurotransmission with fast-scan cyclic voltammetry during reward valuation and risk-based decision making in rats with and without a history of adolescent alcohol intake. We specifically targeted the mesolimbic dopamine system, the site of action for virtually all abused substances. This system, which continuously develops during the adolescent period, is central to both reward processing and risk-based decision making. We report that a history of adolescent alcohol use alters dopamine signaling to risk but not to reward. Thus, a corruption of cost encoding suggests that adolescent alcohol use leads to long-term changes in decision making by altering the valuation of risk.
    Print ISSN: 0027-8424
    Electronic ISSN: 1091-6490
    Topics: Biology , Medicine , Natural Sciences in General
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