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  • 1
    Electronic Resource
    Electronic Resource
    Causes and effects of siltation on the environment of Nepal (1990)
    Springer
    The environmentalist 10 (1990), S. 55-65 
    Details
    ISSN: 1573-2991
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Energy, Environment Protection, Nuclear Power Engineering
    Notes: Summary The present investigation was carried out in 1986, and is based upon about 20 years of river discharge data (1960s to 1980s). There are three important river-systems in Nepal - Sapt Kosi River system in East Nepal, Sapt Gandaki River system in mid- Nepal, and Karnali River system in West Nepal. The average annual highest rainfall is 3,685 mm yr−1, and the greatest mean annual suspended sediment load (1.434 g L−1) was recorded in Sapt Gandaki, while the water discharge from Sapt Kosi (1,747 m3 s−1) was the highest and so also was the silt discharge (0.4 million ton day−1). The correlation between water discharge and silt discharge was found to be statistically highly significant as verified by correlation and regression analyses. An abatement of human interference in the catchment areas of these rivers, and fodder/fuelwood plantation through the active participation of local people in their marginal land to decrease the human pressure on natural forests, are considered the two major easy, effective and economic methods to control the siltation hazard that is causing extensive deterioration to the environment in Nepal.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02239558
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    Paper (German National Licenses)
    Fulltext
  • 2
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    Rad26p regulates the occupancy of histone H2A-H2B dimer at the active genes in vivo (2012)
    Oxford University Press
    Details
    Publication Date: 2012-04-24
    Description: Recently, we have demonstrated a predominant association of Rad26p with the coding sequences but not promoters of several GAL genes following transcriptional induction. Here, we show that the occupancy of histone H2A–H2B dimer at the coding sequences of these genes is not altered following transcriptional induction in the absence of Rad26p. A histone H2A–H2B dimer-enriched chromatin in rad26 is correlated to decreased association of RNA polymerase II with the active coding sequences (and hence transcription). However, the reduced association of RNA polymerase II with the active coding sequence in the absence of Rad26p is not due to the defect in formation of transcription complex at the promoter. Thus, Rad26p regulates the occupancy of histone H2A–H2B dimer, which is correlated to the association of elongating RNA polymerase II with active GAL genes. Similar results are also found at other inducible non- GAL genes. Collectively, our results define a new role of Rad26p in orchestrating chromatin structure and hence transcription in vivo .
    Print ISSN: 0305-1048
    Electronic ISSN: 1362-4962
    Topics: Biology
    Published by Oxford University Press
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  • 3
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    The 19S proteasome subcomplex promotes the targeting of NuA4 HAT to the promoters of ribosomal protein genes to facilitate the recruitment of TFIID for transcriptional initiation in vivo (2012)
    Oxford University Press
    Details
    Publication Date: 2012-03-14
    Description: Previous studies have implicated SAGA (Spt-Ada-Gcn5-acetyltransferase) and TFIID (Transcription factor-IID)-dependent mechanisms of transcriptional activation in yeast. SAGA-dependent transcriptional activation is further regulated by the 19S proteasome subcomplex. However, the role of the 19S proteasome subcomplex in transcriptional activation of the TFIID-dependent genes has not been elucidated. Therefore, we have performed a series of chromatin immunoprecipitation, mutational and transcriptional analyses at the TFIID-dependent ribosomal protein genes such as RPS5 , RPL2B and RPS11B . We find that the 19S proteasome subcomplex is recruited to the promoters of these ribosomal protein genes, and promotes the association of NuA4 (Nucleosome acetyltransferase of histone H4) co-activator, but not activator Rap1p (repressor-activator protein 1). These observations support that the 19S proteasome subcomplex enhances the targeting of co-activator at the TFIID-dependent promoter. Such an enhanced targeting of NuA4 HAT (histone acetyltransferase) promotes the recruitment of the TFIID complex for transcriptional initiation. Collectively, our data demonstrate that the 19S proteasome subcomplex enhances the targeting of NuA4 HAT to activator Rap1p at the promoters of ribosomal protein genes to facilitate the recruitment of TFIID for transcriptional stimulation, hence providing a new role of the 19S proteasome subcomplex in establishing a specific regulatory network at the TFIID-dependent promoter for productive transcriptional initiation in vivo .
    Print ISSN: 0305-1048
    Electronic ISSN: 1362-4962
    Topics: Biology
    Published by Oxford University Press
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  • 4
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    Rrd1p, an RNA polymerase II-specific prolyl isomerase and activator of phosphoprotein phosphatase, promotes transcription independently of rapamycin response (2014)
    Oxford University Press
    Details
    Publication Date: 2014-09-02
    Description: Rrd1p (resistance to rapamycin deletion 1) has been previously implicated in controlling transcription of rapamycin-regulated genes in response to rapamycin treatment. Intriguingly, we show here that Rrd1p associates with the coding sequence of a galactose-inducible and rapamycin non-responsive GAL1 gene, and promotes the association of RNA polymerase II with GAL1 in the absence of rapamycin treatment following transcriptional induction. Consistently, nucleosomal disassembly at GAL1 is impaired in the absence of Rrd1p, and GAL1 transcription is reduced in the rrd1 strain. Likewise, Rrd1p associates with the coding sequences of other rapamycin non-responsive and inducible GAL genes to promote their transcription in the absence of rapamycin treatment. Similarly, inducible, but rapamycin-responsive, non- GAL genes such as CTT1 , STL1 and CUP1 are also regulated by Rrd1p. However, transcription of these inducible GAL and non- GAL genes is not altered in the absence of Rrd1p when the steady-state is reached after long transcriptional induction. Consistently, transcription of the constitutively active genes is not changed in the rrd1 strain. Taken together, our results demonstrate a new function of Rrd1p in stimulation of initial rounds of transcription, but not steady-state/constitutive transcription, of both rapamycin-responsive and non-responsive genes independently of rapamycin treatment.
    Print ISSN: 0305-1048
    Electronic ISSN: 1362-4962
    Topics: Biology
    Published by Oxford University Press
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  • 5
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    Acoustic Emission-Based Health Monitoring of Space Launch System Structures (2019)
    In:  CASI
    Details
    Publication Date: 2019-11-02
    Description: This report documents the technical accomplishments of the project, Acoustic Emission- Based Health Monitoring of Space Launch System Structures, sponsored by the National Aeronautics and Space Administration (Award No. NNM13AA12G). As the project title suggests, the goal of the project was to develop a structural health monitoring (SHM) system for Space Launch System (SLS) vehicles based on acoustic emission (AE) or AE-like signals. Such a system will enhance SLS reliability by identifying the damage locations and type of damage when the damage is initiated. This SHM system would also lead to reduced maintenance costs by enabling ground support equipment to inspect only SLS elements or parts that are likely to be damaged. Finally, it will facilitate lean designs that meet tolerance levels specified by barely detectable damage. By avoiding excess conservatism in this manner, this SHM system will further reduce manufacturing costs of SLS vehicles.
    Keywords: Acoustics
    Type: NASA/CR-2019–220138
    Format: application/pdf
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    NASA TECHNICAL REPORTS
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