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  • 1
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    In:  Geophys. J. Int., Ottawa, Bundesanstalt für Geowissenschaften und Rohstoffe, vol. 165, no. 1, pp. 373-381, pp. 2371
    Publication Date: 2006
    Keywords: Crustal deformation (cf. Earthquake precursor: deformation or strain) ; Tectonics ; Geodesy ; Iran ; Subduction zone ; GJI ; active ; deformation, ; collision, ; Subduction
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  • 2
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    In:  Geophys. J. Int., Kunming, China, AGU, vol. 163, no. 1, pp. 90-105, pp. L14317, (ISSN: 1340-4202)
    Publication Date: 2005
    Keywords: Earthquake ; Aftershocks ; Seismicity ; Fault zone ; Source parameters ; aftershocks, ; Bam ; earthquake ; GJI
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  • 3
    Publication Date: 2015-10-23
    Description: Determining the relationship between folding and faulting in fold and thrust belts is important for understanding the growth of geological structures, the depth extent of seismic slip and consequently the potential earthquake hazard. The 2013 Mw6.2 Khaki-Shonbe earthquake occurred in the Simply Folded Belt of the Zagros Mountains, Iran. We combine seismological solutions, aftershock relocations, satellite interferometry and field observations to determine fault geometry and its relationship with the structure, stratigraphy and tectonics of the central Zagros. We image reverse slip on two along-strike, south-west dipping fault segments. The mainshock rupture initiated at the lower northern end of the larger north-west segment. Based upon the hypocentre and rupture duration, slip on the smaller southern segment is likely aseismic. Both faults verge away from the foreland, towards the high range interior, contrary to the fault geometries depicted in many structural cross-sections of the Zagros. The slip modelled occurred over two mutually exclusive depth ranges above 10km, resulting in long (∼16km), narrow rupture segments (∼7km). Aftershocks cluster in the depth range 3–14km. This indicates reverse slip and coseismic shortening occurred mostly or exclusively in the sedimentary cover, with slip distributions likely to be lithologically controlled in depth by the Hormuz salt at the base of the sedimentary cover (∼10–12km), and the Kazhdumi Formation mudrocks at upper-levels (∼4–5km). Our findings suggest lithology plays a significant role in the depth extent of slip found in reverse faults in folded belts, providing an important control on the potential size of earthquakes.
    Electronic ISSN: 2333-5084
    Topics: Geosciences , Physics
    Published by Wiley on behalf of The American Geophysical Union (AGU).
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  • 4
    Publication Date: 2012-02-23
    Description: SUMMARY On 2005 February 22, the Dahuiyeh-Zarand earthquake, M w 6.5, struck one of the most seismically active regions in south-central Iran, east of Zarand City in Kerman province, causing more than 500 fatalities. The causative fault of the 2005 Dahuiyeh-Zarand earthquake, a reverse fault, striking nearly EW and dipping to the north, was located within a mountainous region and therefore more difficult to identify compared to the range-bounded faults. Its identification, after the 2005 event, is very important for both the estimation of seismic hazard as well as for the damage and fatality functions. We have inverted six three-component near-field strong motion waveforms to obtain the complete earthquake rupture history and slip distribution. Accelerograms are bandpass filtered with 0.2–1.0 Hz, and a length of 15–17 s of the waveforms is inverted. The lack of absolute timing has been successfully overcome by estimating, from the velocity model of the region, the propagation of P and S waves from the epicentre to the stations. The final fault slip model and the estimated source parameters are able to explain the observed waveforms. The rupture is found to be bilateral with a maximum slip of 2.4 m concentrated on two asperities in the west and east sides of the nucleation point at depths of 6–12 km. The western asperity is located to the east of Zarand City and beneath the Dahuiyeh village, which might explain why the Dahuiyeh village was totally destroyed by this earthquake.
    Print ISSN: 0956-540X
    Electronic ISSN: 1365-246X
    Topics: Geosciences
    Published by Oxford University Press on behalf of The Deutsche Geophysikalische Gesellschaft (DGG) and the Royal Astronomical Society (RAS).
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  • 5
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    American Association for the Advancement of Science (AAAS)
    Publication Date: 2002-01-05
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Tatar, Marc -- Rand, David M -- New York, N.Y. -- Science. 2002 Jan 4;295(5552):54-5.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Ecology and Evolutionary Biology, Brown University, Providence, RI 02912, USA. marc_tatar@brown.edu〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/11778030" target="_blank"〉PubMed〈/a〉
    Keywords: *Aging ; Animals ; Caenorhabditis elegans/genetics/growth & development/metabolism/*physiology ; Caenorhabditis elegans Proteins/genetics/physiology ; Cell Nucleus/metabolism ; Diet ; Electron Transport ; Energy Metabolism ; Escherichia coli/metabolism ; Fermentation ; Helminth Proteins/genetics/physiology ; Insulin/metabolism ; Larva/growth & development/metabolism ; *Longevity ; Mitochondria/metabolism ; Mutation ; Reactive Oxygen Species/metabolism ; Receptor, Insulin/genetics/physiology ; Receptors, Cytoplasmic and Nuclear/genetics/physiology ; Signal Transduction ; Ubiquinone/administration & dosage/*metabolism
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 6
    Publication Date: 2001-04-09
    Description: The Drosophila melanogaster gene insulin-like receptor (InR) is homologous to mammalian insulin receptors as well as to Caenorhabditis elegans daf-2, a signal transducer regulating worm dauer formation and adult longevity. We describe a heteroallelic, hypomorphic genotype of mutant InR, which yields dwarf females with up to an 85% extension of adult longevity and dwarf males with reduced late age-specific mortality. Treatment of the long-lived InR dwarfs with a juvenile hormone analog restores life expectancy toward that of wild-type controls. We conclude that juvenile hormone deficiency, which results from InR signal pathway mutation, is sufficient to extend life-span, and that in flies, insulin-like ligands nonautonomously mediate aging through retardation of growth or activation of specific endocrine tissue.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Tatar, M -- Kopelman, A -- Epstein, D -- Tu, M P -- Yin, C M -- Garofalo, R S -- R01 AG16632/AG/NIA NIH HHS/ -- New York, N.Y. -- Science. 2001 Apr 6;292(5514):107-10.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Brown University, Providence, RI 02912, USA., University of Massachusetts, Amherst, MA 01003, USA. Marc_Tatar@Brown.edu〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/11292875" target="_blank"〉PubMed〈/a〉
    Keywords: Aging/*physiology ; Alleles ; Animals ; Carrier Proteins/*genetics/*physiology ; Corpora Allata/*metabolism ; *Drosophila Proteins ; Drosophila melanogaster/genetics/*physiology ; Female ; Fertility ; Genes, Insect ; Genotype ; Insulin/pharmacology ; Juvenile Hormones/metabolism ; Longevity/*physiology ; Male ; Methoprene/pharmacology ; Mutation ; Protein-Tyrosine Kinases/*genetics/*physiology ; *Receptor Protein-Tyrosine Kinases ; Receptor, Insulin/genetics/physiology ; Reproduction ; Signal Transduction ; Superoxide Dismutase/metabolism ; Triglycerides/metabolism ; Vitellogenesis/drug effects
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 7
    Publication Date: 2005-02-05
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Tatar, Marc -- New York, N.Y. -- Science. 2005 Feb 4;307(5710):675; author reply 675.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Brown University, Division of Biology and Medicine, Box G-W, Providence, RI 02912, USA. marc_tatar@brown.edu.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/15692034" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Drosophila Proteins/genetics/*metabolism ; Drosophila melanogaster/genetics/growth & development/*physiology ; Fat Body/*metabolism ; Female ; Forkhead Transcription Factors ; Gene Expression ; *Longevity ; Transcription Factors/genetics/*metabolism
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 8
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    American Association for the Advancement of Science (AAAS)
    Publication Date: 2003-03-01
    Description: Reduced signaling of insulin-like peptides increases the life-span of nematodes, flies, and rodents. In the nematode and the fly, secondary hormones downstream of insulin-like signaling appear to regulate aging. In mammals, the order in which the hormones act is unresolved because insulin, insulin-like growth factor-1, growth hormone, and thyroid hormones are interdependent. In all species examined to date, endocrine manipulations can slow aging without concurrent costs in reproduction, but with inevitable increases in stress resistance. Despite the similarities among mammals and invertebrates in insulin-like peptides and their signal cascade, more research is needed to determine whether these signals control aging in the same way in all the species by the same mechanism.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Tatar, Marc -- Bartke, Andrzej -- Antebi, Adam -- New York, N.Y. -- Science. 2003 Feb 28;299(5611):1346-51.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Ecology and Evolutionary Biology, Brown University, Providence, RI 02912, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/12610294" target="_blank"〉PubMed〈/a〉
    Keywords: *Aging ; Animals ; Caenorhabditis elegans/genetics/physiology ; *Caenorhabditis elegans Proteins ; Caloric Restriction ; Drosophila melanogaster/genetics/physiology ; Endocrine System/*physiology ; Forkhead Transcription Factors ; Gene Expression Regulation ; Growth Hormone/metabolism ; Humans ; Insect Hormones/physiology ; Insulin/*metabolism ; *Longevity ; Mice ; *Signal Transduction ; Somatomedins/*metabolism ; Transcription Factors/genetics/metabolism
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 9
    Publication Date: 2010-08-28
    Description: 〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3985480/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3985480/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Baur, Joseph A -- Chen, Danica -- Chini, Eduardo N -- Chua, Katrin -- Cohen, Haim Y -- de Cabo, Rafael -- Deng, Chuxia -- Dimmeler, Stefanie -- Gius, David -- Guarente, Leonard P -- Helfand, Stephen L -- Imai, Shin-Ichiro -- Itoh, Hiroshi -- Kadowaki, Takashi -- Koya, Daisuke -- Leeuwenburgh, Christiaan -- McBurney, Michael -- Nabeshima, Yo-Ichi -- Neri, Christian -- Oberdoerffer, Philipp -- Pestell, Richard G -- Rogina, Blanka -- Sadoshima, Junichi -- Sartorelli, Vittorio -- Serrano, Manuel -- Sinclair, David A -- Steegborn, Clemens -- Tatar, Marc -- Tissenbaum, Heidi A -- Tong, Qiang -- Tsubota, Kazuo -- Vaquero, Alejandro -- Verdin, Eric -- P01 AG027916/AG/NIA NIH HHS/ -- R00 AG031182/AG/NIA NIH HHS/ -- R01 AG019719/AG/NIA NIH HHS/ -- R01 AG023039/AG/NIA NIH HHS/ -- R01 AG023088/AG/NIA NIH HHS/ -- R01 AG023088-08/AG/NIA NIH HHS/ -- R01 AG024360/AG/NIA NIH HHS/ -- R01 AG028730/AG/NIA NIH HHS/ -- R01 AG028730-05/AG/NIA NIH HHS/ -- R01 HL067724/HL/NHLBI NIH HHS/ -- R01 HL091469/HL/NHLBI NIH HHS/ -- R01 HL102738/HL/NHLBI NIH HHS/ -- R37 AG024360/AG/NIA NIH HHS/ -- Z99 DK999999/Intramural NIH HHS/ -- New York, N.Y. -- Science. 2010 Aug 27;329(5995):1012-3; author reply 1013-4. doi: 10.1126/science.329.5995.1012.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/20798296" target="_blank"〉PubMed〈/a〉
    Keywords: Aging/*physiology ; Animals ; *Caloric Restriction ; Humans ; *Longevity ; Signal Transduction ; Sirtuins/*physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 10
    Publication Date: 2013-05-22
    Description: The role of protein farnesylation in lamin A biogenesis and the pathogenesis of progeria has been studied in considerable detail, but the importance of farnesylation for the B-type lamins, lamin B1 and lamin B2, has received little attention. Lamins B1 and B2 are expressed in nearly every cell type from...
    Print ISSN: 0027-8424
    Electronic ISSN: 1091-6490
    Topics: Biology , Medicine , Natural Sciences in General
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