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  • 1
    Electronic Resource
    Electronic Resource
    Visceral Receptors (1956)
    Palo Alto, Calif. : Annual Reviews
    Annual Review of Physiology 18 (1956), S. 387-408 
    Details
    ISSN: 0066-4278
    Source: Annual Reviews Electronic Back Volume Collection 1932-2001ff
    Topics: Medicine , Biology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1146/annurev.ph.18.030156.002131
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  • 2
    Electronic Resource
    Electronic Resource
    A Look at an Old but Still Current Problem (1963)
    Palo Alto, Calif. : Annual Reviews
    Annual Review of Physiology 25 (1963), S. 1-16 
    Details
    ISSN: 0066-4278
    Source: Annual Reviews Electronic Back Volume Collection 1932-2001ff
    Topics: Medicine , Biology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1146/annurev.ph.25.030163.000245
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  • 3
    Electronic Resource
    Electronic Resource
    Pharmacology in Old and Modern Medicine (1967)
    Palo Alto, Calif. : Annual Reviews
    Annual Review of Pharmacology 7 (1967), S. 1-15 
    Details
    ISSN: 0362-1642
    Source: Annual Reviews Electronic Back Volume Collection 1932-2001ff
    Topics: Medicine , Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1146/annurev.pa.07.040167.000245
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  • 4
    Electronic Resource
    Electronic Resource
    Regulation of blood pressure and hypertension (1957)
    Springer
    Cellular and molecular life sciences 13 (1957), S. 379-380 
    Details
    ISSN: 1420-9071
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Résumé L'auteur résume les mécanismes de l'homéostasie physiologique de la pression artérielle et indique que la réaction à la pression intravasculaire des parois artérielles barosensibles constitue un facteur prédominant de la régulation réflexe de la pression artérielle. Une élévation du seuil d'excitabilité des parois artérielles barosensibles pourrait constituer un élément causal important de l'hypertension artérielle.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02179181
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  • 5
    Electronic Resource
    Electronic Resource
    Substances synaptolytiques, curarisantes et nicotinolytiques (1949)
    Springer
    Cellular and molecular life sciences 5 (1949), S. 213-214 
    Details
    ISSN: 1420-9071
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Diethylaminoethyl ester of phenyl-cyclopentane carboxylic acid (Parpanit) and diaminoethylphenothiazine (2987 R. P., Diparcol), substances paralysing the autonomic synapses, the postsynaptic cholinergic innervation and the motor centers, protect very actively against nicotinic and cholinergic intoxication. Parpanit has no anticholinesterase action, while Diparcol inactivates selectively pseudo-cholinesterasein vitro andin vivo. Curarizing and synaptolytic agents as tubocurarine, methyltubocurarine, diiodoethylate of bis (quinoleyl-8′-1, 5-pentane) (3381 R. P.) and lauryl-dimethyl-aminoethanol (764 L.), protect against nicotinic intoxication, but do not suppress the muscarinic actions of cholinergic agents. Triiodate of tri(tri-ethylammonium-methoxy)-1,2,3-benzene (2559 F.), curarizing, but not synaptolytic substance, protects only against nicotinic convulsions and muscular fasciculations. Tetra-ethyl-ammonium, synaptolytic substance, protects only against synaptic stimulations induced by nicotinic agents. Atropine protects against nicotinic and muscarinic actions of nicotinic substances, but not against nicotinic convulsions and muscular fasciculations. The curarizing and synaptolytic substances as well as atropine do not inhibit cholinesterases. Thus no correlation exists between anticholinesterase, synaptolytic and nicotinolytic properties.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02172502
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  • 6
    Electronic Resource
    Electronic Resource
    Influences de la tubocurarine sur la régulation proprioceptive de la pression artérielle (1946)
    Springer
    Cellular and molecular life sciences 2 (1946), S. 453-454 
    Details
    ISSN: 1420-9071
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Experiments performed on dogs show that: (1) Slow intravenous injection of a clinical dosis of tubocurarine (Intocostrin) does not produce a decrease or only a slight and momentary fall in general arterial blood pressure, although the neuromuscular paralysis is complete. In these conditions, tubocurarine also does not depress the physiological mechanisms of the blood pressure homeostasis. In clinical dosis, tubocurarine thus does not block synaptic conduction. (2) Rapid intravenous injection of a clinical dosis of tubocurarine induces a fall in general blood pressure, which is sometimes momentary, and depresses the mechanisms of blood pressure homeostasis. These influences of tubocurarine are counteracted by ephedrine. (3) Rapid intravenous injection of a large amount of tubocurarine, produces a marked fall in blood pressure and a paralysis of the blood pressure homeostasis. These influences are not counteracted by ephedrine or prostigmine. (4) Prostigmine counteracts, as already known, the neuromuscular depression induced by tubocurarine. This antagonism is however not related to an influence of tubocurarine on the cholinesterase.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02153598
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  • 7
    Electronic Resource
    Electronic Resource
    Influence of Blood Pressure and Blood Flow on the Activity of the Respiratory, Vasomotor and Cardio-Regulatory Centres (1945)
    [s.l.] : Nature Publishing Group
    Nature 156 (1945), S. 750-751 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] THE classical opinion is that changes in the central blood flow and blood pressure affect and regulate in a direct central manner the activities of the respiratory and cardio-vascular centres:decrease of central blood pressure or blood flow was considered to stimulate directly the respiratory ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/156750b0
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  • 8
    Electronic Resource
    Electronic Resource
    Influence des anticholinestérases sur les réflexes d'origine sino-carotidienne (1946)
    Springer
    Cellular and molecular life sciences 2 (1946), S. 260-260 
    Details
    ISSN: 1420-9071
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Experiments performed on dogs show that the very active anticholinesterase diisopropylfluorophosphate (DFD) does not affect either the cardiovascular and respiratory reflexes of carotid sinus origin or the peripheral excitability of the heart vagus nerve. Injection of prostigmine after administration of DFP and previous complete inhibition of the cholinesterases, still induces a slowing of the heart, an increase of vagal excitability, an increase of the peristaltic movements of the intestines and a bronchospasm. These experiments thus do not support the theory of a central or peripheral cholinergic transmission of the cardiovascular and respiratory reflexes induced by stimulation of the carotid sinus pressoceptors. These experiments also show that several pharmacological actions of prostigmine are not related to the anticholinesterase action of this drug.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02156566
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  • 9
    Electronic Resource
    Electronic Resource
    Action du succinate de soude sur la respiration (1947)
    Springer
    Cellular and molecular life sciences 3 (1947), S. 294-295 
    Details
    ISSN: 1420-9071
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Intravenous injection of sodium succinate in dogs anesthetized with chloralosane produces a prolonged stimulation of respiration and decreases the duration of the apnea following pulmonary hyperventilation.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02164180
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  • 10
    Electronic Resource
    Electronic Resource
    Observations expérimentales sur l'apnée acapnique chez le Chien (1947)
    Springer
    Cellular and molecular life sciences 3 (1947), S. 374-375 
    Details
    ISSN: 1420-9071
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Suppression of the chemo-receptors of the carotid sinus and aorta æreas prolongs the acapnic apnea markedly. Animals deprived of their chemo-receptors may even die in apnea. During hyperventilation, the expiratory position of the thorax increases. During the acapnic apnea the inspiratory position returns progressively to normal. During hyperventilation and acapnic apnea, rhythmic changes occur in the tonus of the respiratory muscles. These changes in tonus occur in normal animals as well as in animals deprived of their chemo-receptors and vagi nerves. In normal dogs, hyperventilation and acapnea do not induce a fall in arterial blood pressure. Arterial hypotension occurs, however, during hyperventilation in dogs deprived of their carotid sinus and aortic innervation. During acapnic apnea, the arterial blood pressure rises in animals deprived of their carotid sinus and aortic nerves. A secondary fall of arterial pressure occurs during prolonged acapnic apnea in these animals.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02154725
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