Publication Date:
2014-10-02
Description:
Key Points Absence of STAT3 serine phosphorylation restricts activated K-Ras–driven myeloproliferative disease in a mouse model. A mitochondrial function of STAT3 supports K-Ras–driven, factor-independent growth of myeloid progenitors in vitro.
Print ISSN:
0006-4971
Electronic ISSN:
1528-0020
Topics:
Biology
,
Medicine
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