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  • 1
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    Enterotoxin A production in Staphylococcus aureus: inhibition by glucose (1986)
    Springer
    Details
    Publication Date: 1986-03-01
    Print ISSN: 0302-8933
    Electronic ISSN: 1432-072X
    Topics: Biology
    Published by Springer
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  • 2
    Electronic Resource
    Electronic Resource
    EFFECT OF GLUCOSE ANALOGS ON THE SYNTHESIS OF STAPHYLOCOCCAL ENTEROTOXIN A (1986)
    Oxford, UK : Blackwell Publishing Ltd
    Journal of food safety 8 (1986), S. 0 
    Details
    ISSN: 1745-4565
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Agriculture, Forestry, Horticulture, Fishery, Domestic Science, Nutrition , Process Engineering, Biotechnology, Nutrition Technology
    Notes: Glucose, 2-deoxyglucose (2-DOG), and α-methylglucose (α-MG) inhibited staphylococcal enteroxin A (SEA) synthesis by Staphylococcus aureus 196E whereas β-methylglucose (β-MG) and 3-0-methylglucose (3-0-MG) did not inhibit even at high concentrations. Glucose and β-MG decreased the pH (〈 6.0) whereas the pH with 2-DOG, α-MG was above 8.0 at 24 h. Glucose (at levels not inhibitory to SEA synthesis) potentiated the inhibition of toxin synthesis by 2-DOG and β-MG and the glucose-analog combinations had a decreased pH. The inhibition of SEA synthesis by glucose, glucose analogs, or glucose-analog combinations does not appear to be directly related to a decline in pH. The inhibitory effects shown by glucose and its analog suggest that SEA synthesis may be regulated by a mechanism similar to catabolite repression.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1745-4565.1986.tb00559.x
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  • 3
    Electronic Resource
    Electronic Resource
    ENTEROTOXIN BIOSYNTHESIS BY PROGENY OF REPAIRED HEAT-INJURED CELLS OF STAPHYLOCOCCUS AUREUS (1984)
    Oxford, UK : Blackwell Publishing Ltd
    Journal of food safety 6 (1984), S. 0 
    Details
    ISSN: 1745-4565
    Source: Blackwell Publishing Journal Backfiles 1879-2005
    Topics: Agriculture, Forestry, Horticulture, Fishery, Domestic Science, Nutrition , Process Engineering, Biotechnology, Nutrition Technology
    Notes: By using a method which permitted the selection of repaired cells from a population of heat-injured and noninjured cells of Staphylococcus aureus 196E, we were able to determine that the progeny of repaired cells retained the ability to produce enterotoxin A (SEA). There were large variations in the amount of SEA produced by the progeny of individual colony forming units (CFU) before and after heating. The average amount of SEA produced by the progeny of noninjured and repaired staphylococci were similar and not significantly different.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1111/j.1745-4565.1984.tb00483.x
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  • 4
    Electronic Resource
    Electronic Resource
    Enterotoxin A production in Staphylococcus aureus: inhibition by glucose (1986)
    Springer
    Archives of microbiology 144 (1986), S. 131-136 
    Details
    ISSN: 1432-072X
    Keywords: Phosphoenolpyruvate phosphotransferase system ; Catabolite repression ; Glucose effect ; Cyclic adenosine-3′,5′-monophosphate ; Pleiotrophic mutant ; Staphylococcal enterotoxin A ; Staphylococcus aureus
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Abstract In this study, we investigated the relationship between carbohydrate metabolism and repression of staphylococcus enterotoxin A (SEA) in Staphylococcus aureus 196E and a pleiotrophic mutant derived from strain 196E. The mutant, designated at strain 196E-MA, lacked a functional phosphoenolpyruvate phosphotransferase system (PTS). The mutant produced acid, under aerobic conditions, from only glucose and glycerol. The parent strain contained an active PTS, and aerobically produced acid from a large number of carbohydrates. Prior growth in glucose led to repression of SEA synthesis in the parent strain; addition to the casamino acids enterotoxin production medium (CAS) led to more severe repression of toxin synthesis. The repression was not related to pH decreases produced by glucose metabolism. When S. aureus 196E was grown in the absence of glucose, there was inhibition of toxin production as glucose level was increased in CAS. The inhibition was related to pH decrease and was unlike the repression observed with glucose-grown strain 196E. The inhibition of SEA synthesis in mutant strain 196E-MA was approximately the same in cells grown with or without glucose and was pH related. Repression of SEA synthesis similar to that seen with glucose-grown S. aureus 196E could not be demonstrated in the mutant. In addition, glucose-grown S. aureus 196E neither synthesized β-galactosidase nor showed respiratory activity with certain tricarboxylic acid (TCA) cycle compounds. Glucose-grown strain 196E-MA, however, did not show supressed respiration of TCA cycle compounds; β-galactosidase was not synthesized because the mutant lacked a functional PTS. Cyclic adenosine-3′, 5′-monophosphate did not reverse the repression by glucose of SEA or β-galactosidase synthesis in glucose-grown S. aureus 196E. An active PTS appears to be necessary to demonstrate glucose (catabolite) repression in S. aureus.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00414722
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