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  • 1
    Publication Date: 2013-06-07
    Description: Author(s): X.-D. Cai, C. Weedbrook, Z.-E. Su, M.-C. Chen, Mile Gu, M.-J. Zhu, Li Li, Nai-Le Liu, Chao-Yang Lu, and Jian-Wei Pan Solving linear systems of equations is ubiquitous in all areas of science and engineering. With rapidly growing data sets, such a task can be intractable for classical computers, as the best known classical algorithms require a time proportional to the number of variables N . A recently proposed quan... [Phys. Rev. Lett. 110, 230501] Published Thu Jun 06, 2013
    Keywords: General Physics: Statistical and Quantum Mechanics, Quantum Information, etc.
    Print ISSN: 0031-9007
    Electronic ISSN: 1079-7114
    Topics: Physics
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  • 2
    Publication Date: 2015-03-20
    Description: Author(s): X.-D. Cai, D. Wu, Z.-E. Su, M.-C. Chen, X.-L. Wang, Li Li, N.-L. Liu, C.-Y. Lu, and J.-W. Pan Machine learning, a branch of artificial intelligence, learns from previous experience to optimize performance, which is ubiquitous in various fields such as computer sciences, financial analysis, robotics, and bioinformatics. A challenge is that machine learning with the rapidly growing “big data” ... [Phys. Rev. Lett. 114, 110504] Published Thu Mar 19, 2015
    Keywords: General Physics: Statistical and Quantum Mechanics, Quantum Information, etc.
    Print ISSN: 0031-9007
    Electronic ISSN: 1079-7114
    Topics: Physics
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  • 3
    Publication Date: 1992-10-02
    Description: The 4-kilodalton (39 to 43 amino acids) amyloid beta protein (beta AP), which is deposited as amyloid in the brains of patients with Alzheimer's diseases, is derived from a large protein, the amyloid beta protein precursor (beta APP). Human mononuclear leukemic (K562) cells expressing a beta AP-bearing, carboxyl-terminal beta APP derivative released significant amounts of a soluble 4-kilodalton beta APP derivative essentially identical to the beta AP deposited in Alzheimer's disease. Human neuroblastoma (M17) cells transfected with constructs expressing full-length beta APP and M17 cells expressing only endogenous beta APP also released soluble 4-kilodalton beta AP, and a similar, if not identical, fragment was readily detected in cerebrospinal fluid from individuals with Alzheimer's disease and normal individuals. Thus cells normally produce and release soluble 4-kilodalton beta AP that is essentially identical to the 4-kilodalton beta AP deposited as insoluble amyloid fibrils in Alzheimer's disease.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Shoji, M -- Golde, T E -- Ghiso, J -- Cheung, T T -- Estus, S -- Shaffer, L M -- Cai, X D -- McKay, D M -- Tintner, R -- Frangione, B -- AG05891/AG/NIA NIH HHS/ -- AG06656/AG/NIA NIH HHS/ -- AR02594/AR/NIAMS NIH HHS/ -- New York, N.Y. -- Science. 1992 Oct 2;258(5079):126-9.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Neurology, Gunma University, Japan.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/1439760" target="_blank"〉PubMed〈/a〉
    Keywords: Alzheimer Disease/*cerebrospinal fluid ; Amino Acid Sequence ; Amyloid beta-Peptides/*biosynthesis ; Amyloid beta-Protein Precursor/metabolism ; Animals ; Base Sequence ; Cell Line ; Immunoblotting ; Leukemia, Myeloid/*metabolism ; Molecular Sequence Data ; Neuroblastoma/*metabolism ; Transfection
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 4
    Publication Date: 2012-08-10
    Description: Transferring an unknown quantum state over arbitrary distances is essential for large-scale quantum communication and distributed quantum networks. It can be achieved with the help of long-distance quantum teleportation and entanglement distribution. The latter is also important for fundamental tests of the laws of quantum mechanics. Although quantum teleportation and entanglement distribution over moderate distances have been realized using optical fibre links, the huge photon loss and decoherence in fibres necessitate the use of quantum repeaters for larger distances. However, the practical realization of quantum repeaters remains experimentally challenging. Free-space channels, first used for quantum key distribution, offer a more promising approach because photon loss and decoherence are almost negligible in the atmosphere. Furthermore, by using satellites, ultra-long-distance quantum communication and tests of quantum foundations could be achieved on a global scale. Previous experiments have achieved free-space distribution of entangled photon pairs over distances of 600 metres (ref. 14) and 13 kilometres (ref. 15), and transfer of triggered single photons over a 144-kilometre one-link free-space channel. Most recently, following a modified scheme, free-space quantum teleportation over 16 kilometres was demonstrated with a single pair of entangled photons. Here we report quantum teleportation of independent qubits over a 97-kilometre one-link free-space channel with multi-photon entanglement. An average fidelity of 80.4 +/- 0.9 per cent is achieved for six distinct states. Furthermore, we demonstrate entanglement distribution over a two-link channel, in which the entangled photons are separated by 101.8 kilometres. Violation of the Clauser-Horne-Shimony-Holt inequality is observed without the locality loophole. Besides being of fundamental interest, our results represent an important step towards a global quantum network. Moreover, the high-frequency and high-accuracy acquiring, pointing and tracking technique developed in our experiment can be directly used for future satellite-based quantum communication and large-scale tests of quantum foundations.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Yin, Juan -- Ren, Ji-Gang -- Lu, He -- Cao, Yuan -- Yong, Hai-Lin -- Wu, Yu-Ping -- Liu, Chang -- Liao, Sheng-Kai -- Zhou, Fei -- Jiang, Yan -- Cai, Xin-Dong -- Xu, Ping -- Pan, Ge-Sheng -- Jia, Jian-Jun -- Huang, Yong-Mei -- Yin, Hao -- Wang, Jian-Yu -- Chen, Yu-Ao -- Peng, Cheng-Zhi -- Pan, Jian-Wei -- England -- Nature. 2012 Aug 9;488(7410):185-8. doi: 10.1038/nature11332.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Shanghai Branch, National Laboratory for Physical Sciences at Microscale and Department of Modern Physics, University of Science and Technology of China, Shanghai 201315, China.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/22874963" target="_blank"〉PubMed〈/a〉
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
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  • 5
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    American Association for the Advancement of Science (AAAS)
    Publication Date: 1993-01-22
    Description: The 4-kilodalton amyloid beta protein (A beta), which forms fibrillar deposits in Alzheimer's disease (AD), is derived from a large protein referred to as the amyloid beta protein precursor (beta APP). Human neuroblastoma (M17) cells transfected with constructs expressing wild-type beta APP or a mutant, beta APP delta NL, recently linked to familial AD were compared. After continuous metabolic labeling for 8 hours, cells expressing beta APP delta NL had five times more of an A beta-bearing, carboxyl terminal, beta APP derivative than cells expressing wild-type beta APP and they released six times more A beta into the medium. Thus this mutant beta APP may cause AD because its processing is altered in a way that releases increased amounts of A beta.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Cai, X D -- Golde, T E -- Younkin, S G -- AG06656/AG/NIA NIH HHS/ -- New York, N.Y. -- Science. 1993 Jan 22;259(5094):514-6.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Division of Neuropathology, Case Western Reserve University, Cleveland, OH 44106.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/8424174" target="_blank"〉PubMed〈/a〉
    Keywords: Alzheimer Disease/genetics/metabolism ; Amino Acid Sequence ; Amyloid beta-Peptides/*biosynthesis/genetics ; Amyloid beta-Protein Precursor/*genetics/metabolism ; Base Sequence ; Cloning, Molecular ; Humans ; Molecular Sequence Data ; *Mutagenesis, Site-Directed ; Neuroblastoma ; Oligodeoxyribonucleotides ; Polymerase Chain Reaction/methods ; Transfection ; Tumor Cells, Cultured
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 6
    Publication Date: 1994-05-27
    Description: Normal processing of the amyloid beta protein precursor (beta APP) results in secretion of a soluble 4-kilodalton protein essentially identical to the amyloid beta protein (A beta) that forms insoluble fibrillar deposits in Alzheimer's disease. Human neuroblastoma (M17) cells transfected with constructs expressing wild-type beta APP or the beta APP717 mutants linked to familial Alzheimer's disease were compared by (i) isolation of metabolically labeled 4-kilodalton A beta from conditioned medium, digestion with cyanogen bromide, and analysis of the carboxyl-terminal peptides released, or (ii) analysis of the A beta in conditioned medium with sandwich enzyme-linked immunosorbent assays that discriminate A beta 1-40 from the longer A beta 1-42. Both methods demonstrated that the 4-kilodalton A beta released from wild-type beta APP is primarily but not exclusively A beta 1-40. The beta APP717 mutations, which are located three residues carboxyl to A beta 43, consistently caused a 1.5- to 1.9-fold increase in the percentage of longer A beta generated. Long A beta (for example, A beta 1-42) forms insoluble amyloid fibrils more rapidly than A beta 1-40. Thus, the beta APP717 mutants may cause Alzheimer's disease because they secrete increased amounts of long A beta, thereby fostering amyloid deposition.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Suzuki, N -- Cheung, T T -- Cai, X D -- Odaka, A -- Otvos, L Jr -- Eckman, C -- Golde, T E -- Younkin, S G -- AG06656/AG/NIA NIH HHS/ -- New York, N.Y. -- Science. 1994 May 27;264(5163):1336-40.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Discovery Research Division, Takeda Chemical Industries, Ltd., Ibaraki, Japan.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/8191290" target="_blank"〉PubMed〈/a〉
    Keywords: Alzheimer Disease/genetics ; Amyloid beta-Peptides/chemistry/*secretion ; Amyloid beta-Protein Precursor/chemistry/genetics/*metabolism ; Culture Media, Conditioned ; Enzyme-Linked Immunosorbent Assay ; Humans ; *Mutation ; Neuroblastoma ; Transfection ; Tumor Cells, Cultured
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
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  • 7
    Publication Date: 2015-02-27
    Description: Quantum teleportation provides a 'disembodied' way to transfer quantum states from one object to another at a distant location, assisted by previously shared entangled states and a classical communication channel. As well as being of fundamental interest, teleportation has been recognized as an important element in long-distance quantum communication, distributed quantum networks and measurement-based quantum computation. There have been numerous demonstrations of teleportation in different physical systems such as photons, atoms, ions, electrons and superconducting circuits. All the previous experiments were limited to the teleportation of one degree of freedom only. However, a single quantum particle can naturally possess various degrees of freedom--internal and external--and with coherent coupling among them. A fundamental open challenge is to teleport multiple degrees of freedom simultaneously, which is necessary to describe a quantum particle fully and, therefore, to teleport it intact. Here we demonstrate quantum teleportation of the composite quantum states of a single photon encoded in both spin and orbital angular momentum. We use photon pairs entangled in both degrees of freedom (that is, hyper-entangled) as the quantum channel for teleportation, and develop a method to project and discriminate hyper-entangled Bell states by exploiting probabilistic quantum non-demolition measurement, which can be extended to more degrees of freedom. We verify the teleportation for both spin-orbit product states and hybrid entangled states, and achieve a teleportation fidelity ranging from 0.57 to 0.68, above the classical limit. Our work is a step towards the teleportation of more complex quantum systems, and demonstrates an increase in our technical control of scalable quantum technologies.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Wang, Xi-Lin -- Cai, Xin-Dong -- Su, Zu-En -- Chen, Ming-Cheng -- Wu, Dian -- Li, Li -- Liu, Nai-Le -- Lu, Chao-Yang -- Pan, Jian-Wei -- England -- Nature. 2015 Feb 26;518(7540):516-9. doi: 10.1038/nature14246.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉1] Hefei National Laboratory for Physical Sciences at Microscale and Department of Modern Physics, University of Science and Technology of China, Hefei, Anhui 230026, China [2] CAS Centre for Excellence and Synergetic Innovation Centre in Quantum Information and Quantum Physics, University of Science and Technology of China, Hefei, Anhui 230026, China.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/25719668" target="_blank"〉PubMed〈/a〉
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
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  • 8
    Electronic Resource
    Electronic Resource
    [S.l.] : American Institute of Physics (AIP)
    Physics of Fluids 10 (1998), S. 2249-2259 
    ISSN: 1089-7666
    Source: AIP Digital Archive
    Topics: Physics
    Notes: In nearly isothermal, compressible turbulence of a nondense gas the mass fraction of an embedded passive scalar satisfies the same formal conservation equation as a passive scalar in incompressible turbulence. Direct numerical simulation of this system shows that the compressible turbulence modes are less efficient than the incompressible in transporting scalar spectral content from large to small scales. It is argued that the cause of this outcome is the reduced size of the integral length scale of the compressible velocity components vis á vis that of the incompressible velocity components, and this also explains the experimentally observed ineffectiveness of the dilatational velocity modes in determining scalar flux in homogeneous, compressible turbulence with a uniform mean scalar gradient. © 1998 American Institute of Physics.
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  • 9
    Electronic Resource
    Electronic Resource
    [S.l.] : American Institute of Physics (AIP)
    Physics of Fluids 9 (1997), S. 1754-1763 
    ISSN: 1089-7666
    Source: AIP Digital Archive
    Topics: Physics
    Notes: Direct numerical simulation of decaying, isotropic, compressible turbulence in three dimensions is used to examine the behavior of fluctuations in density, temperature and pressure when the initial conditions include temperature fluctuations larger than pressure fluctuations. The numerical procedure is described elsewhere, the initial turbulent Mach number is subsonic, 0.3 to 0.7, and the initial compressible turbulence is characterized as being in one of three states in which the ratios of initial kinetic energy in the compressible modes to total kinetic energy are, respectively, very small, moderate or nearly unity. Only at the lowest values of initial turbulent Mach number and energy ratio do thermodynamic scalings follow the predictions in the literature. For turbulent Mach numbers above 0.3, or for finite values of the kinetic energy ratio, the scalings are more complex. A relationship between turbulent Mach number, compressible pressure and energy ratio, which has been proposed previously for isothermal problems, appears to hold, on average, for the cases computed in this study, all of which are non-isothermal. © 1997 American Institute of Physics.
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  • 10
    Electronic Resource
    Electronic Resource
    [S.l.] : American Institute of Physics (AIP)
    Physics of Fluids 8 (1996), S. 2555-2557 
    ISSN: 1089-7666
    Source: AIP Digital Archive
    Topics: Physics
    Notes: An asymptotic self-similar solution is obtained for the one-point probability density function (pdf) equation, of a passive scalar with uniform mean gradient in incompressible homogeneous turbulence. It is argued that the same solution should be a valid approximation when turbulence is generated in a high-quality wind tunnel. The asymptotic pdf shape is a unique function of the conditional expectation of the normalized scalar dissipation rate. The mean scalar gradient modifies the scalar pdf shape only if the conditional expected velocity component in the direction of the mean gradient is a nonlinear function of scalar fluctuation value. Experimental data from wind tunnel studies are consistent with the sign and scale of the changes produced by these nonlinearities. © 1996 American Institute of Physics.
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