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  • 1
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    Exchange energies of kapellasite from high-temperature series analysis of the kagome lattice J_{1}-J_{2}-J_{d}-Heisenberg model (2013)
    American Physical Society (APS)
    Details
    Publication Date: 2013-04-04
    Description: Author(s): B. Bernu, C. Lhuillier, E. Kermarrec, F. Bert, P. Mendels, R. H. Colman, and A. S. Wills We present a method to build magnetic models of insulators based on high-temperature expansions by fitting both the magnetic susceptibility and the low-temperature specific heat data. It is applied to the frustrated magnet kapellasite [Cu 3 Zn(OH) 6 Cl 2 ] with the J 1 - J 2 - J d -Heisenberg model on the kagome ... [Phys. Rev. B 87, 155107] Published Wed Apr 03, 2013
    Keywords: Electronic structure and strongly correlated systems
    Print ISSN: 1098-0121
    Electronic ISSN: 1095-3795
    Topics: Physics
    Published by American Physical Society (APS)
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  • 2
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    Spin dynamics in the S=1/2 quantum kagome compound vesignieite, Cu_{3}Ba(VO_{5}H)_{2} (2011)
    American Physical Society (APS)
    Details
    Publication Date: 2011-06-01
    Description: Author(s): R. H. Colman, F. Bert, D. Boldrin, A. D. Hillier, P. Manuel, P. Mendels, and A. S. Wills We report the study of high-quality samples of the frustrated S =1/2 kagome antiferromagnet vesignieite, Cu 3 Ba(VO 5 H) 2 . Neutron powder diffraction measurements evidence the excellence of the kagome lattice and show no sign of a transition to magnetic long-range order. A kink in the susceptibility belo... [Phys. Rev. B 83, 180416] Published Tue May 31, 2011
    Keywords: Magnetism
    Print ISSN: 1098-0121
    Electronic ISSN: 1095-3795
    Topics: Physics
    Published by American Physical Society (APS)
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  • 3
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    Kapellasite: A Kagome Quantum Spin Liquid with Competing Interactions (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-07-21
    Description: Author(s): B. Fåk, E. Kermarrec, L. Messio, B. Bernu, C. Lhuillier, F. Bert, P. Mendels, B. Koteswararao, F. Bouquet, J. Ollivier, A. D. Hillier, A. Amato, R. H. Colman, and A. S. Wills Magnetic susceptibility, NMR, muon spin relaxation, and inelastic neutron scattering measurements show that kapellasite, Cu 3 Zn(OH) 6 Cl 2 , a geometrically frustrated spin- 1/2 kagome antiferromagnet polymorphic with herbertsmithite, is a gapless spin liquid showing unusual dynamic short-range correlatio... [Phys. Rev. Lett. 109, 037208] Published Fri Jul 20, 2012
    Keywords: Condensed Matter: Electronic Properties, etc.
    Print ISSN: 0031-9007
    Electronic ISSN: 1079-7114
    Topics: Physics
    Published by American Physical Society (APS)
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  • 4
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    IrSr_{2}Sm_{1.15}Ce_{0.85}Cu_{2.175}O_{10}: A reentrant spin-glass material (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-04-24
    Description: Author(s): R. H. Colman and A. C. Mclaughlin A new iridium-containing layered cuprate material, Ir 0.825 Sr 2 Sm 1.15 Ce 0.85 Cu 2.175 O 10 , has been synthesized by conventional ambient-pressure solid-state techniques. The material's structure has been fully characterized by Rietveld refinement of high-resolution synchrotron x-ray diffraction data; tilts... [Phys. Rev. B 85, 144419] Published Mon Apr 23, 2012
    Keywords: Magnetism
    Print ISSN: 1098-0121
    Electronic ISSN: 1095-3795
    Topics: Physics
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  • 5
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    Ground state and intrinsic susceptibility of the kagome antiferromagnet vesignieite as seen by ^{51}V NMR (2011)
    American Physical Society (APS)
    Details
    Publication Date: 2011-11-04
    Description: Author(s): J. A. Quilliam, F. Bert, R. H. Colman, D. Boldrin, A. S. Wills, and P. Mendels [Phys. Rev. B 84, 180401] Published Thu Nov 03, 2011
    Keywords: Magnetism
    Print ISSN: 1098-0121
    Electronic ISSN: 1095-3795
    Topics: Physics
    Published by American Physical Society (APS)
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  • 6
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    Discovery and molecular characterization of a Bcl-2-regulated cell death pathway in schistosomes [Cell Biology] (2011)
    National Academy of Sciences
    Details
    Publication Date: 2011-04-27
    Description: Schistosomiasis is an infectious disease caused by parasites of the phylum platyhelminthe. Here, we describe the identification and characterization of a Bcl-2–regulated apoptosis pathway in Schistosoma japonicum and S. mansoni. Genomic, biochemical, and cell-based mechanistic studies provide evidence for a tripartite pathway, similar to that in humans including BH3-only proteins that are inhibited by prosurvival Bcl-2–like molecules, and Bax/Bak-like proteins that facilitate mitochondrial outer-membrane permeabilization. Because Bcl-2 proteins have been successfully targeted with “BH3 mimetic” drugs, particularly in the treatment of cancer, we investigated whether schistosome apoptosis pathways could provide targets for future antischistosomal drug discovery efforts. Accordingly, we showed that a schistosome prosurvival protein, sjA, binds ABT-737, a well-characterized BH3 mimetic. A crystal structure of sjA bound to a BH3 peptide provides direct evidence for the feasibility of developing BH3 mimetics to target Bcl-2 prosurvival proteins in schistosomes, suggesting an alternative application for this class of drugs beyond cancer treatment.
    Print ISSN: 0027-8424
    Electronic ISSN: 1091-6490
    Topics: Biology , Medicine , Natural Sciences in General
    Published by National Academy of Sciences
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  • 7
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    Spin-liquid ground state in the frustrated kagome antiferromagnet MgCu_{3}(OH)_{6}Cl_{2} (2011)
    American Physical Society (APS)
    Details
    Publication Date: 2011-09-07
    Description: Author(s): E. Kermarrec, P. Mendels, F. Bert, R. H. Colman, A. S. Wills, P. Strobel, P. Bonville, A. Hillier, and A. Amato We report muon spin resonance experiments on Mg x Cu 4− x (OH) 6 Cl 2 with x ∼1 , a material isostructural to Herbertsmithite exhibiting regular kagome planes of S =1/2 ions (Cu 2+ ), and therefore a candidate for a spin-liquid ground state. We evidence the absence of any magnetic ordering down to 20 mK ( ∼ J /10 4 )... [Phys. Rev. B 84, 100401] Published Tue Sep 06, 2011
    Keywords: Magnetism
    Print ISSN: 1098-0121
    Electronic ISSN: 1095-3795
    Topics: Physics
    Published by American Physical Society (APS)
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  • 8
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    Spin dynamics and disorder effects in the S=12 kagome Heisenberg spin-liquid phase of kapellasite (2014)
    American Physical Society (APS)
    Details
    Publication Date: 2014-11-05
    Description: Author(s): E. Kermarrec, A. Zorko, F. Bert, R. H. Colman, B. Koteswararao, F. Bouquet, P. Bonville, A. Hillier, A. Amato, J. van Tol, A. Ozarowski, A. S. Wills, and P. Mendels We report Cl35 NMR, ESR, μSR, and specific-heat measurements on the S=12 frustrated kagome magnet kapellasite α-Cu3Zn(OH)6Cl2, where a gapless spin-liquid phase is stabilized by a set of competing exchange interactions. Our measurements confirm the ferromagnetic character of the nearest-neighbor exc... [Phys. Rev. B 90, 205103] Published Tue Nov 04, 2014
    Keywords: Electronic structure and strongly correlated systems
    Print ISSN: 1098-0121
    Electronic ISSN: 1095-3795
    Topics: Physics
    Published by American Physical Society (APS)
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  • 9
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    The transcriptional network for mesenchymal transformation of brain tumours (2009)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2009-12-25
    Description: The inference of transcriptional networks that regulate transitions into physiological or pathological cellular states remains a central challenge in systems biology. A mesenchymal phenotype is the hallmark of tumour aggressiveness in human malignant glioma, but the regulatory programs responsible for implementing the associated molecular signature are largely unknown. Here we show that reverse-engineering and an unbiased interrogation of a glioma-specific regulatory network reveal the transcriptional module that activates expression of mesenchymal genes in malignant glioma. Two transcription factors (C/EBPbeta and STAT3) emerge as synergistic initiators and master regulators of mesenchymal transformation. Ectopic co-expression of C/EBPbeta and STAT3 reprograms neural stem cells along the aberrant mesenchymal lineage, whereas elimination of the two factors in glioma cells leads to collapse of the mesenchymal signature and reduces tumour aggressiveness. In human glioma, expression of C/EBPbeta and STAT3 correlates with mesenchymal differentiation and predicts poor clinical outcome. These results show that the activation of a small regulatory module is necessary and sufficient to initiate and maintain an aberrant phenotypic state in cancer cells.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4011561/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4011561/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Carro, Maria Stella -- Lim, Wei Keat -- Alvarez, Mariano Javier -- Bollo, Robert J -- Zhao, Xudong -- Snyder, Evan Y -- Sulman, Erik P -- Anne, Sandrine L -- Doetsch, Fiona -- Colman, Howard -- Lasorella, Anna -- Aldape, Ken -- Califano, Andrea -- Iavarone, Antonio -- 1RC2CA148308-01/CA/NCI NIH HHS/ -- P20 GM075059/GM/NIGMS NIH HHS/ -- P20GM075059/GM/NIGMS NIH HHS/ -- R01 CA085628/CA/NCI NIH HHS/ -- R01 CA101644/CA/NCI NIH HHS/ -- R01 CA109755/CA/NCI NIH HHS/ -- R01 CA127643/CA/NCI NIH HHS/ -- R01 NS061776/NS/NINDS NIH HHS/ -- R01 NS061776-01A2/NS/NINDS NIH HHS/ -- R01 NS061776-02/NS/NINDS NIH HHS/ -- R01CA085628/CA/NCI NIH HHS/ -- R01CA101644/CA/NCI NIH HHS/ -- R01CA109755/CA/NCI NIH HHS/ -- R01NS061776/NS/NINDS NIH HHS/ -- RC2 CA148308/CA/NCI NIH HHS/ -- U01 CA168426/CA/NCI NIH HHS/ -- U54 CA121852/CA/NCI NIH HHS/ -- U54CA121852/CA/NCI NIH HHS/ -- England -- Nature. 2010 Jan 21;463(7279):318-25. doi: 10.1038/nature08712. Epub 2009 Dec 23.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Institute for Cancer Genetics, Columbia University Medical Center, New York, New York 10032, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/20032975" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Brain Neoplasms/diagnosis/*genetics/*pathology ; CCAAT-Enhancer-Binding Protein-beta/genetics/metabolism ; Cell Differentiation/genetics ; Cell Line, Tumor ; Cell Transformation, Neoplastic/genetics/metabolism/pathology ; Cellular Reprogramming/genetics ; Computational Biology ; *Gene Expression Regulation, Neoplastic ; *Gene Regulatory Networks ; Glioma/diagnosis/genetics/pathology ; Humans ; Mesenchymal Stromal Cells/metabolism/pathology ; Mesoderm/*metabolism/*pathology ; Mice ; Mice, Inbred NOD ; Mice, SCID ; Neoplasm Invasiveness/genetics/pathology ; Neurons/metabolism/pathology ; Prognosis ; Reproducibility of Results ; STAT3 Transcription Factor/genetics/metabolism ; *Transcription, Genetic
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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  • 10
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    Alterations in synaptic strength preceding axon withdrawal (1997)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1997-01-17
    Description: Permanent removal of axonal input to postsynaptic cells helps shape the pattern of neuronal connections in response to experience, but the process is poorly understood. Intracellular recording from newborn and adult mouse muscle fibers temporarily innervated by two axons showed an increasing disparity in the synaptic strengths of the two inputs before one was eliminated. The connection that survived gained strength by increasing the amount of neurotransmitter released (quantal content), whereas the input that was subsequently removed became progressively weaker, because of a reduction in quantal content and a reduction in quantal efficacy associated with reduced postsynaptic receptor density. Once the synaptic strengths of two inputs began to diverge, complete axonal withdrawal of the weaker input occurred within 1 to 2 days. These experiments provide a link between experience-driven changes in synaptic strength and long-term changes in connectivity in the mammalian nervous system.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Colman, H -- Nabekura, J -- Lichtman, J W -- NS 20364/NS/NINDS NIH HHS/ -- NS 34448/NS/NINDS NIH HHS/ -- New York, N.Y. -- Science. 1997 Jan 17;275(5298):356-61.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Anatomy and Neurobiology, Box 8108, Washington University School of Medicine, 660 South Euclid, St. Louis, MO 63110, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/8994026" target="_blank"〉PubMed〈/a〉
    Keywords: Acetylcholine/metabolism ; Action Potentials ; Aging ; Animals ; Animals, Newborn ; Axons/*physiology ; Electric Stimulation ; Evoked Potentials, Motor ; Mice ; Muscle Fibers, Skeletal/physiology ; Muscle, Skeletal/*innervation ; Neuromuscular Junction/*physiology ; Receptors, Cholinergic/metabolism ; *Synaptic Transmission
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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