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  • 1
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    Morphine tolerance: is there evidence for a conditioning model? (1978)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1978-04-21
    Description: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Hayes, R L -- Mayer, D J -- New York, N.Y. -- Science. 1978 Apr 21;200(4339):343-5.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/635595" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Behavior, Animal/physiology ; Conditioning (Psychology)/*physiology ; *Drug Tolerance ; Morphine/*pharmacology ; Rats
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 2
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    Organization of endogenous opiate and nonopiate pain control systems (1982)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1982-06-11
    Description: Research during the past decade has revealed the existence of neural systems that modulate pain transmission. Much of this work has focused on the role of endogenous opiate systems, but recent research indicates the involvement of nonopiate mechanisms as well. In this article, we present data demonstrating that opiate and nonopiate analgesia systems can be selectively activated by different environmental manipulations and describe the neural circuitry involved. Both neural and hormonal pathways and both opiate and nonopiate substances play roles in the complex modulation of pain transmission. The existence and description of these modulatory mechanisms have important clinical implications for the treatment of pain.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Watkins, L R -- Mayer, D J -- DA 00576/DA/NIDA NIH HHS/ -- New York, N.Y. -- Science. 1982 Jun 11;216(4551):1185-92.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6281891" target="_blank"〉PubMed〈/a〉
    Keywords: Acupuncture Therapy ; Brain/physiology ; Electric Stimulation ; Endorphins/*physiology ; Foot ; Hypnosis ; Morphine/pharmacology ; Pain/*physiopathology ; Placebos ; Receptors, Opioid/*physiology ; Spinal Cord/physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 3
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    Evidence for the neuropeptide cholecystokinin as an antagonist of opiate analgesia (1983)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1983-01-21
    Description: The endogenous neuropeptide cholecystokinin, when administered systemically or perispinally, potently antagonizes opiate analgesia produced by foot shock and morphine. Nonopiate foot-shock analgesia is not reduced by this neuropeptide. The spinal cord appears to be a critical site of cholecystokinin action. These experiments suggest a physiological role for cholecystokinin as a specific opiate antagonist in analgesia-mediating systems. A similar mode of action may explain other behavioral effects of cholecystokinin, such as suppression of food intake.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Faris, P L -- Komisaruk, B R -- Watkins, L R -- Mayer, D J -- New York, N.Y. -- Science. 1983 Jan 21;219(4582):310-2.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6294831" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Cholecystokinin/*pharmacology ; Conditioning, Classical ; Morphine/*antagonists & inhibitors ; Pain/*physiopathology ; Receptors, Cell Surface/physiology ; Receptors, Opioid/*drug effects
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 4
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    Potentiation of opiate analgesia and apparent reversal of morphine tolerance by proglumide (1984)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1984-04-27
    Description: Exogenous cholecystokinin selectively antagonizes opiate analgesia, which suggests that endogenous cholecystokinin may act physiologically as an opiate antagonist and may play a role in opiate tolerance. The use of the selective cholecystokinin antagonist proglumide provided a test of these hypotheses in rats that were either inexperienced with or tolerant to opiates. Proglumide potentiated analgesia produced by morphine and endogenous opiates and seemed to reverse tolerance. These results suggest that endogenous cholecystokinin systems oppose the action of opiates.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Watkins, L R -- Kinscheck, I B -- Mayer, D J -- DA 00576/DA/NIDA NIH HHS/ -- New York, N.Y. -- Science. 1984 Apr 27;224(4647):395-6.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6546809" target="_blank"〉PubMed〈/a〉
    Keywords: *Analgesia ; Animals ; Brain/drug effects ; Dose-Response Relationship, Drug ; Drug Synergism ; Drug Tolerance/drug effects ; Endorphins/physiology ; Enkephalin, Methionine/analogs & derivatives/pharmacology ; Glutamine/*analogs & derivatives ; Humans ; Injections, Spinal ; Morphine/*pharmacology ; Proglumide/administration & dosage/*pharmacology ; Rats ; Spinal Cord/drug effects
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 5
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    Potentiation of Opiate Analgesia and Apparent Reversal of Morphine Tolerance by Proglumide (1984)
    American Association for the Advancement of Science
    Details
    Publication Date: 1984-04-27
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science
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