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    Development. Programming the X chromosome (2004)
    American Association for the Advancement of Science (AAAS)
    Details
    Publikationsdatum: 2004-01-31
    Beschreibung: 〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Hajkova, Petra -- Surani, M Azim -- New York, N.Y. -- Science. 2004 Jan 30;303(5658):633-4.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Wellcome Trust Cancer Research UK Gurdon Institute, University of Cambridge, Cambridge CB2 1QR, UK.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/14752149" target="_blank"〉PubMed〈/a〉
    Schlagwort(e): Animals ; Chromosomes, Mammalian/physiology ; *Dosage Compensation, Genetic ; Embryo, Mammalian/*physiology ; *Embryo, Nonmammalian ; *Embryonic and Fetal Development ; Female ; *Gene Expression Regulation, Developmental ; Genes, Homeobox ; Genomic Imprinting ; Histones/metabolism ; Male ; Methylation ; Placenta/*physiology ; Pluripotent Stem Cells/physiology ; Polycomb Repressive Complex 2 ; Proteins/metabolism ; RNA, Long Noncoding ; RNA, Untranslated/metabolism ; Repressor Proteins/metabolism ; X Chromosome/*physiology
    Print ISSN: 0036-8075
    Digitale ISSN: 1095-9203
    Thema: Biologie , Chemie und Pharmazie , Informatik , Medizin , Allgemeine Naturwissenschaft , Physik
    Publiziert von American Association for the Advancement of Science (AAAS)
    Standort Signatur Erwartet Verfügbarkeit
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  • 2
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    Development of gynogenetic eggs in the mouse: implications for parthenogenetic embryos (1983)
    American Association for the Advancement of Science (AAAS)
    Details
    Publikationsdatum: 1983-12-02
    Beschreibung: Mouse eggs with different genetic constitutions were prepared by micromanipulation of fertilized diploids and triploids. The diploid gynogenones, activated by the male gamete which was then removed, developed at best to about the 25-somite stage as did the genetically similar diploid parthenogenones stimulated to develop in the complete absence of the male gamete. The failure of development to term in both cases may be due to homozygosity and does not appear to be due to a lack of extragenetic contribution from spermatozoa.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Surani, M A -- Barton, S C -- New York, N.Y. -- Science. 1983 Dec 2;222(4627):1034-6.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6648518" target="_blank"〉PubMed〈/a〉
    Schlagwort(e): Animals ; Cell Division ; Cytoplasm/physiology ; Embryo Implantation ; Embryo Transfer ; Embryo, Mammalian/*physiology ; Female ; Genes, Lethal ; Homozygote ; Male ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; *Parthenogenesis ; Spermatozoa/physiology
    Print ISSN: 0036-8075
    Digitale ISSN: 1095-9203
    Thema: Biologie , Chemie und Pharmazie , Informatik , Medizin , Allgemeine Naturwissenschaft , Physik
    Publiziert von American Association for the Advancement of Science (AAAS)
    Standort Signatur Erwartet Verfügbarkeit
    BibTip Andere fanden auch interessant ...
    AKTUELLE ARTIKEL
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