Publication Date:
1989-05-19
Description:
T cell vaccination against experimental autoimmune disease is herein shown to be mediated in part by anti-ergotypic T cells, T cells that recognize and respond to the state of activation of other T cells. The anti-ergotypic response thus combines with the previously shown anti-idiotypic T cell response to regulate autoimmunity.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Lohse, A W -- Mor, F -- Karin, N -- Cohen, I R -- NS 23372/NS/NINDS NIH HHS/ -- New York, N.Y. -- Science. 1989 May 19;244(4906):820-2.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Weizmann Institute of Science, Department of Cell Biology, Rehovot, Israel.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/2471264" target="_blank"〉PubMed〈/a〉
Keywords:
Animals
;
Antigens, Bacterial/immunology
;
Autoimmune Diseases/*immunology
;
Concanavalin A/pharmacology
;
Encephalomyelitis, Autoimmune, Experimental/*immunology
;
Hypersensitivity, Delayed
;
Immunization
;
Immunization, Passive
;
Immunoglobulin Idiotypes/immunology
;
Lymphocyte Activation
;
Mycobacterium tuberculosis/immunology
;
Myelin Basic Protein/immunology
;
Rats
;
Rats, Inbred Lew
;
T-Lymphocytes/*immunology
Print ISSN:
0036-8075
Electronic ISSN:
1095-9203
Topics:
Biology
,
Chemistry and Pharmacology
,
Computer Science
,
Medicine
,
Natural Sciences in General
,
Physics
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