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  • 1
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    Resolution of lung inflammation by CD44 (2002)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2002-04-06
    Description: Successful repair after tissue injury and inflammation requires resolution of the inflammatory response and removal of extracellular matrix breakdown products. We have examined whether the cell-surface adhesion molecule and hyaluronan receptor CD44 plays a role in resolving lung inflammation. CD44-deficient mice succumb to unremitting inflammation following noninfectious lung injury, characterized by impaired clearance of apoptotic neutrophils, persistent accumulation of hyaluronan fragments at the site of tissue injury, and impaired activation of transforming growth factor-beta1. This phenotype was partially reversed by reconstitution with CD44+ cells, thus demonstrating a critical role for this receptor in resolving lung inflammation.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Teder, Priit -- Vandivier, R William -- Jiang, Dianhua -- Liang, Jiurong -- Cohn, Lauren -- Pure, Ellen -- Henson, Peter M -- Noble, Paul W -- HL60539/HL/NHLBI NIH HHS/ -- New York, N.Y. -- Science. 2002 Apr 5;296(5565):155-8.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Department of Medicine, Pulmonary and Critical Care Section, Yale University School of Medicine, New Haven, CT 06520, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/11935029" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Antigens, CD44/*physiology ; Apoptosis ; Bleomycin ; Bone Marrow Transplantation ; Bronchoalveolar Lavage Fluid/chemistry/cytology ; Cell Count ; Chemokines/genetics/metabolism ; Chimera ; Humans ; Hyaluronic Acid/analysis/metabolism ; Lung/immunology/metabolism/*pathology ; Lung Diseases, Interstitial/*immunology/metabolism/*pathology ; Macrophages, Alveolar/physiology ; Mice ; Mice, Inbred C57BL ; Neutrophil Infiltration ; Neutrophils ; Phagocytosis ; Phenotype ; Pulmonary Alveoli/immunology/metabolism/pathology ; RNA, Messenger/genetics/metabolism ; Transforming Growth Factor beta/metabolism ; Transforming Growth Factor beta1
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 2
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    Requirement for TNF-alpha and IL-1 alpha in fetal thymocyte commitment and differentiation (1995)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1995-06-30
    Description: CD25 expression occurs early in thymocyte differentiation. The mechanism of induction of CD25 before T cell receptor rearrangement and the importance of this mechanism for T cell development are unknown. In a thymus reconstitution assay, tumor necrosis factor alpha (TNF-alpha) and interleukin-1 alpha (IL-1 alpha), two cytokines produced within the thymic microenvironment, induced CD25 expression on early immature thymocytes. Either TNF-alpha or IL-1 alpha was necessary for further thymocyte maturation and CD4+CD8+ differentiation. In irradiated mice reconstituted with CD117+CD25+ thymocytes, commitment to the T cell lineage was marked by the loss of precursor multipotency.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Zuniga-Pflucker, J C -- Jiang, D -- Lenardo, M J -- New York, N.Y. -- Science. 1995 Jun 30;268(5219):1906-9.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, Bethesda, MD 20892, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7541554" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Cell Differentiation ; Cells, Cultured ; Hematopoietic Stem Cells/*cytology/immunology ; Interleukin-1/pharmacology/*physiology ; Interleukin-7/pharmacology ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; Mice, SCID ; Organ Culture Techniques ; Proto-Oncogene Proteins/biosynthesis ; Proto-Oncogene Proteins c-kit ; Receptor Protein-Tyrosine Kinases/biosynthesis ; Receptors, Colony-Stimulating Factor/biosynthesis ; Receptors, Interleukin-2/*biosynthesis ; Stromal Cells/physiology ; T-Lymphocyte Subsets/cytology/immunology ; T-Lymphocytes/*cytology/immunology ; Thymus Gland/cytology/*embryology/immunology ; Tumor Necrosis Factor-alpha/pharmacology/*physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 3
    Electronic Resource
    Electronic Resource
    Observation of new neutron rich nuclei29F,35,36Mg,38,39Al,40,41Si,43,44P,45–47S,46–49Cl, and49–51Ar from the interactions of 55 MeV/U48Ca+Ta (1989)
    Springer
    The European physical journal 332 (1989), S. 189-193 
    Details
    ISSN: 1434-601X
    Keywords: 21.10.−k ; 07.75. + h ; 25.70.NP ; 27.30. + t ; 27.40. + z
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract After magnetic separation, identification through time of flight and δE×E measurements has allowed the observation of the new nuclei,29F,35,36Mg,38,39Al,40,41Si,43,44P,45,46,47S;46,47,48,49Cl;49,50,51Ar from the interaction of a48Ca beam of 55 MeV/u with tantalum targets.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01289774
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    Paper (German National Licenses)
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