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  • Articles  (27)
  • Springer  (27)
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  • Articles  (27)
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  • 1
    Electronic Resource
    Electronic Resource
    Wild-derived alleles of five allozyme-encoding loci in B10.W mice (1981)
    Springer
    Immunogenetics 13 (1981), S. 173-176 
    Details
    ISSN: 1432-1211
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00524614
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  • 2
    Electronic Resource
    Electronic Resource
    Linkage of Pgm-3 in the house mouse and homologies of three phosphoglucomutase loci in mouse and man (1981)
    Springer
    Biochemical genetics 19 (1981), S. 465-474 
    Details
    ISSN: 1573-4927
    Keywords: phosphoglucomutase ; homology ; linkage ; house mouse ; major histocompatability complex
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology
    Notes: Abstract The discovery of a third phosphoglucomutase locus (Pgm-3) in the house mouse is reported. Three alleles are recognized on the basis of differences in electrophoretic mobility and enzymatic activity. Pgm-3 a (fast mobility and high activity) is present in inbred strain C57BL/10J and 24 other strains; Pgm-3 b (slow mobility and high activity) is present in LP/Pas and six other strains; and Pgm-3 c (no detectable activity in any tissue tested) is present in strain DBA/2J and 14 other strains. Seventy-four recombinant inbred strains derived from progenitors that differed at Pgm-3 were used to study genic linkage. Pgm-3 is on chromosome 9 and is linked to Sep-1, d, Mod-1, and Ltw-3. Gene order and recombination frequencies are estimated as d 3.8±1.8% Pgm-3 2.3±1.2% Mod-1. Substrate specificities and cofactor requirements show that mouse Pgm-1 is homologous with human Pgm-2, mouse Pgm-2 with human Pgm-1, and mouse Pgm-3 with human Pgm-3.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00484619
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  • 3
    Electronic Resource
    Electronic Resource
    An explanation for the rate-of-loading and the duration-of-load effects in wood in terms of fracture mechanics (1982)
    Springer
    Journal of materials science 17 (1982), S. 2831-2840 
    Details
    ISSN: 1573-4803
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: Abstract Wood is one of the few engineering materials for which design codes specify that the applied stress be contingent on the duration of the load. This is in recognition of the fact that the strength of wood appears to degrade with time when under stress. This paper describes a set of experiments in which the kinetics of wood fracture are examined. It is shown that the present results and those of earlier workers can be fully explained by a relatively simple model and mathematical analysis, based on fracture mechanics. According to these results, the delayed failure of wood is caused by subcritical crack growth. The model helps to reveal what must be done to incorporate the duration-of-load effect into timber design in a probabalistic manner.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00644658
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  • 4
    Electronic Resource
    Electronic Resource
    Slow crack growth in graphite (1975)
    Springer
    Journal of materials science 10 (1975), S. 846-856 
    Details
    ISSN: 1573-4803
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics
    Notes: Abstract Slow crack growth tests on isotropic graphite and pyrolytic graphite at room temperature have revealed a stress intensity-crack velocity behaviour similar to the third stage of crack growth in soda-lime glass. Thus, it appears that graphite undergoes failure at room temperature only at large fractions of the critical stress intensity. Considerable difference in fracture behaviour of graphites having different microstructures was observed. Slow crack growth occurred more readily at 500
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01163079
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  • 5
    Electronic Resource
    Electronic Resource
    Fert is on mouse Chromosome 11, not Chromosome 17 (1994)
    Springer
    Mammalian genome 5 (1994), S. 830-830 
    Details
    ISSN: 1432-1777
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00292028
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  • 6
    Electronic Resource
    Electronic Resource
    Location of the mouse mast cell protease 7 gene (Mcpt7) to Chromosome 17 (1994)
    Springer
    Mammalian genome 5 (1994), S. 656-657 
    Details
    ISSN: 1432-1777
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00411467
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  • 7
    Electronic Resource
    Electronic Resource
    New murine polymorphisms detected by random amplified polymorphic DNA (RAPD) PCR and mapped by use of recombinant inbred strains (1994)
    Springer
    Mammalian genome 5 (1994), S. 762-767 
    Details
    ISSN: 1432-1777
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Oligonucleotide primers of random sequence that were 12 bases in length, 58% in GC content, and lacking internal palindromes were designed. By random amplified polymorphic DNA (RAPD) PCR, these primers were used to survey for DNA variations between the progenitors of the mouse AXB and BXA recombinant inbred sets (A/J and C57BL/6J). We identified 17 DNA variants detected by 10 primers. Map positions for these variants were determined by comparing their strain distribution patterns in the AXB, BXA recombinant inbred sets with strain distribution patterns of previously published loci. When necessary, BXD and NXSM recombinant inbred sets were also used. These 17 new loci mapped to 12 chromosomes. The 10 primers were also used to survey 20 inbred mouse strains including the progenitors of other recombinant inbred sets and four mouse strains recently inbred from the wild (CAST/Ei, MOLF/Ei, PERA/Ei, and SPRET/Ei).
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00292009
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  • 8
    Electronic Resource
    Electronic Resource
    Genetic mapping of 40 cDNA clones on the mouse genome by PCR (1994)
    Springer
    Mammalian genome 5 (1994), S. 349-355 
    Details
    ISSN: 1432-1777
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract We recently proposed a new PCR-based genetic marker assay for the mouse genome that exploits sequence differences in the 3′-untranslated region (UTR) of cDNAs between different mouse strains, called “biallelic polymorphic expressed sequence tags (bESTs).” The specific use of 3′-UTR has several advantages: (1) frequent sequence polymorphism between different mouse strains, (2) most commonly uninterrupted by introns, (3) usually unique sequence even among closely related gene family members. In this paper, we identify additional genetic loci defined by bEST and determine their location on the mouse genetic map by using interspecific backross mapping panels between C57BL/6J and Mus spretus. Of 136 markers tested, 86 produced unique PCR products from C57BL/6J and M. spretus genomic DNAs. We then sequenced these 86 PCR products from C57BL/6J and M. spretus and found that 59 markers have sequence polymorphisms. Of these, we mapped 36 by restriction fragment length polymorphism (RFLP) of the PCR products and 4 by length polymorphism (LP) of the PCR products. We discuss the possibility of a large-scale application of this method for cDNA mapping.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00356553
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  • 9
    Electronic Resource
    Electronic Resource
    Maps from two interspecific backcross DNA panels available as a community genetic mapping resource (1994)
    Springer
    Mammalian genome 5 (1994), S. 253-274 
    Details
    ISSN: 1432-1777
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract We established two mouse interspecific backcross DNA panels, one containing 94 N2 animals from the cross (C57BL/6J × Mus spretus)F1 × C57BL/6J, and another from 94 N2 animals from the reciprocal backcross (C57BL/6J × SPRET/Ei)F1 × SPRET/Ei. We prepared large quantities of DNA from most tissues of each animal to create a community resource of interspecific backcross DNA for use by laboratories interested in mapping loci in the mouse. Initial characterization of the genetic maps of both panels has been completed. We used MIT SSLP markers, proviral loci, and several other sequence-defined genes to anchor our maps to other published maps. The BSB panel map (from the backcross to C57BL/6J) contains 215 loci and is anchored by 45 SSLP and 32 gene sequence loci. The BSS panel map (from the backcross to SPRET/Ei) contains 451 loci and is anchored by 49 SSLP loci, 43 proviral loci, and 60 gene sequence loci. To obtain a high density of markers, we used motif-primed PCR to “fingerprint” the panel DNAs. We constructed two maps, each representing one of the two panels. All new loci can be located with a high degree of certainty on the maps at current marker density. Segregation patterns in these data reveal several examples of transmission ratio distortion and permit analysis of the distribution of crossovers on individual chromosomes.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00389540
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  • 10
    Electronic Resource
    Electronic Resource
    Mapping of the ACTH, MSH, and neural (MC3 and MC4) melanocortin receptors in the mouse and human (1994)
    Springer
    Mammalian genome 5 (1994), S. 503-508 
    Details
    ISSN: 1432-1777
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract The melanocortin peptides regulate a wide variety of physiological processes, including pigmentation and glucocorticoid production, and also have several activities in the central and peripheral nervous systems. The melanocortin receptor family includes the melanocytestimulating hormone receptor (MSH-R), adrenocorticotropic hormone receptor (ACTH-R), and two neural receptors, MC3-R and MC4-R. In the human these receptors map to 16q24 (MSH-R), 18p11.2 (ACTH-R), 20q13.2 (MC3-R), and 18q22 (MC4-R). The corresponding locations in the mouse are 8, 18, and 2; a variant for mapping MC4-R has not yet been identified. The data reported here also show that the neural MC3 receptor maps close to a disease locus for benign neonatal epilepsy in human and near the El-2 epilepsy susceptibility locus in the mouse.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00369320
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