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  • Guinea pig  (1)
  • Housing  (1)
  • Lysosomes  (1)
  • Springer  (2)
  • 1
    Electronic Resource
    Electronic Resource
    British weather: Conversation topic or serious health risk? (1996)
    Springer
    International journal of biometeorology 39 (1996), S. 151-155 
    Details
    ISSN: 1432-1254
    Keywords: Housing ; Climate ; Seasonal mortality ; Building ; Energy efficiency
    Source: Springer Online Journal Archives 1860-2000
    Topics: Geography , Physics
    Notes: Abstract Britain is renowned for low indoor winter temperatures and a high rate of excess winter deaths and a causal association has been drawn between the two. A lack of priority given to energy efficiency here is often justified in terms of the mild climate. However, it can be shown that British climatic features may be related to poor health as long as poor standards of construction and insulation prevail. Legislation should therefore be implemented to improve standards both for new housing and refurbishment of much existing housing stock, not only for reasons of energy conservation but also for community health.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01211229
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  • 2
    Electronic Resource
    Electronic Resource
    Are lysosomal enzymes involved in rapid damage in vertebrate muscle cells? (1988)
    Springer
    Cell & tissue research 253 (1988), S. 447-455 
    Details
    ISSN: 1432-0878
    Keywords: Lysosomes ; Muscle damage ; Calcium ; Lysosomotropic agents ; Phospholipase A2 ; Frog (Rana temporaria) ; Guinea pig
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Summary Experiments with lysosomotropic agents suggest that the sarcotubular system subserves some of the functions of the lysosomal apparatus in frog skeletal muscle. Dinitrophenol or A23187 trigger lysosome labilization and myofilament damage in mammalian cardiac muscle. Lysolecithin labilizes isolated liver lysosomes, but has no action following phospholipase A2 activation in vivo. Zinc ions or a pHi of 7.5 do not protect against myofilament damage. In fractions from mammalian cardiac muscle, calcium and calmodulin do not cause lysosomal labilization whereas cGMP does but only at high concentration (10-4 M). It is concluded that lysosomal hydrolases play no significant part in rapid muscle damage. It is suggested that rises in [Ca]i activate two separate pathways causing (i) myofilament damage; (ii) sarcolemmal (and possibly lysosomal) membrane damage via phospholipase A2 and lipoxygenase activity. Dinitrophenol triggers both pathways independently and thus may cause lysosome labilization. The possibility that the sarcoplasmic reticulum is the site generating myofilament damage is discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00222302
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