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  • Diversity indices  (1)
  • Key words: Bone metabolic markers — Predictor — Osteoporosis — Epidemiology — Bone mineral density.  (1)
  • Membrane transport  (1)
  • Springer  (3)
  • Nature Publishing Group
  • 1
    Electronic Resource
    Electronic Resource
    Biochemical Markers of Bone Turnover and Bone Loss at the Lumbar Spine and Femoral Neck: The Taiji Study (1999)
    Springer
    Calcified tissue international 65 (1999), S. 198-202 
    Details
    ISSN: 1432-0827
    Keywords: Key words: Bone metabolic markers — Predictor — Osteoporosis — Epidemiology — Bone mineral density.
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine , Physics
    Notes: Abstract. The purpose of this study was to ascertain whether biochemical markers of bone turnover predict bone loss. The survey was carried out in Taiji, Wakayama Prefecture, Japan. From a list of inhabitants aged 40–79 years, 400 participants (50 men and 50 women in each of four age groups) were selected randomly. Bone mineral density (BMD) was measured, and blood and urine samples of all participants were examined to obtain values for eight biochemical markers: alkaline phosphatase (ALP), bone Gla protein (BGP), type I procollagen (carboxyterminal peptide of type I procollagen; PICP), cross-linked carboxyterminal telopeptide region of type I collagen (ICTP), and urinary excretion of calcium (Ca), phosphate (P), pyridinoline (Pyr), and deoxypyridinoline (D-Pyr). Each marker was evaluated as a predictor of the rate of bone change in lumbar spine and femoral neck BMD over a 3-year period. The value of Pyr was significantly related to the change of lumbar spine BMD in men (P= 0.009), and that of BGP was found to be significant in women (P= 0.045). By contrast, none of the bone markers significantly correlated with bone loss at the femoral neck. The coefficient of determination at the lumbar spine was 5% and 7% at the femoral neck only. We conclude that biochemical markers of bone turnover cannot predict bone loss rates in middle-aged or elderly Japanese men and women over a 3-year period with sufficient accuracy for use in clinical decision making.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s002239900682
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  • 2
    Electronic Resource
    Electronic Resource
    Effects of channel incision on base flow stream habitats and fishes (1994)
    Springer
    Environmental management 18 (1994), S. 43-57 
    Details
    ISSN: 1432-1009
    Keywords: Streams ; Erosion ; Sediment ; Woody debris ; Channel degradation ; Habitat restoration ; Fish ; Diversity indices
    Source: Springer Online Journal Archives 1860-2000
    Topics: Energy, Environment Protection, Nuclear Power Engineering
    Notes: Abstract Channel incision is a widespread phenomenon that results in stream and riparian habitat degradation. Fishes and physical habitat variables were sampled at base flow from three incised stream channels and one reference stream in northwest Mississippi, USA, to quantify incision effects on fish habitat and provide a basis for habitat rehabilitation planning and design. Incised channels were sampled in spring and autumn; the reference channel was sampled only in the autumn. Incised channel habitat quality was inferior to the reference channel despite the presence of structures designed to restore channel stability. Incised channels had physical habitat diversity levels similar to a nonincised reference channel, but contained fewer types of habitat. At base flow, incised channels were dominated by shallow, sandy habitats, moderate to high mean local Froude numbers, and had relatively little organic debris in their beds. In contrast, the reference stream had greater mean water depth, contained more woody debris, and provided more deep pool habitat. Fish assemblages in incised channels were composed of smaller fishes representing fewer species relative to the reference site. Fish species richness was directly proportional to the mean local Froude number, an indicator of the availability of pool habitat.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02393749
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  • 3
    Electronic Resource
    Electronic Resource
    Transport of K+ and other cations across phospholipid membranes by nonesterified fatty acids (1994)
    Springer
    The journal of membrane biology 141 (1994), S. 21-28 
    Details
    ISSN: 1432-1424
    Keywords: Fatty acids ; Liposomes ; Cation transport ; Membrane transport
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Chemistry and Pharmacology
    Notes: Abstract The rate of change of internal pH and transmembrane potential has been monitored in liposomes following the external addition of various cation salts. Oleic acid increases the transmembrane movement of H+ following the imposition of a K+ gradient. An initial fast change in internal pH is seen followed by a slower rate of alkalinization. High concentrations of the fatty acid enhance the rate comparable to that seen in the presence of nigericin in contrast to the effect of FCCP (carbonyl cyanide p-(tri-fluoromethoxy)phenyl hydrazone) which saturates at an intermediate value. The ability of nonesterified fatty acids to catalyze the movement of cations across the liposome membrane increases with the degree of unsaturation and decreases with increasing chain length. Li and Na salts cause a similar initial fast pH change but have less effect on the subsequent slower rate. Similarly, the main effect of divalent cation salts is on the initial fast change. The membrane potential can enhance or inhibit cation transport depending on its polarity with respect to the cation gradient. It is concluded that nonesterified fatty acids have the capability to complex with, and transport, a variety of cations across phospholipid bilayers. However, they do not act simply as proton/cation exchangers analogous to nigericin nor as protonophores analogous to FCCP. The full cycle of ionophoric action involves a combination of both functions.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00232870
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