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  • 1
    Electronic Resource
    Electronic Resource
    Observation of the A 2Π→X 2Σ+ emission in NO2+ (1988)
    College Park, Md. : American Institute of Physics (AIP)
    The Journal of Chemical Physics 89 (1988), S. 6554-6555 
    Details
    ISSN: 1089-7690
    Source: AIP Digital Archive
    Topics: Physics , Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.455377
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  • 2
    Electronic Resource
    Electronic Resource
    Photoion–photoion coincidence study of the fragmentation of the doubly charged N2+2 cation (1988)
    College Park, Md. : American Institute of Physics (AIP)
    The Journal of Chemical Physics 88 (1988), S. 1732-1736 
    Details
    ISSN: 1089-7690
    Source: AIP Digital Archive
    Topics: Physics , Chemistry and Pharmacology
    Notes: Formation and fragmentation of the doubly charged N2+2 cation were studied by using the photoion–photoion coincidence (PIPICO) method. Six electronic states of N2+2 were observed. Their ionization energies were measured with uncertainties of about 0.3 eV, which allowed a tentative assignment of these states. It was furthermore demonstrated that N2+2 dissociates into N+(3P)+N+(3P) whichever initial electronic state is populated.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.454736
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  • 3
    Electronic Resource
    Electronic Resource
    Discovery of an emission in NO++ (1986)
    College Park, Md. : American Institute of Physics (AIP)
    The Journal of Chemical Physics 85 (1986), S. 1316-1321 
    Details
    ISSN: 1089-7690
    Source: AIP Digital Archive
    Topics: Physics , Chemistry and Pharmacology
    Notes: Doubly charged NO++ ions were produced by double photoionization of neutral nitric oxide molecules with the synchrotron radiation from ACO as a photon source of variable energy in the 35–68 eV range. Two stable states of NO++ were observed in the double photoionization spectrum with energies of 38.6 and 40.0 eV. An emission from NO++ was discovered by using a new photoion–photon of fluorescence coincidence (PIFCO) experiment. The fluorescence efficiency was measured as a function of the excitation energy, indicating that the emitting state, whose onset energy was observed at 42.5 eV, is a stable or slowly predissociating state of NO++. On the basis of the comparison with calculated energies, the three stable states of NO++ with energies at 38.6, 40.0, and 42.5 eV were assigned to, respectively, the X 2Σ+, A 2Π, and B 2Σ+ electronic states. The detected emission was ascribed to a B 2Σ+→X 2Σ+ emission around 3170 A(ring).
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1063/1.451217
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  • 4
    Electronic Resource
    Electronic Resource
    Two group I mitochondrial introns in the cob-box and coxI genes require the same MRS1/PET157 nuclear gene product for splicing (1990)
    Springer
    Current genetics 18 (1990), S. 117-124 
    Details
    ISSN: 1432-0983
    Keywords: Yeast ; Mitochondrial RNA splicing ; Nuclear pet - mutant ; Group I introns
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Summary We have studied the role of the product of the nuclear gene PET157 in mitochondrial pre-mRNA splicing. Cytoduction experiments show that a mitochondrial genome deleted for the three introns bI3, aI5 and aI6 is able to suppress the pet157-1 mutation: the strain recovers respiratory competency indicating that the product of the PET157 gene is only required for mitochondrial premRNA splicing. Characterization of the high molecular weight pre-mRNAs which accumulate in the pet157 mutant demonstrate that the product of the PET157 gene is required for the excision of two group I introns bI3 and aI6 (corresponding to aI5β) located in the cob-box and coxI genes respectively. Furthermore, the pet157 mutant strain accumulates the bI3 maturase in the form of a polypeptide of 50K (p50) previously observed in mitochondrial mutants defective in the excision of bI3. We have shown by restriction analysis and allelism tests that the pet157-1 mutation is allelic to the nuclear mrs1 mutation, previously described as specifically blocking the excision of bI3. Finally, revertants obtained by the deletion of bI3 or aI6 from the mitochondrial DNA were isolated from the MRS1 disrupted allele, confirming the involvment of the product of the MRS1/PET157 gene in the excision of the two introns bI3 and aI6.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00312599
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  • 5
    Electronic Resource
    Electronic Resource
    Ion photodesorption from argon multilayers (1998)
    Springer
    Applied physics 66 (1998), S. 527-538 
    Details
    ISSN: 1432-0630
    Keywords: PACS: 79.20; 61.80
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics , Physics
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/s003390050708
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  • 6
    Electronic Resource
    Electronic Resource
    Cloning by functional complementation, and inactivation, of theSchizosaccharomyces pombe homologue of theSaccharomyces cerevisiae geneABC1 (1996)
    Springer
    Molecular genetics and genomics 251 (1996), S. 204-210 
    Details
    ISSN: 1617-4623
    Keywords: Schizosaccharomyces pombe ; Heterologous functional complementation ; gene inactivation ; respiration ; bc1 complex
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Abstract TheSaccharomyces cerevisiae geneABC1 is required for the correct functioning of thebc 1 complex of the mitochondrial respiratory chain. By functional complementation of aS. cerevisiae abc1 − mutant, we have cloned aSchizosaccharomyces pombe cDNA, whose predicted product is 50% identical to the Abc1 protein. Significant homology is also observed with bacterial, nematode, and even human amino acid sequences of unknown function, suggesting that the Abc1 protein is conserved through evolution. The cloned cDNA corresponds to a singleS. pombe geneabc1Sp, located on chromosome II, expression of which is not regulated by the carbon source. Inactivation of theabc1Sp gene by homologous gene replacement causes a respiratory deficiency which is efficiently rescued by the expression of theS. cerevisiae ABC1 gene. The inactivated strain shows a drastic decrease in thebc 1 complex activity, a decrease in cytochromeaa3 and a slow growth phenotype. To our knowledge, this is the first example of the inactivation of a respiratory gene inS. pombe. Our results highlight the fact thatS. pombe growth is highly dependent upon respiration, and thatS. pombe could represent a valuable model for studying nucleo-mitochondrial interactions in higher eukaryotes.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02172919
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  • 7
    Electronic Resource
    Electronic Resource
    Long range control circuits within mitochondria and between nucleus and mitochondria (1980)
    Springer
    Molecular genetics and genomics 179 (1980), S. 469-482 
    Details
    ISSN: 1617-4623
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Summary To uncover the functional circuitry both within the mitochondrial genome and between the mitochondrial and the nuclear genome, we have developed a general method for selecting and characterizing genetically suppressor mutations that restore the respiratory capacity of mit - mitochondrial mutants. Several hundreds of pseudo-wild type revertants due to a second unlinked mutation which suppresses a target mit - mutation were isolated. The suppressor mutations were found located either in the nuclear (abbreviated NAM for ‘nuclear accommodation of mitochondria’) or in the mitochondrial genome (abbreviated MIM for ‘mitochondrial-mitochondrial interaction’). The specificity of action of various suppressors upon some 250 different mit - mutations located in several genes was tested. According to this specificity of action, suppressors were subdivided into two major classes: allele specific or gene specific suppressors. Because the cob-box mitochondrial gene has a mosaic organization, we were able to find a novel third class of extragenic suppressors specific for mit - mutations within the introns of this gene. Four examples of suppressors showing various specificities of action illustrate our approach. (1) a nuclear gene controlling specific alleles of different mitochondrial genes; (2) a nuclear gene controlling selectively one intron of a split mitochondrial gene; (3) a mitochondrial gene controlling specific alleles of different mitochondrial genes; (4) a region in one complex mitochondrial gene which controls selectively one intron of another split mitochondrial gene. Different mechanisms of suppression are discussed stressing the alleviation of splicing deficiencies of intron mutations.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00271736
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  • 8
    Electronic Resource
    Electronic Resource
    SLS1, a newSaccharomyces cerevisiae gene involved in mitochondrial metabolism, isolated as a syntheticlethal in association with anSSM4 deletion (1996)
    Springer
    Molecular genetics and genomics 252 (1996), S. 700-708 
    Details
    ISSN: 1617-4623
    Keywords: Saccharomyces cerevisiae ; Mitochondria ; Integral membrane protein
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology
    Notes: Abstract SSM4 was isolated as a suppressor ofrna14-1, a mutant involved in nuclear mRNA maturation. In order to isolate genes interacting withSSM4, we have searched for mutants that are syntheticlethal in association with anSSM4 deletion. Among the mutants obtained, one, namedsls1-1, shows apet − phenotype. We have cloned and sequenced this gene. It encodes a protein with a calculated molecular mass of 73 kDa. This protein contains a mitochondrial targeting presequence but does not show homology with other known proteins. Deletion ofSLS1 does not affect cell viability on glucose but is lethal on a non-fermentable medium. The Sls1p protein does not appear to be involved in mitochondrial DNA replication, transcription, or in RNA splicing maturation or stability. We have also tagged this protein and localized it in mitochondria. Treatment with alkaline carbonate does not extract this protein from mitochondria, suggesting strongly that it is a mitochondrial integral membrane protein. Thus, theSLS1 gene, encodes a mitochondrial integral membrane protein and is paradoxically synlethal in association with a deletion of theSSM4 gene, which encodes an integral nuclear membrane protein.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02173976
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  • 9
    Electronic Resource
    Electronic Resource
    Fragmentation of doubly charged ammonia cations NH 3 ++ studied by the photoion-photoion coincidence (PIPICO) method (1986)
    Springer
    The European physical journal 4 (1986), S. 57-64 
    Details
    ISSN: 1434-6079
    Keywords: 3300 ; 3520J ; 3690
    Source: Springer Online Journal Archives 1860-2000
    Topics: Physics
    Notes: Abstract Doubly charged NH 3 ++ cations were produced by double photoionization of neutral ammonia molecules by using the synchrotron radiation from ACO as a photon source of variable energy in the 35–49 eV energy range. The fragmentation of NH 3 ++ was studied by the photoion-photoion coincidence (PIPICO) method. NH 3 ++ cations were produced in the $$\tilde X^1 $$ A 1 and $$\tilde B^1 $$ electronic states of which the onset energies were measured at, respectively, 35.4±0.5 eV and 44.5±0.5 eV. It was shown that the NH 3 ++ ions, initially produced in their $$\tilde X^1 $$ A 1 state, rapidly dissociate (in less than 50 ns), into NH 2 + + H+. Furthermore, the comparison with results obtained by other methods indicates that NH 3 ++ ions can either be long-lived (τ〉10 µs) or slowly dissociating (1 µs〈τ〈10 µs) or rapidly dissociating (τ〈50 ns), depending on their geometry and/or internal energy in their $$\tilde X^1 $$ E A 1 electronic state.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01432498
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  • 10
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    Ion photodesorption from argon multilayers (1998)
    Springer
    Details
    Publication Date: 1998-05-01
    Print ISSN: 0947-8396
    Electronic ISSN: 1432-0630
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics , Physics
    Published by Springer
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