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  • damage tensors  (1)
  • mitochondria  (1)
  • Springer  (2)
  • American Chemical Society (ACS)
  • American Geophysical Union
  • American Institute of Physics (AIP)
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Keywords
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  • Springer  (2)
  • American Chemical Society (ACS)
  • American Geophysical Union
  • American Institute of Physics (AIP)
Years
  • 1
    Electronic Resource
    Electronic Resource
    A fracture damage model for Jointed rock masses and its application to the stability analysis of dam abutments (1991)
    Springer
    Acta mechanica solida Sinica 4 (1991), S. 101-116 
    Details
    ISSN: 0894-9166
    Keywords: fracture ; damage mechanics ; stability of dam abutment ; arch dam ; jointed rock mass ; damage tensors
    Source: Springer Online Journal Archives 1860-2000
    Topics: Mechanical Engineering, Materials Science, Production Engineering, Mining and Metallurgy, Traffic Engineering, Precision Mechanics , Physics
    Notes: Abstract Based on continuum damage mechanics, for jointed rock masses, a fracture damage model is presented in this paper. First, the damage tensors are defined through the elastic-flexibility of intact rock and the equivalent elastic-damage flexibility for rock mass. Then, by the self-consistent principle of solid mechanics, the equivalent elastic-damage flexibility tensors involving the interaction between multicracks are deduced. The damage evolution law is proposed involving the mechanism of crack propagation process: frictional sliding, crack kinking, growing of branched tension cracks, interlinking of the microcracks near branched crack tips leading to the breakthrough of macro-cracks and finally the failure of rock mass. Thus the evolution of damage variables reasonably unified with the process of crack propagation is given. Finally, a plastic-brittle damage constitutive relation including brittle coupled strain rate, developed and applied to the stability analysis of complicated rock foundation of a dam in China, is described in this paper.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF02208114
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  • 2
    Electronic Resource
    Electronic Resource
    A Four-Compartment Model for Ca2+ Dynamics: An Interpretation of Ca2+ Decay after Repetitive Firing of Intact Nerve Terminals (2000)
    Springer
    Journal of computational neuroscience 8 (2000), S. 275-298 
    Details
    ISSN: 1573-6873
    Keywords: presynaptic nerve terminals ; calcium decay ; ryanodine-sensitive store ; mitochondria
    Source: Springer Online Journal Archives 1860-2000
    Topics: Computer Science , Medicine , Physics
    Notes: Abstract In the presynaptic nerve terminals of the bullfrog sympathetic ganglia, repetitive nerve firing evokes [Ca2+] transients that decay monotonically. An algorithm based on an eigenfunction expansion method was used for fitting these [Ca2+] decay records. The data were fitted by a linear combination of two to four exponential functions. A mathematical model with three intraterminal membrane-bound compartments was developed to describe the observed Ca2+ decay. The model predicts that the number of exponential functions, n, contained in the decay data corresponds to n − 1 intraterminal Ca2+ stores that release Ca2+ during the decay. Moreover, when a store stops releasing or starts to release Ca2+, the decay data should be fitted by functions that contain one less exponential component for the former and one more for the latter than do the fitting functions for control data. Because of the current lack of a parameter by which quantitative comparisons can be made between two decay processes when at least one of them contained more than one exponential components, we defined a parameter, the overall rate (OR) of decay, as the trace of the coefficient matrix of the differential equation systems of our model. We used the mathematical properties of the model and of the OR to interpret effects of ryanodine and of a mitochondria uncoupler on Ca2+ decay. The results of the analysis were consistent with the ryanodine-sensitive store, mitochondria, and another, yet unidentified store release Ca2+ into the cytosol of the presynaptic nerve terminals during Ca2+ decay. Our model also predicts that mitochondrial Ca2+ buffering accounted for more than 86% of all the flux rates across various membranes combined and that there are type 3 and type 1 and/or type 2 ryanodine receptors in these terminals.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1023/A:1008954127682
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