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  • Articles  (3)
  • Nature Publishing Group  (3)
  • 2000-2004  (3)
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  • Articles  (3)
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  • 1
    Electronic Resource
    Electronic Resource
    Cancer Guarding the guardian? (2004)
    [s.l.] : Nature Publishing Group
    Nature 427 (2004), S. 110-111 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] If there is beauty in simplicity then the p53 protein is unlikely to win any contests. As one of our key defences against cancer, p53 lies at the heart of a massive research effort that has generated an avalanche of papers. The result is a bewildering complexity to virtually every aspect of p53 ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/427110a
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Cancer Pinning a change on p53 (2002)
    [s.l.] : Nature Publishing Group
    Nature 419 (2002), S. 795-797 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] One in three of us will succumb to cancer at some point in our lives. So it's not surprising that p53, a protein that helps to prevent the development of malignancies, is the subject of intense scrutiny. In recent years great progress has been made in understanding how p53 is regulated. Two reports ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/419795a
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  • 3
    Electronic Resource
    Electronic Resource
    Proliferation, cell cycle and apoptosis in cancer (2001)
    [s.l.] : Nature Publishing Group
    Nature 411 (2001), S. 342-348 
    Details
    ISSN: 1476-4687
    Source: Nature Archives 1869 - 2009
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Notes: [Auszug] Beneath the complexity and idiopathy of every cancer lies a limited number of 'mission critical' events that have propelled the tumour cell and its progeny into uncontrolled expansion and invasion. One of these is deregulated cell proliferation, which, together with the obligate compensatory ...
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1038/35077213
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    Paper (German National Licenses)
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