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  • 1
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    Magnetic phase diagram in the Co-rich side of the LCo_{1−x}Fe_{x}AsO (L = La, Sm) system (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-09-06
    Description: Author(s): Y. K. Li, X. F. Xu, C. Cao, C. Y. Shen, Y. K. Luo, Q. Tao, X. Lin, L. Zhang, G. H. Cao, and Z. A. Xu The magnetic phase diagram has been mapped out via the measurements of electronic resistivity, magnetization, and specific heat in the cobalt-based layered L Co 1− x Fe x AsO ( L =La, Sm) compounds. The ferromagnetic (FM) transition at ∼63 K for LaCoAsO is rapidly suppressed upon Fe doping, and ultimately d... [Phys. Rev. B 86, 104408] Published Wed Sep 05, 2012
    Keywords: Magnetism
    Print ISSN: 1098-0121
    Electronic ISSN: 1095-3795
    Topics: Physics
    Published by American Physical Society (APS)
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  • 2
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    Temporal and spatial variations of stem CO2 efflux of three species in subtropical China (2012)
    Oxford University Press
    Details
    Publication Date: 2012-04-13
    Description: Aims Although stem CO 2 efflux is critical to ecosystem carbon and energy balance and its feedback to future climate change, little information is available on stem CO 2 efflux and its responses to temperature, especially in subtropical China. This study aims to (i) evaluate the temporal and spatial variations of stem CO 2 efflux of three species, including oak ( Quercus acutissima Carr.), masson pine ( Pinus massoniana Lamb.) and loblolly pine ( Pinus taeda Linn.) in subtropical China and (ii) analyze the temperature sensitivity of stem CO 2 efflux in the three species based on 2-year field measurements. Methods We measured stem CO 2 efflux and stem temperature (at 3 cm depth) of the three species using the horizontally oriented soil chamber technique from September 2008 to August 2010. We also conducted a 24-h measurement to examine the diurnal variation of stem CO 2 efflux in three consecutive days in April 2009. Important findings The temporal dynamics of stem CO 2 efflux followed the change of the stem temperature in a 3-cm depth with a bell-shaped curve in the three species. Stem temperature explained 77–85% of the seasonal variations of stem CO 2 efflux over the entire study period in the three species. The temperature sensitivity ( Q 10 ) of stem CO 2 efflux was obviously different among the three species with higher Q 10 value found in oak (2.24) and lower values in the coniferous species (1.76 and 1.63). Our results also showed that the Q 10 values of stem CO 2 efflux in all the three species were lower in the growing season than that in the non-growing season, indicating that the growth and maintenance respiration had different temperature responses. Moreover, we found that the temperature-normalized stem CO 2 efflux ( R 10 ) changed greatly between the growing and non-growing seasons in oak and masson pine, but not in loblolly pine. Additionally, we also found that in the non-growing season, the principal factor responsible for the spatial variation of stem CO 2 efflux among the 15 sampling trees was sapwood volume, whereas in the growing season, stem CO 2 efflux was closely related to annual dry-matter production in the three subtropical species.
    Print ISSN: 1752-993X
    Electronic ISSN: 1752-9921
    Topics: Biology
    Published by Oxford University Press
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  • 3
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    Ultrashort pulses and short-pulse equations in 2+1 dimensions (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-08-24
    Description: Author(s): Y. Shen, N. Whitaker, P. G. Kevrekidis, N. L. Tsitsas, and D. J. Frantzeskakis In this paper, we derive and study two versions of the short pulse equation (SPE) in (2+1) dimensions. Using Maxwell's equations as a starting point, and suitable Kramers-Kronig formulas for the permittivity and permeability of the medium, which are relevant, e.g., to left-handed metamaterials and d... [Phys. Rev. A 86, 023841] Published Thu Aug 23, 2012
    Keywords: Quantum optics, physics of lasers, nonlinear optics, classical optics
    Print ISSN: 1050-2947
    Electronic ISSN: 1094-1622
    Topics: Physics
    Published by American Physical Society (APS)
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  • 4
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    Finite-temperature dynamics of matter-wave dark solitons in linear and periodic potentials: An example of an antidamped Josephson junction (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-09-15
    Description: Author(s): Y. Shen, P. G. Kevrekidis, N. Whitaker, N. I. Karachalios, and D. J. Frantzeskakis We study matter-wave dark solitons in atomic Bose-Einstein condensates (BECs) at finite temperatures, under the effect of linear and periodic potentials. Our model, namely, a dissipative Gross-Pitaevskii equation, is treated analytically by means of dark-soliton perturbation theory and the Landau dy... [Phys. Rev. A 86, 033616] Published Fri Sep 14, 2012
    Keywords: Matter waves and collective properties of cold atoms and molecules
    Print ISSN: 1050-2947
    Electronic ISSN: 1094-1622
    Topics: Physics
    Published by American Physical Society (APS)
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  • 5
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    Pressure Effect on the Structural Transition and Suppression of the High-Spin State in the Triple-Layer T^{′}-La_{4}Ni_{3}O_{8} (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-06-09
    Description: Author(s): J.-G. Cheng, J.-S. Zhou, J. B. Goodenough, H. D. Zhou, K. Matsubayashi, Y. Uwatoko, P. P. Kong, C. Q. Jin, W. G. Yang, and G. Y. Shen We report a comprehensive high-pressure study on the triple-layer T ′ -La 4 Ni 3 O 8 with a suite of experimental probes, including structure determination, magnetic, and transport properties up to 50 GPa. Consistent with a recent ab inito calculation, application of hydrostatic pressure suppresses an insu... [Phys. Rev. Lett. 108, 236403] Published Fri Jun 08, 2012
    Keywords: Condensed Matter: Electronic Properties, etc.
    Print ISSN: 0031-9007
    Electronic ISSN: 1079-7114
    Topics: Physics
    Published by American Physical Society (APS)
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  • 6
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    Spatial solitons under competing linear and nonlinear diffractions (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-02-28
    Description: Author(s): Y. Shen, P. G. Kevrekidis, N. Whitaker, and Boris A. Malomed We introduce a general model which augments the one-dimensional nonlinear Schrödinger (NLS) equation by nonlinear-diffraction terms competing with the linear diffraction. The new terms contain two irreducible parameters and admit a Hamiltonian representation in a form natural for optical media. The ... [Phys. Rev. E 85, 026606] Published Mon Feb 27, 2012
    Keywords: Classical physics
    Print ISSN: 1539-3755
    Electronic ISSN: 1550-2376
    Topics: Physics
    Published by American Physical Society (APS)
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  • 7
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    Interferon gamma (IFN-{gamma}) disrupts energy expenditure and metabolic homeostasis by suppressing SIRT1 transcription (2012)
    Oxford University Press
    Details
    Publication Date: 2012-02-28
    Description: Chronic inflammation impairs metabolic homeostasis and is intimately correlated with the pathogenesis of type 2 diabetes. The pro-inflammatory cytokine IFN- is an integral part of the metabolic inflammation circuit and contributes significantly to metabolic dysfunction. The underlying mechanism, however, remains largely unknown. In the present study, we report that IFN- disrupts the expression of genes key to cellular metabolism and energy expenditure by repressing the expression and activity of SIRT1 at the transcription level. Further analysis reveals that IFN- requires class II transactivator (CIITA) to repress SIRT1 transcription. CIITA, once induced by IFN-, is recruited to the SIRT1 promoter by hypermethylated in cancer 1 (HIC1) and promotes down-regulation of SIRT1 transcription via active deacetylation of core histones surrounding the SIRT1 proximal promoter. Silencing CIITA or HIC1 restores SIRT1 activity and expression of metabolic genes in skeletal muscle cells challenged with IFN-. Therefore, our data delineate an IFN-/HIC1/CIITA axis that contributes to metabolic dysfunction by suppressing SIRT1 transcription in skeletal muscle cells and as such shed new light on the development of novel therapeutic strategies against type 2 diabetes.
    Print ISSN: 0305-1048
    Electronic ISSN: 1362-4962
    Topics: Biology
    Published by Oxford University Press
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  • 8
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    Experimental demonstration of a slippage-dominant free-electron laser amplifier (2012)
    American Physical Society (APS)
    Details
    Publication Date: 2012-02-16
    Description: Author(s): X. Yang, Y. Shen, B. Podobedov, Y. Hidaka, S. Seletskiy, and X. J. Wang We report the first experimental demonstration of a slippage-dominant free-electron laser (FEL) amplifier using a 140-fs full width at half maximum broadband seed laser pulse. The evolution of the longitudinal phase space of a laser seeded FEL amplifier in the slippage-dominant regime was experiment... [Phys. Rev. E 85, 026404] Published Wed Feb 15, 2012
    Keywords: Plasma physics
    Print ISSN: 1539-3755
    Electronic ISSN: 1550-2376
    Topics: Physics
    Published by American Physical Society (APS)
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  • 9
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    A steroid receptor-microRNA switch regulates life span in response to signals from the gonad (2012)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 2012-12-15
    Description: Although the gonad primarily functions in procreation, it also affects animal life span. Here, we show that removal of the Caenorhabditis elegans germ line triggers a switch in the regulatory state of the organism to promote longevity, co-opting components involved in larval developmental timing circuits. These components include the DAF-12 steroid receptor, which is involved in the larval stage two-to-stage three (L2-L3) transition and up-regulates members of the let-7 microRNA (miRNA) family. The miRNAs target an early larval nuclear factor lin-14 and akt-1/kinase, thereby stimulating DAF-16/FOXO signaling to extend life. Our studies suggest that metazoan life span is coupled to the gonad through elements of a developmental timer.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3909774/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3909774/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Shen, Yidong -- Wollam, Joshua -- Magner, Daniel -- Karalay, Oezlem -- Antebi, Adam -- R01 AG027498/AG/NIA NIH HHS/ -- T32 GM008231/GM/NIGMS NIH HHS/ -- New York, N.Y. -- Science. 2012 Dec 14;338(6113):1472-6. doi: 10.1126/science.1228967.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Max Planck Institute for Biology of Ageing, Joseph-Stelzmann-Strasse 9b, D-50931 Cologne, Germany.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/23239738" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Caenorhabditis elegans/genetics/growth & development/*physiology ; Caenorhabditis elegans Proteins/genetics/*physiology ; Forkhead Transcription Factors ; Gene Expression Regulation, Developmental ; Germ Cells/metabolism ; Gonads/*metabolism ; Larva/genetics/growth & development/physiology ; Longevity/genetics/*physiology ; MicroRNAs/genetics/*metabolism ; Nuclear Proteins/genetics/physiology ; Proto-Oncogene Proteins c-akt/genetics/physiology ; Receptors, Cytoplasmic and Nuclear/genetics/*physiology ; Receptors, Steroid/genetics/*physiology ; Signal Transduction ; Transcription Factors/genetics/physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 10
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    Interpreting cancer genomes using systematic host network perturbations by tumour virus proteins (2012)
    Nature Publishing Group (NPG)
    Details
    Publication Date: 2012-07-20
    Description: Genotypic differences greatly influence susceptibility and resistance to disease. Understanding genotype-phenotype relationships requires that phenotypes be viewed as manifestations of network properties, rather than simply as the result of individual genomic variations. Genome sequencing efforts have identified numerous germline mutations, and large numbers of somatic genomic alterations, associated with a predisposition to cancer. However, it remains difficult to distinguish background, or 'passenger', cancer mutations from causal, or 'driver', mutations in these data sets. Human viruses intrinsically depend on their host cell during the course of infection and can elicit pathological phenotypes similar to those arising from mutations. Here we test the hypothesis that genomic variations and tumour viruses may cause cancer through related mechanisms, by systematically examining host interactome and transcriptome network perturbations caused by DNA tumour virus proteins. The resulting integrated viral perturbation data reflects rewiring of the host cell networks, and highlights pathways, such as Notch signalling and apoptosis, that go awry in cancer. We show that systematic analyses of host targets of viral proteins can identify cancer genes with a success rate on a par with their identification through functional genomics and large-scale cataloguing of tumour mutations. Together, these complementary approaches increase the specificity of cancer gene identification. Combining systems-level studies of pathogen-encoded gene products with genomic approaches will facilitate the prioritization of cancer-causing driver genes to advance the understanding of the genetic basis of human cancer.〈br /〉〈br /〉〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3408847/" target="_blank"〉〈img src="https://static.pubmed.gov/portal/portal3rc.fcgi/4089621/img/3977009" border="0"〉〈/a〉   〈a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3408847/" target="_blank"〉This paper as free author manuscript - peer-reviewed and accepted for publication〈/a〉〈br /〉〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Rozenblatt-Rosen, Orit -- Deo, Rahul C -- Padi, Megha -- Adelmant, Guillaume -- Calderwood, Michael A -- Rolland, Thomas -- Grace, Miranda -- Dricot, Amelie -- Askenazi, Manor -- Tavares, Maria -- Pevzner, Samuel J -- Abderazzaq, Fieda -- Byrdsong, Danielle -- Carvunis, Anne-Ruxandra -- Chen, Alyce A -- Cheng, Jingwei -- Correll, Mick -- Duarte, Melissa -- Fan, Changyu -- Feltkamp, Mariet C -- Ficarro, Scott B -- Franchi, Rachel -- Garg, Brijesh K -- Gulbahce, Natali -- Hao, Tong -- Holthaus, Amy M -- James, Robert -- Korkhin, Anna -- Litovchick, Larisa -- Mar, Jessica C -- Pak, Theodore R -- Rabello, Sabrina -- Rubio, Renee -- Shen, Yun -- Singh, Saurav -- Spangle, Jennifer M -- Tasan, Murat -- Wanamaker, Shelly -- Webber, James T -- Roecklein-Canfield, Jennifer -- Johannsen, Eric -- Barabasi, Albert-Laszlo -- Beroukhim, Rameen -- Kieff, Elliott -- Cusick, Michael E -- Hill, David E -- Munger, Karl -- Marto, Jarrod A -- Quackenbush, John -- Roth, Frederick P -- DeCaprio, James A -- Vidal, Marc -- F32 GM095284/GM/NIGMS NIH HHS/ -- F32GM095284/GM/NIGMS NIH HHS/ -- K08 CA122833/CA/NCI NIH HHS/ -- K08 HL098361/HL/NHLBI NIH HHS/ -- K08HL098361/HL/NHLBI NIH HHS/ -- K25 HG006031/HG/NHGRI NIH HHS/ -- K25HG006031/HG/NHGRI NIH HHS/ -- P01 CA050661/CA/NCI NIH HHS/ -- P01CA050661/CA/NCI NIH HHS/ -- P50 HG004233/HG/NHGRI NIH HHS/ -- P50HG004233/HG/NHGRI NIH HHS/ -- R01 CA047006/CA/NCI NIH HHS/ -- R01 CA063113/CA/NCI NIH HHS/ -- R01 CA066980/CA/NCI NIH HHS/ -- R01 CA081135/CA/NCI NIH HHS/ -- R01 CA085180/CA/NCI NIH HHS/ -- R01 CA093804/CA/NCI NIH HHS/ -- R01 CA131354/CA/NCI NIH HHS/ -- R01 HG001715/HG/NHGRI NIH HHS/ -- R01CA047006/CA/NCI NIH HHS/ -- R01CA063113/CA/NCI NIH HHS/ -- R01CA066980/CA/NCI NIH HHS/ -- R01CA081135/CA/NCI NIH HHS/ -- R01CA085180/CA/NCI NIH HHS/ -- R01CA093804/CA/NCI NIH HHS/ -- R01CA131354/CA/NCI NIH HHS/ -- R01HG001715/HG/NHGRI NIH HHS/ -- T32 HL007208/HL/NHLBI NIH HHS/ -- T32HL007208/HL/NHLBI NIH HHS/ -- U01 CA141583/CA/NCI NIH HHS/ -- U01CA141583/CA/NCI NIH HHS/ -- England -- Nature. 2012 Jul 26;487(7408):491-5. doi: 10.1038/nature11288.〈br /〉〈span class="detail_caption"〉Author address: 〈/span〉Genomic Analysis of Network Perturbations Center of Excellence in Genomic Science, Dana-Farber Cancer Institute, Boston, Massachusetts 02215, USA.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/22810586" target="_blank"〉PubMed〈/a〉
    Keywords: Adenoviridae/genetics/metabolism/pathogenicity ; Gene Expression Profiling ; Gene Expression Regulation, Neoplastic ; Genes, Neoplasm/*genetics ; Genome, Human/*genetics ; Herpesvirus 4, Human/genetics/metabolism/pathogenicity ; *Host-Pathogen Interactions/genetics ; Humans ; Neoplasms/*genetics/*metabolism/pathology ; Oncogenic Viruses/genetics/metabolism/*pathogenicity ; Open Reading Frames/genetics ; Papillomaviridae/genetics/metabolism/pathogenicity ; Polyomavirus/genetics/metabolism/pathogenicity ; Receptors, Notch/metabolism ; Signal Transduction ; Two-Hybrid System Techniques ; Viral Proteins/genetics/*metabolism
    Print ISSN: 0028-0836
    Electronic ISSN: 1476-4687
    Topics: Biology , Chemistry and Pharmacology , Medicine , Natural Sciences in General , Physics
    Published by Nature Publishing Group (NPG)
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