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  • 1
    Electronic Resource
    Electronic Resource
    Springer
    Review of quantitative finance and accounting 5 (1995), S. 375-392 
    ISSN: 1573-7179
    Keywords: antitakeover devices ; supermajority nonfair price amendments
    Source: Springer Online Journal Archives 1860-2000
    Topics: Economics
    Notes: Abstract Separation of corporate ownership and control creates an environment whereby the agent (management) may pursue self-interests at the expense of the principal (stockholders). One mechanism protecting stockholders from self-interested management is the market for corporate control, or the takeover market. Antitakeover devices impede the operations of this market. This paper reports on the study of one type of antitakeover device, the supermajority nonfair price amendment. This device is particularly onerous, and if antitakeover devices do protect inefficient management as has been alluded to in the literature, then evidence of inefficiency should be observable for companies adopting supermajority nonfair price amendments. To test the above proposition, we examine the difference in performance over a seven-year period (1) Between firms having supermajority nonfair price amendments and a set of matched firms that do not have these devices, and (2) between firms with these amendments and their respective industries. In both tests, performance was lower for the firms adopting these amendments, which suggests that these devices are used to protect inefficient management. Further, the argument that managers of firms which adopt antitakeover devices so that they can take a long-term outlook at the expense of short-term profitability was not supported by the data.
    Type of Medium: Electronic Resource
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  • 2
    ISSN: 1573-7691
    Keywords: Domain decomposition ; nested dissection ; LU-factorization ; time integration ; Newton's method ; spectral elements ; finite elements
    Source: Springer Online Journal Archives 1860-2000
    Topics: Computer Science
    Notes: Abstract Domain decomposition by nested dissection for concurrent factorization and storage (CFS) of asymmetric matrices is coupled with finite element and spectral element discretizations and with Newton's method to yield an algorithm for parallel solution of nonlinear initial-and boundary-value problem. The efficiency of the CFS algorithm implemented on a MIMD computer is demonstrated by analysis of the solution of the two-dimensional, Poisson equation discretized using both finite and spectral elements. Computation rates and speedups for the LU-decomposition algorithm, which is the most time consuming portion of the solution algorithm, scale with the number of processors. The spectral element discretization with high-order interpolating polynomials yields especially high speedups because the ratio of communication to computation is lower than for low-order finite element discretizations. The robustness of the parallel implementation of the finite-element/Newton algorithm is demonstrated by solution of steady and transient natural convection in a two-dimensional cavity, a standard test problem for low Prandtl number convection. Time integration is performed using a fully implicit algorithm with a modified Newton's method for solution of nonlinear equations at each time step. The efficiency of the CFS version of the finite-element/Newton algorithm compares well with a spectral element algorithm implemented on a MIMD computer using iterative matrix methods.
    Type of Medium: Electronic Resource
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  • 3
    ISSN: 1572-9931
    Source: Springer Online Journal Archives 1860-2000
    Topics: Biology , Medicine
    Notes: Abstract Recently, point mutations in superoxide dismutase 1 (SOD1) have been shown to lead to a subset of autosomal dominantly inherited familial amyotrophic lateral sclerosis (ALS). These findings have led to the hypothesis that defects in oxygen radical metabolism may be involved in the pathogenesis of ALS. Therefore, we decided to analyze other enzymes involved in oxygen radical metabolism for possible involvement in other forms of ALS. We report here analysis of two genes encoding the molybdenum hydroxylases aldehyde oxidase (AO) and xanthine dehydrogenase/ oxidase (XDH) for involvement in ALS. Of particular interest, one gene identified as encoding aldehyde oxidase is shown to map to 2q33, a region recently shown to contain a gene responsible for a familial form of ALS with autosomal recessive inheritance (FALS-AR). The AO gene appears to be located within 280,000 bp of simple sequence repeat marker D2S116, which shows no recombination with the FALS-AR locus. The AO gene is highly expressed in glial cells of human spinal cord. In addition, we mapped a gene for XDH to 2p22, a region previously shown to contain a highly homologous but different form of XDH. Neither of these XDH genes appears to be highly expressed in human spinal cord. This evidence suggests that AO may be a candidate gene for FALS-AR.
    Type of Medium: Electronic Resource
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