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  • Disease Models, Animal  (8)
  • American Association for the Advancement of Science (AAAS)  (8)
  • Periodicals Archive Online (PAO)
  • 1980-1984  (8)
  • 1955-1959
  • 1940-1944
  • 1981  (8)
  • 1957
Collection
Keywords
Publisher
  • American Association for the Advancement of Science (AAAS)  (8)
  • Periodicals Archive Online (PAO)
Years
  • 1980-1984  (8)
  • 1955-1959
  • 1940-1944
Year
  • 1
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    Hyperkeratosis induced by sunlight degradation products of the major polybrominated biphenyl in Firemaster (1981)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1981-08-21
    Description: Sunlight photodegradation of 2,2', 4,4', 5,5' -hexabromobiphenyl, the major component of Firemaster, gave a mixture that produces severe hyperkeratosis of the rabbit ear. This component in its pure state does not cause hyperkeratosis. One or more of the four major photolysis products must be responsible for this activity. A similar photodegradation pattern was observed for 2,2', 3,4,4', 5,5' -heptabromobiphenyl, the second largest component of Firemaster.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Patterson, D G -- Hill, R H -- Needham, L L -- Orti, D L -- Kimbrough, R D -- Liddle, J A -- New York, N.Y. -- Science. 1981 Aug 21;213(4510):901-2.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6266016" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; *Biphenyl Compounds/radiation effects ; Chemical Industry ; Disease Models, Animal ; Environmental Exposure ; Keratosis/*chemically induced ; Michigan ; Photochemistry ; *Polybrominated Biphenyls/radiation effects ; Rabbits ; Sunlight
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 2
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    Functional restoration of vision in the cat after long-term monocular deprivation (1981)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1981-09-04
    Description: Recovery of visual acuity was studied in six long-term monocularly deprived cats after removal of the nondeprived eye or reverse lid suture. Although both manipulations improved visual acuity, removal of the nondeprived eye was associated with more rapid recovery and higher find acuity than in reverse suture. These results are in agreement with the known electrophysiological effects of these recovery conditions and are also similar to the effects of reverse occlusion or loss of the nonamblyopic eye in human amblyopes.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Smith, D C -- EYO 7005/EY/NEI NIH HHS/ -- New York, N.Y. -- Science. 1981 Sep 4;213(4512):1137-9.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7268422" target="_blank"〉PubMed〈/a〉
    Keywords: Age Factors ; Amblyopia/physiopathology ; Animals ; Cats ; Disease Models, Animal ; Form Perception/physiology ; Visual Acuity ; Visual Cortex/growth & development/*physiology ; Visual Perception/*physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 3
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    Phototherapy-induced hypocalcemia in newborn rats: prevention by melatonin (1981)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1981-11-13
    Description: When young rats are exposed to white fluorescent light the concentration of calcium in their serum decreases. This effect is prevented by shielding the occiput, by inhibiting corticosterone synthesis, and by exogenous melatonin. Furthermore, the expected hypocalcemic response to cortisol injection is prevented by melatonin. Light-induced hypocalcemia may result from increased calcium uptake by bone when the blocking effect of melatonin decreases after pineal inhibition by transcranial illumination.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Hakanson, D O -- Bergstrom, W H -- New York, N.Y. -- Science. 1981 Nov 13;214(4522):807-9.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6895262" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Animals, Newborn/*radiation effects ; Disease Models, Animal ; Female ; Humans ; Hydrocortisone/antagonists & inhibitors ; Hypocalcemia/etiology/*prevention & control ; Infant, Newborn ; Jaundice, Neonatal/therapy ; Light ; Male ; Melatonin/*pharmacology ; Phototherapy/adverse effects ; Rats ; Spectrum Analysis ; Time Factors
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 4
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    Giant synaptic potential hypothesis for epileptiform activity (1981)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1981-01-16
    Description: According to one hypothesis, the paroxysmal depolarizing shift observed in the penicillin model of epilepsy results from a giant excitatory postsynaptic potential. This hypothesis has recently been questioned, primarily because it has never been subjected to rigorous experimental examination. Four quantitative predictions were derived from this hypothesis and tested in CA3 pyramidal neurons of the hippocampus. The four critical predictions concern the behavior of the paroxysmal depolarizing shift under current- and voltage-clamp conditions as a function of membrane potential. The experiments confirmed all four predictions.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Johnston, D -- Brown, T H -- NS11535/NS/NINDS NIH HHS/ -- NS15772/NS/NINDS NIH HHS/ -- RR-05471-17/RR/NCRR NIH HHS/ -- New York, N.Y. -- Science. 1981 Jan 16;211(4479):294-7.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7444469" target="_blank"〉PubMed〈/a〉
    Keywords: Action Potentials ; Animals ; Disease Models, Animal ; Electric Conductivity ; Epilepsy/*physiopathology ; Guinea Pigs ; Hippocampus/*physiopathology ; In Vitro Techniques ; Membrane Potentials ; Penicillin G ; Synaptic Membranes/physiology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 5
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    Rapid correction of hyponatremia causes demyelination: relation to central pontine myelinolysis (1981)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1981-03-06
    Description: The human demyelinative disorder central pontine myelinolysis may be an iatrogenic disease caused by a rapid rise in serum sodium, usually when hyponatremia is corrected. Rats treated with hypertonic saline after 3 days of vasopressin-induced hyponatremia had demyelinative lesions in the corpus striatum, lateral hemispheric white matter, cerebral cortex, hippocampal fimbria, anterior commissure, thalamus, brainstem tegmentum, and cerebellum. Thus, rapid correction of hyponatremia can lead to demyelinative lesions and may be the cause of central pontine myelinolysis in man.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Kleinschmidt-DeMasters, B K -- Norenberg, M D -- New York, N.Y. -- Science. 1981 Mar 6;211(4486):1068-70.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7466381" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Blood-Brain Barrier ; Brain Diseases/*etiology ; Demyelinating Diseases/*etiology/pathology ; Disease Models, Animal ; Hyponatremia/*complications ; Male ; Pons/*pathology ; Rats
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 6
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    Spontaneous diabetes in rats: destruction of islets is prevented by immunological tolerance (1981)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1981-09-18
    Description: Spontaneous diabetes occurring in "BB" rats (derived from a colony of outbred Wistar rats) is the result of destruction of pancreatic islets by infiltrating mononuclear cells (insulitis) and may be a disease very similar to human juvenile onset diabetes. Both diseases probably have an autoimmune etiology. Evidence is presented that islets transplanted to diabetic BB rats are destroyed by the original disease process. Inoculation of bone marrow from normal (nondiabetes-susceptible) rat donors into neonatal BB recipients usually prevented the development of hyperglycemia.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Naji, A -- Silvers, W K -- Bellgrau, D -- Barker, C F -- AM19525/AM/NIADDK NIH HHS/ -- AM26007/AM/NIADDK NIH HHS/ -- CA18640/CA/NCI NIH HHS/ -- New York, N.Y. -- Science. 1981 Sep 18;213(4514):1390-2.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6791286" target="_blank"〉PubMed〈/a〉
    Keywords: Animals ; Autoimmune Diseases/*immunology ; Bone Marrow Transplantation ; Diabetes Mellitus, Experimental/*immunology ; Disease Models, Animal ; Graft Rejection ; Immune Tolerance ; Islets of Langerhans/*immunology ; Islets of Langerhans Transplantation ; Rats
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 7
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    beta-Endorphin: possible involvement in the antihypertensive effect of central alpha-receptor activation (1981)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1981-01-02
    Description: Clonidine and L-alpha-methylnoradrenaline (but not D-alpha-methylnoradrenaline) increase the release of a substance with beta-endorphin immunoreactivity from slices of brainstem of spontaneously hypertensive rats, but not that of normotensive rats. It was reported earlier that opiate antagonists inhibit the hypotensive action of clonidine and alpha-methyldopa in spontaneously hypertensive but not in normotensive rats and that beta-endorphin has hypotensive effects of its own. Together, these findings indicate that release of beta-endorphin by central alpha-receptor agonists may contribute to the antihypertensive action of these drugs.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Kunos, G -- Farsang, C -- Ramirez-Gonzales, M D -- New York, N.Y. -- Science. 1981 Jan 2;211(4477):82-4.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/6108611" target="_blank"〉PubMed〈/a〉
    Keywords: Adrenergic alpha-Agonists/*pharmacology ; Animals ; Brain Stem/*metabolism ; Clonidine/pharmacology ; Disease Models, Animal ; Endorphins/*metabolism ; Hypertension/*physiopathology ; Immunoassay ; Male ; Naloxone/pharmacology ; Nordefrin/pharmacology ; Rats
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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  • 8
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    Hyperthermia-induced seizures in the rat pup: a model for febrile convulsions in children (1981)
    American Association for the Advancement of Science (AAAS)
    Details
    Publication Date: 1981-08-28
    Description: Seizures were produced in rat pups by ambient hyperthermia. Seizure threshold temperatures, measured rectally and intracerebrally, increased between 2 and 10 days of age. Electrocortical paroxysmal discharges were confirmed in hyperthermic 6- and 10-day-old pups. The increasing resistance to hyperthermic seizures with maturation and the electroencephalographic changes induced by hyperthermia are similar to those in young children.〈br /〉〈span class="detail_caption"〉Notes: 〈/span〉Holtzman, D -- Obana, K -- Olson, J -- NS 16256/NS/NINDS NIH HHS/ -- New York, N.Y. -- Science. 1981 Aug 28;213(4511):1034-6.〈br /〉〈span class="detail_caption"〉Record origin:〈/span〉 〈a href="http://www.ncbi.nlm.nih.gov/pubmed/7268407" target="_blank"〉PubMed〈/a〉
    Keywords: Age Factors ; Animals ; Brain/physiopathology ; Disease Models, Animal ; Electroencephalography ; Fever/*complications ; Rats ; Seizures/*physiopathology ; Seizures, Febrile/*physiopathology
    Print ISSN: 0036-8075
    Electronic ISSN: 1095-9203
    Topics: Biology , Chemistry and Pharmacology , Computer Science , Medicine , Natural Sciences in General , Physics
    Published by American Association for the Advancement of Science (AAAS)
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