Publication Date:
2018-03-29
Description:
Key Points IL-18 distinguishes susceptibility to MAS amongst hyperferritinemic and autoinflammatory diseases. Excess IL-18 in NLRC4 gain-of-function mice derives from intestinal epithelia, and free IL-18 promotes experimental MAS.
Print ISSN:
0006-4971
Electronic ISSN:
1528-0020
Topics:
Biology
,
Medicine
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